ライフサイエンスコーパス: noradrenaline

1 inal cord and is the major source of central noradrenaline.

2 creases extracellular levels of dopamine and noradrenaline.

3 adrenergic projections and are responsive to noradrenaline.

4 red both the presence of adult-born cell and noradrenaline.

5 amine agonist ligands such as adrenaline and noradrenaline.

6 ne oxidase A (MAOA) that is known to degrade noradrenaline.

7 of VLPO neurons, which are also inhibited by noradrenaline.

8 nd neurotensin, as well as acetylcholine and noradrenaline.

9 stems releasing the monoamines serotonin and noradrenaline.

10 s in mice by lowering the bioavailability of noradrenaline.

11 tamate reversed the constriction produced by noradrenaline.

12 levated blood pressures and increased plasma noradrenaline.

13 cularly the catecholamines dopamine (DA) and noradrenaline.

14 Similar effects were produced with noradrenaline.

15 LPS) thermogenesis, but a normal response to noradrenaline.

16 Noradrenaline (0.005 mg ml(-1)) induced much lower sweat

17 Topical noradrenaline (1 nM) blunted ROV in YM and YF to levels

18 shold stimulation of alphaARs in young mice (noradrenaline; 10(-9) m) depressed ROV most effectively

19 e of alpha7 nAChR in the modulation of [(3)H]noradrenaline ([(3)H]NA) release from rat hippocampal sl

20 trol sites (control, 38.2 +/- 0.5 degrees C; noradrenaline, 37.7 +/- 0.4 degrees C, P < 0.05, n = 5).

21 ia increased plasma adrenaline (26-fold) and noradrenaline (5-fold) in shams but elicited no change i

22 were significantly suppressed by hyperoxia (noradrenaline 50.7 +/- 5.2%, adrenaline 62.6 +/- 3.3%, c

23 Dialysis with noradrenaline abrogated this rise.

24 Here we assess the roles of noradrenaline, acetylcholine, and dopamine within a sing

25 stimulated in a store-independent manner by noradrenaline acting on alpha-adrenoceptors and by diacy

26 Spillover and subsequent pooling of noradrenaline activated distal alpha2-receptors, which p

27 Here, we show that noradrenaline activates alpha2-adrenergic receptors to c

28 Noradrenaline acts through adrenergic receptors (ARs), o

29 ular resistance, in plasma concentrations of noradrenaline, adrenaline and vasopressin, and in the ma

30 erived factor, non-esterified fatty acids or noradrenaline (all P > 0.05).

31 Noradrenaline also evoked TRPC1 channel activity and ass

32 he options searched by rats, suggesting that noradrenaline also plays a role in balancing exploration

33 For example, locus coeruleus noradrenaline (also known as norepinephrine) (LC-NE) neu

34 a2-adrenoreceptor in complex with a covalent noradrenaline analog and a conformationally selective an

35 ed cells with the capacity to accumulate the noradrenaline analog metaiodobenzylguanidine (MIBG).

36 ut the greatest effort has been in preparing noradrenaline analogs, such as norepinephrine, (11)C-met

37 in synaptic AMPAR phenotype was mediated by noradrenaline and action potential prolongation.

38 e wake- and sleep-promoting neuromodulators, noradrenaline and adenosine, respectively.

39 Noradrenaline and adrenaline dose-dependently suppressed

40 Plasma noradrenaline and adrenaline levels rose rapidly with dr

41 sure myography to assess the contribution of noradrenaline and ATP to sympathetic neurotransmission i

42 their synaptic transmission, the effects of noradrenaline and clonidine were tested on the capsaicin

43 current evidence that synergistic action of noradrenaline and cortisol enables emotional stimuli to

44 The closely related transporters for noradrenaline and dopamine depend on SEC24D.

45 led, study that examined specific effects of noradrenaline and dopamine on both metacognition and per

46 fects of the catecholamine neurotransmitters noradrenaline and dopamine on prefrontal cortical functi

47 sine, the corresponding residue found in the noradrenaline and dopamine transporters, switched the SE

48 associated with region-dependent changes in noradrenaline and GABA levels in key areas of the limbic

49 ement with these findings, the beta-agonists noradrenaline and isoproterenol stimulate OEA production

50 NSSs for dopamine, noradrenaline and serotonin are targeted by the psychost

51 e neurons, resulting in increased release of noradrenaline and serotonin in forebrain areas involved

52 a double dissociation for the involvement of noradrenaline and serotonin in human cognition.

