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1 to induce DNA damage associated apoptosis in non-small cell lung carcinoma.
2 l tissue confirmed RS/DJ-1 overexpression in non-small cell lung carcinoma.
3  G1 cyclins D1 and E is reported in invasive non-small cell lung carcinoma.
4 ction of treatment response in patients with non-small cell lung carcinoma.
5 al microRNA (miR) expression is described in non-small cell lung carcinoma.
6 eatic carcinoma as well as in the H125 human non-small-cell lung carcinoma.
7 urred in patients with refractory breast and non-small-cell lung carcinoma.
8 d DUR index in 13 patients with grade 3 or 4 non-small-cell lung carcinoma.
9 ossibility of distinguishing small-cell from non-small-cell lung carcinoma.
10 ients with non-metastatic surgically treated non-small-cell lung carcinoma.
11  that has been investigated in patients with non-small-cell lung carcinoma.
12 e of angiogenesis) as a prognostic factor in non-small-cell lung carcinoma.
13 r p53 mutation or both (P = 0.01) in primary non-small cell lung carcinomas.
14 ll lung carcinomas and to a lesser extent in non-small cell lung carcinomas.
15 tor-positive gliomas, breast carcinomas, and non-small cell lung carcinomas.
16 und in the human bronchial epithelium and in non-small cell lung carcinomas.
17 ChR) stimulate growth of both small cell and non-small cell lung carcinomas.
18              MAGE-A4 was expressed in 48% of non-small cell lung carcinomas.
19 ed with the development of a subset of human non-small cell-lung carcinomas.
20  whose expression is lost in >50% of primary non-small-cell lung carcinomas.
21 arization in a series of 500 primary stage I non-small-cell lung carcinomas.
22 ), pancreatic duct carcinoma (4 of 4 cases), non-small cell lung carcinoma (5 of 5 cases), soft tissu
23 2 small cell lung cancer cell lines, 6 of 18 non-small cell lung carcinomas, 9 of 22 breast tumors, a
24 f 42) of breast carcinomas, 45% (5 of 11) of non-small cell lung carcinomas, 90% (9 of 10) of colon c
25 ted here demonstrate that treatment of human non-small cell lung carcinoma and bladder carcinoma cell
26 s, which are widely used in the treatment of non-small cell lung carcinoma and other late-stage tumor
27 p14ARF in 53 human cell lines and 86 primary non-small cell lung carcinomas and correlated this with
28 m A (RASSF1A) was observed in 40% of primary non-small cell lung carcinomas and in several tumor cell
29 of-the-art, mutant Kras-driven GEMMs--one of non-small-cell lung carcinoma and another of pancreatic
30 L/TRAIL sensitivity in pancreatic carcinoma, non-small-cell lung carcinoma and melanoma cell lines, a
31        MTDIA treatment inhibited A549 (human non-small cell lung carcinoma) and H358 (human bronchiol
32 l tissues but has been described in gliomas, non-small cell lung carcinomas, and breast carcinomas, h
33 p11.3, which is lost in approximately 60% of non-small cell lung carcinomas, and exhibits growth-supp
34 tomas, H-69 small cell lung carcinomas, H-23 non-small cell lung carcinomas, and MDA-MB-468 breast ca
35 static mesothelioma (n = 2), chemorefractory non-small-cell lung carcinoma, and bronchioloalveolar ca
36 rins, expressed on the cell surface of human non-small cell lung carcinomas, are used here as a targe
37  including childhood acute myeloid leukemia; non-small cell lung carcinoma; arthrogryposis multiplex
38 nstructed a genome-wide transcriptome map of non-small cell lung carcinomas based on gene-expression
39 mal growth factor receptor found on gliomas, non-small cell lung carcinomas, breast carcinomas, and o
40 ally effective in vivo against NCI-H23 human non-small-cell lung carcinomas, but this modality was su
41 evaluated against the COR-L23 parental human non small cell lung carcinoma cell line and the COR-L23/
42  show that siRNA-mediated TRPC1 depletion in non small cell lung carcinoma cell lines induced G(0)/G(
43 , mice were inoculated subcutaneously with a non-small cell lung carcinoma cell line and treated with
44 beta)-induced COX-2 expression in H358 human non-small cell lung carcinoma cell line by blocking CDK2
45 human liver AKN-1 cell line and in the human non-small cell lung carcinoma cell line, A-549, the abil
46                  However, 24-h exposure of a non-small cell lung carcinoma cell line, NCI H157, to 10
47  3p12, and a 3p14.2 homozygous deletion in a non-small cell lung carcinoma cell line.
