ライフサイエンスコーパス: normal control

1 etween the two groups (i.e., glaucoma versus normal control).

2 and patients with femoral neck fracture (as normal control).

3 f juvenile rats (hyperglycemic, ketotic, and normal control).

4 nd II plasma tested was less than 40% of our normal control.

5 nts possessing different FUS mutations and a normal control.

6 i disease caused by mutations in GRK1, and 1 normal control.

7 group showed increased levels comparable to Normal-Control.

8 n their peripheral blood, in comparison with normal controls.

9 n participants with early AD and cognitively normal controls.

10 wedish AD cases, and 707 Swedish cognitively normal controls.

11 ith early, diffuse SSc compared with that in normal controls.

12 epair for isolated MR, were compared with 13 normal controls.

13 ncreased in PDAC patients by comparison with normal controls.

14 : Age and sex matched healthy individuals as normal controls.

15 umented dogs, 8 with pacing-induced HF and 6 normal controls.

16 n capillaries of diabetic animals but not in normal controls.

17 quire data from reference samples or matched normal controls.

18 d BRB (with normal fundus or initial DR) and normal controls.

19 ls of diabetic animals (P<0.01), compared to normal controls.

20 ere no false positives in the 25 age-matched normal controls.

21 losive disorder compared with psychiatric or normal controls.

22 fficient to distinguish these specimens from normal controls.

23 atients whose disease was in remission or in normal controls.

24 ein, in some cases more than 50% relative to normal controls.

25 ic leukemia, whereas it was not expressed in normal controls.

26 sample of 200 angle closure subjects and 302 normal controls.

27 ntly decreased in pups with NEC, compared to normal controls.

28 in athletes with comparable hypertrophy and normal controls.

29 tia and compare them to those of cognitively normal controls.

30 dneys of mice with diabetes than in those of normal controls.

31 cts with mild cognitive impairment (MCI) and normal controls.

32 ure with FMR, heart failure without FMR, and normal controls.

33 Ten Sprague-Dawley rats served as normal controls.

34 ons, rather than hypothesis-rejection versus normal controls.

35 m the majority of MDS patients compared with normal controls.

36 ere 60-fold higher in patients with AKI than normal controls.

37 -regulated genes in IPF lungs, compared with normal controls.

38 ma9Vdelta2 T cells was reduced compared with normal controls.

39 le-nucleobase lesions than the corresponding normal controls.

40 and 73 individually age- and gender-matched normal controls.

41 s, 11 with organic movement disorders and 12 normal controls.

42 obe of AD cases and age-matched, cognitively normal controls.

43 LC cell lines and tissues as compared to the normal controls.

44 VCDR were genotyped in 876 OAG cases and 883 normal controls.

45 rt DCM subjects, and 29 related mutation (-) normal controls.

46 significantly higher in VF myocytes than in normal controls.

47 f glycosylation (CDG) patients compared with normal controls.

48 evident comparing lamellar bone formation to normal controls.

49 how increased HOXA9 expression compared with normal controls.

50 e measured and compared with the age-matched normal controls.

51 se (HD) patients in comparison with those of normal controls.

52 mained unchanged in comparison with those of normal controls.

53 ease, had poorer quality of life compared to normal controls.

54 iety of pediatric cancer lines compared with normal controls.

55 monkeys (Macaca nemestrina) and two visually normal controls.

56 the submacular RPE on OCT when compared with normal controls.

57 tabolic syndrome compared with metabolically normal controls.

58 men and 21 women) were age- and sex-matched normal controls.

59 ontotemporal dementia against neurologically normal controls.

60 nochoroidopathy and 37 eyes of 37 historical normal controls.

61 adults with poorly controlled asthma and 10 normal controls.

62 poral dementia (FTD) and matched cognitively normal controls.

63 c profiles of PD were clearly different from normal controls.

64 ures are indistinguishable from age-matched, normal controls.

65 rment and AD dementia compared to clinically normal controls.

66 tients with acoustic neuromas compared to 24 normal controls.

67 ndemented controls compared with age-matched normal controls.

68 of seizures, and 19 age-matched, cognitively normal controls.

