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1                              In all cases of nodular sclerosis (132), mixed cellularity (34), lymphoc
2 iatric cases (two lymphocyte-predominant, 25 nodular sclerosis, 16 mixed cellularity, 5 lymphocyte de
3 -rich (81-fold; 95% CI, 30- to 177-fold) and nodular sclerosis (4.6-fold; 95% CI, 2.9- to 7.0-fold) a
4 RS cells expressed BCL-2 in 359 (65%) of 551 nodular sclerosis, 67 (47%) of 143 mixed cellularity, an
5 rall, 6 (3 T and 3 null) of 49 ALCL and 3 (2 nodular sclerosis and 1 mixed cellularity) of 72 HD show
6                  In glomeruli where advanced nodular sclerosis and global sclerosis were dominant, IL
7  These associations were significant for the nodular sclerosis and mixed cellularity subtypes but wer
8          Similar associations were found for nodular sclerosis and mixed cellularity subtypes.
9                                              Nodular sclerosis and mixed-cellularity subtypes had sim
10                  Risk was decreased for both nodular sclerosis and other subtypes.
11 hat HLA class II loci predispose to sporadic nodular sclerosis HD (NSHD).
12              Bulk disease, "B" symptoms, and nodular sclerosis histology were risk factors for inferi
13 thyroidism (thyroid radiation dose, sex, and nodular sclerosis histology), the risk of hypothyroidism
14 logical subtypes (odds ratio [OR] for MC vs. nodular sclerosis histology, 3.2; 95% confidence interva
15                                              Nodular sclerosis Hodgkin lymphoma (NSHL) and primary me
16  loci within the HLA region are observed for nodular sclerosis Hodgkin lymphoma (rs9269081, HLA-DPB1*
17 th Reed-Sternberg cells in 30 of 35 cases of nodular-sclerosis, lymphocyte-depletion, and mixed-cellu
18      Histologic distribution was as follows: nodular sclerosis (n = 64), lymphocyte predominant (n =
19 pecific mortality but only for patients with nodular sclerosis (NS) histologic subtype (hazard ratio
20  low stage (stage I/II) at presentation with nodular sclerosis (NS) histology predominating in 80% of
21 ession of eotaxin and MDC was highest in the nodular sclerosis (NS) subtype.
22  B2M-deficient cases encompassed most of the nodular sclerosis subtype cases and only a minority of m
23 ologic features, it clinically resembles the nodular sclerosis subtype of classical Hodgkin lymphoma
24 mainly confined to the mixed cellularity and nodular sclerosis subtypes.
25 wo of three lymphocyte-predominant, 26 of 39 nodular, sclerosis, two of two mixed cellularity, and tw
26 ssumed dendritic shapes, particularly in the nodular sclerosis type, producing an interdigitating mes
27 astic cells of Hodgkin's disease, notably in nodular sclerosis type.

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