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1 e TS (+0.19 +/- 0.08%) compared with that in nonatherosclerotic (-0.10 +/- 0.13%) and control (-0.09
2 neous coronary artery dissection (SCAD) is a nonatherosclerotic acute coronary syndrome for which opt
3 idase proteins (gp91phox, Nox1, and Nox4) in nonatherosclerotic and atherosclerotic human coronary ar
4 nal antiserum was performed on a total of 83 nonatherosclerotic and atherosclerotic human coronary ar
5 ined ACE expression immunohistochemically in nonatherosclerotic and diseased human coronary arteries.
6 43+/-0.02 at induction versus 0.35+/-0.02 in nonatherosclerotic animals (P=0.031) and no further incr
7 andardized uptake value mean, 0.10+/-0.01 in nonatherosclerotic animals versus 0.20+/-0.03 [P=0.002]
8           In contrast to previous studies of nonatherosclerotic animals, the results indicate that in
9 e marrow-derived progenitor cells from young nonatherosclerotic ApoE-/- mice prevents atherosclerosis
10     Fibromuscular dysplasia (FMD) is a rare, nonatherosclerotic arterial disease for which the molecu
11 or basal NFkappaB activity in SMC in vivo as nonatherosclerotic arteries did not contain nuclear p65
12 ested the hypothesis that human atheroma and nonatherosclerotic arteries express the two Cox isoforms
13                                           In nonatherosclerotic arteries, ACE immunoreactivity was fo
14 rarchical clustering of genes and miRs among nonatherosclerotic arteries, stable, and unstable plaque
15 ntimal expansion has been reduced in vivo in nonatherosclerotic balloon-injured arteries by transfer
16 ts, n = 4), whereas it only infected 2 of 14 nonatherosclerotic blood vessel explants (SV, n = 10; IM
17 e cells residing in both atherosclerotic and nonatherosclerotic blood vessels were free of active CMV
18 section (SCAD) is an increasingly recognized nonatherosclerotic cause of acute coronary syndrome.
19 s from injured atherosclerotic and uninjured nonatherosclerotic common femoral arteries (CFA) in chol
20 ther aberrant angiogenesis also occurs under nonatherosclerotic conditions, we performed femoral arte
21 position was seen in aortas of the wild-type nonatherosclerotic control mice.
22 as stable lesions showed only the precursor; nonatherosclerotic control segments were negative for bo
23 ild-type C57BL/6 mice served as sham-treated nonatherosclerotic controls.
24 uptake in atherosclerotic animals but not in nonatherosclerotic controls.
25 t or with premounted stents were inflated in nonatherosclerotic coronary arteries of either young dom
26   Minimal extracellular PLTP was detected in nonatherosclerotic coronary arteries, but extracellular
27                                           In nonatherosclerotic coronary arteries, superoxide was pre
28                                              Nonatherosclerotic coronary artery ring segments were un
29  of specific binding of [3H]-leukotrienes to nonatherosclerotic coronary artery.
30               In contrast, only 1 (4%) of 24 nonatherosclerotic coronary specimens showed any evidenc
31 the expression of 11 000 murine genes in the nonatherosclerotic descending aorta by Affymetrix gene c
32  strong c-FLIP immunoreactivity in SMCs from nonatherosclerotic diffuse intimal thickening and in the
33 t not all, prospective studies report higher nonatherosclerotic disease rates in people with low seru
34 ether low cholesterol is causally related to nonatherosclerotic disease risk.
35 ibromuscular dysplasia is a noninflammatory, nonatherosclerotic disease that has been reported in alm
36 bserved associations of low cholesterol with nonatherosclerotic disease.
37 binding activity in both atherosclerotic and nonatherosclerotic human coronary arteries.
38 is monoclonal expansion can also be found in nonatherosclerotic intima and media.
39                                              Nonatherosclerotic intima contained extensive deposits o
40 chemic stroke, including atherosclerotic and nonatherosclerotic ischemic stroke subtypes.
41                                   The common nonatherosclerotic, large-vessel arteriopathies affectin
42 eriopathies in the context of other systemic nonatherosclerotic, large-vessel vascular disease (e.g.
43                                Compared with nonatherosclerotic-MSCs, atherosclerotic-MSCs displayed
44 as performed on atherosclerotic (n = 87) and nonatherosclerotic (n = 51) coronary arterial segments f
45 ents (75%) due to atherosclerotic (n=12) and nonatherosclerotic (n=3) causes.
46 ere isolated from atherosclerotic (n=38) and nonatherosclerotic (n=42) donors.
47  Arterial stiffness leads to LV mass through nonatherosclerotic pathways in mice.
48 gh ABI might lead to greater LV mass through nonatherosclerotic pathways.
49 dependent and -independent vasodilation in a nonatherosclerotic peripheral conduit artery of 26 elder
50 thelial regeneration after balloon injury in nonatherosclerotic rabbits.
51                                 In VSMC from nonatherosclerotic regions, RT-PCR analysis revealed an
52                                  We analyzed nonatherosclerotic repair arteries gathered at coronary
53 s the intimal response to arterial injury in nonatherosclerotic rodents and rabbits and inhibits the
54                                              Nonatherosclerotic samples contained neither HSP.
55                                              Nonatherosclerotic SCAD patients were prospectively foll
56              Thromboangiitis obliterans is a nonatherosclerotic segmental inflammatory disease that a
57 -rich atherosclerotic lesions and 26 normal, nonatherosclerotic segments) were exposed to heparinized
58     In contrast, GPI-PLD was not detected in nonatherosclerotic segments.
59                                              Nonatherosclerotic spontaneous coronary artery dissectio
60 untries have long revealed a predominance of nonatherosclerotic stroke, hypertensive heart disease, n
61 iovascular Data Registry enhanced pediatric, nonatherosclerotic structural heart disease and congenit
62 solate actively infected none of six sets of nonatherosclerotic SV explants at 5 to 7 days after inoc
63 nted in 2 or 3 main coronary arteries of 136 nonatherosclerotic swine and examined by light microscop
64  14) had 5-fold higher levels of TIMP-3 than nonatherosclerotic tissue (n= 10).
65 as detected in the endothelial cell layer of nonatherosclerotic vessels but was absent from the endot
66  reduced in human plaques when compared with nonatherosclerotic vessels.

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