53 nd found a significant decrease in levels of noradrenaline and the serotonin metabolite 5-hydroxyindo

54 ated pressurized MAs preconstricted 50% with noradrenaline and treated with guanethidine (to inhibit

55 enal function, plasma renin activity, plasma noradrenaline and vasopressin concentration, and serum l

56 plasma levels of catecholamines (adrenaline, noradrenaline) and biomarkers reflecting endothelial dam

57 sruption of the catecholamines (dopamine and noradrenaline) and review the efficacy of catecholaminer

58 re neuromodulatory factors such as dopamine, noradrenaline, and brain-derived neurotrophic factor [3-

59 mice resulted in reduced striatal serotonin, noradrenaline, and dopamine concentrations and a signifi

60 drugs (terlipressin, midodrine, octreotide, noradrenaline, and dopamine; alone or in combination) wi

61 cular conductance, increasing jugular venous noradrenaline, and falling arterial carbon dioxide tensi

62 ons of cortisol, vasopressin, adrenaline and noradrenaline, and falls in the fetal : maternal ratio o

63 ns that release the monoamines serotonin and noradrenaline, and local vessel dilation is induced by g

64 rictor; terlipressin; midodrine; octreotide; noradrenaline; and norepinephrine.

65 tested against the removal time of 5-HT and noradrenaline applied by microinjection to the NTS.

66 Since the major source of noradrenaline are neurons in the locus coeruleus, we hyp

67 lators such as acetylcholine, serotonin, and noradrenaline are powerful regulators of neocortical act

68 in hippocampus, and suggest novel roles for noradrenaline as a modulator of the hippocampal search p

69 ls of insulin, cortisol, growth hormone, and noradrenaline, as well as hypoglycemic symptoms and cogn

70 T-type channels is significantly elevated by noradrenaline at 8 h and 24 h.

71 nal inhibitory effect through the actions of noradrenaline at spinal alpha2 -adrenoceptors, although

72 In the presence of siderophore, noradrenaline augmented transferrin utilization by B. br

73 a cell type selective manner, with 5-HT and noradrenaline being limited to presynaptic cells, GABA b

74 onal substrate and cofactor availability for noradrenaline biosynthesis.

75 Older patients had high plasma noradrenaline but attenuated adrenaline release with hig

76 abnormalities did not depend on the loss of noradrenaline but were abolished by the antiparkinsonian

77 To address whether noradrenaline can down-regulate TRPV1 channel activity i

78 Noradrenaline can modulate multiple cellular functions i

79 e neurons was found in locus coeruleus/A5/A7 noradrenaline cell groups, whereas the extent of neurona

80 the locus coeruleus and adjoining A5 and A7 noradrenaline cell groups.

81 9 versus 3.4 +/- 5.6 ng/mL*h, P = 0.03), and noradrenaline concentration (544 +/- 334 versus 402 +/-

82 that was evidenced by increased circulating noradrenaline concentration had little effect on HR, pot

83 f MAOA reversed the age-related reduction in noradrenaline concentration in adipose tissue, and resto

84 Circulating noradrenaline concentration increased from 0.9 +/- 0.4 n

85 .001) and reduced adrenaline (P < 0.001) and noradrenaline concentrations (P < 0.001).

86 cant hyperthermia, associated with increased noradrenaline concentrations in brown adipose tissue.

