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1 n in 49 men (26 without diabetes and 23 with non-insulin-dependent diabetes mellitus).
2 ing cause of both morbidity and mortality in non-insulin dependent diabetes mellitus.
3 h may reduce the risk of vascular disease in non-insulin dependent diabetes mellitus.
4 esistance and is the biggest risk factor for non-insulin-dependent diabetes mellitus.
5 ionship of ob gene expression to obesity and non-insulin-dependent diabetes mellitus.
6 c oxide-mediated vasodilation is impaired in non-insulin-dependent diabetes mellitus.
7 riants of IRS-1, especially in patients with non-insulin-dependent diabetes mellitus.
8 etic (ZDF/Gmifa) rats were used as models of non-insulin-dependent diabetes mellitus.
9 with IIP and MDI is effective in controlling non-insulin-dependent diabetes mellitus.
10 iabetes, tropical calcific pancreatitis, and non-insulin-dependent diabetes mellitus.
11 have been proposed as a potential cause for non-insulin-dependent diabetes mellitus.
12 forearm resistance vessels of patients with non-insulin-dependent diabetes mellitus.
13 role of islet amyloid in the pathogenesis of non-insulin-dependent diabetes mellitus.
14 b gene in these animal models of obesity and non-insulin-dependent diabetes mellitus.
15 hyperglycemia in both insulin-dependent and non-insulin-dependent diabetes mellitus.
16 ed vasodilation is abnormal in patients with non-insulin-dependent diabetes mellitus.
17 contribute to the insulin resistance seen in non-insulin-dependent diabetes mellitus.
18 as are a class of drugs widely used to treat non-insulin-dependent diabetes mellitus.
19 ay be useful in the treatment of obesity and non-insulin-dependent diabetes mellitus.
20 of obesity, including insulin resistance and non-insulin-dependent diabetes mellitus.
21 the insulin resistance syndrome and risk of non-insulin-dependent diabetes mellitus.
22 hibits a phenotype that includes obesity and non-insulin-dependent diabetes mellitus.
23 to the most common complication of obesity, non-insulin-dependent diabetes mellitus.
24 significantly reduce the risk of developing non-insulin-dependent diabetes mellitus.
25 o the impaired insulin secretion observed in non-insulin-dependent diabetes mellitus.
26 del of the insulin resistance of obesity and non-insulin-dependent diabetes mellitus.
27 an important role in the pathophysiology of non-insulin-dependent diabetes mellitus.
28 -HD genes result in genetic diseases such as non-insulin-dependent diabetes mellitus.
29 condition similar to adult-onset obesity and non-insulin-dependent diabetes mellitus.
30 ole in the insulin resistance of obesity and non-insulin-dependent diabetes mellitus.
31 death for any MPS result than patients with non-insulin-dependent diabetes mellitus.
32 e production characteristic of patients with non-insulin-dependent diabetes mellitus.
33 ms, and its levels are increased in serum in non-insulin-dependent diabetes mellitus.
34 non-full sibs in a genetic linkage study of non-insulin-dependent diabetes mellitus.
35 sion may be modulated in disease states like non-insulin-dependent diabetes mellitus.
36 dividuals with impaired glucose tolerance or non-insulin-dependent diabetes mellitus.
37 for type I (insulin-dependent) and type II (non-insulin-dependent) diabetes mellitus.
