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1  level of </=2 IU per deciliter, or </=2% of normal levels).
2 oxp3- effector T cells (Teffs) remained at a normal level.
3 py and the return of the platelet count to a normal level.
4  the epithelial barrier function to a nearly normal level.
5 t-induced secretion of bFGF and VEGF to near normal levels.
6 NAT2 expression in rTg4510 tauopathy mice to normal levels.
7 XCL13 at relapse including 3 with previously normal levels.
8 ntact time between secretion and EGJ towards normal levels.
9 tored M3R-stimulated insulin release to near normal levels.
10 lial damage (thrombomodulin) markers to near-normal levels.
11 ation patient lymphoblasts restores MeCP2 to normal levels.
12 ors and increased expression of TGF-beta1 to normal levels.
13 stablished measure of muscle damage, to near-normal levels.
14 ly restores p70S6K activity and cell size to normal levels.
15 ted basally and were increased by insulin to normal levels.
16 ths after transplantation, but did not reach normal levels.
17  thylakoid membrane complexes accumulated to normal levels.
18  by restoring penile PDE5 gene expression to normal levels.
19 class switching from IgM to IgG1 at 5-10% of normal levels.
20 le were still present at 50% (or greater) of normal levels.
21 reas renal EPO was down-regulated to 6.7% of normal levels.
22 "liking" reactions to approximately one-half normal levels.
23 : then, a year or so later, they declined to normal levels.
24 ostnatal proliferation and spatial memory to normal levels.
25 eficient animals express slow fiber genes at normal levels.
26 o I-SceI-dependent class switching at almost normal levels.
27 tructure brought the PLP1/DM20 ratio to near normal levels.
28 c triglyceride levels were fully restored to normal levels.
29 main-containing inositol 5'-phosphatase 1 to normal levels.
30 pair, the extension rate recovered to nearly normal levels.
31 ry state induced by periadolescent MK-801 to normal levels.
32   The mitochondrial DNA was also detected at normal levels.
33 rapid weakening of activity to near or below normal levels.
34 mokines, and antimicrobial peptides) to near-normal levels.
35 DL-C and apoB into sd-LDL and lb-LDL to near normal levels.
36 ey hyperaccumulate copper up to 50-fold over normal levels.
37 lity transition pore opening was restored to normal levels.
38 rogramming efficiency of somatic FA cells to normal levels.
39 rs in lymph nodes and spleen were at or near normal levels.
40 id not affect the growth CRC cell lines with normal levels.
41 ormed abnormal complexes but were present at normal levels.
42 decrease in IL excitability was increased to normal levels.
43 s likely to have periodontitis as those with normal levels.
44 utant nodules increased nitrogen fixation to normal levels.
45 nt with dichloroacetate restored PDH flux to normal levels (0.018 +/- 0.002 s(-1)), reversed diastoli
46 <240 U/L), albumin level, 2.6 g/dL (26 g/L) (normal level, 3.5-5.0 g/dL [35-50 g/L]), and creatinine
47    Interestingly, c-Jun protein rebounded to normal levels 4 h following U0126 exposure but not after
48 or overall survival compared with those with normal level (60.0% vs. 83.6%, log-rank test, p = 0.035)
49  drugs that might restore gene expression to normal level across the Br and T modules.
50 r1p are antiprion system components at their normal levels, acting with Hsp42.
51 sumption after 1 h of exposure, returning to normal levels after 16 h.
52 respiratory allergic patients that return to normal levels after steroid administration.
53      Post-RYGB, 49 proteins were returned to normal levels after surgery.
54 ed increased mortality that declines to near-normal levels after the first several years, but in our
55 ction, increasing NS1 SUMOylation beyond its normal levels also exerted a negative effect on its IFN-
56 ibrin generation to approximately 78% of its normal level and hence improve clot formation under dilu
57 y), disrupted by IR injury, were restored to normal level and the induction of ventricular tachycardi
58 orms, but those for lean mass remained below normal levels and diminished significantly over time (P
59 -DG in the regenerating fibers reaches up to normal levels and lasts for >4 weeks, but no up-regulati
60  TLR2 signaling restored the inflammation to normal levels and the ability to clear Lm in Pxr(-/-) mi
61  the protein product was expressed at nearly normal levels and was functional in the NKTCL cell lines
62 etter restoration of complete blood count to normal level, and significantly longer median survival a
63  Csf1r were expressed on the cell surface at normal levels, and bound CSF1, but were not able to sust
64 PS I content and activities were restored to normal levels, and cells again produced thylakoids that
65 cy of cell division when DamX is produced at normal levels, and inhibiting cell division when DamX is
66 ls, increased serum levels of ALT (even high-normal levels) are associated with markers of cardiovasc
67 degree of disease activity, and increased to normal levels as a result of anti-TNF-alpha therapy.
