ライフサイエンスコーパス: nontransgenic

1 c (0.053 +/- 0.020 min(-1); n = 32 mice) and nontransgenic (0.056 +/- 0.029 min(-1); n = 17 mice) ani

2 transgenic (0.78 +/- 0.26 mm, P = .01), and nontransgenic (1.0 +/- 0.19 mm, P = .002) mice.

3 and thus palmitoyl-coenzyme A recycling, in nontransgenic (4.5+/-2.3 micromol/min per gram dry weigh

4 to pressure overload and oxidative stress in nontransgenic adult mouse hearts.

5 Compared with nontransgenic age-matched control littermates, rTg(tau(P

6 rons derived from various brain regions from nontransgenic and genetically engineered mice and rats c

7 and stroma of untreated or estrogen-treated nontransgenic and HPV-transgenic mice.

8 Additionally, we fed nontransgenic and K8 overexpressing mice (K8tg) with the

9 Adult male nontransgenic and myocyte-restricted HO-1 transgenic mic

10 al doses of 10 mg/kg trichostatin A (TSA) in nontransgenic and SMA model mice resulted in increased l

11 ate-salt model of neurogenic hypertension in nontransgenic and syn-hACE2 mice overexpressing ACE2 in

12 We performed adoptive transfers of both nontransgenic and TCR-transgenic OVA(257-264)-specific (

13 age, cell turnover was reduced with aging in nontransgenic and TGF-alpha mice, indicating that some g

14 lanine, and fructose remained similar in the nontransgenic and transgenic fruits.

15 tified in isolated hearts, from normal mice (nontransgenic) and mice with cardiac-specific overexpres

16 Atrial JPH2 levels in WT-JPH2 transgenic, nontransgenic, and JPH2 knockdown mice correlated negati

17 2CTF), and describe a novel disease-relevant nontransgenic animal model of AD.

18 ew representative analogs were assessed in a nontransgenic animal model of Alzheimer's disease (AD),

19 A nontransgenic animal model that still develops hallmarks

20 ice may be used as a simple, toxin-free, and nontransgenic animal model to study PD-related nigrostri

21 gnitude higher in transgenic animals than in nontransgenic animals 2 to 4 weeks postinfection, and th

22 inhibition of these neurons in freely moving nontransgenic animals has not been possible.

23 ells accumulated in inflamed joints, even in nontransgenic animals.

24 red with genetically matched and age-matched nontransgenic animals.

25 isease-prone HLA-B7 transgenic or wild-type (nontransgenic) animals.

26 antly reduced polyp load (60%) compared with nontransgenic Apc(Min+) littermates.

27 addition, RLK pathways are ideal targets for nontransgenic approaches, such as synthetic molecules, p

28 nced (anergic) immunoglobulin transgenic and nontransgenic B cells but not by transiently stimulated

29 , onward transmission to both transgenic and nontransgenic birds was prevented.

30 However, nontransgenic, Bt and BtHr cotton had similar yields ove

31 In nontransgenic CD4 T cells, blocking IL-4Ralpha with Abs

32 nt as well as lean control Tg-fMCD(Skel) and nontransgenic control mice were treated with Shield-1 an

33 In nontransgenic control mice, oxidative stress was coincid

34 ls of CA-IX, Glut1, and VEGF than tubules in nontransgenic control mice.

35 ischemic reserve capacity when compared with nontransgenic control mice.

36 nits were indistinguishable from age-matched nontransgenic control mice.

37 NA expression below approximately 50% of the nontransgenic control.

38 with increased macrophage uPA expression or nontransgenic controls (all apolipoprotein E-null [Apoe(

39 a a bacterial artificial chromosome (TG) and nontransgenic controls (Cont).

40 ize of MI was similar between Tg-DN-Mst1 and nontransgenic controls (NTg).

41 Compared with nontransgenic controls and singly transgenic mice expres

42 human APOE3 or APOE4 to the levels of their nontransgenic controls and the significant decrease of i

43 maize events had a greater grain yield than nontransgenic controls under both drought stress and wel

44 Relative to nontransgenic controls, calcium dynamics were significan

45 Compared with nontransgenic controls, hAPP mice had significantly fewe

46 Compared with nontransgenic controls, neither transgenic model exhibit

47 persistently oxidized glucose compared with nontransgenic controls, while exhibiting supranormal per

48 uridine (BrdU), Ki-67, and c-Myc relative to nontransgenic controls.

49 differ significantly between transgenic and nontransgenic controls.

