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1  Previous mapping experiments between EL and nonepileptic ABP mice identified, and a congenic strain
2 standing of mechanisms underlying paroxysmal nonepileptic as well as some epileptic disorders.
3 er of cortical neurons compared with the two nonepileptic baboons.
4 tic drugs (AEDs) are commonly prescribed for nonepileptic conditions, including migraine headache, ch
5  seizures or PDs but not during electrically nonepileptic epochs.
6 ic seizures, single unprovoked seizures, and nonepileptic events from those with new-onset epilepsy.
7 t that SWDs and associated immobility may be nonepileptic in healthy outbred rats and reflect instead
8                      Noninjected control and nonepileptic monkeys with injections into the middle and
9                             In addition, the nonepileptic paroxysmal movement disorder hyperekplexia
10 e anticonvulsants, and is among the first in nonepileptic patients, suggesting that sudden anticonvul
11 epilepsy models but has not been observed in nonepileptic rodents, suggesting that FRs are associated
12 ere more frequent in epileptic compared with nonepileptic rodents; however, this feature showed limit
13 ncephalography remains the gold standard for nonepileptic seizure diagnosis.
14 nt video electroencephalography to establish nonepileptic seizure diagnosis.
15                          The misdiagnosis of nonepileptic seizure is costly to patients, the healthca
16                                              Nonepileptic seizure patients remain one of the most cha
17 es, such as frontal lobe seizures, may mimic nonepileptic seizure semiology.
18 re that could benefit them.The first step in nonepileptic seizure treatment is proper diagnosis.
19                           The methodology in nonepileptic seizure treatment trials is examined, descr
20 e number of publications about (psychogenic) nonepileptic seizures (NES) over the past two decades.
21 burdens from diagnostic delay of psychogenic nonepileptic seizures (PNES) requires prompt referral fo
22 er, seizures type, also known as psychogenic nonepileptic seizures (PNES).
23 wledge of longer term outcome in psychogenic nonepileptic seizures (PNESs) patients is limited; we kn
24 between convulsive epileptic and psychogenic nonepileptic seizures (PNESs).
25  studies assessing patients with psychogenic nonepileptic seizures and developments in treatment.
26                                              Nonepileptic seizures are best conceptualized and referr
27                      Finally, realizing that nonepileptic seizures are in a spectrum of somatoform di
28                                Patients with nonepileptic seizures are prescribed antiepileptic drugs
29                                              Nonepileptic seizures are seizure-like symptoms that occ
30                                              Nonepileptic seizures occur in 10 to 20% of children who
31 cribed antiepileptic drugs that do not treat nonepileptic seizures, have multiple laboratory tests pe
32                This review will characterize nonepileptic seizures, identify associated factors, prop
33                                  Psychogenic nonepileptic seizures, the most common conversion disord
34 r equipped to treat the underlying causes of nonepileptic seizures.
35 that more clearly establish the diagnosis of nonepileptic seizures.
36 BA in epilepsy patients at seizure onset and nonepileptic sites, cortical lesions, and from patients
37 ty from an early hyperexcitatory to a mature nonepileptic state.
38                           In the left-handed nonepileptic subjects, there was high posttest probabili
39 r alpha1 subunit per postsynaptic density in nonepileptic VP was 6.1 +/- 3.7, for alpha3 subunit in R

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