68 esponse to relative hypoxia and recovered to normal levels at the end of blood vessel formation.
69                     POT1(CP)was expressed at normal levels, bound TPP1 and telomeres, and blocked ATR
70                 Restoration of p75ECD to the normal level by brain delivery of the gene encoding huma
71 KO triple mice reduced the liver size to the normal level by decreasing lipid storage in both hepatoc
72 BP1-knockdown Huh7.5 cells was restored to a normal level by downregulation of either p53 or BCCIP.
73 the peripheral T cells were repopulated to a normal level by syngeneic bone marrow transplantation.
74 01) despite recovery of touch sensitivity to normal levels by 6 months.
75 served when the BLMAP was decreased to below normal levels by an infusion of sodium nitroprusside.
76 ted that recognition memory had recovered to normal levels by Day 3.
77     Remarkably, returning Mtor and Rictor to normal levels by deleting one allele of Mtor and one all
78  patient's fibroblasts were also restored to normal levels by exogenous expression of wild-type MRPS7
79 ine co-transport stimulation was reversed to normal levels by ketotifen.
80 y reduced endocytosis, which was restored to normal levels by PLS3 overexpression.
81   The salt tolerance of sos2 was restored to normal levels by wild-type SOS2, but not by a mutated fo
82 in cancer cell lines to approximately 50% of normal levels causes a delay in the cell cycle and accum
83 st, thymus markers were delayed but achieved normal levels, concurrent with complete loss through apo
84 cells restored alveolar fluid clearance to a normal level, decreased inflammation, and were associate
85 nthesis restored cytokine production to near-normal levels, delayed ACS progression, and extended hos
86 th nocodazole restored Plk1 activity to near normal levels, demonstrating that MTs also contribute to
87 g extracellular matrix stiffness relative to normal levels disrupts junctional integrity and increase
88 expression pattern of these mediators toward normal levels during 4 weeks of treatment.
89  function continues to improve and may reach normal levels during the first posttransplant year.
90     CD3 T-cell counts gradually recovered to normal levels during this period but CD8 T cells recover
91  these circulating microRNAs decrease toward normal levels during treatment with N-acetyl cysteine (N
92  can be manipulated to enhance GSIS to supra-normal levels even in the face of defective mitochondria
93 mice with PPARgamma expression at 25% of the normal level exhibited high autoantibody levels and deve
94 tored dopamine neuron population activity to normal levels following CMS; LHb inactivation had no res
95                 Counts did not usually reach normal levels for age, but patients were able to clear p
96 sed BMI, but not sex, influences recovery to normal levels for the markers studied, possibly indicati
97 uals who maintained or decreased their BP to normal levels had the lowest remaining LTR for CVD, 22%
98                                         This normal-level Hsp104 curing is promoted by Sti1p, Hsp90,
99 onditionally reduce ATR expression to 10% of normal levels in adult mice to compare the impact of thi
100 ed mice, DHT implants restored SV weights to normal levels in alphaERKO mice but not in WT mice, sugg
101 he enhanced response to PM(2.5) plus O(3) to normal levels in an established model of chronic allergi
102 ted glucagon and corticosterone secretion to normal levels in diabetic rats.
103 ome that is elevated in PWID, can regress to normal levels in former injection drug users who are HCV
104 levels were elevated on average 5-fold above normal levels in hematologic patients receiving voricona
105 (<1 year) T1D patients tested, but return to normal levels in individuals diabetic for >1 year.
106 s DEmiRs and target DEGs to their respective normal levels in inflammation, mitochondrial function an
107  model, Mybl2 knockdown by RNAi to 20-30% of normal levels in multipotent hematopoietic progenitors r
108 iac hypertrophy (PCH) and restoring GDF11 to normal levels in old mice rescued PCH.
109 me activity for 6 mo at approximately 30% of normal levels in one animal, and in excess of normal lev
110                   These parameters return to normal levels in osteoclasts derived from double mutant
111 irus (HIV) infection but can recover to near normal levels in patients who spontaneously control vire
112 Serum thrombopoietin (TPO) was maintained at normal levels in Pf4-Cre-positive Jak2(f/f) mice, consis
113 ated in Tbx1(+/-) embryos and is restored to normal levels in Tbx1(+/-);Trp53(+/-) embryos.
