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1 most common cancer type among men (excluding nonmelanoma skin cancer).
2 and 98 patients developed cancer (excluding nonmelanoma skin cancer).
3 mary cause of skin cancer (both melanoma and nonmelanoma skin cancer).
4 rt members, 730 reported 802 SMNs (excluding nonmelanoma skin cancers).
5 global increase in incidence of melanoma and nonmelanoma skin cancer.
6 se may offer a viable therapeutic option for nonmelanoma skin cancer.
7 nscreen use to prevent actinic keratoses and nonmelanoma skin cancer.
8 adiation in sunlight is the primary cause of nonmelanoma skin cancer.
9 t ranged from 48.7% for myeloma to 31.4% for nonmelanoma skin cancer.
10 llowing UV irradiation, the primary cause of nonmelanoma skin cancer.
11 the major risk factor for the development of nonmelanoma skin cancer.
12 c has been associated with increased risk of nonmelanoma skin cancer.
13 psoriasis patients are at increased risk for nonmelanoma skin cancer.
14 on plays a critical role in the induction of nonmelanoma skin cancer.
15 ith no previous history of cancer other than nonmelanoma skin cancer.
16 global increase in incidence of melanoma and nonmelanoma skin cancer.
17 sion, which contribute to the development of nonmelanoma skin cancer.
18 %]), attributable to the higher incidence of nonmelanoma skin cancer.
19 organ transplants have an increased risk for nonmelanoma skin cancer.
20 quitous in skin and has been associated with nonmelanoma skin cancer.
21 porine have an increased risk for developing nonmelanoma skin cancer.
22 ell carcinomas (MCCs), an aggressive form of nonmelanoma skin cancer.
23 anding the pathogenesis of wound healing and nonmelanoma skin cancer.
24 aging nor a higher incidence for sun-induced nonmelanoma skin cancer.
25 lignancies, 233 benign meningiomas, and 1856 nonmelanoma skin cancers.
26 the most prevalent mutations found in human nonmelanoma skin cancers.
27 n phenotypes and development of melanoma and nonmelanoma skin cancers.
28 or subsequent malignancies, meningiomas, and nonmelanoma skin cancers.
29 ic receptors as a new treatment modality for nonmelanoma skin cancers.
30 a-HPVs) may contribute to the development of nonmelanoma skin cancers.
31 ent of precancerous skin lesions and certain nonmelanoma skin cancers.
32 than heterosexual women to report having had nonmelanoma skin cancer (2001-2005 CHIS: aOR, 0.56; 95%
36 ctrum strikingly similar to that reported in nonmelanoma skin cancer and characteristic of DNA damage
38 present in sunlight is the primary cause of nonmelanoma skin cancer and has been implicated in the d
42 aviolet A dramatically increases the risk of nonmelanoma skin cancer and prior exposure to psoralen+u
43 vation was clear for all main cancers except nonmelanoma skin cancer and was stronger for cancers of
44 e and effective in reducing the rates of new nonmelanoma skin cancers and actinic keratoses in high-r
46 of breast cancer or other cancers (excluding nonmelanoma skin cancer), and we completed a personal ba
48 feature of diseases like psoriasis, eczema, nonmelanoma skin cancer, and melanoma where differentiat
49 ve an increased incidence of viral warts and nonmelanoma skin cancer, and the presence of HPV DNA in
50 rs, six non-MDS hematologic malignancies, 39 nonmelanoma skin cancers, and 68 cases of MDS/acute myel
53 ecent dramatic increases in the incidence of nonmelanoma skin cancers are largely attributable to hig
54 1273 men had any malignant neoplasm (except nonmelanoma skin cancer), as compared with 1293 in the p
55 ohort who had no cancer diagnosis (excluding nonmelanoma skin cancer) at the start of follow-up and r
56 ts an avoidable risk factor for melanoma and nonmelanoma skin cancer - both of which may be lethal.