38 dds ratio, 11.2; P = 0.04), and 6 of 43 with non-insulin-dependent diabetes mellitus (14%; odds ratio
39 rearm resistance vessels of 21 patients with non-insulin-dependent diabetes mellitus and 23 matched h
40 s associated with greatly increased risks of non-insulin-dependent diabetes mellitus and cardiovascul
41 s are a characteristic pathologic feature of non-insulin-dependent diabetes mellitus and contain isle
42 resistant (IR) offspring of individuals with non-insulin-dependent diabetes mellitus and eight age-we
43 dence of such complications in patients with non-insulin-dependent diabetes mellitus and hypertension
44 n important adjunct in the treatment of both non-insulin-dependent diabetes mellitus and insulin-depe
45 aired in patients with insulin-dependent and non-insulin-dependent diabetes mellitus and restored by
46 ortunity to screen the gene in patients with non-insulin-dependent diabetes mellitus and/or obesity f
47 ncidence of adult hypertension, incidence of non-insulin-dependent diabetes mellitus, and prevalence
48 disease that accompanies obesity and related non-insulin-dependent diabetes mellitus, and that the ad
50 obesity, hypertension, hyperlipidaemia, and non-insulin-dependent diabetes mellitus are set against
52 ng individuals already at increased risk for non-insulin-dependent diabetes mellitus because of obesi
53 forearm resistance vessels of patients with non-insulin-dependent diabetes mellitus can be improved
54 nset diabetes of the young (MODY), a form of non-insulin-dependent diabetes mellitus characterized by
55 m the Finland-United States Investigation of Non-Insulin-Dependent Diabetes Mellitus (FUSION) Genetic
56 m the Finland-United States Investigation of Non-Insulin-Dependent Diabetes Mellitus (FUSION) study.
57 m the Finland-United States Investigation of Non-Insulin-Dependent Diabetes Mellitus Genetics study a
58 eting exercise, IR offspring of parents with non-insulin-dependent diabetes mellitus had (i) normal r
59 hile it is well established that people with non-insulin dependent diabetes mellitus have defects in
60 nsorineural hearing loss, childhood obesity, non-insulin-dependent diabetes mellitus, hyperlipidemia
62 to improve insulin resistance in obesity and non-insulin-dependent diabetes mellitus in both rodents
63 nvestigation of agouti's role in obesity and non-insulin-dependent diabetes mellitus in mice holds si
64 y be critical in the long-term management of non-insulin-dependent diabetes mellitus in obese patient
65 .1 region linked with insulin resistance and non-insulin-dependent diabetes mellitus in the Pima Indi
66 d with increased risk of type 2 diabetes, or non-insulin-dependent diabetes mellitus, in Mexican Amer
67 he abnormalities associated with early stage non-insulin-dependent diabetes mellitus, including gluco
68 both type 1 (insulin-dependent) and type 2 (non-insulin-dependent) diabetes mellitus, including isle
69 e hypothesis that endothelial dysfunction in non-insulin dependent diabetes mellitus is the consequen
72 r an autosomal dominant, early-onset form of non-insulin-dependent diabetes mellitus (maturity-onset
73 hepatic glucose production in obesity and/or non-insulin-dependent diabetes mellitus may reflect resi
75 the development of one form of early-onset, non-insulin-dependent diabetes mellitus, MODY1, as well
76 tropical calcific pancreatitis (n = 15), and non-insulin-dependent diabetes mellitus (n = 43) and con
78 opment of several chronic diseases including non-insulin dependent diabetes mellitus (NIDDM) and coro
79 and fetal thinness have been associated with non-insulin dependent diabetes mellitus (NIDDM) and insu
80 tudy tested the hypothesis that persons with non-insulin dependent diabetes mellitus (NIDDM) have gre
81 ales) suffering from periodontal disease and non-insulin dependent diabetes mellitus (NIDDM) were ran
87 ew the effect of weight on the prevalence of non-insulin-dependent diabetes mellitus (NIDDM) and of i
88 controls and obese subjects with or without non-insulin-dependent diabetes mellitus (NIDDM) during n
90 ontributing to postprandial hyperglycemia in non-insulin-dependent diabetes mellitus (NIDDM) has not
