ライフサイエンスコーパス: nodular

1 classified by MRI as infiltrative and 81 as nodular.

2 <50 years]), superficial spreading subtype (nodular, 0.5 [0.2-1.0]; lentigo maligna, 0.4 [0.2-0.7];

3 The iris tumor was nodular (16/21, 76%) or diffuse (5/21, 24%).

4 92; 15%), cutaneous (84; 14%), and pulmonary nodular (47; 8%).

5 eterogeneous, including maculopapular (82%), nodular (6%), and diffuse cutaneous (12%) mastocytosis.

6 A-mut) and associated with primary pigmented nodular adrenocortical disease (PPNAD) and increased ste

7 er; however, their role in primary pigmented nodular adrenocortical disease (PPNAD) has not been inve

8 Primary pigmented nodular adrenocortical disease (PPNAD) is associated wit

9 ng PRKAR1A mutations cause primary pigmented nodular adrenocortical disease (PPNAD) or Carney complex

10 o altered kinase activity, primary pigmented nodular adrenocortical disease, and tumors of the thyroi

11 er features reminiscent of primary pigmented nodular adrenocortical disease, histiocytic and epitheli

12 stology (hazard ratio, 0.19 for desmoplastic/nodular and 45.97 for large-cell/anaplastic medulloblast

13 Furthermore, we show that the nodular and digitate silica structures at El Tatio that

14 potential significance of the characteristic nodular and mm-scale digitate opaline silica structures

15 to be approximately 3-fold more abundant in nodular and tumor tissue than in control tissue.

16 Female sex,nodular and unclassified or other histologic subtypes, i

17 pes: superficial spreading, lentigo maligna, nodular, and acral lentiginous.

18 f BCC and histological subtype (superficial, nodular, and infiltrating) in the confocal mosaics.

19 l markings, consolidation, and ground-glass, nodular, and reticular opacity), distribution, and exten

20 Types of CM included superficial spreading, nodular, and unclassified in 5106 cases among 3206 men a

21 iffuse anterior in 80% (n = 28), followed by nodular anterior 11.4% (n = 4), and necrotizing in 8.6%

22 ck of corneal involvement, papillomatous and nodular appearance, microscopic multifocality, and posit

23 Eight CAN biopsies showed nodular arteriolar hyalinization and one was positive fo

24 renal gland as normal, smoothly enlarged, or nodular at initial CT examination.

25 This high prevalence of nodular autonomy usually results in a further increase i

26 id activity which, in the long term, reduces nodular autonomy.

27 cessful elimination of superficial and early nodular basal cell carcinoma (BCC) in 2 cases using RCM

28 The recurrence rate of periocular nodular basal cell carcinoma (PNBCC) following treatment

29 mod compared with surgery for superficial or nodular basal cell carcinoma at low-risk sites in our no

30 (superficial basal cell carcinoma, 6 weeks; nodular basal cell carcinoma, 12 weeks) or excisional su

31 normal morphology or, in a few cases, small nodular basal laminar deposits.

32 ent option for small low-risk superficial or nodular basal-cell carcinoma dependent on factors such a

33 s of resting hair follicles in mice, induced nodular BCC development from a small subset of cells in

34 ke tumors from interfollicular epidermis and nodular BCC-like tumors from hair follicle stem cells.

35 cases of NM, 134 of invasive non-NM, 115 of nodular benign melanocytic tumors, and 135 of nodular no

36 17 patients with treatment-naive noncavitary nodular bronchiectatic MAC lung disease.

37 atment regimen for patients with noncavitary nodular bronchiectatic MAC lung disease.

38 ded for the initial treatment of noncavitary nodular bronchiectatic Mycobacterium avium complex (MAC)

39 were low body mass index (BMI), radiographic nodular-bronchiectatic (NB) pattern, and increase in the

40 complex lung disease, 55% women and 53% with nodular/bronchiectatic disease, received a mean of 16.1

41 constitutively phosphorylated at Ser-380 in nodular but not superficial spreading melanoma and did n

42 were infiltrative and two of 27 tumors were nodular) but none were found in HCV patients (P = .001).

