ライフサイエンスコーパス: non-melanoma skin cancer

1 tis, viral warts, molluscum contagiosum, and non-melanoma skin cancer.

2 asal alar lobule after two-layer excision of non-melanoma skin cancer.

3 story of photosensitizing medication use and non-melanoma skin cancer.

4 as for the identification of target cells in non-melanoma skin cancer.

5 facing to reduce or prevent aging-associated non-melanoma skin cancer.

6 A confers an increased risk for melanoma and non-melanoma skin cancer.

7 are novel loci conferring susceptibility to non-melanoma skin cancer.

8 ated by UV irradiation, the primary cause of non-melanoma skin cancer.

9 omponent in the development of aging-related non-melanoma skin cancer.

10 e development and progression of UVB-induced non-melanoma skin cancer.

11 tologists treat actinic keratoses to prevent non-melanoma skin cancer.

12 s a significant factor in the development of non-melanoma skin cancer.

13 dministration of ARD on at least one type of non-melanoma skin cancer.

14 HPV8 and 77) are suspected to be involved in non-melanoma skin cancer.

15 arge, population-based case-control study of non-melanoma skin cancer.

16 ll-recognized etiologic factor for cutaneous non-melanoma skin cancer.

17 ynamic therapy (PDT) is widely used to treat non-melanoma skin cancer.

18 mental factors contribute to pathogenesis of non-melanoma skin cancers.

19 43; and with skin cancer (Bowen's disease or non-melanoma skin cancer), 378.

20 oming available, especially for treatment of non-melanoma skin cancer and Barrett's oesophagus, and i

21 cell carcinoma (SCC) is the most aggressive non-melanoma skin cancer and is dramatically increased i

22 dent cancer cases were documented (excluding non-melanoma skin cancer and non-aggressive prostate can

23 r transmission from donors with a history of non-melanoma skin cancer and selected cancers of the CNS

24 ln399gln) is associated with a lower risk of non-melanoma skin cancer and suggest that the etiology o

25 nts as evidenced by the fact that 80% of all non-melanoma skin cancers are diagnosed in patients over

26 treatment of pre-cancerous skin lesions and non-melanoma skin cancers are not completely effective.

27 issue and ease of observation, acceptance of non-melanoma skin cancers as model carcinomas has been h

28 TP53 is an accepted UVR target in human non-melanoma skin cancer, but is not thought to have a m

29 for the majority of the approximately 10,000 non-melanoma skin cancer deaths in the United States ann

30 s, leading to more than one million cases of non-melanoma skin cancer diagnosed annually in the Unite

31 ity for all incident cancer cases, excluding non-melanoma skin cancers, diagnosed between 2002 and 20

32 's lymphoma (SIR=28.56, 95% CI, 7.68-73.11), non-melanoma skin cancer (estimated SIR> or =3.16) and f

33 We have demonstrated that non-melanoma skin cancers express functional purinergic

34 The rising incidence and morbidity of non-melanoma skin cancers has generated great interest i

35 an early warning sign of progression toward non-melanoma skin cancer, if ignored.

36 tions in p53 were detected in 11/23 (48%) of non melanoma skin cancers in renal allograft recipients

37 en shown to contribute to the development of non-melanoma skin cancer in humans.

38 om an incident survey of all newly diagnosed non-melanoma skin cancer in New Hampshire, and controls

39 een implicated in the increased incidence of non-melanoma skin cancer in transplant recipients, most

40 Another group of HPVs causes non-melanoma skin cancers in genetically predisposed or

41 trum of HPV types are also commonly found in non-melanoma skin cancers in immunocompromised individua

42 Non-melanoma skin cancer is a disease primarily afflicti

43 The development of extensive and severe non-melanoma skin cancer is an extremely common complica

44 Diagnosis of non-melanoma skin cancer is made clinically and confirme

45 mulation of genetic change and behaviour for non-melanoma skin cancer is not straightforward.

46 This approach is limited because non-melanoma skin cancer is predominantly formed on body

47 Non-melanoma skin cancer is the most common cancer world

48 Currently, the only effective treatment for non-melanoma skin cancer is the removal of the tumors af

49 The incidence of keratinocyte-derived (non-melanoma) skin cancers is increasing rapidly.

50 For example, unlike non-melanoma skin cancers, melanoma is not restricted to

51 iagnosed with cancer before 1986 (other than non-melanoma skin cancer, n=2076) and those with missing

52 This approach is limited because non-melanoma skin cancer (NMSC) is predominantly formed

53 Non-melanoma skin cancer (NMSC) is the most common malig

54 Non-melanoma skin cancer (NMSC) represents a significant

55 sue injury, represents a clinical marker for non-melanoma skin cancer (NMSC) risk.

56 genes was related to EMAST in a series of 61 non-melanoma skin cancer (NMSC) tumors.

57 Given the high incidence of non-melanoma skin cancer (NMSC), a preventative interven

58 sociation between UVB and the development of non-melanoma skin cancer (NMSC), controlling for known c

59 type are risk factors for the development of non-melanoma skin cancer (NMSC), including basal cell ca

60 buting factor in ultraviolet B (UVB)-induced non-melanoma skin cancer (NMSC), which consists primaril

61 genes are associated with susceptibility to non-melanoma skin cancer (NMSC).

62 iseases is associated with decreased risk of non-melanoma skin cancer (NMSC).

63 r of sunburns, tanning ability and number of non-melanoma skin cancers (NMSCs) among 10 183 European

64 Non-melanoma skin cancers (NMSCs) are among the most com

65 Non-melanoma skin cancers (NMSCs) are the most common ma

66 73 malignancies other than non-melanoma skin cancer occurred (SIR 0.9 [95% CI 0.7-1

67 =50% size of alar subunit) after excision of non-melanoma skin cancer on the alar lobule.

68 Non-melanoma skin cancer represents the most common canc

69 HPV DNA was detected in 15 of 20 (75%) non-melanoma skin cancer, seven of 17 (41.2%) dysplastic

70 Non-melanoma skin cancer, the most common neoplasia afte

71 rincipal aetiological factor associated with non-melanoma skin cancer, the most prevalent group of ma

72 mouse model of UVB-induced inflammation and non-melanoma skin cancer to further define sex discrepan

73 st examples of video-mosaics of melanoma and non-melanoma skin cancers, to demonstrate potential clin