114 (citZ) transcript was present at higher-than-normal levels in the citB null mutant, due at least in p
115 tability and levels of KCC2 and NKCC1 toward normal levels in the lumbar spinal cord.
116 enerated mice in which Mecp2 remains at near normal levels in the nervous system, but is severely dep
117 , assessed by western blot, was below 15% of normal levels in the nine mutation carriers in whom this
118 ormal levels in one animal, and in excess of normal levels in three animals.
119              Return of Dyrk1a copy number to normal levels in Ts65Dn mice rescued the appendicular bo
120                       Reducing hematocrit to normal levels in tubular Vegfa-deficient mice resulted i
121 in IL-17-producing cells was not restored to normal levels in wild-type and Blimp-1CKO-mixed bone mar
122 aneously restoring fibrinogen (to 88% of its normal level) in diluted blood can restore fibrin genera
123 s that express APP and processing enzymes at normal levels is not well understood.
124 s likely to have periodontitis as those with normal levels (&lt;/=199 mg/dL), and those with STC >/=240
125 creatinine level, 1.7 mg/dL (150.3 mumol/L) (normal level, &lt;1.2 mg/dL [<106.1 mumol/L]).
126 l level, <300 U/L); amylase level, 1435 U/L (normal level, &lt;140 U/L); lactate dehydrogenase level, 25
127  phosphatase level of 85 U/L (1.42 mukat/L) (normal level, &lt;142 U/L [2.37 mukat/L]), a serum creatini
128 0.8-2.0), a urine protein level of 15 mg/dL (normal level, &lt;20 mg/dL), a total serum calcium level of
129  U/L); lactate dehydrogenase level, 253 U/L (normal level, &lt;240 U/L), albumin level, 2.6 g/dL (26 g/L
130 ge, 1.1-1.3 mmol/L); lipase level, 2423 U/L (normal level, &lt;300 U/L); amylase level, 1435 U/L (normal
131 glutamyl transferase (77 U/L [1.28 mukat/L]; normal level, &lt;55 U/L [<0.92 mukat/L]) and C-reactive pr
132 -reactive protein (97.1 mg/L [924.8 nmol/L]; normal level, &lt;8 mg/L [<76.2 nmol/L]]) levels were chron
133 phorylase activity rose from undetectable to normal levels (median 697 nmol/h/mg protein, range 262-1
134     ABCB4(S320F) homozygosity, with half the normal level of ABCB4, is the tipping point between more
135                                       A high-normal level of ALT was associated with higher levels of
136 onse (HBV DNA level <2000 IU/L, persistently normal level of ALT).
137 ration with densely aligned collagen fibers, normal level of cellularity, and functional restoration.
138  ERPs of a mouse line expressing 150% of the normal level of cone M-opsin with those of WT mice revea
139                                 Therefore, a normal level of CtpA activity is critical for T3SS funct
140 vels of aSyn prevent Cdc5 from maintaining a normal level of GTP-bound Rho1, which is an essential GT
141    Here we show that Plc1p is required for a normal level of histone acetylation; plc1Delta cells tha
142                                   However, a normal level of LTP can be generated by repetitive 100-H
143 geo/beta-geo)), a hypomorph with ~10% of the normal level of Pikfyve protein.
144 , such as TACI(+/-) mice, expressed half the normal level of TACI on their B cells and exhibited simi
145 ith Crohn's disease, and have shown that the normal levels of a key antimicrobial peptide produced by
146  anti-inflammatory eicosanoids, and restored normal levels of acylcarnithins but not of sphingomyelin
147 es (OR = 0.04, 95% CI 0.005-0.49; P = 0.01), normal levels of alanine aminotransferase at the end of
148 HBV) DNA who were positive for HBeAg and had normal levels of alanine aminotransferase were randomly
149 ith chronic HBV infection, high viral loads, normal levels of alanine aminotransferase, and therapy w
150 )-positive patients with high viral load and normal levels of alanine aminotransferase.