50 Vgp120tg mice expressing or lacking CCR5 and nontransgenic controls.

51 reased insulin sensitivity compared to obese nontransgenic controls.

52 -hiPSCs was increased 100-fold above that of nontransgenic controls.

53 Transgenic cotton had higher yield than nontransgenic cotton for any given number of insecticide

54 largely because higher insecticide use with nontransgenic cotton improved control of key pests.

55 lar effects of cultivation of transgenic and nontransgenic cotton on biodiversity.

56 tical rates of fluorescence yield decline as nontransgenic cotton.

57 LUT4 gene and promoter (hGLUT4 TG) and their nontransgenic counterparts (NT) were fed either a contro

58 ve of infection, were observed in all of the nontransgenic cows but in none of the transgenic animals

59 ere administered to three transgenic and ten nontransgenic cows.

60 We demonstrate that IL-60 enables nontransgenic expression of an entire bacterial operon i

61 i-glomerular basement disease model, whereas nontransgenic FcRgamma(-/-) mice were completely protect

62 yogenic differentiation when cocultured with nontransgenic fetal cardiomyocytes (>18 000 EGFP(+) cell

63 r proteinuria and 75% lower albuminuria than nontransgenic FHH littermates.

64 ant in Bt cotton and Bt maize fields than in nontransgenic fields managed with insecticides.

65 fection with all three types of viruses than nontransgenic FVB mice.

66 turnover was enhanced overall compared with nontransgenic glands, indicating that aberrant cell turn

67 ls mature, a higher proportion expresses the nontransgenic H chain allele.

68 TAC induced a 25% increase in nontransgenic heart size but only a 7% increase in ssTnI

69 ation in hearts of Pim-wt mice versus 26% in nontransgenic hearts after infarction challenge.

70 iptional profiling of Galphaq transgenic and nontransgenic hearts by Illumina RNA sequencing and Affy

71 Differential mRNA expression in Galphaq and nontransgenic hearts correlated well between microarrays

72 xpression augmented TAG turnover 3-fold over nontransgenic hearts, despite similar fractions of acety

73 -energy phosphate content similar to that in nontransgenic hearts, providing evidence for greater ene

74 basal and beta-adrenergic stress compared to nontransgenic hearts, with a reduction in maximal Ca(2+)

75 Four weeks after ligation, nontransgenic HF mice exhibited postinfarction left vent

76 diac matrix with cardiomyocytes derived from nontransgenic human induced pluripotent stem cells and g

77 ansgenic infected mice relative to those for nontransgenic infected mice.

78 study using a wild gourd and transgenic and nontransgenic introgressives, we measured the effects of

79 transgenic mouse islets develop amyloid but nontransgenic islets do not, a broad spectrum MMP inhibi

80 oped much less severe proteinuria than their nontransgenic Lamb2-deficient littermates; the level of

81 function, and remodeling were compared with nontransgenic littermate control (NLC) and wild-type (WT

82 s of myocardial fibers from Tg-CTGF mice and nontransgenic littermate control (NLC) mice were discern

83 after TAC, swim-trained transgenic GRK5 and nontransgenic littermate control mice exhibited similar

84 KChIP2 from ventricles between p90RSK-Tg and nontransgenic littermate control mice were similar, as a

85 Cardiac-specific GRK5 transgenic mice and nontransgenic littermate control mice were subjected to

86 ter I/R in DN-RSK (0.9+/-0.2%) compared with nontransgenic littermate controls (6.2+/-2.6%).

87 reperfusion from 46.9+/-5.6% area at risk in nontransgenic littermate controls to 26.0+/-4.2% in DN-R

88 chain (alphaMHC)-BMP10 transgenic hearts and nontransgenic littermate controls using Affymetrix mouse

89 Beginning on day of life 75, TG9 mice and nontransgenic littermate controls were given a daily 10

90 ergic excitation to the same extent as their nontransgenic littermate controls, as a result of the ex

91 Compared with nontransgenic littermate controls, monocytes of TLR10 tr

92 in size and macrophage content compared with nontransgenic littermate controls.

93 toimmune alopecia susceptibility relative to nontransgenic littermate controls.

94 Nontransgenic littermate mice were not affected by this

95 s the PKA phosphorylation sites) and matched nontransgenic littermates (NTG) on a CD1 background.

