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1 and CBH II) preferentially cleaves from the nonreducing end.
2 ccharides with Delta-4,5-unsaturation at the nonreducing end.
3 only lack the galactose residue at the Le(a) nonreducing end.
4 y added to the fifth xylose residue from the nonreducing end.
5 ched to the phosphoethanolamine group at the nonreducing end.
6 eavage occurred from the reducing end or the nonreducing end.
7 to a growing chain of polysialic acid at the nonreducing end.
8 acid (HA) by addition to the reducing or the nonreducing end.
9 tment with beta-glycosidases that act at the nonreducing end.
10 nzyme contain a C4 gemdiol/keto group at the nonreducing end.
11 ainly to the second glucose residue from the nonreducing end.
12 ly degraded from the reducing end toward the nonreducing end.
13 ivity and degrades the chitin chain from the nonreducing end.
14 Sia alpha 2-6Gal or Sia alpha 2-3Gal at the nonreducing end.
15 erified to a variety of mycolic acids at the nonreducing ends.
16 ific sulfates and monosaccharides from their nonreducing ends.
17 of the terminal trisaccharide, having at the nonreducing end a GlcNAc or GalNAc, and bound them to BS
18 1 at the residues immediately located at the nonreducing end, allowing the formation of an N-Ac domai
19 ing N-acyl and O-acetyl modifications at the nonreducing end and a critical 6-O-sulfate at the reduci
20 igosaccharides containing a galactose at the nonreducing end and a propargyl group at the reducing en
21 ccharides of N-glycan with a GlcNAc at their nonreducing end and found that the extended sugar moiety
22 ontaining an unsaturated uronate unit at the nonreducing end and two contiguous AT-binding sequences
23 binding subsites (-I, -II, and -III) for the nonreducing end and two glucose binding subsites (+I and
26 oside hydrolase Family 6 (GH6) CBHs act from nonreducing ends by an inverting mechanism and are prese
27 of glycosyl hydrolases (GH5) and by Cel6A, a nonreducing-end cellobiohydrolase from family GH6 with t
30 >/=1/10,000), but only vaccines enriched for nonreducing end de-N-acetyl residues by treatment with e
31 n and minimally to an altered binding of the nonreducing-end DEF trisaccharide to the native serpin c
32 results suggest that glycosidases acting on nonreducing ends digest large amounts of xyloglucan in w
33 erminal 4,5-unsaturated glucuronic acid, the nonreducing end disaccharide moiety does not interact wi
35 also confirming that growth occurred at the nonreducing end following initiation on phosphatidylglyc
37 h the azido group at the C-6 position of the nonreducing end fucose could elicit a strong IgG immune
38 d alpha2-3- or alpha2-6-linked to a terminal nonreducing end galactose, poly-LacNAc extended core-3 O
39 harides contained N-acetylglucosamine at the nonreducing ends, galactosylation was judged to be ineff
40 plex with an oligosaccharide acceptor with a nonreducing end GlcNAc that has a beta1-6-glycosidic lin
42 sthydrolysis state in which the newly formed nonreducing end has already left the substrate binding p
43 ndicated that ligands bound to E2 with their nonreducing ends in position -2, consistent with the pos
45 and masked as a 1,6-anhydro sugar, while the nonreducing end is activated as a free alkyne and masked
46 type that could be explained by an excess of nonreducing ends leading to a reinforced xyloglucan netw
47 stic of neuraminic acid, particularly at the nonreducing end, may be important for processing and pre
48 s contain neither the galactose at the Le(a) nonreducing end nor the fucose at the Le(x) reducing end
49 ide evidence that NS domains residing at the nonreducing end (NRE) are, on average, longer than those
50 ohydrate-binding module (CBM) that binds the nonreducing end of beta-1,3-glucan chains, and an unchar
51 te displace the 6-hydroxymethyl group of the nonreducing end of both acarbose and D-gluco-dihydroacar
53 ution reveals that the GlcNAc residue at the nonreducing end of chitobiose makes extensive hydrophobi
54 n (GH18(C)) and releases chitobiose from the nonreducing end of chitooligosaccharides, therefore func
57 samines present as monosaccharides or at the nonreducing end of odd-numbered oligosaccharide substrat
58 ronate or beta-D-manuronate residues, at the nonreducing end of oligo-alginates in an exolytic fashio
59 the unsaturated Delta4,5 uronic acid at the nonreducing end of oligosaccharides resulting from prior
63 ucing an azido-containing sialic acid to the nonreducing end of the galactosides through a sialyltran
65 lternate additions of Glc and GlcUA onto the nonreducing end of the growing polysaccharide chain.
66 l" antithrombin binding site and also at the nonreducing end of the molecule, which is reported in in
68 initiated by subsite 1 interactions with the nonreducing end of the oligosaccharide substrate, minimi
71 luronic acid blocks from the reducing to the nonreducing end of the polymer following the initial ran
73 4, where the (GlcNAc)2 group arises from the nonreducing end of the substrate and is formed as the be
78 meric enzyme that releases fructose from the nonreducing end of various oligosaccharides and essentia
79 aditionally as releasing cellobiose from the nonreducing ends of cellulose, but this definition is in
80 by W58L was confirmed from both reducing and nonreducing ends of CNP-labeled oligosaccharide substrat
83 and 2-O-sulfo-iduronic acid residues at the nonreducing ends of the glycans as co-receptors for Shh.
86 ith its C-terminus free and the GPI with its nonreducing end phosphoethanolamine bearing a free amino
90 and (GlcNAc)4 where dimers cleaved from the nonreducing end reflect the most common binding and hydr
92 deletion of reducing-end residues G and H or nonreducing-end residue D produced variable losses in pe
94 accumulated heparan sulfate exhibits unique nonreducing end structures with terminal N-sulfoglucosam
95 n bonding between Asn(462) and xylose at the nonreducing end subsite +2 was important for the higher
96 at 355 nm, producing fragment ions from the nonreducing end that do not contain the appended fluorop
99 The enzyme sulfates the substrate from the nonreducing end toward the reducing end consecutively, l
100 hin sulfate (ECS), was designed to mimic the nonreducing end trisaccharide unit DEF of the sequence s
102 -O-, 5-O- and 3,5-di-O-linked alpha-Araf and nonreducing end-units of alpha-Araf, Arap, beta-Galp and
103 ed cellodextrins modified at the reducing or nonreducing ends upon incubation with cellulose and cell
104 alogues with modification at the reducing or nonreducing end were synthesized using chemoenzymatic me
105 investigated on cellopentaose labeled at the nonreducing end with 14C, and cellooligosaccharides redu
106 hand, synthesis from the reducing end to the nonreducing end yielded the mannopentaose with the desir
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