87 Under normoxaemic conditions plasma noradrenaline concentrations were lower in AD fetuses th

88 Adrenaline and noradrenaline concentrations were lower in the CW group

89 dorsal horn receives a dense innervation of noradrenaline-containing fibers that originate from pont

90 These results suggest that endogenous NO and noradrenaline contribute to the temperature threshold of

91 eptors and reveal a novel mechanism by which noradrenaline controls synaptic strength and plasticity

92 Adrenaline and noradrenaline correlated with syndecan-1 (r = 0.38, P <

93 e investigated potential mechanisms by which noradrenaline could directly influence the survival of a

94 Noradrenaline crucially modulates emotional memory.

95 The area under the noradrenaline curve (relative to the normoxia response)

96 Noradrenaline-dependent plasticity affects the output of

97 Furthermore, noradrenaline did not elicit normal increases in lymphat

98 usion predicted that the functional range of noradrenaline diffusion was nearly fivefold greater in t

99 ent of corticotropin-releasing factor (CRF), noradrenaline, dopamine, glutamate, kappa/dynorphin, and

100 derived from the monoamine neurotransmitters noradrenaline, dopamine, serotonin, and histamine, and t

101 y facilitating the inhibitory control of the noradrenaline-excited neurons, histamine may inhibit the

102 normally be under the inhibitory control of noradrenaline-excited neurons.

103 Conversely, on the noradrenaline-excited VLPO neurons, histamine depolarize

104 The endogenous neurotransmitter noradrenaline exerts anti-inflammatory and neuroprotecti

105 Culturing rat pinealocytes in noradrenaline for 24 h induced a low-voltage activated t

106 B. bronchiseptica can use the catecholamine noradrenaline for growth on transferrin.

107 pharmacologically (tyramine, which displaces noradrenaline from axon terminals) induced vasoconstrict

108 signal in response to nocturnal delivery of noradrenaline from sympathetic neurons.

109 ll show that in the dorsal cochlear nucleus, noradrenaline functions to simultaneously reduce spontan

110 r dopamine, 5-hydroxytryptamine (serotonin), noradrenaline, GABA and glycine.

111 ransmission mediated by serotonin, dopamine, noradrenaline, glycine and GABA (gamma-aminobutyric acid

112 such as serotonin, dopamine, histamine, and noradrenaline have important and varied physiological fu

113 ling from the stress hormones adrenaline and noradrenaline, have a lower incidence of prostate cancer

114 The addition of adrenaline, noradrenaline, hydrocortisone, or dexamethasone to lipop

115 hetic discharge, resulting in the release of noradrenaline in brown adipose tissue and white adipose

116 Superfused ATP in retina or noradrenaline in cerebellum resulted in constriction of

117 ent and animal studies points to the role of noradrenaline in dopaminergically insensitive aspects of

118 Responses to noradrenaline in endothelium-intact and endothelium-denu

119 ision making and uncovers a specific role of noradrenaline in energizing behavior to face challenges.

120 suggest that modulation of TRPV1 channels by noradrenaline in nociceptive neurons is a mechanism wher

121 secreting the catecholamines adrenaline and noradrenaline in the 'fight-or-flight' response.

122 Transcription is recruited by noradrenaline in the hippocampus.

123 eptor signaling via adrenergic nerve-derived noradrenaline in the prostate stroma is critical for act

124 In contrast, increasing the release of noradrenaline in the rat locus ceruleus prolonged the du

125 ceptor agonist, which decreases the level of noradrenaline in vivo, has an interesting effect in huma

126 e invertebrate counterpart of adrenaline and noradrenaline, in synaptic and behavioral plasticity in

127 ce of Ca2+ channel activation and attenuated noradrenaline-induced G protein modulation.

128 ed synaptic transmission and also attenuated noradrenaline-induced G protein modulation.