57 bronchus, and lung; malignant skin melanoma; nonmelanoma skin cancer; breast; cervical; uterine; ovar
58 Administration approved for the treatment of nonmelanoma skin cancers, but it has limited activity ag
59 s that beta-HPV infections may contribute to nonmelanoma skin cancer by increasing the likelihood tha
61 ity of Pennsylvania, who were diagnosed with nonmelanoma skin cancer, dermatophytosis, acne rosacea,
62 a causative role in ODC up-regulation during nonmelanoma skin cancer development by binding to and st
65 eningiomas, and 1.71 (95% CI, 0.88-3.33) for nonmelanoma skin cancers for survivors with reference ch
71 The primary end point was the number of new nonmelanoma skin cancers (i.e., basal-cell carcinomas pl
73 ated with a dose-dependent increased risk of nonmelanoma skin cancer in patients treated for psoriasi
74 play an important part in the development of nonmelanoma skin cancer in psoralen + ultraviolet A-trea
76 12-month intervention period, the number of nonmelanoma skin cancers in the 6-month postintervention
77 o, 386 participants who had had at least two nonmelanoma skin cancers in the previous 5 years to rece
79 same cumulative dose, which may explain why nonmelanoma skin cancer incidence depends more strongly
85 rved being central nervous system (n = 344), nonmelanoma skin cancer (n = 278), digestive (n = 105),
86 mon type of previous cancer in the donor was nonmelanoma skin cancer (n=776) followed by central nerv
88 le of mitochondrial DNA (mtDNA) deletions in nonmelanoma skin cancer (NMSC) and in cutaneous photoagi
89 erum 25-hydroxyvitamin D levels with risk of nonmelanoma skin cancer (NMSC) and melanoma, we evaluate
91 Protective effects of UV-B radiation against nonmelanoma skin cancer (NMSC) are exerted via signaling
92 ndependently reviewed the DSCMs for residual nonmelanoma skin cancer (NMSC) before and after a brief
93 on-exposed survivors who developed an SN1 of nonmelanoma skin cancer (NMSC) had a cumulative incidenc
96 aimed to determine the risk of melanoma and nonmelanoma skin cancer (NMSC) in patients with IBD and
100 ot have a greater than expected incidence of nonmelanoma skin cancer (NMSC) or other cancers, whereas
101 th 95% CIs for any incident cancer excluding nonmelanoma skin cancer (NMSC) were 1.06 (95% CI, 1.02-1
103 e intake and risks of skin cancer (overall), nonmelanoma skin cancer (NMSC), and basal cell carcinoma
104 d higher risk than HIV-uninfected persons of nonmelanoma skin cancer (NMSC), defined as basal cell ca
107 duced immunosuppression is a risk factor for nonmelanoma skin cancer (NMSC), particularly squamous ce
108 ng Medicare billing codes and categorized as nonmelanoma skin cancer (NMSC), viral-linked and "other"
109 in a single patient were counted, except for nonmelanoma skin cancer (NMSC), where only the first was
113 cies (malignancies), including and excluding nonmelanoma skin cancer (NMSC); all malignancies were ex
114 in a case-control study of 191 patients with nonmelanoma skin cancer (NMSC; 81 SCC and 110 basal cell
116 V) have been suspected to be carcinogenic in nonmelanoma skin cancers (NMSC), but the basis for poten
119 ous basal cell and squamous cell carcinomas (nonmelanoma skin cancers (NMSCs)), data are insufficient
120 ght to be involved in the initiation of some nonmelanoma skin cancers (NMSCs), particularly in patien
121 des further evidence that for primary facial nonmelanoma skin cancers (NMSCs), recurrence rates with
123 with briakinumab vs. placebo, respectively), nonmelanoma skin cancers (NMSCs; four vs. zero squamous
124 ent SMNs were thyroid cancer, breast cancer, nonmelanoma skin cancer, non-Hodgkin's lymphoma, and acu
126 squamous cell carcinoma (SCC) (often termed nonmelanoma skin cancer or keratinocyte carcinoma [KC])
127 et A is associated with an increased risk of nonmelanoma skin cancer, particularly squamous cell carc
128 lomaviruses (HPVs) have been associated with nonmelanoma skin cancers, particularly in immunocompromi
129 hree patients who underwent Mohs surgery for nonmelanoma skin cancers presented between 2 and 4 weeks
130 an papilloma virus infections and associated nonmelanoma skin cancers, providing additional genetic a
131 a US population-based case-control study of nonmelanoma skin cancer, randomly selected from drivers'
132 that can reduce mortality from melanoma and nonmelanoma skin cancer, screening holds the greatest pr
133 found moderately increased SIR estimates for nonmelanoma skin cancer, smoking-related cancers, and Ho
136 e exposure to sunlight increases the risk of nonmelanoma skin cancer, the avoidance of all direct sun
137 iolet (UV) irradiation is the major cause of nonmelanoma skin cancer, the most common form of cancer
138 identified Zmiz1 as a candidate oncogene in nonmelanoma skin cancer through a transposon mutagenesis
139 were adjusted based on age, sex, history of nonmelanoma skin cancer, US geographic region, and popul
140 tocols and disability, dermatitis, melanoma, nonmelanoma skin cancer, viral skin diseases, and fungal
143 the 10 most common cancers in the US and of nonmelanoma skin cancer was not increased with TNFalpha
147 role of the Fragile Histidine Triad gene in nonmelanoma skin cancer, we have used reverse transcript
149 .44-4.27) with placebo/dexamethasone; IRs of nonmelanoma skin cancers were 2.40 (95% CI, 1.33-4.33) a
151 d who had no prior history of cancer (except nonmelanoma skin cancer) were followed prospectively for
152 the incidence of all-type cancer (excluding nonmelanoma skin cancers), which was evaluated using Kap
153 a (MCC) is a highly malignant neuroendocrine nonmelanoma skin cancer, which is associated with the Me
154 IN OUTCOME MEASURES: Total cancer (excluding nonmelanoma skin cancer), with prostate, colorectal, and
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