92 s of the pathophysiology of renal disease in non-insulin-dependent diabetes mellitus (NIDDM) have bee
93 andidate responsible for the pathogenesis of non-insulin-dependent diabetes mellitus (NIDDM) in human
94 ompound troglitazone, which is used to treat non-insulin-dependent diabetes mellitus (NIDDM) in man,
95 tle, if any, impact on the long-term risk of non-insulin-dependent diabetes mellitus (NIDDM) in the g
97 is that severe periodontitis in persons with non-insulin-dependent diabetes mellitus (NIDDM) increase
102 resistance in the offspring of parents with non-insulin-dependent diabetes mellitus (NIDDM) is the b
106 ivity remains impaired in skeletal muscle of non-insulin-dependent diabetes mellitus (NIDDM) patients
107 measure of insulin sensitivity in studies of non-insulin-dependent diabetes mellitus (NIDDM) risk, bu
109 gated the role of MIF in the pathogenesis of non-insulin-dependent diabetes mellitus (NIDDM) using MI
110 iabetes mellitus (IDDM) and 15 patients with non-insulin-dependent diabetes mellitus (NIDDM) were mat
111 sulin-dependent diabetes mellitus (IDDM) and non-insulin-dependent diabetes mellitus (NIDDM) were ran
112 abetes mellitus ($1996 per QALY), those with non-insulin-dependent diabetes mellitus (NIDDM) who use
113 al placebo regimen in over 600 patients with non-insulin-dependent diabetes mellitus (NIDDM) who were
114 pendent diabetes mellitus, 26% of those with non-insulin-dependent diabetes mellitus (NIDDM) who were
115 R was 5.4 micrograms/minute for persons with non-insulin-dependent diabetes mellitus (NIDDM) who were
116 isposal is a common finding in patients with non-insulin-dependent diabetes mellitus (NIDDM), as well
117 sensitivity in obesity and in patients with non-insulin-dependent diabetes mellitus (NIDDM), by acti
118 orted positive result from a genome scan for non-insulin-dependent diabetes mellitus (NIDDM), Hanis e
119 g schizophrenia, bipolar affective disorder, non-insulin-dependent diabetes mellitus (NIDDM), inflamm
120 th insulin resistance syndrome (IRS) such as non-insulin-dependent diabetes mellitus (NIDDM), obesity
121 insulin resistance have been associated with non-insulin-dependent diabetes mellitus (NIDDM), obesity
122 s (18 to 97 years of age) with hypertension, non-insulin-dependent diabetes mellitus (NIDDM), recent
142 been proved to be caused by mtDNA mutations; non-insulin-dependent diabetes mellitus (NIDDM); and hyp
146 d to diseases with late age at onset such as non-insulin-dependent diabetes mellitus, psychiatric dis
148 had comparable defects in glucose clearance, non-insulin dependent diabetes mellitus subjects also ha
149 Therefore, nondiabetic (lean and obese) and non-insulin dependent diabetes mellitus subjects were st
150 ith islets from rodent models of obesity and non-insulin-dependent diabetes mellitus, suggesting the
151 s have suggested linkage of Type 2 diabetes (non-insulin-dependent diabetes mellitus) susceptibility
152 cept natives of Alaska, have a prevalence of non-insulin-dependent diabetes mellitus that is two to s
153 ated in a model of type 2 diabetes mellitus (non-insulin-dependent diabetes mellitus), the ob/ob mous
154 nge affect the risk for diabetes, especially non-insulin-dependent diabetes mellitus, the authors exa
156 hts (lean to very obese) and with or without non-insulin-dependent diabetes mellitus to examine the r
157 lin-dependent diabetes mellitus (type 1) and non-insulin dependent diabetes mellitus (type 2) after l
159 vascular morphology and function in type 2 (non-insulin-dependent) diabetes mellitus (type 2D), smal
160 pancreatic diseases in Bangladesh, including non-insulin-dependent diabetes mellitus, was undertaken.
161 ent with mixed connective tissue disease and non-insulin-dependent diabetes mellitus who developed an
162 may portend an increase in the incidence of non-insulin-dependent diabetes mellitus with important p
163 the presence of enhanced oxidative stress in non-insulin dependent diabetes mellitus, with recent dat
164 tify the genetic basis for susceptibility to non-insulin-dependent diabetes mellitus within the conte
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