129 Acute noradrenaline-induced hyperthermia requires UCP1 but not

130 lation at these synapses, demonstrating that noradrenaline-induced suppression of glutamate release i

131 The noradrenaline-induced T-type channel mediated an increas

132 Acting via the beta(3)-adrenergic receptors, noradrenaline induces lipolysis in white adipocytes, whe

133 We investigated whether noradrenaline induces plasticity at the glomerulus.

134 We propose that noradrenaline influences learning of uncertain events ar

135 Low-dose noradrenaline infusion alone shifted the axon reflex to

136 renergic blockade, with and without low-dose noradrenaline infusion, also abolished the axon reflex r

137 etylium tosylate); and low-dose (0.1 microM) noradrenaline infusion.

138 these findings, we propose that in the VLPO, noradrenaline-inhibited neurons may normally be under th

139 e-excited neurons, histamine may inhibit the noradrenaline-inhibited neurons, resulting in excitation

140 model emphasizes the role of focal glutamate-noradrenaline interactions in creating functional hotspo

141 Noradrenaline is believed to support cognitive flexibili

142 ves extensive noradrenergic projections, and noradrenaline is released in the hippocampus in response

143 eruleus, we hypothesized that alterations in noradrenaline levels are a consequence of stress or dama

144 Several studies report that noradrenaline levels are altered in the central nervous

145 Since reduced noradrenaline levels could be permissive for increased i

146 mpathetic vasomotor tone (P < 0.001) and the noradrenaline levels in cerebrospinal fluid and plasma (

147 these results suggest that methods to raise noradrenaline levels or increase locus coeruleus functio

148 levels in sympathetic fibers and bone marrow noradrenaline levels rose (P<0.05, respectively), associ

149 human ovarian cancer samples, high tumoural noradrenaline levels were correlated with high pSrc(Y419

150 ease in astrocyte activation, a reduction in noradrenaline levels, and neuronal stress indicated by h

151 glomerular filtration rate and plasma renin, noradrenaline, lipopolysaccharide binding protein, tropo

152 n nociceptive neurons is a mechanism whereby noradrenaline may suppress incoming noxious stimuli at t

153 Amine neurotransmitters, such as noradrenaline, mediate arousal, attention, and reward in

154 ron firing following a stressor depends on a noradrenaline-mediated mechanism.

155 Results: Noradrenaline-mediated vasoconstriction was increased in

156 Noradrenaline mediates the effects of different hormones

157 in several pro-oxidant molecules involved in noradrenaline metabolism.

158 ly low-quality evidence supported the use of noradrenaline, midodrine plus octreotide, and dopamine p

159 Noradrenaline modulates global brain states and diverse

160 Noradrenaline (NA) and adrenaline (ADR) effluxes were mo

161 pressed significantly reduced levels of both noradrenaline (NA) and NA-synthesizing dopamine beta-hyd

162 ypothesis that forearm vascular responses to noradrenaline (NA) and tyramine (TYR) are related to SNA

163 pinealocytes is changed by culturing them in noradrenaline (NA) as a surrogate for the night signal.

164 responsible for the 'distal' effect by which noradrenaline (NA) blocks exocytosis in the beta-cell we

165 The adrenoceptor agonist noradrenaline (NA) caused a dose-related reduction in ev

166 us ceruleus (LC) degeneration and subsequent noradrenaline (NA) deficiency in early Alzheimer's disea

167 at the sensitization of cutaneous vessels to noradrenaline (NA) during direct skin cooling seen in yo

168 direct role of M-channels in the release of noradrenaline (NA) from SNs.

169 and cognitive function, the specific role of noradrenaline (NA) in AD is not well understood.

170 The role of prefrontal noradrenaline (NA) in higher order flexibility indexed t

171 ations of monoamine neurotransmitter such as noradrenaline (NA) in living cells with simple, sensitiv

172 Current models propose that noradrenaline (NA) increases excitation of mitral/tufted

173 essory OB (AOB), we have recently shown that noradrenaline (NA) increases GABA inhibitory input on to

174 chanism for long-term potentiation ABSTRACT: Noradrenaline (NA) is a neuromodulator that can effect l

175 ell recordings from identified SDH neurones, noradrenaline (NA) or serotonin (5HT) were briefly appli