96 significantly weaker with age compared with nontransgenic littermates and exhibited typical myopathi

97 iferation in bone marrow and spleen, whereas nontransgenic littermates and hMRP8-NPM transgenic mice

98 had little to no effect on ERG responses in nontransgenic littermates and other retinal degeneration

99 developed normally in comparison with their nontransgenic littermates but had a suppressed rate of h

100 enic mice survived significantly longer than nontransgenic littermates in response to a lethal tumor

101 This phenotype was not observed in nontransgenic littermates or in mice expressing an hP23H

102 At 2 to 3 months of age, ssTnI mice or their nontransgenic littermates underwent aortic constriction

103 omatic and symptomatic PrP-SCA7-92Q mice and nontransgenic littermates was compared.

104 Age-matched nontransgenic littermates were controls.

105 o(+/-)), and the eyes of transgenic mice and nontransgenic littermates were exposed for 2.5 minutes t

106 estricted dominant-interfering p53 and their nontransgenic littermates were treated with doxorubicin

107 tablished beta-amyloid peptide pathology and nontransgenic littermates were treated with either alpha

108 Tg2576 mice and their nontransgenic littermates were treated with simvastatin

109 nally, infection of both transgenic mice and nontransgenic littermates with HSV-1 revealed no differe

110 diac gene transcription differs from that of nontransgenic littermates, primarily in the expression o

111 and the TMA decreased with age compared with nontransgenic littermates, ruling out the SR as a source

112 In contrast to nontransgenic littermates, such animals had B-cell-restr

113 In contrast to nontransgenic littermates, which develop lymphocytic spo

114 asmacytoid DCs (pDCs) in transgenic, but not nontransgenic littermates, without elimination of spleni

115 epared from G93A-SOD1 embryos and from their nontransgenic littermates.

116 hibited a distinct immunoprofile compared to nontransgenic littermates.

117 orebrains of Tg-Abeta+Tau mice compared with nontransgenic littermates.

118 acquisition of the MWM task when compared to nontransgenic littermates.

119 te uptake into peripheral organs compared to nontransgenic littermates.

120 ed with wild-type ERalpha transgenic mice or nontransgenic littermates.

121 nd compared their diabetes susceptibility to nontransgenic littermates.

122 nse to azoxymethane (AOM) when compared with nontransgenic littermates.

123 duction and dilated ducts when compared with nontransgenic littermates.

124 significantly different between SA mice and nontransgenic littermates.

125 a transgenic mice when compared with that of nontransgenic littermates.

126 constitutively active Gqalpha compared with nontransgenic littermates.

127 cell population and comparing it to that of nontransgenic littermates.

128 other TCRBV gene segments when compared with nontransgenic littermates.

129 crypt foci and no tumors, in comparison with nontransgenic littermates.

130 ce, nor did it affect tumor dissemination in nontransgenic littermates.

131 um and membrane depolarization compared with nontransgenic littermates.

132 emyelinated or remyelinated axons than their nontransgenic littermates.

133 sing HuWtSOD1, but not to cells derived from nontransgenic littermates.

134 on with influenza viruses of human origin as nontransgenic littermates.

135 DDC feeding induced MBs in nontransgenic livers, but MBs were rarely seen in any of

136 RGS5 expression in nontransgenic MA was induced after activation of either

137 Adoptive transfer of nontransgenic macrophages partially restored neutrophil

138 easured by Akt phosphorylation compared with nontransgenic macrophages.

139 he male sterile phenotype, producing stable, nontransgenic male sterility.

140 l-mesenchymal transformation was frequent in nontransgenic malignancies.

141 PyroGlu-3 Abeta is further present in two nontransgenic mammalian models of cerebral amyloidosis,

142 ocal Lesions in Genomes (TILLING) provides a nontransgenic method for reverse genetics that is widely

143 icant increase in infarct size compared with nontransgenic mice (from 36.9+/-2.5% to 50.9+/-4.3%).

144 We compared data obtained from control nontransgenic mice (NTG) with a transgenic mouse model e

145 protein transgenic mice (CRPtg) compared to nontransgenic mice (NTG).

146 ACE2 activity in the cerebrospinal fluid of nontransgenic mice after deoxycorticosterone acetate-sal

147 reduction also protected both transgenic and nontransgenic mice against excitotoxicity.

148 TLR7 agonist administration to nontransgenic mice also drove type I IFN-dependent emerg

149 own to induce alphaS pathology in the CNS of nontransgenic mice and alphaS transgenic mice, albeit wi

150 ansgenic male mice at 6 months compared with nontransgenic mice and female hIAPP transgenic mice.

