ライフサイエンスコーパス: nonesterified fatty acid

1 line, triglycerides, cholesteryl esters, and nonesterified fatty acids).

2 iators, lysophosphatidylcholine and oxidized nonesterified fatty acid.

3 nge in the pattern of total, esterified, and nonesterified fatty acids.

4 rations, and were negatively associated with nonesterified fatty acids.

5 ators, lyso-phosphatidylcholine and oxidized nonesterified fatty acids.

6 is likely due to decreased beta-oxidation of nonesterified fatty acids.

7 was accounted for by MAP, triglycerides, and nonesterified fatty acids.

8 creases cholesterol synthesis and release of nonesterified fatty acids.

9 orary storage site for energy in the form of nonesterified fatty acids.

10 erol (12 +/- 3 to 258 +/- 47 micromol/l) and nonesterified fatty acid (194 +/- 10 to 540 +/- 80 micro

11 tion into triglyceride-rich lipoproteins and nonesterified fatty acid, AEE, and muscle markers were s

12 racteristic decrease from baseline in plasma nonesterified fatty acids after a mixed meal was inhibit

13 The d31-palmitate appearance in nonesterified fatty acid and very-low-density lipoprotei

14 Nonesterified fatty acids and lipid peroxidation were in

15 atty acyl chains from phospholipids to yield nonesterified fatty acids and lysophospholipids.

16 ease plasma concentrations of both TGRLs and nonesterified fatty acids and meal 2 to increase TGRLs o

17 In contrast, the liver pool sizes of nonesterified fatty acids and triglycerides were not alt

18 r epididymal fat pads, lower blood levels of nonesterified fatty acids and triglycerides, and higher

19 Lipolysis (glycerol, nonesterified fatty acids) and endogenous glucose produc

20 ous glucose production, lipolysis (glycerol, nonesterified fatty acid), and glycogenolysis (lactate)

21 ty lipoproteins, cholesterol, triglycerides, nonesterified fatty acids, and leptin, whereas adiponect

22 ced physical activity; increased circulating nonesterified fatty acids; and increased IMCLs, diacylgl

23 Nonesterified fatty acids are key intermediates in cellu

24 plasma glucose, branched chain amino acids, nonesterified fatty acids, beta-hydroxybutyrate, and uri

25 ese results also suggest that esterified and nonesterified fatty acids can bind to and regulate prote

26 The nonesterified fatty acid concentration was significantly

27 ion, it increased lipid oxidation and plasma nonesterified fatty acid concentrations compared with HF

28 Nonesterified fatty acid concentrations were lower up to

29 serum insulin, glucose, triacylglycerol, and nonesterified fatty acid concentrations were measured, a

30 ts in increased serum levels of glycerol and nonesterified fatty acids, consistent with increased lip

31 to a meal produced TGRL that was enriched in nonesterified fatty acids, decreased IRF-1 expression, i

32 zed de novo in the liver from carbohydrates, nonesterified fatty acids derived from adipose tissue, n

33 ied fatty acids derived from adipose tissue, nonesterified fatty acids derived from the spillover of

34 ucagon-like peptide 1, insulin, glucose, and nonesterified fatty acids determined for 4 h.

35 Metabolic profiling of plasma nonesterified fatty acids discovered that palmitic acid

36 mass spectrometry was used to analyze free (nonesterified) fatty acid (FFA) and triacylglycerol flux

37 ed at 10-min intervals; blood triglycerides, nonesterified fatty acids, glucose, lactate, inflammator

38 Pathologically increased nonesterified fatty acids have widely been viewed as a k

39 beta-cell membrane phospholipids to release nonesterified fatty acids, including AA, and inhibiting

40 ome in db mice contributed high-glucose- and nonesterified fatty acid-induced osteoblast apoptosis th

41 the percentage of small dense LDL; glucose; nonesterified fatty acids; insulin; and the homeostasis

42 Adipose release of nonesterified fatty acids into plasma decreased by 53% a

43 n of glucagon secretion, reduction in plasma nonesterified fatty acid level, decrease in the load of

44 ratio (P = 0.013), and, surprisingly, higher nonesterified fatty acid levels (P = 0.01).

45 ion decreases serum triacylglycerol (TG) and nonesterified fatty acid levels and improves insulin sen

46 rization associated with reduced circulating nonesterified fatty acid levels and normal glucose homeo

47 Fasting increased plasma nonesterified fatty acid levels in both lean and obese r

48 olysis but did not result in increased serum nonesterified fatty acid levels or ectopic TAG storage.

49 nd beta cell volume without affecting plasma nonesterified fatty acid levels, strongly suggesting tha

50 ks, without significant alteration of plasma nonesterified fatty acid levels.