176 We simultaneously measured noradrenaline (NA) overflow at the blood vessel adventit

177 -Dawley (SD) rats and acetylcholine (ACh) or noradrenaline (NA) release to field stimulation was meas

178 femoral artery infusions of tyramine (evokes noradrenaline (NA) release), phenylephrine (alpha1-agoni

179 Although noradrenaline (NA) reuptake inhibitors are shown to incr

180 Moreover, bath application of noradrenaline (NA) significantly depolarizes URS motoneu

181 Noradrenaline (NA) strengthens memory (re)consolidation.

182 onspecific odor cues coincide with surges of noradrenaline (NA) triggered by intensely arousing socia

183 raded intradermal microdialysis infusions of noradrenaline (NA) were measured during GnRH antagonist,

184 ation of endocytosis following exocytosis by noradrenaline (NA), a physiological inhibitor of insulin

185 Neurotransmitters, such as noradrenaline (NA), and neuropeptides, including nocicep

186 timulated release of preloaded radiolabelled noradrenaline (NA), dopamine (DA) and serotonin (5-HT) i

187 Noradrenaline (NA, non-selective alphaAR agonist) constr

188 lar conductance (FVC) to increasing doses of noradrenaline (NA; 2, 4 and 8 ng (100 ml)(-1) min(-1)) b

189 ons and that other neurotransmitter systems [noradrenaline (NE) and serotonin (5-HT)] were increased

190 Neuromodulatory systems such as noradrenaline (NE), acetylcholine (ACh), and serotonin (

191 thod that appeared not to be associated with noradrenaline nerve fibres.

192 , reduced or augmented the evoked release of noradrenaline neurotransmitter from superior cervical ga

193 of the lateral calf for graded infusions of noradrenaline (norepinephrine) (NA; 10(-12) to 10(-2) m)

194 investigate the signalling pathway involved, noradrenaline (norepinephrine) and the beta(3)-adrenergi

195 rmally grown sheep fetuses following a 7-day noradrenaline (norepinephrine) infusion.

196 al beta-adrenergic receptor stimulation with noradrenaline (norepinephrine; NA, 50 mul, 250 muM) was

197 , which emphasizes the qualitative effect of noradrenaline of updating the representation of the envi

198 Epigenetic regulation by noradrenaline offers a novel mechanism for long-term pot

199 First, I will review the effects of noradrenaline on GABAergic networks.

200 th an emphasis on the quantitative effect of noradrenaline on stimulus processing.

201 blocked with KN-93, the inhibitory effect of noradrenaline on the capsaicin-activated current was gre

202 The inhibitory effect of noradrenaline on the capsaicin-activated current was not

203 Here, we studied the effects of noradrenaline on the functional connectivity of GABAergi

204 vestigated the role of nitric oxide (NO) and noradrenaline on the temperature threshold for the axon

205 The inhibitory effect of noradrenaline on TRPV1 channels was dependent on calcium

206 o major stress neuromodulators, cortisol and noradrenaline, on loss aversion during financial decisio

207 98 +/- 20%, an effect reproduced by chronic noradrenaline or CL 316 243.

208 Noradrenaline or clonidine inhibited the capsaicin-activ

209 Application of noradrenaline or of the selective beta-adrenergic agonis

210 enic T cells that had been preincubated with noradrenaline or splenocytes harvested from stressed mic

211 Importantly, infusion of noradrenaline or vasopressin to normalize the chronic hy

212 d vasoactive therapy-dopamine, and if needed noradrenaline) or intracranial pressure-targeted therapy

213 on-dopamine systems, such as those involving noradrenaline, or by dopamine systems outside the SNC.

214 hat increased sympathetic nerve activity and noradrenaline outflow associated with hypertension may b

215 h low-quality evidence supporting the use of noradrenaline over placebo (4.17, 1.37-12.50) and over m

216 Older trauma patients had markedly higher noradrenaline (p < .001) but an attenuated increase in a

217 Arterial adrenaline (P < 0.05) and venous noradrenaline (P < 0.05) were higher with hypoxia during

218 ertebrate homolog of mammalian adrenaline or noradrenaline, plays important roles in modulating behav

219 lb (MOB) is richly targeted by LC fibers and noradrenaline profoundly influences MOB circuitry and od

220 and increase 3-methoxy-4-hydroxyphenylglycol/noradrenaline ratio.

