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1     Furthermore, activating granule cells in non-epileptic animals evoked acute seizures of increasin
2                                   In normal, non-epileptic animals, these strongly inhibited dentate
3 were increased in epileptic mice compared to non-epileptic animals.
4 m contralateral non-seizure-generating MTL ("non-epileptic") areas.
5 ine the portrayal of epilepsy, seizures, and non-epileptic attack disorder in 62 movies produced over
6 y of life and other psychosocial issues; and non-epileptic attack disorder.
7 (iii) conversion symptoms (motor, sensory or non-epileptic attacks) (18%).
8 e diagnosis and investigation of psychogenic non-epileptic attacks, drop attacks, falls, syncope and
9               Other associated symptoms were non-epileptic attacks, migraine, fatigue and sleep paral
10 ow-up; five of whom had initial diagnoses of non-epileptic attacks.
11 ilepsy rats from Strasbourg (GAERS), and its non-epileptic control (NEC) strain.
12  epileptic rats when compared to age-matched non-epileptic control rats.
13 mygdalas and hippocampi were conducted in 50 non-epileptic controls (age 7-79 years) and 50 patients
14 ment of these mice with GABAA antagonists at non-epileptic doses causes a persistent post-drug recove
15 at least 10 years of age (7367 artefact-free non-epileptic electrodes), whereas a younger group inclu
16 l autonomic manifestations were dismissed as non-epileptic events of other diseases.
17 gnificantly higher burst rates compared with non-epileptic hippocampal neurons (both p = 0.01).
18 erence in burst firing between epileptic and non-epileptic hippocampal neurons was observed.
19  between epileptic MTL neurons compared with non-epileptic MTL neurons during SWS (p = 0.04) and REM
20 t disorder characterized by a combination of non-epileptic myoclonic jerks and dystonia.
21 al synchrony was found between epileptic and non-epileptic neurons during Aw.
22 in resulted in a 90% clinical improvement in non-epileptic paroxysmal manifestations and a normalised
23 patients with GLUT1-DS (7-47 years old) with non-epileptic paroxysmal manifestations.
24 50 epileptic patients with ictal deja vu, 50 non-epileptic patients attending general neurology clini
25  hyperandrogenemia), this was not evident in non-epileptic primates.
26      Some evidence suggests that psychogenic non-epileptic seizures (PNES) are associated with increa
27                                  Psychogenic non-epileptic seizures (PNES) are classified with other
28 of 260 consecutive patients with psychogenic non-epileptic seizures (PNES), 5-10 years after diagnosi
29  studies of long-term outcome in psychogenic non-epileptic seizures (PNES), and none of long-term hea
30 n the treatment of patients with psychogenic non-epileptic seizures and generates ideas for future re
31 While the diagnostic features of psychogenic non-epileptic seizures have been better characterized in
32 d that, first, patients tend to transit from non-epileptic to epileptic states more often than contro

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