69 ive CNV due to AMD, non-neovascular AMD, and normal controls.

70 opa metabolites and biliverdin than those of normal controls.

71 nary artery vasculopathy with 35 age-matched normal controls.

72 ity of Pittsburgh (U. Pitt., n = 70) than in normal controls.

73 pective study of individuals with CRSwNP and normal controls.

74 decreased hippocampal activity, relative to normal controls.

75 e to TE was higher than that measured in the normal controls (1.07%).

76 asmablast proportions (3.1 +/- 0.8%) than in normal controls (1.3 +/- 0.3%) (p < 0.03), and the exten

77 nd 73 +/- 26), compared with nonrejectors or normal controls (10.6 +/- 7.9 or 10.7 +/- 6.5, P=0.001).

78 In comparison with data from normal controls, 12 patients had normal (11)C-HED PET, 5

79 2.05 mum) was significantly less compared to normal control (245 +/- 15.87 mum; P = .004).

80 es (34 adults, 51 children) and 110 visually normal controls (44 adults, 66 children).

81 The cohort consisted of 483 subjects: 105 normal controls (55 men; 50 women; and median age, 14 ye

82 tly higher in patients with chronic HCV than normal controls (65.9 % vs 28.7 %, respectively).

83 12 normal controls, 7 patients with primary open angle glau

84 d in 8943 US AD cases, 10 480 US cognitively normal controls, 862 Swedish AD cases, and 707 Swedish c

85 (aged 53.7 +/- 9.8 years) and 30 eyes of 15 normal controls (aged 50.7 +/- 9.8 years).

86 Fifty-one strabismic children and 130 normal controls ages 3 to 17 years participated.

87 modulator (GSM) in neurons derived from both normal control and 3 PS1 mutations (A246E, H163R, and M1

88 RNA in MLL-rearranged AML compared with both normal control and non-MLL-rearranged AML.

89 Normal rats were used as normal control and were divided into NC and NR group fed

90 obtained at autopsy, from 18 neurologically normal controls and 14 individuals who had primary restl

91 r thickness (CMT) were measured using OCT in normal controls and 3 months postoperatively in children

92 Group comparisons between normal controls and AD patients showed no significant ov

93 CD8(+) T cells, reaching levels observed in normal controls and also modestly increased the number o

94 ant difference expression (p < 0.05) between normal controls and gastric cancer patients, including 7

95 Compared to both normal controls and hypertensive heart disease patients,

96 sis had higher serum levels of IFNgamma than normal controls and patients with ET showed higher IFNga

97 icited by HDACi 4b in human fibroblasts from normal controls and patients with Huntington's disease (

98 ethod, we compare a person with amnesia with normal controls and we compare people with induced happy

99 Of these, 42 subjects had IED, 40 were normal controls, and 50 were clinical psychiatric contro

100 e cancer were more efferocytic compared with normal controls, and CXCL5 serum levels were higher in m

101 stinguish different disease types as well as normal controls, and highlight the importance of cell co

102 and occipital cortex compared to clinically normal controls, and in probable DLB, the uptake in thes

103 fold (P=0.005) higher in MR patients than in normal controls, and LV ejection fraction was 64+/-7% (5

104 ferent types of patients (Meniere's disease, normal controls, and other otopathologic conditions) and

105 sentation of a noise mask to the FE, as in a normal control animal.

106 differential methylation calling method when normal controls are not available.

107 ta and right carotid artery as compared with normal control arteries (mean TBR = 1.95 +/- 0.51 vs. 1.

108 ta and right carotid artery as compared with normal control arteries (mean TBR = 1.95 +/- 0.51 vs. 1.

109 umes, lower RVEF and global area strain than normal controls as expected.

110 DNA content can be detected in the brains of normal controls, as well as in Rett syndrome patients.

111 amsters were classified into eight groups: a normal control, atherogenic control and six other experi

112 ne tumours plus eight human and three murine normal controls, augmented with matched whole-genome, RN

113 olar I disorder in the euthymic state and 25 normal controls balanced for age and gender.