221 administered 40 mg atomoxetine, a selective noradrenaline re-uptake inhibitor to 25 patients with Pa

222 ants and the newer, safer class of serotonin/noradrenaline re-uptake inhibitors, for example duloxeti

223 inals are found in the glomerular layer, but noradrenaline receptors do not seem to acutely modulate

224 oreceptors in brain in vivo, here applied to noradrenaline receptors in rat brain.

225 Instead, noradrenaline reduces associative plasticity by selectiv

226 raction of resistance vessels is enhanced by noradrenaline release along perivascular sympathetic ner

227 slow LC is due to a combination of increased noradrenaline release and decreased activity of both NOS

228 a glutamate "priority" signal that modulates noradrenaline release depending on arousal state.

229 We conclude that noradrenaline release from LC has persistent effects on

230 cy is explained by presynaptic inhibition of noradrenaline release in the pPVN, due to increased endo

231 central opioid mechanism that auto-inhibits noradrenaline release in the PVN.

232 ibit vasoconstrictor responses to endogenous noradrenaline release via tyramine in the skeletal muscl

233 rm potentiation in vivo and on AMPA-mediated noradrenaline release were measured to predict its poten

234 o intra-arterial tyramine (evokes endogenous noradrenaline release), phenylephrine (alpha1-agonist) a

235 lanation: that priority itself evokes phasic noradrenaline release.

236 been attributed to the beneficial effects of noradrenaline released into the amygdala attributable to

237 roteins on binding the agonists adenosine or noradrenaline, respectively.

238 In healthy volunteers, inhibition of central noradrenaline reuptake improved response inhibition but

239 tcomes with serotonin reuptake inhibiting or noradrenaline reuptake inhibiting antidepressants and av

240 Parkinson's disease found that the selective noradrenaline reuptake inhibitor atomoxetine could impro

241 f action of methylphenidate, infusion of the noradrenaline reuptake inhibitor atomoxetine into these

242 erefore examined the effect of the selective noradrenaline reuptake inhibitor atomoxetine on response

243 The noradrenaline reuptake inhibitor reboxetine attenuates e

244 this approach using atomoxetine, a selective noradrenaline reuptake inhibitor that modulates the pref

245 Atomoxetine (ATO), a selective noradrenaline reuptake inhibitor, had no effect on respo

246 ti-obesity drug sibutramine, a serotonin and noradrenaline reuptake inhibitor.

247 tonin reuptake inhibitors (SSRIs), serotonin-noradrenaline reuptake inhibitors (SNRIs) and tricyclic

248 NNTs were 6.4 (95% CI 5.2-8.4) for serotonin-noradrenaline reuptake inhibitors, mainly including dulo

249 s overall, serotonin reuptake inhibitors, or noradrenaline reuptake inhibitors, or differential respo

250 ain for tricyclic antidepressants, serotonin-noradrenaline reuptake inhibitors, pregabalin, and gabap

251 aturated as a consequence of a dysfunctional noradrenaline reuptake mechanism.

252 vealed that this may be due to dysfunctional noradrenaline reuptake.

253 serotonin-reuptake inhibitors and serotonin-noradrenaline-reuptake inhibitors, are effective, and th

254 er responses to sub-EC(50) concentrations of noradrenaline revealed oscillatory Ins(1,4,5)P(3), DAG,

255 ee of sympathetic nervous system activation (noradrenaline, rho = 0.26, P = 0.05; heart rate variabil

256 nes of the molecular clock through circadian noradrenaline secretion by the sympathetic nervous syste

257 regulation; (iv) neurotransmitter (dopamine, noradrenaline, serotonin) expressions, transporter or re

258 We found that noradrenaline simultaneously reduced spontaneous inhibit

259 Noradrenaline strongly inhibited the activity of TRPV1 c

260 lability of BH(4) and/or tyrosine may impair noradrenaline synthesis and contribute to the attenuated

261 nse inhibition, whereas manipulations of the noradrenaline system did.