151 rtensive patients with hyperaldosteronism or normal levels of aldosterone exhibited increased activit
152 e channels but normal surface expression and normal levels of alpha and gamma subunit-activating clea
153 the relationship between low-normal and high-normal levels of ALT and an extended panel of cardiovasc
154 ts with CHB, undetectable serum HBV DNA, and normal levels of aminotransferases after long-term (4 or
155                Cells lacking CnrN accumulate normal levels of AprA and CfaD.
156 res of respiratory physiology but maintained normal levels of arterial oxygen saturation.
157 dispersed in axons, thereby maintaining near normal levels of ATP even in boutons lacking mitochondri
158 ation carriers were healthy and demonstrated normal levels of atrial natriuretic peptide.
159 efective in fusion activity and yet retained normal levels of attachment to cell surface heparan sulf
160 bnormalities in gait and balance and exhibit normal levels of basal locomotion.
161                     We demonstrate here that normal levels of beta1 chain limit formation of integrin
162  (BBB), has an essential role in maintaining normal levels of brain BCAAs.
163                 The [URE3] variants cured by normal levels of Btn2p and Cur1p all have low seed numbe
164 n2 cur1 double mutant are cured by restoring normal levels of Btn2p and Cur1p, with both proteins nee
165 rranged T cell receptor alphabeta transgene, normal levels of CD4 single-positive cells were produced
166 SCM) and CD4(+) central memory T cells, with normal levels of CD4(+) T(SCM) proliferation, and lack o
167                                 In contrast, normal levels of CD4(+)IL-21(+) T cells were found in SI
168 press a low level of total CD79b protein but normal levels of CD79a and IgM protein.
169              B cell-deficient mice developed normal levels of CD8(+) effector T cell responses early
170 ivo, Fib(AEK) mice were generated that carry normal levels of circulating fibrinogen but lack the cap
171      Ablation of Erfe in Th3/+ mice restored normal levels of circulating hepcidin at 6 weeks of age,
172 activate Stat5 during late pregnancy despite normal levels of circulating serum prolactin and pituita
173                                  Higher-than-normal levels of circulating triglycerides are a risk fa
174 eficient B cells lack the ability to undergo normal levels of class switch recombination and somatic
175 ally, our model predicted that restoring the normal levels of clotting factors II, IX, and X while si
176                    Affected individuals have normal levels of coagulation factor V (FV) activity, but
177 eins were differentially affected, with near normal levels of cytochrome c oxidase subunit2 and Nad7
178  reveals that both proteins are required for normal levels of cytosine methylation and hydroxymethyla
179 s that dictate the ability of DAT to sustain normal levels of DA clearance, we pursued a forward gene
180               Whole brain homogenates showed normal levels of DISC1 protein, but decreased binding of
181  by 4-fold in Elmo1(-/-) lymphocytes despite normal levels of Dock2 mRNA.
182  morpholino, with the ability to induce near-normal levels of dystrophin, and restores function in bo
183 or apoptosis and atrophy and maintained near-normal levels of electroretinographic responses.
184 ynchronization as measures that characterize normal levels of excitability and quantify any deviation
185                         We conclude that, at normal levels of expression during bacterial infection,
186  lacking DdPpk1 accumulate approximately 50% normal levels of extracellular polyphosphate, suggesting
187                       Patients with MPGN had normal levels of factor H, and structural analysis of th
188 n certain rare genetic contexts, maintaining normal levels of FGFR2 signaling is important for human
189 e decay of mRNAs, but rather for maintaining normal levels of functional tRNAs and protein synthesis
190 females presented abnormal levels of Ga2 but normal levels of Gb3, supporting the importance of analy
191          Although Enpp1 (-/-) mice generated normal levels of germinal center B cells and plasmablast
192 wever, MEK1DD-expressing organoids exhibited normal levels of growth and retained apicobasal polarity
193  reperfusion injury, even in the presence of normal levels of GSH.
194                                              Normal levels of H3 T118ph are important because it is r
195 ut containing intact H2Bub enzymes that have normal levels of H3K4me3, suggesting that monoubiquitina
196 t with the GNAT proactivator SPV106 restored normal levels of H3K9Ac and H3K14Ac, reduced DNA CpG hyp
197                                              Normal levels of HDL-C are not an independent risk facto
198 ceiving cognitive behaviour therapy achieved normal levels of health anxiety compared with those in t
199 xchange or infusion may transiently maintain normal levels of hematologic measures but does not treat
200                             Knockin mice had normal levels of hepatic fat on a chow diet, but when ch
201 NA production and therefore fails to secrete normal levels of Hla at early phases of growth.
202  HIF1, but not HIF2 target genes in spite of normal levels of hypoxia-inducible transcription factor
203 nd that IFN-lambdaR1-deficient mice produced normal levels of IFN, robust NK cell responses, but grea
204                    MHCII(-/-) DC10 expressed normal levels of IL-10, but, nevertheless, were unable t
205        Further, ASC-deficient mice displayed normal levels of IL-1beta in genital secretions.
206  Patients with cells available for study had normal levels of IL-2-induced STAT5 phosphorylation.
207 ic cells (BMDCs) from TTP(-/-) mice produced normal levels of IL12/23p40.