43 ; of the remaining 6 that were classified as nodular by MRI, 5 histologically demonstrated stage II o

44 ve cusps undergo retraction, stiffening, and nodular calcification.

45 g studies showed a presacral solid mass with nodular calcifications.

46 phs of 50 patients with 55 confirmed primary nodular cancers (mean diameter, 20 mm) as well as 30 pat

47 ibitor of metalloproteinases 1 in metastatic nodular compared with metastatic superficial spreading m

48 ma lines and metastatic tissues derived from nodular compared with superficial spreading melanoma.

49 histologically desmoplastic; the other 2 had nodular components.

50 ctive lymphoid hyperplasia (BRLH) (38 [5%]), nodular conjunctivitis (31 [4%]), dermoid (30 [4%]), and

51 of 12 patients had focal low SI that was not nodular (contour deforming) over a region in and around

52 pterygia (vascular component) and Salzmann's nodular degeneration (corneal component).

53 bed in more recent case series of Salzmann's nodular degeneration and diffuse keratoconjunctival prol

54 d at the periphery of discoid platelets into nodular densities consistent with clustered alphaIIbbeta

55 tumor for analysis, including 38 classic, 30 nodular desmoplastic, and 18 large-cell anaplastic (LCA)

56 atient's postablation CT images showed focal nodular diaphragmatic thickening.

57 Twenty-four (47%) patients had nodular disease with bronchiectasis and 27 (53%) had upp

58 ocyte sedimentation rate, rheumatoid factor, nodular disease, modified Health Assessment Questionnair

59 d for total thyroidectomy for goiter, benign nodular disease, suspected thyroid cancer, or known thyr

60 s, there was cellular atypia consistent with nodular dysplasia.

61 ed States and had endoscopic evidence of non-nodular dysplastic BE </=8 cm in length.

62 imaging, with thick peripheral, septal, and nodular enhancement in all cases.

63 studies revealed multiple punctate foci with nodular enhancement in the brain and multifocal cystic l

64 nvolving the cervicothoracic spinal cord and nodular enhancement of the cauda equina.

65 Lesions with initially nodular enhancement were smaller than those initially en

66 ial response by EASL criteria and peripheral nodular enhancement, respectively.

67 Conclusions and Relevance: Papular and nodular eruptions with scale, as well as mucosal erosion

68 ruli and expanded mesangium with diffuse and nodular expansion of mesangial matrix.

69 Analysis of primary clinical samples of nodular fasciitis confirmed the activation of a Jak1-STA

70 Chromosomal translocations in nodular fasciitis result in USP6 overexpression, leading

71 ne cyst (ABC), and the related benign lesion nodular fasciitis.

72 , which recapitulate key features of ABC and nodular fasciitis; however, the identity of USP6's relev

73 eosinophilic angiocentric fibrosis, reactive nodular fibrous pseudotumor, sclerosing mesenteritis, an

74 MR imaging in another patient showed nodular focal low SI that was suspicious for cancer and

75 tensity (SI): normal, suspicious for cancer (nodular focal low SI), or indeterminate (focal low SI th

76 us gingivitis, is a rare condition involving nodular gingival enlargement with ulceration and periodo

77 ney involvement and histologically exhibited nodular glomerular involvement.

78 Morphologically, smoking-associated nodular glomerulosclerosis closely resembles diabetic ne

79 sufficient to induce Kimmelstiel-Wilson-like nodular glomerulosclerosis in mice through a process tha

80 supports that the entity known as idiopathic nodular glomerulosclerosis is not idiopathic.

81 rogates renal insufficiency and the diabetic nodular glomerulosclerosis phenotype of diabetic Sema3a(

82 use mesangial sclerosis (focally approaching nodular glomerulosclerosis), focal arteriolar hyalinosis

83 in of function in eNOS(-/-) mice resulted in nodular glomerulosclerosis, mesangiolysis, microaneurysm

84 teinuria, renal insufficiency, and extensive nodular glomerulosclerosis, mimicking advanced DN in hum

85 racellular matrix including both diffuse and nodular glomerulosclerosis, tubulointerstitial fibrosis,

86 mesangiolysis, and focal segmental and early nodular glomerulosclerosis.