262 blunted methamphetamine and fully inhibited noradrenaline thermogenesis, but an increased febrile re

263 rs in mature BAT, increasing the response to noradrenaline through enhanced p38MAPK/CREB signaling an

264 ting sympathoadrenal activation (adrenaline, noradrenaline), tissue/endothelial cell/glycocalyx damag

265 for the enzyme responsible for conversion of noradrenaline to adrenaline.

266 nergic inputs may constitute a mechanism for noradrenaline to modulate incoming noxious stimuli in th

267 o the host NE stress hormones adrenaline and noradrenaline to modulate virulence.

268 of monoamine oxidase A, restored hippocampal noradrenaline to normal levels (from 60 to 97% of vehicl

269 isms underlying somatodendritic dopamine and noradrenaline transmission and found that the extent of

270 studies indicate the involvement of CRF and noradrenaline transmission in bed nucleus of stria termi

271 al, we have engineered HSV1716 to convey the noradrenaline transporter (NAT) gene (HSV1716/NAT), whos

272 is compensated for by the expression of the noradrenaline transporter (NAT) gene, and not the seroto

273 t of human SK-N-BE(2c) neuroblastoma and UVW/noradrenaline transporter (NAT) glioma cells.

274 erm DNA damage and supraadditive toxicity to noradrenaline transporter (NAT)-expressing cells and xen

275 d by the serotonin transporter (SERT) or the noradrenaline transporter (NET) inhibitors citalopram an

276 G to spheroids and xenografts expressing the noradrenaline transporter.

277 osine hydroxylase expression and that of the noradrenaline transporter; and enhanced expression and s

278 A mutation in the norepinephrine (noradrenaline) transporter gene prompted further genetic

279 C activation was also found with recombinant noradrenaline transporters and in rat hippocampal slices

280 d PET with (11)C-MeNER to map the density of noradrenaline transporters in groups of patients with PD

281 )C-MeNER) is a highly selective inhibitor of noradrenaline transporters, and PET studies suggest that

282 essin-treated patients, and 33% (range 6-40) noradrenaline-treated patients with reversal of hepatore

283 ency of Ca(2+) oscillations induced by 1 muM noradrenaline was 150% higher than in the wild-type.

284 Furthermore, noradrenaline was an independent predictor of <24-hours

285 Noradrenaline was as effective as terlipressin for the t

286 tion was enhanced in male CF mice, and renal noradrenaline was increased in female CF mice.

287 Inhibition of capsaicin-activated current by noradrenaline was mediated by GTP binding proteins, and

288 The effect of noradrenaline was reproduced by clonidine and antagonize

289 while total peripheral resistance and plasma noradrenaline were not similarly lower; upright total ac

290 neuroendocrine (NE) hormones adrenaline and noradrenaline were reported to display cross-talk for th

291 i O157:H7, the catecholamines adrenaline and noradrenaline were shown to act synergistically with a b

292 disease, cortical and spinal cord levels of noradrenaline were significantly reduced versus control

293 um-22 attenuated the removal of 5-HT but not noradrenaline, whereas desipramine had the reverse actio

294 The second group comprised adrenaline and noradrenaline which displayed higher intrinsic activity

295 e loss of other neurotransmitters, including noradrenaline, which is linked to impulsivity and respon

296 engaging the stress hormone/neurotransmitter noradrenaline, which mediates and modulates the consolid

297 e, sympathetic nerve fibers released surplus noradrenaline, which signaled bone marrow niche cells to

298 ific immune cells are activated by increased noradrenaline, while others are in fact suppressed.

299 Terlipressin with albumin and noradrenaline with albumin are both superior to midodrin

300 Thus, cotransmission of dopamine and noradrenaline with either GABA or glutamate appears to b