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1  sympathetic neurons than in an electrically nonexcitable cell line, and provide a framework for futu
2                                   Unlike the nonexcitable cell membranes that are ubiquitous in all d
3 at ryanodine receptors are expressed in some nonexcitable cell types and furthermore suggest that the
4 CE) is a major Ca(2+) influx pathway in most nonexcitable cell types and is activated by any stimulus
5 ecules that play roles in both excitable and nonexcitable cell types and with or without the pore-for
6    Only 2 (HeLa and LLC-PK1 cells) out of 11 nonexcitable cell types examined expressed ryanodine rec
7 discuss sodium channel expression in diverse nonexcitable cell types, including astrocytes, NG2 cells
8 ryanodine receptors in several excitable and nonexcitable cell types.
9 easing the susceptibility to apoptogens in a nonexcitable cell.
10 lum (ER) are a major Ca(2+) entry pathway in nonexcitable cells and are essential for T cell activati
11 is the predominant Ca(2+) entry mechanism in nonexcitable cells and controls a variety of physiologic
12 ical, and molecular properties as I(CRAC) in nonexcitable cells and its rate of activation during rep
13        Possible roles for these molecules in nonexcitable cells are acute cell-volume regulation and,
14 ion potential (AP)-generating cells, whereas nonexcitable cells are generally considered as barriers
15 ransmission; their structure and function in nonexcitable cells are not well-defined.
16  cyclase type 8 (AC8) is activated by CCE in nonexcitable cells but is not responsive to other forms
17  junctional resistance between excitable and nonexcitable cells during cardiac action potential propa
18 o the role of voltage-gated Ca2+ channels in nonexcitable cells during development.
19 t models for agonist-activated Ca2+ entry in nonexcitable cells focus on the capacitative mechanism w
20 ium entry or store-operated calcium entry in nonexcitable cells is a process whereby the activation o
21 e intracellular Ca(2+) concentration of many nonexcitable cells is regulated by calcium store release
22 ole of Ca2+ in stimulus-response coupling in nonexcitable cells is still not well understood.
23                          Ca(2+) signaling in nonexcitable cells is typically initiated by receptor-tr
24                                      Whether nonexcitable cells may modulate excitable cell function
25 -7)) are widely distributed in excitable and nonexcitable cells of vertebrates.
26 pacitative (or store-operated) Ca2+ entry in nonexcitable cells represents a switching between two di
27  identity of the calcium channels present in nonexcitable cells such as T lymphocytes.
28 nnels are expressed in various excitable and nonexcitable cells supporting important cellular respons
29                  Astrocytes are electrically nonexcitable cells that communicate by means of Ca(2+) s
30 activated pathway for the entry of Ca(2+) in nonexcitable cells that is entirely separate from the wi
31 t and that the channel probably functions in nonexcitable cells to depolarize membrane potential and/
32  sustained Ca2+ signals seen in a variety of nonexcitable cells under conditions of maximal stimulati
33 The extracellular potential of excitable and nonexcitable cells with respect to ground is approximate
34 mportant role in agonist-evoked secretion in nonexcitable cells, although this has not been confirmed
35  coordinating local activity of electrically nonexcitable cells, because identical patterns of ATP re
36                We have determined that, like nonexcitable cells, both neonatal and adult cardiomyocyt
37                                           In nonexcitable cells, depletion of endoplasmic reticulum C
38                                      In many nonexcitable cells, depletion of the inositol 1,4, 5-tri
39   Here we show that T lymphocytes, which are nonexcitable cells, express both regulatory beta and por
40 ysiological significance of this behavior in nonexcitable cells, in which the primary mechanism of Ca
41 dentification of Orai as the SOCE channel in nonexcitable cells, investigation of Orai function in ne
42                                           In nonexcitable cells, stimulation by high agonist concentr
43                                           In nonexcitable cells, the predominant mechanism for regula
44                                           In nonexcitable cells, there are currently two models for i
45                                           In nonexcitable cells, we had previously established that C
46  L-type voltage-gated Ca2+ channel CaV1.2 in nonexcitable cells.
47 r molecular counterpart for Ca(2+) influx in nonexcitable cells.
48 n electrical signaling in nerve, muscle, and nonexcitable cells.
49 or Ca(2+) signal generation in excitable and nonexcitable cells.
50 and membrane potential in both excitable and nonexcitable cells.
51 g electrical signaling in both excitable and nonexcitable cells.
52 he Ca2+-sensitive phosphatase calcineurin in nonexcitable cells.
53 a(2+) signaling events in both excitable and nonexcitable cells.
54 esents the principal Ca2+ entry pathway into nonexcitable cells.
55 ich is a major Ca(2+) influx pathway in most nonexcitable cells.
56 ue to follow dynamic changes in potential in nonexcitable cells.
57 iphosphate (IP3)-mediated Ca2+ liberation in nonexcitable cells.
58 with respect to store-operated Ca2+ entry in nonexcitable cells.
59 nnels) are widely expressed in excitable and nonexcitable cells.
60 t Ca(2+) influx pathway in oocytes and other nonexcitable cells.
61 d in the sustained phase of calcium entry in nonexcitable cells.
62 a(2+) are sufficient for exocytosis in these nonexcitable cells.
63 f whether they are expressed in excitable or nonexcitable cells.
64 zing the membrane potential of excitable and nonexcitable cells.
65 lux for stimulus-secretion coupling in these nonexcitable cells.
66 apes the profile of the Ca2+ signal in these nonexcitable cells.
67 tant in the physiology of both excitable and nonexcitable cells.
68 y be the primary mechanism for Ca2+ entry in nonexcitable cells.
69 ol many important processes in excitable and nonexcitable cells.
70  calcium (SOC) influx in platelets and other nonexcitable cells.
71 + channels in nuclear signal transduction in nonexcitable cells.
72 ssed at the plasma membrane of excitable and nonexcitable cells.
73 e and identify ryanodine receptors (RyRs) in nonexcitable mouse parotid acini.
74                                              Nonexcitable muscle membrane after direct muscle stimula
75                      We investigated whether nonexcitable muscle membrane indicates fast-twitch myofi
76         We suggest that electrophysiological nonexcitable muscle membrane predicts preferential type
77                                Patients with nonexcitable muscle membranes (n = 15) showed smaller me
78  of 95 nM) and its predominant expression in nonexcitable tissues of adult animals.
79        Our findings show how channels act in nonexcitable tissues to regulate stem cells and may lead
80  studied, including cells from excitable and nonexcitable tissues, such as the nervous and cardiovasc
81 hysiology in a wide variety of excitable and nonexcitable tissues.
82 very little expression was detected in other nonexcitable tissues.
83  in sympathetic neurons than in electrically nonexcitable tsA201 cells.

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