114 comparative real sample analysis of healthy normal (Control), benign and ovarian cancer patients wit

115 in idiopathic narcolepsy-cataplexy and with normal control brains.

116 ased in eosinophilic subjects as compared to normal controls, but were similar between the eosinophil

117 c ALS, other neurodegenerative diseases, and normal controls, C4F6-immunoreactive inclusions were not

118 as present in 79% of FECD cases and in 3% of normal controls cases (p<0.001).

119 ter constitutive nuclear levels of Nrf2 than normal control CD34(+) cells because of an imbalance bet

120 Compared with age-matched normal controls, CD7(+) lymphocytes, NK cells, and CD16

121 s obtained from asthmatic donors compared to normal control cells.

122 tween malignant hyperthermia-susceptible and normal controls cells.

123 g Initiative (ADNI) database-198 cognitively normal controls (CN), 321 mild-cognitive impairment (MCI

124 neurofibromin immunolabeling was detected in normal control corneal endothelium, but was absent in co

125 versus LMNA-wild-type DCM (DCM(LMNAWT)), and normal controls (CTRL(LMNAWT)).

126 ed with renal tubules from kidney samples of normal controls, cysts in kidney samples from patients w

127 We studied normal control dogs (control, n=16) and 3 canine AF-mode

128 biopsy material, with subject assignment to normal control, drug-treated control, and autophagic myo

129 nvasive hemodynamic data were collected from normal controls, dyssynchronous HF and CRT models.

130 l brushings and sputum were obtained from 25 normal controls, eight mild/no inhaled corticosteroids (

131 ucoma suspects (6 eyes from 3 subjects), and normal control eyes (16 eyes from 9 normal subjects).

132 A total of 43 diabetic and 11 normal control eyes were evaluated with OCTA.

133 A cohort of diabetic and normal control eyes were imaged with a prototype SS-OCT

134 Compared to normal controls, eyes with pLPC may present a higher pro

135 activation of ATR with hydroxyurea than did normal control fibroblasts.

136 re WM differences between the simple T2h and normal control for both preterm and full-term neonates,

137 on was assessed in HFpEF, and compared to 50 normal controls free of cardiovascular disease and to 44

138 her in the group with CP (85.7%) than in the normal control group (73.3%; P <0.03).

139 h hippocampal damage (moderate/severe) and a normal control group.

140 h "moderate" hippocampal damage (MHD), and a normal control group.

141 ed into three groups (group 1: Sham-operated normal controls; group 2: Ischemia-reperfusion injury wi

142 whereas it decreased in glaucoma control and normal control groups by 2.0 mm Hg (P = .003) and 2.1 mm

143 enia, bipolar disorder, major depression and normal controls (>700 subjects).

144 UGR offspring were similar to those found in normal controls in a geriatric cohort.

145 tagonistic signals that were even lower than normal controls in the double CAM alpha(1)A/B-AR mice fo

146 VHs exhibited reduced GM volumes relative to normal controls in the left superior temporal gyrus, fro

147 2 diabetes without clinical retinopathy and normal controls, in order to assess possible increased m

148 FL) thickness in a series of PD patients and normal controls, in order to assess possible retinal ana

149 of 162 samples were analyzed, composed of 91 normal control individuals (51 male, 33 female, and 7 no

150 s with cross-sectional data from cognitively normal control individuals (age 65-89 years) who were ne

151 In normal control individuals and in people with type 1 dia

152 set representing the spectrum of cognitively normal controls, individuals with mild cognitive impairm

153 GS: In all drug-treated subjects, but not in normal controls, LC3 and p62 showed punctate staining ch

154 se (MHBD) activity of only 6 (+/-5) % of the normal control level.

155 ene expression profiling was performed for 4 normal (control) livers as well as 8 background liver an

156 Staining for K19 in normal controls, livers with "minimal change" PBC, CHC,

157 nsferase expression were compared in IPF and normal control lung tissues.

158 0.2 log deg(2), P < 0.01) and right eyes of normal controls (mean = 0.12 log deg(2), P </= 0.01).