48 ung cancer in 13 small cell carcinoma and 17 non-small cell lung carcinoma cell lines and in 68 micro
49 G1 arrest and rapid apoptosis in three human non-small cell lung carcinoma cell lines with wild-type
50         When reintroduced into nonexpressing non-small cell lung carcinoma cell lines, this gene, nam
51 ted with the cytotoxicity of meayamycin B in non-small cell lung carcinoma cell lines.
52 ed levels of Bcl-2 and Bcl-XL proteins and a non-small-cell lung carcinoma cell line (NCI-H460).
53  play no direct role in the MDR phenotype in non-small cell lung carcinoma cells and that their cellu
54 sms by which EZH2 expression is regulated in non-small cell lung carcinoma cells by oncogenic KRAS.
55 erent administration schedules in A549 human non-small cell lung carcinoma cells in vitro.
56 ng overexpression of the mda-7 gene in human non-small cell lung carcinoma cells in vivo and its effe
57        In concordance, cultured SCLC but not non-small cell lung carcinoma cells showed no or extreme
58 arcinoma) and H358 (human bronchioloalveolar non-small cell lung carcinoma cells) xenograft tumor gro
59  in xenograft models based on human NCI-H460 non-small cell lung carcinoma cells.
60 dria and induced apoptosis in p53-null H1299 non-small cell lung carcinoma cells.
61 ced growth inhibition and apoptosis in human non-small cell lung carcinoma cells.
62 al interaction between MTOR and ponatinib in non-small cell lung carcinoma cells.
63  diaminobutanoic acid (9) against A549 human non-small-cell lung carcinoma cells in culture were 23 a
64                             In patients with non-small cell lung carcinoma, differential display show
65                                        Human non-small cell lung carcinoma expressed varying levels o
66 eletion at the FHIT locus in a series of 106 non-small cell lung carcinomas for which a full clinical
67                            Here, using human non-small-cell lung carcinoma H1299 cells, we investigat
68 knocking down SSRP1 or Spt16 levels in human non-small cell lung carcinoma (H1299) cells.
69               Recent advances in therapy for non-small cell lung carcinoma have shown that a personal
70 on or suppression of pulmonary metastases of non-small cell lung carcinoma in both prophylactic and t
71  of cancer-related mortality worldwide, with non-small-cell lung carcinomas in smokers being the pred
72                     A 60-year-old man with a non-small cell lung carcinoma (large cell type) presente
73 on in multiple tumor types, the RB-defective non-small cell lung carcinoma line H2009 and its RB-reco
74  sensitivities persisted in an additional 22 non-small cell lung carcinoma lines (ras mutations, n =
75 as detected in 40% of the tumors, whereas in non-small cell lung carcinomas, lymphomas, and head and
76 ressed in human hepatocellular carcinoma and non-small cell lung carcinoma, may facilitate cancer cel
77  of malignant cell lines including leukemia, non-small cell lung carcinoma, melanoma, and breast canc
78                          Using a Kras-driven non-small cell lung carcinoma mouse model, we found that
79 have demonstrated that some other aggressive non-small-cell lung carcinomas (n-SCLC) do not have angi
