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3 l (progesterone, dehydroepiandrosterone) and non-steroidal antagonists (verapamil, PSC 833, LY335979,
8 d genetic pathway and uniquely revealed that non-steroidal anti-inflammatories (NSAIDs) rank just as
9 elevant concentrations of ibuprofen (IBF), a non-steroidal anti-inflammatory agent and prevalent envi
10 gense-1IC50/anti-cyclooxygense-2IC50>1) than non-steroidal anti-inflammatory agent ibuprofen (0.44) d
12 prodrugs can easily be conjugated to another non-steroidal anti-inflammatory agent, leading to easy s
14 neurocognitive sequelae from irradiation and non-steroidal anti-inflammatory agents can restore neuro
19 xylate transport process that may be used by non-steroidal anti-inflammatory and fluoroquinolone anti
20 drugs currently on the market, including the non-steroidal anti-inflammatory blockbuster drug celecox
21 of a systemic antibiotic (doxycycline) and a non-steroidal anti-inflammatory drug (ibuprofen), admini
25 isease (GERD), Barrett's esophagus (BE), and non-steroidal anti-inflammatory drug (NSAID) bleeding pr
28 oubts about the cardiovascular safety of the non-steroidal anti-inflammatory drug (NSAID) rofecoxib,
30 e show that inhibition of endogenous PGE2 by non-steroidal anti-inflammatory drug (NSAID) treatment i
32 high or standard doses compared with remote non-steroidal anti-inflammatory drug (NSAID) use or cele
33 al surgery and to evaluate the effect of the non-steroidal anti-inflammatory drug (NSAID), ibuprofen,
34 yptamine-gallic acid hybrid molecule against non-steroidal anti-inflammatory drug (NSAID)-induced gas
37 tion that the carboxylate group of an acidic non-steroidal anti-inflammatory drug can bind to a COX e
43 ne of the drugs was sulindac, a prototypical non-steroidal anti-inflammatory drug with established ch
45 e treatment, we examined the efficacy of the non-steroidal anti-inflammatory drug, flurbiprofen, to s
46 with lumiracoxib (59 events [0.65%]) and the non-steroidal anti-inflammatory drugs (50 events [0.55%]
47 plications was 1.09% (95% CI 0.82-1.36) with non-steroidal anti-inflammatory drugs (64 events) versus
48 udy was designed to determine if non-aspirin non-steroidal anti-inflammatory drugs (NANSAIDs) are ass
50 us reports in the literature have shown that non-steroidal anti-inflammatory drugs (NSAID) may affect
52 of dose and regularity of aspirin use, other non-steroidal anti-inflammatory drugs (NSAID), and the e
54 ors (i.e., TGF-beta 1, PDGF, and IGF-1), and non-steroidal anti-inflammatory drugs (NSAIDs) (indometh
55 sms that underlie the antitumour activity of non-steroidal anti-inflammatory drugs (NSAIDs) against c
58 ase-2 selective inhibitors and non-selective non-steroidal anti-inflammatory drugs (NSAIDs) are assoc
63 As an additional pharmacologic approach, non-steroidal anti-inflammatory drugs (NSAIDs) can reduc
66 e interviewed about use of aspirin and other non-steroidal anti-inflammatory drugs (NSAIDs) during th
68 X)-2 selective inhibitors versus traditional non-steroidal anti-inflammatory drugs (NSAIDs) has not b
70 ies for reducing gastrointestinal effects of non-steroidal anti-inflammatory drugs (NSAIDs) have been
73 horesis (FASS) for the determination of five non-steroidal anti-inflammatory drugs (NSAIDs) in bovine
74 cocorticoid therapy, hydroxychloroquine, and non-steroidal anti-inflammatory drugs (NSAIDs) in the tr
80 of Helicobacter pylori in patients receiving non-steroidal anti-inflammatory drugs (NSAIDs) is unclea
81 cal trials demonstrate that long-term use of non-steroidal anti-inflammatory drugs (NSAIDs) markedly
82 chemopreventive effect for aspirin and other non-steroidal anti-inflammatory drugs (NSAIDs) on colore
83 e clinically important errors: non-selective non-steroidal anti-inflammatory drugs (NSAIDs) prescribe
84 ogical evidence suggests that chronic use of non-steroidal anti-inflammatory drugs (NSAIDs) reduces t
89 ies respectively), while anti-infectives and non-steroidal anti-inflammatory drugs (NSAIDs) were freq
91 tly statin drugs, low-dose aspirin and other non-steroidal anti-inflammatory drugs (NSAIDs), and even
93 inical trials have suggested that the use of non-steroidal anti-inflammatory drugs (NSAIDs), includin
96 increasing prevalence of individuals taking non-steroidal anti-inflammatory drugs (NSAIDs), there is
100 drugs in four therapeutic areas: analgesia (non-steroidal anti-inflammatory drugs [NSAIDs]), diabete
101 umstantial evidence that binding of heme and non-steroidal anti-inflammatory drugs alters the conform
102 rall population, 0.55% (50/9127) of those on non-steroidal anti-inflammatory drugs and 0.65% (59/9117
104 Although Helicobacter pylori and use of non-steroidal anti-inflammatory drugs are common causes,
109 that individuals taking aspirin and related non-steroidal anti-inflammatory drugs have reduced risk