159 active effector cells that are refractory to normal control mechanisms, resulting in persistent and d

160 Compared with normal controls, MF skin displayed increased mRNA levels

161 unts, and lower mean corpuscular volume than normal control mice, a phenotype that becomes more evide

162 roke compared with the recolonization with a normal control microbiota.

163 Compared with a normal control, MTX increased the density of osteoclasts

164 ion profiling between juvenile DM muscle and normal control muscle revealed 27 genes with a significa

165 In this cross-sectional study, normal controls (n = 145) from the Alzheimer's Disease N

166 affected eyes were studied and compared with normal controls (n = 15).

167 affected eyes were studied and compared with normal controls (n = 15).

168 opsy specimens from age-matched, cognitively normal controls (n = 23) and AD (n = 22) cases, across m

169 cts diagnosed with amnestic MCI (n = 12) and normal controls (n = 23) received standard brain (18)F-F

170 y randomly assigning Wistar rats (n = 28) to normal controls (n = 4), CDAA diet with vehicles, and CD

171 inical research criteria for PSP (n = 33) to normal controls (n = 46) and patients meeting criteria f

172 sis of very mild AD (n = 23) and cognitively normal controls (n = 64) who were enrolled in longitudin

173 thout asthma or sinusitis (HES; n = 18), and normal controls (n = 8).

174 AD dementia (n = 19) patients and clinically normal controls (n = 95) from an epidemiological cohort

175 ressure [PASP] >/=40 mm Hg) were compared to normal controls (n=12) and patients with primary pulmona

176 NTN1 receptors compared with histologically normal controls (n=28).

177 nondemented patients (10 LPD, 10 RPD) and 11 normal control (NC) adults.

178 with noncirrhotic liver disease (DC); and 88 normal control (NC) healthy volunteers.

179 intracellular calcium concentration in both normal control (NC) rats and DCP rats, but the sensitivi

180 26 normal control (NC), 37 AD, and 24 patients with PSP par

181 lated tau concentrations were compared in 47 normal controls (NC), 8 asymptomatic gene carriers (NC2)

182 rks in RIS subjects, demographically matched normal controls (NC), and relapsing-remitting (RR) MS pa

183 switching are increased compared with AR and normal controls (NC).

184 ary disorders (BHD) and 20 from age-matched "normal controls"(NC).

185 CSU patients, 10 AD patients, and 10 healthy normal controls (NCs) and measured contents of FBPs (pyr

186 a from 29 chronic SZ patients and 21 matched normal controls (NCs) performing a PRL task in an MRI sc

187 d cognitive impairment (MCI), in cognitively normal controls (NCs), and in cognitively normal apolipo

188 using western blots of postmortem ACC in: 1) normal controls (NCs, n=13) vs subjects with SZ (n=25);

189 Heavy drinkers (HD, N=16, 16 males) and normal controls (NM, N=24, 14 males) were tested after p

190 f the basal ganglia, playing a major role in normal control of behavior and in the pathophysiology of

191 These plants retained normal control of degradation.

192 ainstem and spinal cord for normal lifespan, normal control of heart rate, and respiratory response t

193 ansporter's absence from these cells for the normal control of insulin secretion.

194 of covert attention and is necessary for the normal control of spatial attention during perceptual ju

195 ritis (CAIA) in mice; the mechanisms whereby normal control of the AP is overcome and injury develops

196 lish that somatic mutations in DP-1 uncouple normal control of the E2F pathway, and thus define a new

197 patients with symptomatic AD and cognitively normal controls over time.

198 phagic myopathy group compared to either the normal control (p<0.001) or the drug-treated control gro

199 ficantly higher in patients with cHL than in normal controls (P < .0001).

200 ed MPTP hemispheres relative to those of the normal controls (P < 0.00005) but was reduced (P < 0.05)

201 aseline in 57.6% of DED patients vs.10.5% of normal controls (P < 0.0001).

202 was not different from age- and sex-matched normal controls (P = .46) but was significantly better t

203 ave the dominant TLR7 rs179010-T allele than normal controls (p = 0.0435).

204 a was in the capillaries, compared to 47% in normal controls (P<0.01).

205 ; P < .001) in transgenic mice compared with normal, control pancreata of WT mice (mean intensity, 0.

