ライフサイエンスコーパス: nordihydroguaiaretic acid

1 including rapamycin, 17-alpha-estradiol, and nordihydroguaiaretic acid.

2 lfonamide dihydrochloride], indomethacin, or nordihydroguaiaretic acid.

3 y inhibited by caffeic acid, resveratrol and nordihydroguaiaretic acid.

4 Similarly, inhibition of lipoxygenase with nordihydroguaiaretic acid (1 micromol/L), eicosatetrayno

5 Treatment with the lipoxygenase inhibitor nordihydroguaiaretic acid (10 microM) did not affect tum

6 by AA or DHA was unaffected by extracellular nordihydroguaiaretic acid (10 microM), indomethacin (10

7 of the AT1 receptor-subtype of AngII, and by nordihydroguaiaretic acid (50 microM), which at this con

8 Neither nordihydroguaiaretic acid (a lipoxygenase inhibitor) nor

9 Nordihydroguaiaretic acid, a lipoxygenase inhibitor, pre

10 The addition of nordihydroguaiaretic acid, a lipoxygenase inhibitor, sig

11 Herein we report that nordihydroguaiaretic acid, a pan inhibitor of lipoxygena

12 Nordihydroguaiaretic acid, an inhibitor of the lipoxygen

13 ed the effects of 5-lipoxygenase inhibitors (nordihydroguaiaretic acid and AA861) on IL-1 beta-induce

14 clusion of the characteristic LOX inhibitors nordihydroguaiaretic acid and eicosatetraynoic acid redu

15 nked lignans, whose representatives, such as nordihydroguaiaretic acid and its congeners, have potent

16 The AP-1 inhibitor, nordihydroguaiaretic acid, and a c-Fos small interfering

17 e-1-oxide, the antioxidants desferrioxamine, nordihydroguaiaretic acid, and Amytal, and by the enhanc

18 A LOX inhibitor, nordihydroguaiaretic acid, attenuated production of 15(S

19 ffects were not prevented by indomethacin or nordihydroguaiaretic acid, blockers of cyclooxygenase an

20 The addition of 13-HODE to the nordihydroguaiaretic acid-containing whole blood perfusa

21 ne expression is inhibited by tetra-O-methyl nordihydroguaiaretic acid (M(4)N) and that M(4)N is like

22 The transcription inhibitor tetra-O-methyl nordihydroguaiaretic acid (M4N) was found to arrest the

23 Investigation of the LOX inhibitors nordihydroguaiaretic acid, N-(4-chlorophenyl)-N-hydroxy-

24 Neutralizing ROS with nordihydroguaiaretic acid (NDGA) abrogated cell death in

25 Our labs have synthesized and investigated nordihydroguaiaretic acid (NDGA) derivatives and have es

26 of neurons with 8-bromo-cAMP (8-Br-cAMP) or nordihydroguaiaretic acid (NDGA) had no effect on the re

27 Nordihydroguaiaretic acid (NDGA) was also characterized

28 Nordihydroguaiaretic acid (NDGA) was observed by Ono et

29 agent that does not inhibit COX enzymes; and nordihydroguaiaretic acid (NDGA), a lipoxygenase inhibit

30 the metabolism of AA by cyclo-oxygenase, nor nordihydroguaiaretic acid (NDGA), an inhibitor of the li

31 We found that the antioxidants nordihydroguaiaretic acid (NDGA), catechol, glutaryl pro

32 igated the effects of a hypolipidemic agent, nordihydroguaiaretic acid (NDGA), on host lipid/fatty ac

33 (Dex), mepacrine (Mep), indomethacin (Indo), nordihydroguaiaretic acid (Ndga), phenylephrine (Pe), so

34 Indomethacin and nordihydroguaiaretic acid (NDGA), potent inhibitors of t

35 ere treated with the lipoxygenase inhibitor, nordihydroguaiaretic acid (NDGA), the 5-lipoxygenase inh

36 lipoxygenase or inhibiting its activity with nordihydroguaiaretic acid or luteolin.

37 se inhibitors 5,8,11-eicosatrienoic acid and nordihydroguaiaretic acid partially prevented MIN6 cell

38 trathecal (IT) delivery of the LOX inhibitor nordihydroguaiaretic acid prevented the carrageenan-evok

39 ng enzymes, only the lipoxygenase inhibitor, nordihydroguaiaretic acid reduced EGF-mediated ROS accum

40 S, pyrrolidine dithiocarbamate, pyruvate, or nordihydroguaiaretic acid, reduced both cellular oxidant

41 n diet-associated constituents, curcumin and nordihydroguaiaretic acid, reversibly targets UGTs causi

42 t with the antioxidants N-acetylcysteine and nordihydroguaiaretic acid, suggesting a role for oxidati

43 The lipoxygenase inhibitor nordihydroguaiaretic acid, the specific leukotriene D4 (

44 AA was, at best, only partially inhibited by nordihydroguaiaretic acid treatment.

45 ause incubation with 5-LO inhibitors (AA861, nordihydroguaiaretic acid, zileuton) but not a flavoenzy