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1 iators without affecting baseline mechanical nociceptive threshold.
2 ed the enhancement without altering baseline nociceptive threshold.
3 the spinal cord leads to a chronic decreased nociceptive threshold.
4 on velocity, resting membrane potential, and nociceptive threshold.
5 but had no significant effect on the normal nociceptive threshold.
6 than 2 h) increase in mechanical and thermal nociceptive thresholds.
7 eurons and partial restoration of behavioral nociceptive thresholds.
8 -term removal of NA afferents did not affect nociceptive thresholds.
9 with opioid-mediated elevations in maternal nociceptive thresholds.
10 restored thermal and ameliorated mechanical nociceptive thresholds.
11 and SNAP significantly increased mechanical nociceptive thresholds.
12 ough it had no effect on baseline mechanical nociceptive thresholds.
14 Restoring central NA normalized both the nociceptive threshold and morphine efficacy, which is co
15 contributes only modestly to determining the nociceptive threshold and that its antinociceptive effec
16 t, the (beta)arr2-KO mice have greater basal nociceptive thresholds and markedly enhanced sensitivity
17 e gestation or its hormonal simulation, when nociceptive thresholds are elevated by approximately 70%
19 vioral data showed that minocycline restored nociceptive thresholds, at which time spinal microglial
20 und stress exhibited no change in mechanical nociceptive threshold, but showed a marked increase in h
21 idence for tonic modulation of basal thermal nociceptive thresholds by the spinal cannabinoid system.
23 n, characterized by a decrease in mechanical nociceptive threshold (hyperalgesia), arises through act
24 ind paw induced a decrease in the mechanical nociceptive threshold (hypernociception), which was asso
27 y, engaging the A3AR mechanism did not alter nociceptive thresholds in non-neuropathy animals and the
28 he absence of injury, thermal and mechanical nociceptive thresholds increased 2 weeks post-treatment
30 d to surgical tail resections and mechanical nociceptive thresholds (MNT) were measured in the acute
31 and heterozygous (+/-) counterparts; thermal nociceptive thresholds obtained in +/+ and +/- mice did
32 TX) or naltriben (NTB) substantially reduces nociceptive thresholds of gestation (day 20) and HSP (da
33 -Arg-Thr-Pen-Thr-NH2 (CTAP) has no effect on nociceptive thresholds of gestational day 20, as was pre
34 ain in the absence of a stimulus and reduced nociceptive thresholds so that normally innocuous stimul
35 If endogenous cannabinoids modulate basal nociceptive thresholds, then alterations in this system
36 s became hyperresponsive and when behavioral nociceptive thresholds to mechanical and thermal stimuli
38 e P-mediated chronic hyperalgesia (decreased nociceptive threshold) to thermal, but not mechanical, s
40 s became hyperresponsive and when behavioral nociceptive thresholds were decreased to both mechanical
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