110 monly observed lack of efficacy of classical non-steroidal anti-inflammatory drugs in the treatment o
112 horylation of serine 121 in cells exposed to non-steroidal anti-inflammatory drugs leads to HSP90 dis
113 s have consistently demonstrated that common non-steroidal anti-inflammatory drugs may protect agains
115 effects of tetracyclines, and the effect of non-steroidal anti-inflammatory drugs on decreasing alve
116 activity by the administration of PTH and/or non-steroidal anti-inflammatory drugs or COX-2 selective
117 ofile of such therapies precluded the use of non-steroidal anti-inflammatory drugs or other enzyme in
119 biotics, antimotility agents, narcotics, and non-steroidal anti-inflammatory drugs should not be give
120 en, statin, antihypertensive medications and non-steroidal anti-inflammatory drugs therapy) as well a
123 duction in ulcer complications compared with non-steroidal anti-inflammatory drugs without an increas
124 al anesthetics, ketamine, acetaminophen, and non-steroidal anti-inflammatory drugs) analgesic drugs c
126 ns, diuretics, antivirals, methotrexate, and non-steroidal anti-inflammatory drugs) is organic anion
127 e attack therapies (eg, simple analgesics or non-steroidal anti-inflammatory drugs) or specific agent
128 ooxygenase site, which is the target for the non-steroidal anti-inflammatory drugs, and a spatially d
130 er complications compared with non-selective non-steroidal anti-inflammatory drugs, but evidence is l
131 tory and analgesic properties to traditional non-steroidal anti-inflammatory drugs, but with signific
132 e show that fatty acids, prostaglandins, and non-steroidal anti-inflammatory drugs, compounds that ar
133 tal cancer, and is a key molecular target of non-steroidal anti-inflammatory drugs, compounds that re
135 treatments have been suggested, ranging from non-steroidal anti-inflammatory drugs, drugs for neuropa
137 okine blockade, and further investigation of non-steroidal anti-inflammatory drugs, in the chemopreve
138 ma-secretase modulators (GSMs), such as some non-steroidal anti-inflammatory drugs, is a promising th
139 ation pharmacotherapies, which have included non-steroidal anti-inflammatory drugs, must take into ac
140 COX2 inhibitor lumiracoxib compared with two non-steroidal anti-inflammatory drugs, naproxen and ibup
141 cated PUD was associated with current use of non-steroidal anti-inflammatory drugs, oral steroids or
142 cal studies have shown that long-term use of non-steroidal anti-inflammatory drugs, statins, histamin
143 In this study we evaluate the effect of non-steroidal anti-inflammatory drugs, such as indometha
144 mized placebo-controlled trials, traditional non-steroidal anti-inflammatory drugs, such as naproxen,
156 ut the potential role of these drugs as oral non-steroidal anti-inflammatory therapy for asthma and C
159 the cleavage of all its substrates, certain non-steroidal antiinflammatory drugs (NSAIDs) have been
160 ct and isolates at 100 mug/mL was similar to non-steroidal antiinflammatory drugs aspirin, ibuprofen
161 established anticoagulants like heparin and non-steroidal antiinflammatory drugs may have a solid ba
162 ls in many non-neuronal cells are blocked by non-steroidal antiinflammatory drugs that are derivative
163 ositive breast cancer that has progressed on non-steroidal aromatase inhibitors (NSAIs) is unclear.
164 preserves bone in postmenopausal women, but non-steroidal aromatase inhibitors accelerate bone loss
167 The system uses a commercially available non-steroidal ecdysone agonist, RH5992 (tebufenozide), a
168 ution of the steroid hormone ecdysone or the non-steroidal ecdysone analog RH5992 for either fetal ca
170 al products that were previously reported as non-steroidal estrogen receptor antagonists with in vitr
171 Steroidal dexamethasone is as effective as non-steroidal ibuprofen for preventing or controlling po
174 lpha was activated in vivo by inclusion of a non-steroidal ligand in the diet, the expression of the
175 here is a great interest in the discovery of non-steroidal ligands able to bind to the ligand-binding
176 l companies have been developing a series of non-steroidal ligands that consist of different chemical
177 proteins occurred when 25-OH-cholesterol or non-steroidal LXR agonists (GW3965, TO-901317) were used
178 ed the renoprotective effects of Ly, a novel non-steroidal mineralocorticoid receptor (MR) blocker, t
179 nhibitory potencies of several steroidal and non-steroidal oestrogen analogues were examined and thei
182 itor interaction, a variety of steroidal and non-steroidal phosphate esters were synthesized and test
186 in D signaling, we report discovery of novel non-steroidal small-molecule compounds that activate the
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