206 ve mild cognitive impairment, stable MCI and Normal Control participants).

207 nd included a total of 308 participants (181 normal control participants, 65 healthy siblings, and 62

208 for differences in neural activation across normal control participants, patients with schizophrenia

209 encoding of novel stimuli when compared with normal control participants.

210 roke patients without diabetes compared with normal control patients, while in stroke patients with d

211 control patients with diabetes compared with normal control patients.

212 Compared with normal controls, patients with PH had a 2-fold increase

213 ined cell cycle markers in IBM compared with normal control, polymyositis (PM) and non-inflammatory d

214 After generating reference values in normal controls, QGE(LMNA) was performed in 311 consecut

215 tory verbal hallucinations (AVHs) to that of normal controls remain controversial.

216 lower than those of the age- and sex-matched normal controls [respective means (SDs) =20.15 (6.40) &

217 ts served as controls (Steatotic-Control and Normal-Control, respectively; n=20 for each group).

218 In contrast, Betz cells of AD patients and normal controls retain cellular integrity in the motor c

219 t (13 individuals with PKD1 mutations and 18 normal controls) revealed that of 2008 ELV proteins, 9 (

220 ytokines in patients with HCC, compared with normal controls, revealing a network of potential mechan

221 ssion profiling assay of 85 human AML and 15 normal control samples, we show that among 48 miRNAs tha

222 r patient specimens compared with the paired normal control samples.

223 in MLL-rearranged AML samples compared with normal control samples.

224 alamic volumes of the patients with AVHs and normal controls showed that the area under the curve was

225 Subjects with MCI, compared with normal controls, showed differential associations of olf

226 GF-beta1 by these lymphocytes in contrast to normal control specimens.

227 Participants (n = 42 normal controls) spent 3 consecutive nights in the labor

228 elevated sweat chloride levels compared with normal control subjects (14.5 +/- 7.77 mEq), indicating

229 19-54 years; mean age, 41.2 years) and eight normal control subjects (age range, 22-56 years; mean ag

230 tive impairment (n = 13), and 21 cognitively normal control subjects (ages 41-76, nine male).

231 LVH (mean age, 56.41 +/- 2.78 years), and 12 normal control subjects (mean age, 55.67 +/- 3.08 years)

232 h congenital AS (median age 16 years) and 27 normal control subjects (median age 16 years) were evalu

233 We compared normal control subjects (n = 52), a septic shock no cort

234 ested a cohort of POAG patients (n= 158) and normal control subjects (n= 82), both from Iowa, for ASB

235 acuities of 20/40 or better, and 10 visually normal control subjects aged 22 to 65 years.

236 28 subjects with knee OA risk factors and 33 normal control subjects ages 45-55 years, with a body ma

237 pants; the discovery phase study enrolled 51 normal control subjects and 183 HF patients, and the val

238 were explored in an independent sample of 36 normal control subjects and 35 unaffected siblings durin

239 and bronchoalveolar lavage (BAL) cells from normal control subjects and untreated HIV-1-infected ind

240 ns are actively involved when neurologically normal control subjects name visually presented objects,

241 ozygous subjects (28 +/- 7 years), and eight normal control subjects pair-matched to the heterozygous

242 h asthma (19 with and 15 without EIB) and 10 normal control subjects to examine in vivo differences i

243 The PD control cases and normal control subjects were matched first for age at de

244 iment, 17 patients with schizophrenia and 24 normal control subjects were presented in 3 T magnetic r

245 fficient cystic fibrosis (PS-CF), PI-CF, and normal control subjects, all with normal glucose toleran

246 n was lower in PI-CF compared with PS-CF and normal control subjects, and glucagon-like peptide 1 and

247 the PFC in schizophrenia, compared with the normal control subjects, but not in bipolar disorder.

248 d differentiate gastric cancer patients from normal control subjects, dramatically (p < 0.05).

249 d glucagon responses compared with PS-CF and normal control subjects, indicating reduced beta-cell se

250 In normal control subjects, Nt of SST-IR neurons varied acc

251 ary motor cortices isolated from post-mortem normal control subjects, patients with familial ALS (fAL

252 analyses as well as exclusion testing of 500 normal control subjects, we demonstrated that this genet

253 patients with diabetes without stroke and 30 normal control subjects.

