ライフサイエンスコーパス: noninflamed

1 eased proopiomelanocortin mRNA in cells from noninflamed and inflamed LN.

2 Human gingival tissues from noninflamed and inflamed sites were also analyzed by RT-

3 tive expression of hBD-1 mRNA was similar in noninflamed and inflamed tissues obtained from each of f

4 c fusion protein administration does not, in noninflamed animals, change basal pain sensitivity nor t

5 lymphocytes already reside within the normal/noninflamed aorta before the onset atherosclerosis as a

6 he differences between inflamed and adjacent noninflamed areas in the rat model suggest a tissue grad

7 olonic mucosa revealed minimal expression in noninflamed areas, with striking upregulation under coli

8 for the recruitment of Th1 effector cells to noninflamed as well as inflamed intestine.

9 measured HLA-A2 binding between inflamed and noninflamed cohorts.

10 In noninflamed colons of wild-type mice not receiving tegas

11 ubsets to enter and persist in non-lymphoid, noninflamed compartments, we examined the migration and

12 study, we demonstrate that murine LECs under noninflamed conditions actively scavenge and cross-prese

13 Under noninflamed conditions, C3aR and C5aR protein and mRNA w

14 l tissues such as skin and gut under normal, noninflamed conditions.

15 d UC biopsies and was the sole form found in noninflamed cont.

16 CD compared with ulcerative colitis (UC) and noninflamed control (cont) patients, and were more abund

17 rated little reactivity, as did tissues from noninflamed control brains.

18 erosing panencephalitis, but not in IgG from noninflamed control tissue.

19 f inflammatory bowel disease patients versus noninflamed controls.

20 zed dissociation to mCRP in inflamed but not noninflamed cremaster muscle.

21 ater adhesion of MB(sLex) to inflamed versus noninflamed endothelium (P = 0.0081).

22 dhesion to inflamed endothelium, relative to noninflamed endothelium, under in vitro flow conditions

23 samples) and one group of four age-matched, noninflamed enucleated globes (control samples) were use

24 otomously as having inflamed fat (n = 78) or noninflamed fat (n = 31) on the basis of the presence (+

25 In contrast, obese subjects with noninflamed fat exhibited a mixed clinical phenotype wit

26 Cr and CXCL10:Cr significantly distinguished noninflamed from inflamed biopsies (area under the curve

27 HBD-2 mRNA was also expressed in 14 of 15 noninflamed gingival tissue samples.

28 0 [3.3, 13.7], P = 0.002, n = 9) compared to noninflamed histology (1.4 [0.4, 4.2], normal and inters

29 a protective role of capsule in inflamed and noninflamed hosts.

30 abel macrophages in the medullary sinuses of noninflamed human lymph node.

31 In noninflamed human skin during steady state, there are th

32 in a restricted subset of dermal vessels in noninflamed human skin from three different sites.

33 al vessels could serve to recruit T cells to noninflamed human skin.

34 ed gene expression signatures in inflamed vs noninflamed intestinal and rectal tissues collected from

35 ific localization of CD4 T cells into normal noninflamed islets.

36 average was lower than that in inflamed and noninflamed joints from dogs in group A.

37 rom 1.5 percentage injected dose per gram in noninflamed joints to 22.6 percentage injected dose per

38 percentage injected dose per gram (%ID/g) in noninflamed joints to 32.1 %ID/g in severely inflamed jo

39 antigens is dependent on KCs as well as on a noninflamed liver microenvironment, thereby providing me

40 as increased only in inflamed paw tissue and noninflamed LN-immune cells.

41 Normal (from noninflamed lung), Th1-type (induced by the pulmonary em

42 onlymphoid peripheral site (i.e., the normal/noninflamed lung).

43 naive and activated CD8+ T cells within the noninflamed lungs and quantitated the partitioning of ad

44 Twelve patients also had noninflamed meibomian gland dysfunction (MGD).

45 already present in the adventitia of normal/noninflamed mouse aortas.

46 rophages contribute to tissue homeostasis in noninflamed mucosa through profound down-regulation of p

47 subclasses); however, anti-TNP responses in noninflamed mucosal tissues of mice with colitis exhibit

48 further observed to roll continuously along noninflamed murine dermal endothelium in vivo.

49 In contrast, cells from noninflamed muscle contained negligible amounts of CCL19

50 tissue from patients with juvenile DM and in noninflamed muscle tissue from control subjects were exa

51 Both the inflamed and noninflamed muscle tissue had equivalent levels of CXCL1

52 In contrast, in noninflamed muscle, plasmacytoid DCs were scarce and did

53 Biopsies were classified as noninflamed (n=21), inflamed (borderline changes or abov

54 Lymphocyte trafficking into normal/noninflamed or atherosclerosis-prone aortas was partiall

55 paired behavioral profile than children with noninflamed or non-T cell activated immune profiles.

56 rption, and hemoglobin response to SF) among noninflamed, outpatient populations.

57 Noninflamed patients in BMI Q5 formed the reference grou

58 nt analyses, the all-cause mortality risk in noninflamed patients was higher only in the lowest BMI q

59 was associated with higher BMI quintiles in noninflamed patients.

60 lamed patients, this effect was mitigated in noninflamed patients.

61 the pulmonary vascular compartment into the noninflamed pulmonary interstitium.

62 immunohistochemical analyses of inflamed and noninflamed regions of pancreatic tissue from patients w

63 rylated RelA and IkappaBalpha in inflamed vs noninflamed regions.

64 smooth muscle cells in colitic compared with noninflamed regions.

65 ncreased in regions of colitis compared with noninflamed regions.

66 Six-month urinary CCL2: Cr distinguished noninflamed renal tissue (normal, fibrosis) from IF+i wi

67 mucosa of inflamed IBD specimens compared to noninflamed sections from the same patient, establishing

68 They also travel to noninflamed skin and peripheral LNs and remain in elevat

69 chanisms directing trafficking of T cells to noninflamed skin are less well characterized.

70 akly expressed or absent in keratinocytes of noninflamed skin but induced during acute inflammation.

71 ssion of these molecules on blood vessels in noninflamed skin provides the basis for a model of cutan

72 There are T cells within normal, noninflamed skin that most likely conduct immunosurveill

73 Noninflamed skin venules support constitutive leukocyte

74 cimens and additional controls consisting of noninflamed skin.

75 novel in vivo method, we report that in the noninflamed state, most transfused RBCs were consumed by

76 lymphocyte migration to the intestine in the noninflamed steady state, their role during inflammation

77 L-1beta released significantly more END from noninflamed than from inflamed LN-immune cells.

78 DP47GS accumulation ranged from 1.6 %ID/g in noninflamed tissue to 12.0 %ID/g in severely inflamed jo

79 In noninflamed tissue, IL-8 expression is low but can be ra

80 reduced compared with those from autologous noninflamed tissue.

81 However, resting Tregs from noninflamed tissues exhibit little suppressor activity,

82 trate that dermal fibroblasts and vessels of noninflamed tissues express SDF-1.

83 terized by CX(3)CR1-dependent recruitment to noninflamed tissues.

84 wn to direct blood cell trafficking to other noninflamed tissues.

85 , primary and secondary lymphoid organs, and noninflamed tissues.