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1 imary vasculitides, especially polyarteritis nodosa.
2 with a diagnosis of cutaneous polyarteritis nodosa.
6 Kawasaki disease, and treating polyarteritis nodosa, but most reported feeling comfortable treating c
7 ases of systemic and cutaneous polyarteritis nodosa, consistent with autosomal recessive inheritance.
10 munodeficiency, liver disease, trichorrhexis nodosa, facial dysmorphism, hypopigmentation, and cardia
11 ia for Wegener granulomatosis, polyarteritis nodosa, giant-cell arteritis, and hypersensitivity vascu
13 s-related hospitalizations for polyarteritis nodosa, hypersensitivity vasculitis, Wegener's granuloma
16 nt fevers, livedo reticularis, polyarteritis nodosa, lacunar ischemic strokes, and intracranial hemor
17 itides with neuropathy include polyarteritis nodosa, microscopic polyangiitis (MPA), rheumatoid vascu
18 with Wegener's granulomatosis, polyarteritis nodosa, microscopic polyangiitis, and Churg-Strauss synd
19 hereas the three patients with polyarteritis nodosa or small-vessel vasculitis were homozygous for th
21 polymyositis, dermatomyositis, polyarteritis nodosa, or scleroderma who had an emergent or urgent hos
22 purpura (HSP) and macroscopic polyarteritis nodosa (PAN) as early manifestations of IgA kappa multip
26 tively common manifestation of polyarteritis nodosa (PAN), using the modern classification of PAN pul
27 - females developed mesenteric polyarteritis-nodosa (PAN)-like vasculitis in their life span, some as
28 sis, inflammatory myopathy, or polyarteritis nodosa rather than rheumatoid arthritis, and were less l
29 tis, giant cell arteritis, and polyarteritis nodosa) to atherosclerosis, display remarkable target ti
30 adenosine deaminase, can cause polyarteritis nodosa vasculopathy with highly varied clinical expressi
31 case of idiopathic vasculitis, polyarteritis nodosa, which even today is the basis of our understandi
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