254 Noncontrast studies were completed in the normal control subjects.

255 al pathway was used by our 24 neurologically normal control subjects.

256 ILD but not subjects without lung disease or normal control subjects.

257 ted in a separate group of schizophrenia and normal control subjects.

258 sed as having AD had greater WMH volume than normal control subjects.

259 n, glucose was higher in PI-CF compared with normal control subjects.

260 date and putamen of nine TS and nine matched normal control subjects.

261 losive disorder compared with psychiatric or normal control subjects.

262 rent in cerebrospinal fluid (CSF) from young normal controls, suggesting that the amount of apoE isof

263 r lavage (BAL) fluid of 10 asthmatics and 15 normal controls taken before, at day four during and 6 w

264 ur adult PTSD patients and 23 trauma-exposed normal controls (TENC) underwent 4T MRS of the left and

265 lity of life of 307 glaucoma patients and 76 normal controls that were frequency matched to the age a

266 splantation in Steatotic-Control (P=0.06 vs. Normal-Control), the VSOP-NO group showed increased leve

267 Compared with the epithelial thickness in normal control, the epithelial thickness in LSCD patient

268 isons were performed: In one comparison, two normal controls--those of the women with negative (benig

269 consistantly greater in HCC when compared to normal control tissue (p-value = 0.06) and, in general,

270 to adjacent liver tissue (p-value =0.08) and normal control tissue (p-value =0.02).

271 f the larger tumors as well as from adjacent normal control tissue and microsatellite instability (MS

272 ered abundance in MYC lymphoma compared with normal control tissue by statistical analysis with a fal

273 emethylated in patient-derived compared with normal control tissue-derived epithelial cells.

274 obtained from allergic tissue compared with normal control tissue.

275 py was a logarithmic function of ROI area in normal control tissues (aorta, psoas) and in mathematica

276 NF, RRM2, MKI67, and C12orf5 (when including normal control tissues).

277 of this variant in AD cases and cognitively normal controls to determine whether this variant will s

278 ts in cancer studies, they are often used as normal controls to identify genes differentially express

279 ging eyelid severity levels, ranging from 1 (normal control) to 4 (severe sagging).

280 h the ASDs relative to 12,544 neurologically normal controls, to find potentially druggable genetic t

281 Furthermore, in normal controls, TRNT1 protein levels are 10-fold lower

282 s from 13 clinically infected patients and 7 normal controls using clinical findings as the gold stan

283 False-positive rate in 52 normal controls was 2%.

284 patients with symptomatic AD and cognitively normal controls was performed.

285 a large cohort of 1,098 HCC patients and 835 normal controls, we constructed a diagnostic prediction

286 gton State and 644 unrelated, neurologically normal controls, we examined whether PD was associated w

287 c cholestasis as diseased controls and seven normal controls, we identified 15 genes uniquely express

288 Patients from a dry eye clinic and normal controls were assessed by Schirmer's test for tea

289 Cognitively normal controls were classified using the longitudinal c

290 Human FECD samples and normal controls were examined for p21 expression by immu

291 the eyes of amblyopes compared with visually normal controls were found.

292 Twenty-nine CRAO cases at acute phase and 33 normal controls were included.

293 set depression (LOD), 32 LOD patients and 39 normal controls were recruited and underwent resting-sta

294 e four dNTP pools do not differ in size from normal controls, whereas during quiescence, the dCTP and

295 nature that differentiated IPF patients from normal controls, which may allow for accurate diagnosis

296 osis of early symptomatic AD and cognitively normal controls who were enrolled in longitudinal studie

297 Cognitively normal controls whose CSF VILIP-1, tau, or p-tau181 leve

298 ) relative to that observed in myotubes from normal control (wild-type and/or heterozygous) myotubes.

299 duration of 17.5 months were compared to 20 normal controls with (18)F FDG PET and resting state fMR

300 ied 112 subjects: 92 patients with PH and 20 normal controls with 3D wall motion tracking for RV end-