ライフサイエンスコーパス: noncoronary

1 sociated with an increased odds of prevalent noncoronary and generalized atherosclerosis independentl

2 he deformation modes of the left, right, and noncoronary aortic root regions during isovolumic contra

3 rity of patients from either the left or the noncoronary aortic sinus of Valsalva.

4 FMD must be identified in at least one other noncoronary arterial territory to attribute any coronary

5 ntimal hyperplasia after balloon dilation of noncoronary arteries in small-animal models, suggesting

6 During follow-up (median: 502 days), noncoronary artery bypass graft (CABG) bleeding occurred

7 roportions for the primary outcome (death or noncoronary artery bypass graft protocol major bleeding

8 The primary outcome was the rate of noncoronary artery bypass graft related thrombolysis in

9 nal diagnosis in 127 patients (15%); cardiac noncoronary artery disease, in 124 (14%).

10 mug/L]; P<0.001) than patients with cardiac noncoronary artery disease.

11 ocardial infarction (AMI) from acute cardiac noncoronary artery disease.

12 ted patients with AMI and those with cardiac noncoronary artery disease.

13 een patients with AMI and those with cardiac noncoronary artery disease.

14 Noncoronary cardiac abnormalities are associated with co

15 We explored the association of noncoronary cardiac abnormalities with coronary artery d

16 bypass surgery and in patients who also had noncoronary cardiac disease.

17 ntly available or under development to guide noncoronary cardiac interventional procedures, and sugge

18 ment of improved imaging techniques to guide noncoronary cardiac interventions.

19 This review provides an overview of the noncoronary cardiac structures that can be evaluated wit

20 y, in fact, identify a potentially treatable noncoronary cause for chest pain.

21 and esophagitis, is a potentially treatable noncoronary cause for chest pain.

22 ediction model that accounts for deaths from noncoronary causes among older adults provided well-cali

23 Deaths from noncoronary causes largely exceeded the number of CHD ev

24 low probability of CAD focuses on diagnosing noncoronary causes of chest pain.

25 rdial infarction compared with patients with noncoronary CP.

26 right coronary cusp (RCC) in 14 (31.8%), the noncoronary cusp (NCC) in 1 (2.3%), and at the junction

27 ting patterns were observed in the right and noncoronary cusp fusion patients.

28 n of a left coronary artery (right cusp, 13; noncoronary cusp, one).

29 stenotic) and 3 with fusion of the right and noncoronary cusp.

30 Clinically important noncoronary diagnoses that did not explain patient sympt

31 ACS patients presenting to the ED provided a noncoronary diagnosis that explained the presenting comp

32 group 2 had late gadolinium enhancement in a noncoronary distribution, and no subjects in groups 3 an

33 The inclusion of noncoronary end points in this tool expands the range of

34 terior flow jets (n = 11), whereas right and noncoronary fusion gave rise to left-handed helical flow

35 ivation is a common feature in patients with noncoronary heart disease with a history of VF, and its

36 fractionation analysis in 266 patients with noncoronary heart disease.

37 milar density of innervation, except for the noncoronary leaflet of the aortic valve in which the inn

38 ostic relevance, as the fusion of right- and noncoronary leaflets (R-N) is associated with a greater

39 Coronary SMCs, however, were distinct from noncoronary medial cells, which displayed greater phenot

40 l operation in which a portion of the aortic noncoronary (n = 3) or right coronary (n = 3) leaflet wa

41 t was associated with a higher prevalence of noncoronary (odds ratio: 1.55; 95% confidence interval:

42 aggregation and inflammation in coronary and noncoronary patients.

43 e not homogeneous among the left, right, and noncoronary regions.

44 fusion patterns (right-left, RL versus right-noncoronary, RN) and expression of aortopathy.

45 us had rS pattern in lead I, and VT from the noncoronary sinus had a notched R wave in lead I.

46 ences in integrin expression on coronary and noncoronary SMCs may account for cellular differences.

47 ion on freshly isolated porcine coronary and noncoronary SMCs revealed that coronary SMCs express sig

48 d smooth muscle myosin heavy chain), whereas noncoronary SMCs showed marked phenotypic heterogeneity.

49 and to compare the responses of coronary and noncoronary SMCs to stimulation.

50 SMCs (human and porcine) were distinct from noncoronary SMCs, showing poor adhesion and spreading, a

51 ress significantly less alpha(5)beta(1) than noncoronary SMCs, whereas the expression of total beta(1

52 onary smooth muscle cells (SMCs) differ from noncoronary SMCs.

53 n of coronary SMCs to the levels observed in noncoronary SMCs.

54 utside the pre-defined period, or involved a noncoronary stent.

55 detector CT can be used to evaluate numerous noncoronary structures in the same examination.

56 This study investigated noncoronary vascular calcification and its influence on

57 Relatively little is known about noncoronary vascular calcification.

58 in therapy is beneficial among patients with noncoronary vascular disease (such as congestive heart f

59 Noncoronary vascular disease and clinically important va