1                         These increases were nonosmotic and glucose specific, in that the exposure to

2 dose reductase, which prevents PKC-dependent nonosmotic NF-kappaB activation, may be a useful approac

3 ars to be requisite for glucose-specific and nonosmotic regulation of AR2 in the RPE cell in vitro.

4            Arterial underfilling induces the nonosmotic stimulation of arginine vasopressin and upreg

5                                        Under nonosmotic stress conditions, the SLN1 sensor kinase is