ライフサイエンスコーパス: normal control group

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1 h hippocampal damage (moderate/severe) and a normal control group.

2 h "moderate" hippocampal damage (MHD), and a normal control group.

3 n girls with other disorders or those in the normal control group.

4 gnificant correlations were found within the normal control group.

5 were similar in the two study groups and the normal control group.

6 r atrophy group diminished compared with the normal control group.

7 he LND patients was observed compared to the normal control group.

8 roup of subjects with SQCP aged 2-18 y and a normal control group.

9 iduals with primary RLS and a neurologically normal control group.

10 s (at least r=0.76, P<0.0001) but not in the normal control group.

11 miliar and stranger faces in both autism and normal control groups.

12 ve aphasia, along with neurodegenerative and normal control groups.

13 l controls (normal control group ] [NC1] and normal control group 2 [NC2]) (n = 14 for each).

14 exposure of the liver: group 1 (n = 7) were normal controls; group 2 (n = 7) had hepatic fibrosis.

15 ed into three groups (group 1: Sham-operated normal controls; group 2: Ischemia-reperfusion injury wi

16 ly affected families, their relatives, and a normal control group (214 subjects).

17 her in the group with CP (85.7%) than in the normal control group (73.3%; P <0.03).

18 sponse to hypoglycemia between the H and the normal control group after fasting for 48 h (H 29,639 +/

19 voxel [(18)F]dopa K(i)(o) values between the normal control group and each Parkinson's disease group

20 ntly lower cortisol levels than girls in the normal control group at all 3 sampling times.

21 evation in both the patient group and in the normal control group, but no evidence of a differential

22 whereas it decreased in glaucoma control and normal control groups by 2.0 mm Hg (P = .003) and 2.1 mm

23 wley rats into three groups: group N was the normal control; group C was given 40 mg/kg body weight p

24 The normal control group exhibited clot times of 5.7+/-0.3 m

25 ed significantly from that in the stroke and normal control groups, giving an odds ratio of 2.51 (95%

26 n pattern in five patients compared with the normal control groups; in all five patients this atypica

27 were significantly lower in IDCM than in the normal control group (MFAU: 134 +/- 44 vs. 213 +/- 49 nm

28 U were significantly higher in IDCM than the normal control group (MGU: 247 +/- 63 vs. 125 +/- 64 nmo

29 on with 2 groups of matched normal controls (normal control group ] [NC1] and normal control group 2

30 7 in the RA group, compared with 0.86 in the normal control group (P = 0.0002 for both).

31 Compared to a CAG-normal control group, the CAG-expanded group demonstrate

32 patients with and without limb apraxia and a normal control group to examine preprogramming and respo

33 system atrophy patients as compared with the normal control group, with average reductions of 61% in

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