87 sis, such as Graves' disease and functioning nodular goiters, there are more than 20 less common caus

88 ts in an increase in the prevalence of toxic nodular goitre and hyperthyroidism in populations.

89 gs are not generally used long term in toxic nodular goitre, because of the high relapse rate of thyr

90 oidism is Graves' disease, followed by toxic nodular goitre.

91 s characterized by either the development of nodular granulomatous lesions in the peribronchial regio

92 at replaced the hepatic lobule or histologic nodular growth in the portal triad that effaced adjacent

93 patterns and 1 histologically demonstrated a nodular growth pattern.

94 classified as exhibiting an infiltrative or nodular growth pattern.

95 growth in the sinusoidal spaces, whereas MRI nodular growth patterns corresponded to stage II/III his

96 tients [90.5% males, 89.2% cirrhotics, 89.2% nodular HCC, median age 63 (34-84) years] underwent 111

97 ia with megalencephaly (20), periventricular nodular heterotopia (61), and pachygyria (47).

98 DCX and LIS1), persons with periventricular nodular heterotopia (FLNA), and persons with pachygyria

99 f origin and form persistent periventricular nodular heterotopia (PH).

100 developmental disorders with periventricular nodular heterotopia (PNH) are etiologically heterogeneou

101 Polymicrogyria (PMG) and periventricular nodular heterotopia (PNH) are two developmental brain ma

102 is possible in patients with periventricular nodular heterotopia (PVNH) to detect abnormal fiber proj

103 l migration disorder, called periventricular nodular heterotopia (PVNH; refs. 3-6).

104 lformations, polymicrogyria, periventricular nodular heterotopia and diffuse megalencephaly without c

105 dles syndrome, with X-linked periventricular nodular heterotopia and FG syndrome (Omim, 305450).

106 patients with epilepsy from periventricular nodular heterotopia and matched healthy controls.

107 unction of human FLNA causes periventricular nodular heterotopia in females and is generally lethal (

108 n locations normally seen in periventricular nodular heterotopia on MRI.

109 gration disorders, including periventricular nodular heterotopia, subcortical band heterotopia and li

110 izencephaly, and 15 cases of periventricular nodular heterotopia.

111 elected subgroup of patients with unilateral nodular heterotopia.

112 mpassed cysts (70), hemangiomata (37), focal nodular hyperplasia (FNH) (23), adenomata (47), and 20 l

113 Focal nodular hyperplasia (FNH) and hepatocellular adenoma (HC

114 Follicular nodular hyperplasia (FNH) is a common benign liver tumor

115 Nonoperative management of focal nodular hyperplasia (FNH) is an accepted paradigm in adu

116 Focal nodular hyperplasia (FNH), hepatocellular adenoma (HCA),

117 h nodular regenerative hyperplasia and focal nodular hyperplasia (FNH), which finally evolved to a gi

118 logically indeterminate for adenoma or focal nodular hyperplasia (FNH): (1) continue to observe with

119 (n = 6), cavernous hemangioma (n = 4), focal nodular hyperplasia (n = 2), hamartoma (n = 1), and meta

120 ice developed liver nodules that were either nodular hyperplasia or hepatocellular carcinoma (HCC).

121 s (42.2%) had adenomas, 29 (19.7%) had focal nodular hyperplasia, 25 (17.0%) had hemangiomas, 11 (7.5

122 ding a rate-limiting step between simple and nodular hyperplasia, the latter of which is marked by th

123 Similarly, in nodular hyperplasia, which is the most severe form of SH

124 ve thrombosis, and lack of visibility; focal nodular hyperplasia-like nodules (six [14%] of 42 vs 0 [

125 evels in H-HCA, IHCA, b-HCA, UHCA, and focal nodular hyperplasia.