51 Nonesterified fatty acids may influence mitochondrial fu

52 andial responses in plasma concentrations of nonesterified fatty acid (meal x time, P = 0.00014), tri

53 ution exerts a major influence on endogenous nonesterified fatty acid metabolism, which may in turn m

54 opic hormone (ACTH), cortisol, glucagon, and nonesterified fatty acid (NEFA) concentrations were not

55 ndrial function independent of reductions in nonesterified fatty acid (NEFA) concentrations.

56 VLDL particle and TG transport rates, plasma nonesterified fatty acid (NEFA) flux, and sources of fat

57 2)H(2)]palmitic acid to investigate systemic nonesterified fatty acid (NEFA) incorporation into VLDL

58 arin (0.5 U x kg(-1) x min(-1)) to clamp the nonesterified fatty acid (NEFA) levels during hyperinsul

59 Elevation of plasma nonesterified fatty acid (NEFA) levels has been shown in

60 widely regarded as monitors of intracellular nonesterified fatty acid (NEFA) levels.

61 Nonesterified fatty acid (NEFA) release was suppressed a

62 rd quantitative methods for determination of nonesterified fatty acid (NEFA) species are still missin

63 Determinants of insulin sensitivity based on nonesterified fatty acid (NEFA) suppression after oral g

64 ied BAT oxidative metabolism and glucose and nonesterified fatty acid (NEFA) turnover in 6 healthy me

65 ive metabolism and perfusion and glucose and nonesterified fatty acid (NEFA) turnover were determined

66 Plasma nonesterified fatty acids (NEFA) at elevated concentrati

67 Although nonesterified fatty acids (NEFA) have been positively as

68 gy for simultaneous quantitative analysis of nonesterified fatty acids (NEFA) species in biofluids is

69 ipose tissue lipolysis produces glycerol and nonesterified fatty acids (NEFA) that serve as energy so

70 ntrol subjects, but the rates of delivery of nonesterified fatty acids (NEFA) were downregulated, res

71 mechanism involves leakage of albumin-bound nonesterified fatty acids (NEFAs) across the damaged glo

72 Impaired suppression of plasma nonesterified fatty acids (NEFAs) after glucose ingestio

73 We studied the effects of nonesterified fatty acids (NEFAs) and adipokines on acin

74 Experimental elevation of nonesterified fatty acids (NEFAs) impairs endothelial fu

75 HDL, and LDL cholesterol; triglycerides; and nonesterified fatty acids (NEFAs) in a total of 139 OT1D

76 tissue increases lipolysis and the entry of nonesterified fatty acids (NEFAs) in the liver, whereas

77 issue there was significant uptake of plasma nonesterified fatty acids (NEFAs) in the postprandial bu

78 t growth factor 21 (FGF21), adiponectin, and nonesterified fatty acids (NEFAs) may be involved in ami

79 in conscious dogs to determine the effect of nonesterified fatty acids (NEFAs) on net hepatic glucose

80 Preliminary data suggest that plasma nonesterified fatty acids (NEFAs) raise plasma ANGPTL4 c

81 Autonomic symptom scores, lipid oxidation, nonesterified fatty acids (NEFAs), and glycerol response

82 isotopes for 4 days to label and track serum nonesterified fatty acids (NEFAs), dietary fatty acids,

83 High plasma concentrations of nonesterified fatty acids (NEFAs), transported bound to

84 rgans to circulating triglycerides (TGs) and nonesterified fatty acids (NEFAs), ultimately leading to

85 ic than hGV but releases a lower quantity of nonesterified fatty acids (NEFAs).

86 (VLDL)-triacylglycerols and plasma free FA [nonesterified fatty acids (NEFAs)] were analyzed by usin

87 Adipo-IR (fasting nonesterified fatty acids [NEFAs] x fasting insulin) was

88 r (P < 0.05), whereas glucose (P < 0.05) and nonesterified fatty acids (P < 0.0001) were higher.

89 occurred with elevated glucose, insulin, and nonesterified fatty acids peak after lunch.

90 fasting plasma insulin (r = 0.60, P < 0.05), nonesterified fatty acid (r = 0.63, P < 0.02), and gluco

91 y deplete [Ca(2+)](m) and thus contribute to nonesterified fatty acid-responsive mitochondrial dysfun

92 of glucose, lactate, and ketones and higher nonesterified fatty acids than wild type (WT) littermate

93 es results in the liberation of glycerol and nonesterified fatty acids that are released into the vas

94 n the dose-response curve for suppression of nonesterified fatty acids versus insulin levels in the N

95 This fall in nonesterified fatty acid was accompanied by a fall in th

96 Neither lipolysis nor flux of plasma nonesterified fatty acids were altered compared with bas

97 fied non-HDL-cholesterol, triglycerides, and nonesterified fatty acids, with a minimum effective dose