80  (MCF-7), colorectal adenocarcinoma (HT-29), non-small cell lung carcinoma (NCI-H460), and glioblasto
81 ) arm of chromosome 3 is a common finding in non-small cell lung carcinoma (NSCLC) and is postulated
82 ng both small cell lung carcinoma (SCLC) and non-small cell lung carcinoma (NSCLC) and the second on
83 w that VCP is significantly overexpressed in non-small cell lung carcinoma (NSCLC) as compared to nor
84 sed to identify chromosomal imbalances in 20 non-small cell lung carcinoma (NSCLC) biopsies and cell
85           Microarray data from cell lines of Non-Small Cell Lung Carcinoma (NSCLC) can be used to loo
86  in tobacco-related lung disease, stimulates non-small cell lung carcinoma (NSCLC) cell growth and su
87 onectin (Fn) stimulates the proliferation of non-small cell lung carcinoma (NSCLC) cell growth throug
88 expressed EGFR at levels comparable with the non-small cell lung carcinoma (NSCLC) cell line A549, as
89 ombesin receptor subtype 3 [BRS-3]) in human non-small cell lung carcinoma (NSCLC) cell lines and bro
90                Because the majority of human non-small cell lung carcinoma (NSCLC) cell lines are res
91 ntegrative genomic and proteomic analysis of non-small cell lung carcinoma (NSCLC) cell lines reveale
92  kinase inhibitor PF2341066 in MET-amplified non-small cell lung carcinoma (NSCLC) cell lines to iden
93  loss of DLC-1 protein expression identifies non-small cell lung carcinoma (NSCLC) cell lines whose g
94             This method was applied to human non-small cell lung carcinoma (NSCLC) cell lines, embedd
95 FN), a matrix glycoprotein, stimulates human non-small cell lung carcinoma (NSCLC) cell proliferation
96 e previously showed that nicotine stimulates non-small cell lung carcinoma (NSCLC) cell proliferation
97 or cell proliferation and apoptosis in human non-small cell lung carcinoma (NSCLC) cells in vitro and
98 to be a potent inducer of apoptosis in human non-small cell lung carcinoma (NSCLC) cells through a nu
99 reductase, the silencing of which sensitized non-small cell lung carcinoma (NSCLC) cells to the cytot
100 S1 was also essential for PML degradation in non-small cell lung carcinoma (NSCLC) cells, and PML and
101                      In studies performed in non-small cell lung carcinoma (NSCLC) cells, we found th
102  in various tumor cell lines including human non-small cell lung carcinoma (NSCLC) cells, which are r
103 nd discover the molecular targets of FHIT in non-small cell lung carcinoma (NSCLC) cells.
104 ated death worldwide, and among this cancer, non-small cell lung carcinoma (NSCLC) comprises the majo
105 tablished that PKCvarepsilon is required for non-small cell lung carcinoma (NSCLC) growth in vitro as
106       Here, we demonstrate that HH-dependent non-small cell lung carcinoma (NSCLC) growth is sensitiv
107 gnosis significance of TIL subpopulations in non-small cell lung carcinoma (NSCLC) have thus far not
108 ted from static and parametric PET images of non-small cell lung carcinoma (NSCLC) in order to provid
109 have demonstrated that NE differentiation in non-small cell lung carcinoma (NSCLC) is not uncommon.