151 Finally, and in contrast to studies in nontransgenic mice and in mice expressing PS1HWT, ECL2 n

152 Nontransgenic mice and mice with cardiomyocyte-restricte

153 fibrosis in K18 R89C as compared with WT and nontransgenic mice and resulted in increased messenger R

154 d activity were found to decline with age in nontransgenic mice as well as in hIAPP transgenic female

155 When expressed in nontransgenic mice at levels sufficient to drive Abeta42

156 gnaling was inhibited in doxorubicin-treated nontransgenic mice but not in doxorubicin-treated MHC-CB

157 d neuronal alterations could be simulated in nontransgenic mice by excitotoxin challenge and prevente

158 ich are predisposed to MDB formation, and in nontransgenic mice by feeding the porphyrinogenic compou

159 Nontransgenic mice exhibited reduced left ventricular sy

160 0 mice had similar abnormalities relative to nontransgenic mice in spatial and nonspatial learning an

161 disease in K14E5 mice differed from that in nontransgenic mice in that benign tumors converted from

162 Eight-week-old core-E1-E2, core, and nontransgenic mice inbred on the FVBxC57Bl/6 background

163 urdens were not significantly different from nontransgenic mice infected with either Ed N-522D or par

164 eoxycorticosterone acetate-salt treatment in nontransgenic mice led to significant increases in blood

165 Analysis in nontransgenic mice showed that thioacetamide and CCl(4)

166 r, these autoantibodies were not observed in nontransgenic mice subjected to 4 chronic injury models.

167 rphosphorylation of endogenous mouse MAPT in nontransgenic mice that is further enhanced in mice lack

168 We also observed age-matched nontransgenic mice to study normal effects of aging on s

169 ontransgenic mice, whereas neither K14E5 nor nontransgenic mice treated with the promoting agent 12-O

170 lls and pericytes from brain microregions of nontransgenic mice using CD31 and CD13 as surface marker

171 man tau strains by intracerebrally injecting nontransgenic mice with pathological tau enriched from h

172 ly inhibited (7% in transgenic versus 40% in nontransgenic mice).

173 Here, we found that in wild-type nontransgenic mice, a single intrastriatal inoculation o

174 stic cervical disease than similarly treated nontransgenic mice, although no frank cancers were detec

175 Compared to control nontransgenic mice, as well as transgenic mice expressin

176 e serum of TLR10 transgenic mice compared to nontransgenic mice, but did not affect mouse survival in

177 Compared with nontransgenic mice, HO-1 transgenic HF mice exhibited si

178 ervical carcinogenesis compared with that in nontransgenic mice, indicating that activities of E7 bes

179 transgenic mice vs. approximately 15.5 h in nontransgenic mice, indicating that huGYPA on mature RBC

180 cdk5 inhibitor was administered to very old, nontransgenic mice, inhibition of cdk5 reduced Abeta lev

181 In nontransgenic mice, knockdown of EphB2 mediated by short

182 Nontransgenic mice, or mice that over express either hum

183 e in hypertrophy in both Tg-GSK-3beta-DN and nontransgenic mice, Tg-GSK-3beta-DN exhibited better lef

184 Compared with nontransgenic mice, the CNS of AstroCD24TG mice had high

185 Compared to nontransgenic mice, transgenic mouse hearts showed signi

186 developed more papillomas than like-treated nontransgenic mice, whereas neither K14E5 nor nontransge

187 r inclusions in K8-overexpressing but not in nontransgenic mice, without causing liver injury.

188 ness and collagen accumulation compared with nontransgenic mice, yet IL-5 intestine transgenic mice d

189 nic mice consume excess calories relative to nontransgenic mice, yet they weigh less.

190 The uptake into brain was minimal in nontransgenic mice.

191 03) were significantly reduced compared with nontransgenic mice.

192 sis and less lymphocyte infiltration than in nontransgenic mice.

193 iac-specific overexpression of Sirt1 than in nontransgenic mice.

194 can be used to differentiate vessel types in nontransgenic mice.

195 ced mortality, compared to >30% mortality in nontransgenic mice.

196 s in anatomically connected brain regions in nontransgenic mice.

197 similar to those seen in doxorubicin-treated nontransgenic mice.

198 display a baseline phenotype consistent with nontransgenic mice.

199 ronment of HPV-transgenic mice compared with nontransgenic mice.

200 arcomas have not been observed previously in nontransgenic mice.