126 ves' disease and the inability to cure toxic nodular hyperthyroidism with antithyroid drugs alone, ra

127 Other important causes include toxic nodular hyperthyroidism, due to the presence of one or m

128 y, becoming nodular if small (82%) or nearly nodular if larger (18%).

129 ike lesions enhanced centripetally, becoming nodular if small (82%) or nearly nodular if larger (18%)

130 Pulmonary nodules and nodular infiltrates occur frequently during treatment of

131 nsolidation, tree-in-bud pattern, upper lobe nodular infiltration and cavitation.

132 aled that cavity, tree-in-bud and upper lobe nodular infiltration has significant association with sm

133 lymphadenopathy, consolidation, collapse and nodular infiltration in the upper lobe.

134 Cavity, tree- in-bud pattern and upper lobe nodular infiltration were highly associated with smear p

135 ologic findings of 6 patients with CVID with nodular/infiltrative lung disease who had biopsy specime

136 nd neck (29.4% vs 8.7%; P < .001) and of the nodular, lentigo maligna, or acral lentiginous histologi

137 We hypothesized that nodular lesions abolish otolith-perceptual integration,

138 All initially nodular lesions enhanced centrifugally, whereas initiall

139 A clinical examination revealed nodular lesions in the ears and a lump in the subcutaneo

140 considered in the differential diagnosis of nodular lesions or sinus tracts present in the axillae,

141 Nodular lesions were present in 3 patients.

142 of the three cases had unilateral, multiple nodular lesions with smooth borders accompanied by a hyd

143 nimals infected by 10(4) parasites presented nodular lesions, while those infected with 10(6) parasit

144 rular lesions that include mesangiolysis and nodular lesions.

145 pattern of BA, BASM, presence of ascites and nodular liver appearance at KPE, and early postoperative

146 ), ascites > 20 mL (HR: = 1.90, P = 0.0230), nodular liver appearance compared to firm (HR: = 1.61, P

147 haracterized by the emergence of spontaneous nodular liver lesions in approximately 50% of male mice

148 All 34 radiographs showed a nodular lung cancer that was apparent in retrospect but

149 Nodular lymphocyte Hodgkin lymphoma (NLPHL) is a rare di

150 Nodular lymphocyte predominant Hodgkin lymphoma (NLPHL)

151 Nodular lymphocyte predominant Hodgkin lymphoma (NLPHL)

152 dominant (LP) cells from tissues involved by nodular lymphocyte predominant Hodgkin lymphoma (NLPHL).

153 is subdivided into a classical variant and a nodular lymphocyte predominant variant which are charact

154 sal expression of CD20 by malignant cells in nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

155 Nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

156 Nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

157 res and clinical data from 423 patients with nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

158 number of reports have shown a propensity of nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

159 The optimal treatment of stage IA nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL)

160 and outcome for patients with advanced-stage nodular lymphocyte-predominant Hodgkin lymphoma (NLPHL).

161 Neoplastic cells of nodular lymphocyte-predominant Hodgkin lymphoma were neg

162 before treatment initiation owing to having nodular, lymphocyte-predominant Hodgkin's lymphoma and t

163 h sinopulmonary infections, lymphadenopathy, nodular lymphoid hyperplasia and viremia due to cytomega

164 A model that resembles nodular MAC disease was established in C57 (bg+/bg+) mic

165 s in vivo, including solid acinar, and solid nodular malignancies as well as cystic hyperplasia.

166 onography (EUS) that showed a 3- x 2-cm flat nodular mass with an 8-mm ulcer in the angularis.

167 pear as a rapidly growing cervical, painless nodular mass.

168 nd routine chest roentgenogram shows pleural nodular masses.

169 taneous mastocytosis/plaque mastocytosis, 22 nodular mastocytosis, and nine diffuse cutaneous mastocy

170 present in regions of decreased apoptosis in nodular medulloblastoma.

171 tic differentiation similar to that in human nodular medulloblastomas with activated Shh signaling.

