ライフサイエンスコーパス: non-dividing cell

1 levels of DRAP1 expression in differentiated non dividing cells.

2 CF remains essential for TAD organization in non-dividing cells.

3 tinuous accumulation of repeat expansions in non-dividing cells.

4 s of dGTP or GTP found in either dividing or non-dividing cells.

5 viability, homeostasis and DNA synthesis in non-dividing cells.

6 fficient long-term infection of dividing and non-dividing cells.

7 limitations, with good ability to transfect non-dividing cells.

8 (HIV-1), like other lentiviruses, can infect non-dividing cells.

9 tissues, the H2A-1 gene is expressed in many non-dividing cells.

10 is the ability of the virus to replicate in non-dividing cells.

11 of those prevailing either in cycling or in non-dividing cells.

12 eneration of mutant proteins in dividing and non-dividing cells.

13 nd the nuclear import of the viral genome in non-dividing cells.

14 or contains no viral genes and can transduce non-dividing cells.

15 upply the NLS(s) that enable HIV-1 to infect non-dividing cells.

16 g as replacement histone genes in long-lived non-dividing cells.

17 evelopment of gene-targeting applications in non-dividing cells.

18 Hypotonic stress to flattened, non-dividing cells activated no additional current.

19 ar macrophages as terminally differentiated, non-dividing cells and underscores biological difference

20 other lentiviruses are capable of infecting non-dividing cells and, therefore, need to be imported i

21 BRCA2 is not expressed in the non-dividing cells, and expression is cell cycle stage-d

22 tion of most mammalian cell types, including non-dividing cells, and features are included that give

23 ions reveal a role of heterochronic genes in non-dividing cells, and provide an example of cell-auton

24 In general, non-dividing cells are likely to have low cellular dNTP

25 thrin-coated vesicles occurs continuously in non-dividing cells, but is shut down during mitosis, whe

26 llow dividing cells to be distinguished from non-dividing cells by a greater than two-fold increase i

27 herefore, SAMHD1 expression, particularly in non-dividing cells, can restrict retroviral infections s

28 man immunodeficiency virus type 1 (HIV-1) in non-dividing cells critically depends on import of the v

29 man immunodeficiency virus type 1 (HIV-1) in non-dividing cells depends critically on import of the v

30 upport long-term expression of transgenes in non-dividing cells, exhibiting a decreased risk of inser

31 ral preintegration complex to the nucleus of non-dividing cells following virus entry.

32 hough H3.3-YFP deposition stably remained in non-dividing cells for days after IFN stimulation, it wa

33 The efficacy of in vivo transduction of non-dividing cells has been demonstrated in a wide varie

34 Cilia are found on most non-dividing cells in the human body and, when faulty, c

35 iviral (HIV)-based vector that can transduce non-dividing cells in vitro and deliver genes in vivo.

36 for robust DNA knock-in in both dividing and non-dividing cells in vitro and, more importantly, in vi

37 in activity in the double mutant converted a non-dividing cell into a novel highly proliferating cell

38 The data indicate that YycG activity in non-dividing cells is suppressed by its interaction with

39 ocytes of Xenopus laevis, as an example of a non-dividing cell, is exclusive to the nuclear pore comp

40 s that could produce revertant phenotypes in non-dividing cells of both pro- and eukaryotes, we note

41 ve cells in the late embryonic brainstem are non-dividing cells, presumably immature oligodendrocytes

42 (HIV-1) contributes to viral replication in non-dividing cells, specifically those of the myeloid li

43 iently deliver siRNAs into both dividing and non-dividing cells, stem cells, zygotes, and their diffe

44 ortant mechanism for repairing DNA damage in non-dividing cells such as neurons.

45 iency virus type-1 (HIV-1) is able to infect non-dividing cells such as tissue macrophages productive

46 y type 1 (HIV-1) have the capacity to infect non-dividing cells such as tissue macrophages.

47 s, are capable of efficiently replicating in non-dividing cells (terminally differentiated macrophage

48 oliferation it is also strongly expressed in non-dividing cells undergoing DNA synthesis and repair.

49 Although expansions can accrue in non-dividing cells, we also show that cell cycle arrest

50 Importantly, the rates of expansion in non-dividing cells were at least as high as those of pro

51 includes access of the pDNA to the nuclei of non-dividing cells where the presence of an intact nucle

52 urrent tools are inefficient, especially for non-dividing cells, which compose most adult tissues.