208 d production of normal levels of virions and normal levels of infectivity in the complete absence of
209                  Although Il18(-/-) mice had normal levels of inflammatory monocytes, their NK cells
210    Titrating ventilatory support to maintain normal levels of inspiratory effort may prevent changes
211 defects, SMA Schwann cells failed to express normal levels of key extracellular matrix proteins, incl
212 uring mitosis and that p150N is required for normal levels of Ki-67 accumulation on the PCL.
213 from some female strains, were necessary for normal levels of male investigatory behavior of female s
214           Notably, TDP2 is also required for normal levels of many gene transcripts in developing mou
215  cells, tend to be multinucleate, accumulate normal levels of mass and protein per nucleus, and form
216 ls displayed normal morphology and contained normal levels of mast cell proteases before and after de
217 expressing the human MECP2 gene at twice the normal levels of MeCP2 (Tg1; Collins et al., 2004).
218 therapeutic approaches must restore close to normal levels of MECP2.
219  mice contained increased numbers of LC with normal levels of MHC and costimulatory molecules and T-c
220 ound to be linearly decreasing with even low-normal levels of microalbuminuria.
221 nerate mouse hearts expressing 6- to 16-fold normal levels of microRNA (miR)-143, miR-378, and miR-49
222  female mice developed normally and produced normal levels of milk protein, suggesting a defect in de
223 NA deficient oocytes but not in oocytes with normal levels of mitochondrial DNA.
224 e born to an iron-deficient mother contained normal levels of mitochondrial iron and no ferritin; the
225 t from the mother contained low ferritin and normal levels of mitochondrial iron.
226 ts contained less ferritin than controls but normal levels of mitochondrial iron.
227 Bazooka/Par-3, which in turn is required for normal levels of mobile E-cadherin.
228                Ample evidence indicates that normal levels of monoamines in the hippocampus, amygdala
229                   CS cells, in contrast, had normal levels of mutagenesis despite their TCR deficienc
230 fertile adults, with the capacity to produce normal levels of myeloid and lymphoid lineage cells.
231  immature and mature B cell subsets and have normal levels of naive serum Abs but altered levels of n
232                           Despite expressing normal levels of Ncc mRNA, these mice had lower levels o
233 te with brain-adjacent stem cells to promote normal levels of neurogenesis.
234 g factors is required for the maintenance of normal levels of overlapping mRNA transcripts.
235                                              Normal levels of PE can decline with age in the brain.
236 and showed that DVAP is required to maintain normal levels of phosphoinositides.
237 ve determined that this gene is required for normal levels of PME activity and homogalacturonan methy
238 otes show loss of enzymatic activity despite normal levels of precursor protein, and manifest a more
239 vity had often fully recovered, or overshot, normal levels of productivity in both high- and low-dive
240 rmed, with transduced cells exhibiting supra-normal levels of protein expression and ex vivo migratio
241                                              Normal levels of RDS and the unglycosylated RDS binding
242 able to generate variants despite expressing normal levels of RecA.
243 r are required at the preloaded promoter for normal levels of recruitment and activity of the general
244                  Excitatory neurons regained normal levels of response, while narrow-spiking (inhibit
245 genesis defect, allowing them to reestablish normal levels of ribosome production and cell proliferat
246  with IBS (with visceral hypersensitivity or normal levels of sensitivity).
247 s reduced their visceral hypersensitivity to normal levels of sensitivity.
248 gth sleB [sleB(FL)] and ypeB [ypeB(FL)], but normal levels of SleB(FL) in spores required normal spor
249 exogenous TGF-beta in vitro, they maintained normal levels of Smad2 phosphorylation under steady-stat
250      As a reciprocal experiment, we restored normal levels of SMN in the muscle with low SMN levels i
251 we used a mouse mutant engineered to express normal levels of SNAP-25 but only SNAP-25a.