172 ive nodularity, four of 20 with desmoplastic/nodular medulloblastomas, and one of 108 with other subt

173 Superficial spreading melanoma (SSM) and nodular melanoma (NM) are believed to represent sequenti

174 Nodular melanoma (NM) is a rapidly progressing potential

175 es, superficial spreading melanoma (SSM) and nodular melanoma (NM).

176 ole for activated RSK1 in the progression of nodular melanoma and suggest that melanoma originating f

177 cal vigilance is warranted for patients with nodular melanoma and those with the thickest tumors.

178 Nodular melanoma cells were more sensitive to RSK1 inhib

179 Nodular melanoma occurred more frequently as amelanotic/

180 An association was seen with the nodular melanoma subtype (vs superficial spreading [refe

181 5 BRAF mutation in superficial spreading and nodular melanomas, but add further evidence that this mu

182 stologic subtypes, superficial spreading and nodular melanomas, differ in their speed of dermal invas

183 icial spreading melanomas, three of 11 (27%) nodular melanomas, two of 13 (15%) acral lentiginous mel

184 The nodular mesenchyme expresses the related bHLH factors Ha

185 oth 89% (95% CI, 67 to 100) for desmoplastic/nodular (n = 11), 61% (95% CI, 51 to 71) and 75% (95% CI

186 odular benign melanocytic tumors, and 135 of nodular nonmelanocytic tumors were scored for dermoscopy

187 tive correlating features of pigmented NM vs nodular nonmelanoma were peripheral black dots/globules,

188 of the chest revealed multiple thick-walled nodular opacities throughout both lungs.

189 Nodular opacities, reticular opacities, pleural effusion

190 or indeterminate (focal low SI that was not nodular or contour deforming or diffuse low SI).

191 hat appear to predict the presence of either nodular or diffuse peritoneal pathology.

192 The MRI growth patterns were classified as nodular or diffuse.

193 if they had histologically confirmed primary nodular or superficial basal-cell carcinoma at low-risk

194 : thyroiditis (OR = 0.58, p = 1.4 x 10(-5)), nodular (OR = 0.76, p = 3.1 x 10(-5)) and multinodular (

195 daily for 6 weeks (superficial) or 12 weeks (nodular), or surgical excision with a 4 mm margin.

196 DPO was defined as 10 or more bilateral nodular ossifications (definition 1) or as one or more l

197 ne or more lobes with five or more bilateral nodular ossifications (definition 2).

198 e describe P aeruginosa-induced locoregional nodular panniculitis as a distinct entity.

199 3 new cases of P aeruginosa-induced multiple nodular panniculitis without septicemia and describe com

200 nas aeruginosa-induced locoregional multiple nodular panniculitis without septicemia is an underrepor

201 classes of anthelminthic drugs in the swine nodular parasite Oesophagostomum dentatum.

202 CR was achieved in 25% of 177 patients, and nodular partial remission and partial remission were ach

203 ate was 95%, with complete remission in 72%, nodular partial remission in 10%, partial remission due

204 he CR rate was 70% (95% CI, 63% to 76%), the nodular partial remission rate was 10%, and the partial

205 7%) achieved a complete response (CR), one a nodular partial remission, and 10 patients a partial rem

206 6%, including 12% complete responses and 12% nodular partial remissions.

207 ients who achieved complete response (CR) or nodular partial response (nPR).

208 sponse rate was 52% (complete remission, 8%; nodular partial response, 4%; partial response, 40%).

209 a complete response, one patient (3%) had a nodular partial response, and 17 (55%) patients had a pa

210 omplete response with residual cytopenia, 7% nodular partial response, and 43% partial response.

211 e response (CR) rate, defined as complete or nodular partial response, was significantly greater with

212 26 (41%) complete responses (CRs), 14 (22%) nodular partial responses (nodular PRs), and 18 (28%) pa

213 %, with 20% complete responses (CRs) and 20% nodular partial responses.

214 ancement, and two lesions showed a prevalent nodular pattern.