201 unction and less fibrosis and apoptosis than nontransgenic mice.

202 pared with age-matched transgenic female and nontransgenic mice.

203 e of Pseudomonas from the lung compared with nontransgenic mice.

204 inoma progression compared with like-treated nontransgenic mice.

205 but also enhanced learning and memory in the nontransgenic mice.

206 oducing mice to the level of standard-housed nontransgenic mice.

207 els between caerulein-treated transgenic and nontransgenic mice.

208 modified lipoproteins than macrophages from nontransgenic mice.

209 han transgenic mice expressing core alone or nontransgenic mice.

210 -AD mice to the level of that in age-matched nontransgenic mice.

211 gy to bidirectionally control nociceptors of nontransgenic mice.

212 rain homogenates from human tauopathies into nontransgenic mice.

213 tion of TSPO ligands Ro5-4864 and PK11195 in nontransgenic mice.

214 when compared with IRS2(WT)-beta mice or to nontransgenic mice.

215 e Treg lineage in TCR transgenic mice and in nontransgenic mice.

216 mutant, decreased protein import by >50% in nontransgenic mitochondria from spinal cord, but not fro

217 had no noticeable fitness load compared with nontransgenic mosquitoes when fed on noninfected mice.

218 fecundity and lower mortality) than sibling nontransgenic mosquitoes.

219 t mosquitoes have a selective advantage over nontransgenic mosquitoes.

220 s or co-delivered with zinc-deficient SOD to nontransgenic motor neurons.

221 We now report that nontransgenic mouse AHNPCs transduced with retroviruses

222 -RTN3 mice would ever naturally occur in the nontransgenic mouse brain, we targeted our examination t

223 orCS1:SorL1, and SorCS1:Vps35 complexes from nontransgenic mouse brain.

224 exes during size exclusion chromatography of nontransgenic mouse lens extracts prepared without chemi

225 Nontransgenic mouse lens proteins incubated with purifie

226 Here, we present a nontransgenic mouse model in which spontaneous metastase

227 In nontransgenic mouse myofibrils, the Ca(2+) sensitivity o

228 ad a 2.4-fold higher Ca(2+) sensitivity than nontransgenic mouse myofibrils.

229 EMD57033 was indistinguishable from that of nontransgenic myofibrils.

230 We describe two routes to generate nontransgenic naive human ES cells (hESCs).

231 se genetic changes were more vulnerable than nontransgenic nematodes to mitochondrial complex I inhib

232 ) or Cu(II)), or etoposide compared with the nontransgenic nematodes.

233 In contrast, nontransgenic neonatal CD8(+) T cells when transferred i

234 roinsulin II from a transgene in K cells and nontransgenic NOD mice (controls); pancreas and duodenum

235 creased selection of Tregs was also found in nontransgenic NOD mice in fetal through adult stages.

236 As controls, nontransgenic NOD.H2(h4) mice similarly injected with in

237 AD mouse model of AD, compared with those in nontransgenic (non-Tg) controls, at 6, 8, and 14 mo of a

238 old transgenic (TgCRND8) (Tg) mice and their nontransgenic (non-Tg) littermates were entered in the s

239 nsmission of different tau strains occurs in nontransgenic (non-Tg) mice, and to investigate whether

240 ncrease of NAD(P)H levels and redox state in nontransgenic (non-Tg) neurons until middle age, followe

241 wo-photon imaging to study Ca2+ signaling in nontransgenic (NonTg) and several AD mouse models (PS1KI

242 hysiological recordings in young 3xTg-AD and nontransgenic (NonTg) hippocampal slices, we show that i

243 c Alzheimer's mice (3xTg-AD) and age-matched nontransgenic (nonTg).

244 AM14 site-directed BCR transgenic mice into nontransgenic normal recipients and elicited an EF respo

245 row of humanized NSG-SGM3 mice compared with nontransgenic NSG recipients.

246 MCK-PGC1alpha mice and nontransgenic (NT) littermates were fed a high-fat diet

247 analysis of brains from transgenic (TG) and nontransgenic (NT) mice 5 days after infection identifie

248 in voluntary cage wheel performance between nontransgenic (NTG) and HCM male mice were seen.

249 y or excessive memory generalization in both nontransgenic (NTG) and Tg2576 mice.

250 line effects in the TG rabbits compared with nontransgenic (NTG) littermates.