252            These findings suggest that supra-normal levels of sociability and verbal functioning may
253                         Scn2b null atria had normal levels of sodium current density compared with wi
254 variant that had enhanced Sse expression and normal levels of SpeB production was identified (the var
255 smitter release onto these neurons maintains normal levels of synaptic activity.
256 ion of one copy of the Icosl allele restores normal levels of T(fh) and GC B cells in Taci(-/-) mice.
257 schizophrenia, ChC cartridge boutons contain normal levels of the 67 kDa isoform of glutamic acid dec
258      AADC deficient patients fail to produce normal levels of the monoamine neurotransmitters dopamin
259 the absence of Siw14p are cured by restoring normal levels of the protein.
260 re, in vivo, CTNNBL1 is required to maintain normal levels of the Prp19 complex and to facilitate the
261 se-mediated mRNA decay pathway, resulting in normal levels of the truncated plakoglobin.
262 60M) strain are cured by restoration of just normal levels of the WT Hsp104.
263 ing that LCMT-1 is important for maintaining normal levels of these subunits.
264  growth factor-21 live longer than mice with normal levels of this hormone.
265 with IL-2 immune complexes failed to restore normal levels of Tregs or to ameliorate disease.
266      Genetic disruption of PGC1alpha rescued normal levels of type I fiber markers MyH7 and myoglobin
267                                  Concomitant normal levels of urinary albumin, IgM and RBP identified
268   Interestingly, male UTX KO embryos express normal levels of UTY and survive until birth.
269 glycosylation, we demonstrated production of normal levels of virions and normal levels of infectivit
270               The presence of P-selectin and normal levels of VPS33B and VPS16B in Nbeal2(-/-) platel
271 y be a key regulator in maintaining correct, normal levels of Wnt signalling.
272 PrP after amplification in mice coexpressing normal levels of wt and anchorless PrP(C).
273  mice that express inactive plasmin (Pm) but normal levels of zymogen Pg (PAI-1(-/-)/Pg(S743A/S743A))
274 egulation on Day 5 and Day 9 but returned to normal level on Day 13 and Day 17.
275                 For drug concentrations at a normal level or above, the sample can be diluted up to 1
276 ive lignin peroxidase genes are expressed at normal levels or even higher in the laccase triple mutan
277 nteracted such that methylation returns to a normal level over four generations.
278 deficient group compared with the group with normal levels (P=0.019).
279 ion, in that the target remains present at a normal level prior to induction and is then rapidly depl
280                   Restoring Aurora B to near-normal levels rescues mitotic phenotypes in cells lackin
281  were sharply reduced by 39%, 64%, or 81% of normal levels, respectively.
282 hment of GILZ expression in patients' DCs to normal levels restores their capacity to activate allerg
283 pression of two Msh3 polymorphic variants at normal levels showed no detectable change in expansions,
284 phase even if stratospheric ozone returns to normal levels, so that climate conditions conducive to w
285  restored F81Y S-opsin protein expression to normal levels, suggesting that ligand binding in the ER
286 e than 20% gamma'-Fg, the upper limit of the normal level, the delay in lysis was magnified.
287  and cytoskeletal proteins remained at their normal levels, the major peripheral membrane proteins sp
288 er, the median CD4 cell count remained below normal levels throughout follow-up period and the propor
289     BGL was dichotomized for elevated versus normal levels using a cutoff value of >7.0 mmol/l.
290 nockdown (by approximately 85% compared with normal levels), using lentiviral shRNA, dramatically att
291 xia (AIH) restores phrenic nerve activity to normal levels via enhanced phrenic long-term facilitatio
292                Although viral DNA is made at normal levels, viral capsid accumulation in the nucleus
293        An ATPase-defective Msh3 expressed at normal levels was as defective in expansions as Msh3-/-
294                                        Lower normal levels were associated with faster clinical progr
295 e methyltransferase Set1 is restored to near normal levels when SIN3 is also reduced.
296        The early ART group (n = 33) achieved normal levels, whereas the later ART group (n = 30) cont
297 ried in isolation from one-half to two-times normal levels while subjects with simulated amputation w
298 mic pressure gradient (PSG) was decreased to normal level, while total bilirubin (TBIL) and liver fun
299 cardiomyocyte proliferation and recovered to normal levels within 1-week time.
300 ium cyclosilicate reduced serum potassium to normal levels within 48 hours; compared with placebo, al

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