110                                              Non-small cell lung carcinoma (NSCLC) is promoted by the
111                                              Non-small cell lung carcinoma (NSCLC) is the leading cau
112 lthough the Tumor-Node-Metastasis staging of non-small cell lung carcinoma (NSCLC) is the most effect
113 LKB1 tumor suppressor is a frequent event in non-small cell lung carcinoma (NSCLC) leading to the act
114 to EGF receptor (EGFR)-targeted therapies in non-small cell lung carcinoma (NSCLC) led to investigati
115 he EGF receptor (EGFR) are effective in most non-small cell lung carcinoma (NSCLC) patients whose tum
116 othymidine ((18)F-FLT) PET in advanced-stage non-small cell lung carcinoma (NSCLC) patients with an a
117 an be targeted to overcome the resistance of non-small cell lung carcinoma (NSCLC) to the AKT inhibit
118  (GHRH) MZ-J-7-138 and JV-1-92 on H460 human non-small cell lung carcinoma (NSCLC) xenografted orthot
119 isms of action were investigated in NCI-H838 non-small cell lung carcinoma (NSCLC) xenografted s.c. i
120 promise to treat inoperable locally-advanced non-small cell lung carcinoma (NSCLC), a disease poorly
121 loss at chromosome 17q in the development of non-small cell lung carcinoma (NSCLC), and a number of k
122 umor-driving genes in patients with advanced non-small cell lung carcinoma (NSCLC), especially in tho
123 on characteristic of many cancers, including non-small cell lung carcinoma (NSCLC), head and neck squ
124 in pancreatic ductal adenocarinoma (PDAC) or non-small cell lung carcinoma (NSCLC), respectively, but
125 e overall survival in never smokers who have non-small cell lung carcinoma (NSCLC), we conducted a co
126 f deletion involving region 8p21-22 in human non-small cell lung carcinoma (NSCLC), we examined alter
127            As JNK is frequently activated in non-small cell lung carcinoma (NSCLC), we investigated t
128 his study was to characterize the effects of non-small cell lung carcinoma (NSCLC)-associated mutatio
129                            The presence of a non-small cell lung carcinoma (NSCLC)-related gene or ge
130 cated in the growth and progression of human non-small cell lung carcinoma (NSCLC).
131  PET metrics and histopathologic response in non-small cell lung carcinoma (NSCLC).
132 erozygosity (LOH) on chromosome 3 at 3p21 in non-small cell lung carcinoma (NSCLC).
133 me 11q22-23 is observed at high frequency in non-small cell lung carcinoma (NSCLC).
134 in both small cell lung carcinoma (SCLC) and non-small cell lung carcinoma (NSCLC).
135 ng anaplastic large-cell lymphoma (ALCL) and non-small cell lung carcinoma (NSCLC).
136 dazole ((18)F-FMISO) uptake in patients with non-small cell lung carcinoma (NSCLC).
137 rmal tissue pairs from Chinese patients with non-small cell lung carcinoma (NSCLC).
138 alent in epithelial cancers, particularly in non-small cell lung carcinoma (NSCLC).
139 correlated with prognosis and progression of non-small cell lung carcinoma (NSCLC).
140  normal tissue samples from 17 patients with non-small cell lung carcinoma (NSCLC).
141 otherapy in patients with radically resected non-small cell lung carcinoma (NSCLC).
142 romising new therapeutic agents for treating non-small cell lung carcinoma (NSCLC).
143 adioresistance in several cancers, including non-small cell lung carcinoma (NSCLC).
144  (TRAIL) is capable of inducing apoptosis in non-small cell lung carcinoma (NSCLC).
145 static disease to the brain in patients with non-small cell lung carcinoma (NSCLC).
146 BMP-2 is highly expressed in the majority of non-small cell lung carcinomas (NSCLC) but not in normal
147 vated in some lung tumors, the dependence of non-small cell lung carcinomas (NSCLC) for HH activity h
148 on protein is found in approximately 3-7% of non-small cell lung carcinomas (NSCLC).
149 on is increased in lung cancer, particularly non-small cell lung carcinomas (NSCLC).
150                                              Non-small-cell lung carcinoma (NSCLC) accounts for 85% o
151                      Our current study using non-small-cell lung carcinoma (NSCLC) cell lines, animal
152  to its inhibitors and restores autophagy in non-small-cell lung carcinoma (NSCLC) cells with a TKI-s
153 roves the outcomes of patients with advanced non-small-cell lung carcinoma (NSCLC) harbouring epiderm
154              As therapy for locally advanced non-small-cell lung carcinoma (NSCLC) improves, brain me
155                                              Non-small-cell lung carcinoma (NSCLC) is among the deadl
156 ified the elevated level of EAPII protein in non-small-cell lung carcinoma (NSCLC) patients and NSCLC
157 identified that predict clinical response of non-small-cell lung carcinoma (NSCLC) patients to gefiti
158 ment of second- and third-line patients with non-small-cell lung carcinoma (NSCLC) with the epidermal
159 ceptor (EGFR) is frequently overexpressed in non-small-cell lung carcinoma (NSCLC), and EGFR inhibito
160 ions in small-cell lung carcinoma (SCLC) and non-small-cell lung carcinoma (NSCLC), the extent of suc
161  is more accurate than CT for the staging of non-small-cell lung carcinoma (NSCLC).
