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2 iatric cases (two lymphocyte-predominant, 25 nodular sclerosis, 16 mixed cellularity, 5 lymphocyte de
3 -rich (81-fold; 95% CI, 30- to 177-fold) and nodular sclerosis (4.6-fold; 95% CI, 2.9- to 7.0-fold) a
4 RS cells expressed BCL-2 in 359 (65%) of 551 nodular sclerosis, 67 (47%) of 143 mixed cellularity, an
5 rall, 6 (3 T and 3 null) of 49 ALCL and 3 (2 nodular sclerosis and 1 mixed cellularity) of 72 HD show
7 These associations were significant for the nodular sclerosis and mixed cellularity subtypes but wer
13 thyroidism (thyroid radiation dose, sex, and nodular sclerosis histology), the risk of hypothyroidism
14 logical subtypes (odds ratio [OR] for MC vs. nodular sclerosis histology, 3.2; 95% confidence interva
16 loci within the HLA region are observed for nodular sclerosis Hodgkin lymphoma (rs9269081, HLA-DPB1*
17 th Reed-Sternberg cells in 30 of 35 cases of nodular-sclerosis, lymphocyte-depletion, and mixed-cellu
19 pecific mortality but only for patients with nodular sclerosis (NS) histologic subtype (hazard ratio
20 low stage (stage I/II) at presentation with nodular sclerosis (NS) histology predominating in 80% of
22 B2M-deficient cases encompassed most of the nodular sclerosis subtype cases and only a minority of m
23 ologic features, it clinically resembles the nodular sclerosis subtype of classical Hodgkin lymphoma
25 wo of three lymphocyte-predominant, 26 of 39 nodular, sclerosis, two of two mixed cellularity, and tw
26 ssumed dendritic shapes, particularly in the nodular sclerosis type, producing an interdigitating mes
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