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1 nvolving cysteine replacement of potentially noncritical amino acid residues, followed by chemical mo
2 ffects, occur at a single residue in a small noncritical amino-terminal proteolytic domain of transpo
3 nst cathepsin B cleavage by N-methylation of noncritical backbone NH groups or by dipeptide mimetic s
5 the burden of critical illness, educate our noncritical care colleagues about these interventions, a
6 ator utilization ratios in critical care and noncritical care locations and described event character
8 straints, all p < .01) and critical care vs. noncritical care physicians (42% vs. 24% for sedation an
9 to 11.79 per 1,000 ventilator days, whereas noncritical care unit rates ranged from 0 to 14.86 per 1
10 Although children with both critical and noncritical CHD had poorer outcomes, those with critical
13 line but increases with increasing levels of noncritical coronary stenosis because of adaptive change
15 base-pair combinations that are possible at noncritical DNA positions (from -6 to -4, from -2 to +2,
18 0 examinations of HMH, 22 of SAI, and 124 of noncritical findings was obtained resulting in 2583 repr
21 ceive exceptional services compared with the noncritical group (adjusted odds ratio, 1.46; 95% confid
22 rence, 249.6/100,000; 95%CI, 89.7-409.6) and noncritical heart defects (7306.9/100,000 [401/5488]; pr
23 ects but did have an increased prevalence of noncritical heart defects compared with infants of nonpr
24 eeclampsia was significantly associated with noncritical heart defects in offspring, and preeclampsia
25 io for incident dementia was 1.4 following a noncritical illness hospitalization (95% CI, 1.1 to 1.7;
27 s had no hospitalization, 1287 had 1 or more noncritical illness hospitalizations, and 41 had 1 or mo
28 cases of dementia among those with 1 or more noncritical illness hospitalizations, and 5 cases of dem
36 in1 inhibitors was designed by replacing the noncritical residues within the cyclic peptide ring with
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