215 We identified parameters that produce nodular patterns that resemble, in size and distribution

216 tly shorter survival and developed extensive nodular peritoneal dissemination with hemorrhagic asciti

217 ic gave a high sensitivity (>98.0%) for both nodular pigmented and nonnodular pigmented melanoma but

218 opsies from 3 patients with maculopapular or nodular post-kala-azar dermal leishmaniasis (PKDL).

219 ieved in 70%, partial remission (PR) in 18%, nodular PR in 3%, for an overall response of 92%.

220 melanoma and that Tiam1 may activate Rac in nodular presentations.

221 uding 30 partial responses (PRs; 47%), three nodular PRs (5%), and one complete response (1.6%).

222 s (CRs), 14 (22%) nodular partial responses (nodular PRs), and 18 (28%) partial responses (PRs).

223 modulated CFRs and SSs in ipsilateral uvula-nodular Purkinje cells, demonstrating that the primary v

224 flammation to extreme hepatocellular injury, nodular regeneration, and bile duct proliferation.

225 long-term safety concerns when implicated in nodular regenerative hyperplasia (NRH) of the liver and

226 Nodular regenerative hyperplasia (NRH) was seen in 9 pat

227 lar remodeling; Notch1 knockout mice develop nodular regenerative hyperplasia (NRH).

228 tion and tetralogy of Fallot associated with nodular regenerative hyperplasia and focal nodular hyper

229 is, liver fibrosis, portal hypertension, and nodular regenerative hyperplasia are discussed in this r

230 stopathological analysis of liver revealed a nodular regenerative hyperplasia.

231 rtension due to periportal liver fibrosis or nodular regenerative hyperplasia.

232 One CFLD patient had nodular regenerative hyperplasia.

233 MR imaging criterion for tumor was a focal nodular region of reduced signal intensity at T2-weighte

234 hese glands as normal, smoothly enlarged, or nodular, respectively.

235 ng along the sinusoids in PIR-B(-/-) mice vs nodular restricted localization in WT mice.

236 Ds was associated with idiopathic diffuse or nodular scleritis with a high degree of scleral inflamma

237 Patients with idiopathic diffuse or nodular scleritis with a high degree of scleral inflamma

238 Ds was associated with idiopathic diffuse or nodular scleritis with a low degree of scleral inflammat

239 Patients with idiopathic diffuse or nodular scleritis with a low degree of scleral inflammat

240 ment with IMT was associated with diffuse or nodular scleritis with associated systemic disease (OR =

241 ent with BRMs was associated with diffuse or nodular scleritis with associated systemic disease (OR =

242 Patients with diffuse or nodular scleritis with associated systemic disease may r

243 9, P < 0.001) and with idiopathic diffuse or nodular scleritis without ocular complications (OR = 3.1

244 nce decreased with declining CD4 counts, but nodular sclerosing decreased more precipitously than mix

245 failure, and kidney biopsy analysis showed a nodular sclerosing GN with extensive focal global glomer

246 histological examination diagnosed Hodgkin's nodular sclerosing histological subtype disease has been

247 With more severe immunosuppression, nodular sclerosing HL becomes infrequent, explaining the

248 ligand/9p24.1 amplification is restricted to nodular sclerosing HL, the cHL subtype most closely rela

249 Nodular sclerosing Hodgkin lymphoma (NSHL) is a distinct

250 LBCL and, in fact, closely resemble those of nodular sclerosing Hodgkin lymphoma (NSHL).

251 e-B-cell lymphoma that is closely related to nodular sclerosing Hodgkin's lymphoma.

252 lastic syndrome caused by stage 2A, grade I, nodular sclerosing Hodgkin's lymphoma.

253 ted tumors were reexamined and classified as nodular-sclerosing (no component of papillary carcinoma)

254 cal behavior and histopathologic features of nodular-sclerosing and papillary tumors were assessed.

255 months) was greater than after resection of nodular-sclerosing lesions (33.5 months, P = 0.013).

256 holangiocarcinoma to that of the more common nodular-sclerosing subtype.