251 itutively active (CA) PKN (Tg-CAPKN) than in nontransgenic (NTg) mice (15+/-5 versus 38+/-5%, P<0.01)

252 e was similarly accelerated by Iso in Tg and nontransgenic (Ntg) myocytes.

253 ardial strips of the following mouse models: nontransgenic (NTG), effective null for cMyBP-C (t/t), w

254 cific BisANS incorporation sites on GAPDH in nontransgenic (NTg), G93A, and H46R/H48Q mice using liqu

255 y labeled single actin filaments compared to nontransgenic or transgenic wild-type (Tg-WT) control mi

256 gene expression was compared in R89C versus nontransgenic or wild-type K18-overexpressing mice.

257 umerous (6 +/- 3 transgenic versus 2 +/- 1.5 nontransgenic papillomas per mouse), yet they were more

258 single-positive thymocytes was also seen in nontransgenic PKCtheta(-/-) mice.

259 In situ hybridizations of tissue from nontransgenic plants revealed that the expression of all

260 nsertion flowered significantly earlier than nontransgenic plants, supporting the identity between Ta

261 rrelates with b1 silencing in transgenic and nontransgenic plants.

262 tion of RTBV RNA and viral DNA compared with nontransgenic plants.

263 he RNAi plants had fewer oil bodies than the nontransgenic plants.

264 the genome and generate precisely modified "nontransgenic" plants.

265 atoes; and in none of the volunteers who ate nontransgenic potatoes.

266 biochemical assays and functional studies in nontransgenic protoplasts.

267 In this study, by using a nontransgenic rat model of sporadic AD generated by intr

268 In summary, we provide a novel nontransgenic rat model with high endogenous ghrelin pla

269 transgenic and control (HLA-B7-transgenic or nontransgenic) rats.

270 s were adoptively transferred into syngeneic nontransgenic recipients and their fate in the periphera

271 tected by immunoblot and by inoculation into nontransgenic recipients.

272 In contrast, if tumors were implanted in nontransgenic SCID mice adjacent to a patch of transplan

273 ls and stroma, tumor cells were implanted in nontransgenic SCID mice that received a bone marrow tran

274 ter retransplantation at 14 days to control (nontransgenic) secondary BALB/c recipients, with or with

275 ing CPCs, with 36% of the CPCs versus 73% in nontransgenic sections.

276 tly increased adult survivorship relative to nontransgenic sibling controls.

277 genic kernels weighed the same on average as nontransgenic siblings, with normal endosperm starch and

278 branching and spikelet density compared with nontransgenic siblings.

279 icant difference in survivorship relative to nontransgenic siblings.

280 script accumulation in response to culturing nontransgenic soybean explants on the medium used to ind

281 ssed responses by naive TCR75 T cells and by nontransgenic spleen cells stimulated with anti-CD3.

282 and function of orphan genes, and identify a nontransgenic strategy for modulating protein levels in

283 ChR alpha-chain peptide that dominated young nontransgenic T cell responses in vitro.

284 data indicate that TCR transgenic as well as nontransgenic T(EM) differentiate into T(CM) in the abse

285 ess mature and turned over more rapidly than nontransgenic T2 cells, exhibiting neither conventional

286 Nontransgenic TAC developed diastolic dysfunction (65% i

287 ting the increase in anaplerosis observed in nontransgenic TAC hearts.

288 Isolated perfused hearts from nontransgenic TAC mice showed reduced cardiac function a

289 Contrasting nontransgenic TAC, ssTnI TAC significantly increased glu

290 and at a rate higher than those observed in nontransgenic, TGF-beta1-transgenic, or Delta(beta)RII-t

291 was significantly greater in Tg-Nox4 than in nontransgenic, the LV weight/tibial length was not signi

292 Five mouse lines were used: nontransgenic, those that overexpress wild-type K18 or t

293 lants (0 d in culture) was found to resemble nontransgenic tissue that had been induced for SE by bei

294 d species, but given the 'y' number of other nontransgenic traits that breeders also need to assemble

295 gs were made with wild-type (T-cell receptor nontransgenic) Tregs, and (iii) the B29 human homolog is

296 The nontransgenic w(1118) Drosophila line was used as a cont

297 ts, transgenic mosquitoes gradually replaced nontransgenics when mosquitoes were maintained on mice i

298 es in keratin expression, respectively, with nontransgenic wild-type (WT) mice.

299 had twofold increase in superoxide levels in nontransgenic (wild-type [WT]) diabetic mice compared wi

300 (+) regulatory T cells (Tregs) compared with nontransgenic WT mice.