162 d clinical outcome in patients with resected non-small-cell lung carcinoma (NSCLC; n = 248).
163  Immunohistochemical analysis of a cohort of non-small-cell lung carcinomas (NSCLC) indicated that 15
164 CLL); (4) normal versus colon tumor; and (5) Non-Small-Cell-Lung-Carcinoma (NSCLC) versus renal sampl
165 cember 2013, solid tumour samples (including non-small-cell lung carcinoma [NSCLC], colorectal carcin
166  the MAP kinase pathway activation common in non-small cell lung carcinomas (NSCLCs) and to extend th
167 ase (d-DT), have protumorigenic functions in non-small cell lung carcinomas (NSCLCs) but have AMPK-ac
168 eration and maintenance of Kras(G12V)-driven non-small cell lung carcinomas (NSCLCs).
169 tly observed in human cancers, but rarely in non-small cell lung carcinomas (NSCLCs).
170                                              Non-small-cell lung carcinomas (NSCLCs), which represent
171 ithelial adhesion and are often expressed in non-small-cell lung carcinomas (NSCLCs).
172 ng tumor cell lines, and 98 resected primary non-small-cell lung carcinomas (NSCLCs).
173 ous cell carcinoma, the two major classes of non-small-cell lung carcinomas (NSCLCs).
174 e antitumor activity of Taxol in a MDR human non-small cell lung carcinoma nude mouse xenograft model
175                                              Non-small-cell lung carcinomas of the superior sulcus, f
176                                              Non-small cell lung carcinoma patients are frequently tr
177 nated melanoma patients and three metastatic non-small cell lung carcinoma patients vaccinated with a
178 in anaplastic lymphoma kinase (ALK)-positive non-small-cell lung carcinoma patients, progression duri
179 rly results in patients with ROS1-rearranged non-small-cell lung carcinoma, recently emerging clinica
180 yzed, we observed that the majority (74%) of non-small cell lung carcinoma samples exhibited a signif
181  a subset of primary human cancers including non-small cell lung carcinoma, squamous cell skin carcin
182                                              Non-small cell lung carcinoma still maintains a poor 5-y
183 d that PPP2R2A was commonly downregulated in non-small cell lung carcinomas, suggesting that PPP2R2A
184 in 2014 to treat gastric adenocarcinomas and non-small cell lung carcinomas, targets vascular endothe
185                    Strikingly, patients with non-small cell lung carcinoma that had received WGP trea
186                       Finally, we found that non-small-cell lung carcinoma that presented a cytonucle
187  of combined-modality therapy for stage IIIA non-small cell lung carcinoma, the signs and symptoms of
188                                        Using non-small cell lung carcinoma to illustrate this approac
189  as in a panel of established small cell and non-small cell lung carcinoma tumor cell lines.
190 n (>50%) of DAL-1 was measured in 39 primary non-small cell lung carcinoma tumors as compared with pa
191 ome 18p11.3, which is lost in 38% of primary non-small cell lung carcinoma tumors.
192 nt response to chemotherapy in patients with non-small cell lung carcinoma using functional diffusion
193   A phase II trial of patients with advanced non-small-cell lung carcinoma using the 72-h infusion ev
194                Twenty-one of the twenty-four non-small-cell-lung carcinomas we analyzed express IWS1.
195        One hundred consecutive patients with non-small cell lung carcinoma were referred for CT evalu
196 endocrine small cell lung carcinomas, 4 of 4 non-small cell lung carcinomas with neuroendocrine pheno
197 zed Axl monoclonal antibodies that attenuate non-small cell lung carcinoma xenograft growth by downre
198 s-dependent human PANC-1 pancreatic and A549 non-small-cell lung carcinoma xenografts in nude mice, e

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