ライフサイエンスコーパス: nocturnal asthma

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1 with nocturnal asthma, and patients with non-nocturnal asthma.

2 ory processes that occur in the patient with nocturnal asthma.

3 the morbidity and mortality associated with nocturnal asthma.

4 ght in nocturnal asthma as compared with non-nocturnal asthma.

5 estry appear to be independent predictors of nocturnal asthma.

6 esence in the proximal and distal airways in nocturnal asthma.

7 hat of control subjects or patients with non-nocturnal asthma and did not change significantly at 4:0

8 l asthma was less than that of patients with nocturnal asthma and rose significantly at 4:00 A.M. (p

9 M. in normal control subjects, patients with nocturnal asthma, and patients with non-nocturnal asthma

10 ntributes to population-level differences in nocturnal asthma appears to be largely independent of lu

11 increased in the alveolar tissue at night in nocturnal asthma as compared with non-nocturnal asthma.

12 Patients with nocturnal asthma demonstrate circadian variations in air

13 Patients with nocturnal asthma demonstrate the largest daytime cytokin

14 Symptoms of nocturnal asthma, exercise-related symptoms, BHR symptom

15 patients with nocturnal worsening of asthma (nocturnal asthma) exhibit increased parenchymal inflamma

16 Nocturnal asthma is a common presentation and is associa

17 er numbers of exacerbations, the presence of nocturnal asthma, more severe bronchial hyperresponsiven

18 Eleven patients with nocturnal asthma (NA) and 10 patients with non-nocturnal

19 Eleven patients with nocturnal asthma (NA) and 10 patients with non-nocturnal

20 l blood mononuclear cells from patients with nocturnal asthma (NA) exhibit reduced steroid responsive

21 s parenchymal inflammation, 10 subjects with nocturnal asthma (NA), four subjects with non-nocturnal

22 cturnal asthma (NA) and 10 patients with non-nocturnal asthma (NNA) underwent two bronchoscopies with

23 cturnal asthma (NA) and 10 patients with non-nocturnal asthma (NNA) were studied.

24 octurnal asthma (NA), four subjects with non-nocturnal asthma (NNA), and four normal control subjects

25 appropriate use of medications can minimize nocturnal asthma or allergic symptoms that might disrupt

26 A.M. in control subjects or in patients with nocturnal asthma (p > 0.05, both cases).

27 as also associated with an increased risk of nocturnal asthma (P = 0.007).

28 ever, there are few epidemiologic studies of nocturnal asthma, particularly in minority populations.

29 uate the association of various factors with nocturnal asthma, such as self-identified race/ethnicity

30 Nocturnal asthma symptoms were assessed by questionnaire

31 he relationship between genetic ancestry and nocturnal asthma symptoms.

32 We have shown in nocturnal asthma that alveolar tissue eosinophils are in

33 okine response to melatonin of patients with nocturnal asthma was greater than that of control subjec

34 , the cytokine response of patients with non-nocturnal asthma was less than that of patients with noc

35 Five patients with documented nocturnal asthma were studied in both a vertical and a h

36 5% confidence interval, 2.24-2.93) to report nocturnal asthma when compared with European American in

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