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1 c coding of TFS and ENV following permanent, noise-induced hearing loss.
2 air cell and synaptic ribbon loss as well as noise-induced hearing loss.
3 dBA over 24 hr) in 2013 and were at risk of noise-induced hearing loss.
4 these Kv2.2KO mice were more susceptible to noise-induced hearing loss.
5 ated between the acute and chronic phases of noise-induced hearing loss.
6 promised hearing sensitivity and potentiated noise-induced hearing loss.
7 d enhance the resistance of the inner ear to noise-induced hearing loss.
8 nd elucidated their protective roles against noise-induced hearing loss.
9 support to a role of GPx1 activity in acute noise-induced hearing loss.
10 be considered a target in the prevention of noise-induced hearing loss.
11 e did, however, show increased resistance to noise-induced hearing loss.
12 oxygen species (ROS) and auditory damage in noise-induced hearing loss.
15 udy provides new insights into mechanisms of noise-induced hearing loss and suggests novel interventi
16 rs may be at risk of permanent, irreversible noise-induced hearing loss and that, for many individual
18 57BL/6J mouse is a model for presbycusis and noise-induced hearing loss because of its age-related he
19 noise intensity dependent and contributes to noise-induced hearing loss by mediating the loss of inne
20 ence of the antioxidant glutathione (GSH) on noise-induced hearing loss by using l-buthionine-[S,R]-s
24 nd efficacy of ebselen for the prevention of noise-induced hearing loss in young adults in a phase 2
29 formation and potential energy depletion in noise induced hearing loss (NIHL), we measured the effec
34 of endogenous glutathione (GSH) potentiates noise-induced hearing loss (NIHL), whereas replenishment
37 e Isl1 overexpression protected the ear from noise-induced hearing loss (NIHL): both ABR threshold sh
40 ffect of the long-term inhibition of MMPs on noise-induced hearing loss was further confirmed using t
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