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2 udies were designed to determine whether the nonadrenergic inotropic and vasodilator drug amrinone, w
3 ads to a significant decrease in an airway's nonadrenergic noncholinergic inhibitory (NANCi) response
7 he exact role of carbon monoxide (CO) in the nonadrenergic, noncholinergic (NANC) relaxation is not k
14 tensions in response to different agents and nonadrenergic, noncholinergic nerve stimulation by elect
16 ia adrenergic mechanisms and could occur via nonadrenergic, noncholinergic nitrergic neural and/or en
18 hanisms of action, and indicating that other nonadrenergic, noncholinergic relaxant neurotransmitters
19 demonstrate defects in gastric emptying and nonadrenergic, noncholinergic relaxation of pyloric musc
20 ation of hexamethonium significantly reduced nonadrenergic, noncholinergic relaxation, the catalytic
21 thmatic patients may reflect upregulation of nonadrenergic, noncholinergic, NO-producing nerves in ai
22 duced by neuronal NO synthase (nNOS) mediate nonadrenergic/noncholinergic (NANC) intestinal relaxatio
23 Calcitonin gene-related peptide (CGRP) is a nonadrenergic/noncholinergic (NANC) peptide with vasodil
25 trations, the P2X1 antagonists abolished the nonadrenergic, purinergic component of neurogenic contra
26 thetic innervation undergoes a switch from a nonadrenergic to cholinergic and peptidergic phenotype.
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