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1 = 0.47; P </= .01), with most concordant for normal flora.
2 across all samples, predominantly reflecting normal flora.
3 in vaginal wash samples than did women with normal flora.
4 demonstrate inflammation in response to the normal flora.
5 and the symbiotic relation we have with our normal flora.
6 between a natural product of the intestinal normal flora and important epithelial inflammatory and p
10 l pathogen Candida albicans is a part of the normal flora but it also causes systemic candidiasis if
12 tervention on a healthy vaginal environment (normal flora confirmed by Gram stain with no candidiasis
13 ontal pathogens, opportunistic pathogens, or normal flora did not differ descriptively between groups
20 ginal wash and serum samples from women with normal flora, intermediate flora, and BV (determined by
23 bowel disease, the loss of tolerance to the normal flora may be due in part to inappropriate toll-li
25 Gallibacterium anatis is a member of the normal flora of avian hosts and an important causative a
28 us lugdunensis is often found as part of the normal flora of human skin but has the potential to caus
31 istic fungal pathogen and a component of the normal flora of the gastrointestinal tract, is a frequen
32 Streptococcus intermedius is a member of the normal flora of the mouth but is also an opportunistic p
33 e of this study was to compare the effect of normal flora on inflammation in mice in which the IL-10
35 S gene is contained by invasive isolates and normal-flora strains in a region that contains genes inv
36 uced lactobacilli were less likely to regain normal flora than were those whose lactobacilli were una
37 he reduction in the number of members of the normal flora that interfere with the growth of pathogens
38 city for the pathogen without disturbing the normal flora, the low chance of bacterial resistance to
39 city for the pathogen without disturbing the normal flora, the low chance of bacterial resistance to
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