257 Patients with papillary and nodular-sclerosing tumors had similar demographics, oper

258 better outcome compared with the more common nodular-sclerosing type.

259 -rich (81-fold; 95% CI, 30- to 177-fold) and nodular sclerosis (4.6-fold; 95% CI, 2.9- to 7.0-fold) a

260 pecific mortality but only for patients with nodular sclerosis (NS) histologic subtype (hazard ratio

261 low stage (stage I/II) at presentation with nodular sclerosis (NS) histology predominating in 80% of

262 Similar associations were found for nodular sclerosis and mixed cellularity subtypes.

263 Nodular sclerosis and mixed-cellularity subtypes had sim

264 Risk was decreased for both nodular sclerosis and other subtypes.

265 Bulk disease, "B" symptoms, and nodular sclerosis histology were risk factors for inferi

266 thyroidism (thyroid radiation dose, sex, and nodular sclerosis histology), the risk of hypothyroidism

267 logical subtypes (odds ratio [OR] for MC vs. nodular sclerosis histology, 3.2; 95% confidence interva

268 Nodular sclerosis Hodgkin lymphoma (NSHL) and primary me

269 loci within the HLA region are observed for nodular sclerosis Hodgkin lymphoma (rs9269081, HLA-DPB1*

270 B2M-deficient cases encompassed most of the nodular sclerosis subtype cases and only a minority of m

271 ologic features, it clinically resembles the nodular sclerosis subtype of classical Hodgkin lymphoma

272 ent a discovered phenomenon of titanium nano-nodular self-assembly that occurs during physical vapor

273 The discovered titanium nano-nodular self-structuring has been proven feasible on bio

274 ns), eczematous, urticarial, papular, and/or nodular skin lesions were seen.

275 from eczematous or urticarial to papular or nodular skin lesions.

276 scence, and eyes with superficial ODD showed nodular staining.

277 PAVICs grown in OST+TGF-beta1 produced nodular structures staining positive for calcium content

278 r), stiffness, and formation of macroscopic, nodular structures with calcification in the VIC-laden h

279 Nodular subcutaneous and generalized fat necrosis, due t

280 Analysis showed that the desmoplastic/nodular subtype was a favorable factor in predicting sur

281 derly were more common in men, mostly of the nodular subtype, and located in the head and neck region

282 less commonly than superficial spreading or nodular subtypes.

283 ay N-fixation deficiency (25%) and increased nodular superoxide content.

284 nt IV (27 [64%] of 42 vs 10 [24%] of 42) and nodular surface (37 [88%] of 42 vs seven [17%] of 42) we

285 Formation and remodeling of the nodular thickenings at the closure points of the leaflet

286 ludes condensation, elongation, formation of nodular thickenings, and remodeling of tension-resistant

287 apping reveals abnormal fiber projections in nodular tissue suggestive of abnormal organization of wh

288 to the presence of abnormal fiber tracks in nodular tissue.

289 red the pattern of pulmonary metastases from nodular to diffuse and facilitated disease progression.

290 (desmoplastic/nodular versus nondesmoplastic/nodular) to stratify patients for therapy by risk of rel

291 with well-defined tumor margins (P = .013), nodular tumor enhancement (P = .021), and gross appearan

292 Acral lentiginous and nodular tumors, male sex, tumor site on the scalp or nec

293 le tumors developed into more advanced multi-nodular tumors, whereas the female tumors remain uniform

294 lloblastomas (23 classic, eight desmoplastic-nodular, two large cell, one anaplastic), 17 ependymomas

295 All other HCCs were of a nodular type, with similar nodule sizes in the two group

296 endothelial transmigration, MMP16 supported nodular-type growth of adhesive collagen-surrounded mela

297 ological subtypes, including superficial and nodular variants, raising the possibility that morpholog

298 ill use histopathologic typing (desmoplastic/nodular versus nondesmoplastic/nodular) to stratify pati

299 Differences among patients with nodular versus smooth liver surfaces in the proportion w

300 I-VII (central zone, CZ) and posterior IX-X (nodular zone, NZ).