ライフサイエンスコーパス: normal mouse

1 ssues of the extraocular muscle cones in the normal mouse.

2 R gene was amplified from all tissues of the normal mouse.

3 lar distribution similar to that seen in the normal mouse.

4 In normal mouse AMs, the anti-MARCO mAb ED31 also showed im

5 tudied in detail striatal GDNF production in normal mouse and after damage of DA neurons with MPTP.

6 Upon the basis of its rapid clearance from normal mouse and baboon brain tissues, [N-methyl-(11)C]2

7 r markers have previously been identified in normal mouse and human aorta.

8 etaiotaomicron, a prominent component of the normal mouse and human intestinal microflora.

9 Similarly, insulin secretion was impaired in normal mouse and human islets that were cultured with di

10 We demonstrate its application to normal mouse and human marrow, to murine models of marro

11 Normal mouse and human osteoblasts were cocultured with

12 to isolate the native myostatin complex from normal mouse and human serum.

13 n vivo, and compared results with those from normal mouse and human tissues.

14 In normal mouse and rat liver, CYP26 mRNA was barely detect

15 Further, PS A stimulated proliferation of normal mouse and rat lymphocytes differentially.

16 In the normal mouse and rat retina, COX-2 immunoreactivity is p

17 bit TF-positive endothelium in the wild-type normal mouse and the normal human hemoglobin (HbA)-expre

18 Examination of normal mouse and tumor tissues showed that E4G10 bound t

19 ry response in inducing tumor apoptosis in a normal mouse and tumor-bearing mouse model.

20 uorescent membrane dye DiO and injected into normal mouse anterior chamber.

21 RT-PCR showed cftr mRNA expression in the normal mouse apical incisor tissue but not in the CFTR k

22 e (ApoE +/-), or no copies (ApoE -/-) of the normal mouse ApoE gene.

23 a gene expression pattern common to PAs and normal mouse astrocytes and neural stem cells from these

24 Normal mouse B cells and a variety of B lymphoma cells r

25 2 augments the proliferation and survival of normal mouse B cells and dramatically accelerates lympho

26 no clear evidence for SHC has been found in normal mouse B cells, transgenic mice carrying high-copy

27 was examined in the WEHI-231 B lymphoma and normal mouse B cells.

28 been shown to increase BCR responsiveness in normal mouse B cells.

29 tant form of human fibrillin-1 (C1663R) on a normal mouse background.

30 one levels by approximately 30% and restored normal mouse body weight patterns.

31 The in vivo labeling studies, where normal mouse bone marrow cells (i.e. high turnover) were

32 n accelerate granulocytic differentiation of normal mouse bone marrow cells induced by G-CSF and GM-C

33 ak3 accelerates monocytic differentiation of normal mouse bone marrow cells stimulated with cytokines

34 Furthermore, we show that incubation of normal mouse bone marrow cells with a JAK3-specific inhi

35 reen for general toxicity of shRNAs, we used normal mouse bone marrow cells.

36 ominant-negative activity (RARalpha403) into normal mouse bone marrow generates hematopoietic growth

37 ficantly increase colony-forming capacity of normal mouse bone marrow progenitor cells alone and, par

38 on-induced immortalization/transformation of normal mouse bone marrow progenitor cells in vitro.

39 Immature B cells from normal mouse bone marrow were not responsive to the supp

40 the 32Dcl3 cell line, which is derived from normal mouse bone marrow, is non-tumorigenic and diploid

41 In the normal mouse, both at basal hematopoiesis or following m

42 on protein (PrP(C)) substrate molecules from normal mouse brain by treatment with PNGase F and used r

43 shing a productive infection in at least one normal mouse brain cell type.

44 The level of Trim32 expression in normal mouse brain exceeds that observed in skeletal mus

45 the somatosensory cortex on one side of the normal mouse brain resulted in mirror infection restrict

46 Comparison of the transcriptomes of normal mouse brain with those of wild-type-RV-infected m

47 tal enrichment alters gene expression in the normal mouse brain, and modulates the course of several

48 rain penetrations and fast washouts from the normal mouse brain.

49 study the distribution of the protein in the normal mouse brain.

50 erved in choroid plexus and leptomeninges in normal mouse brain.

51 f the corresponding viral particles into the normal mouse brain.

52 ion was not detectable in the luminal ECs of normal mouse carotid arteries but was rapidly induced af

53 eptin receptor (ObR) is expressed on resting normal mouse CD4(+), CD8(+), B cells, and monocyte/macro

54 mor xenografts in mice with little effect on normal mouse cells or tissues.

55 We show that, in normal mouse cells, MHC I molecules present a peptide en

56 city of the PrP121-231 fragments to cultured normal mouse cerebellar cells.

57 interior face of the rhombic lip throughout normal mouse cerebellar development.

58 ition, serum from a GABHS mouse reacted with normal mouse cerebellum in nondenaturing Western blots a

59 rossly overexpress IRS-1, when compared with normal mouse cerebellum.

60 The (18)F-FDG rate constants of normal mouse cerebral cortex were determined.

61 ypical values of (18)F-FDG rate constants of normal mouse cerebral cortex were estimated and used for

62 pression of GnRH receptors was assessed in a normal mouse cholangiocyte cell line (NMC), sham, and BD

63 Polarized normal mouse cholangiocytes (NMCs) lacking endogenous AQ

64 ltiprotein complex at the apical membrane of normal mouse cholangiocytes, with proteins that negative

65 P-selectin (apoE(-/-) P(-/-)) that were fed normal mouse chow.

66 Normal mouse colon epithelium lacks Paneth cells, a key

67 al and cancer colon cell lines as well as in normal mouse colon.

68 ferences were seen in the neutropenia versus normal mouse comparison or by schedule.

69 lagen probably exists near the limbus of the normal mouse cornea that may help promote biomechanical

70 Likewise, S100A4 antigen was not found in normal mouse corneas.

71 demonstrate that PAM14 is not essential for normal mouse development and cell cycle control.

72 strate that Gadd45gamma is not essential for normal mouse development and hematopoiesis, possibly due

73 inase activity of Raf-1 is not essential for normal mouse development and that Raf-1 plays a key role

74 ull mice indicate that it is dispensable for normal mouse development as well as cell growth in cultu

75 Therefore, during normal mouse development the axes of the blastocyst, whi

76 (hESCs) have the capacity to participate in normal mouse development when transplanted into gastrula

77 as 4A, like H- and N-Ras, is dispensable for normal mouse development, at least in the presence of fu

78 ab3 does not result in detectable defects in normal mouse development, hematopoiesis, or immune syste

79 Thus, although LPA(2) is not essential for normal mouse development, it does act redundantly with L

80 t XLF functionally overlaps with DNA-PKcs in normal mouse development, promotion of genomic stability

81 hypomorphic and that Dicer is essential for normal mouse development.

82 argeting and find RIP3 to be dispensable for normal mouse development.

83 pression pattern of cyclins E1 and E2 during normal mouse development.

84 otochord, and dorsal somite, is required for normal mouse development.

85 ore able to support human sperm binding than normal mouse eggs.

86 they interfere with assembly of full-length normal mouse elastin.

87 cation of beta-catenin and transformation of normal mouse embryo fibroblasts (colony formation in sof

88 ction of S phase after growth stimulation of normal mouse embryo fibroblasts, inhibition of cyclin E/

89 nsformed mouse embryo fibroblasts but not in normal mouse embryo fibroblasts.

90 In the normal mouse embryo, Bmp4 is expressed in mesenchymal ce

91 of apoB-containing lipoproteins, but also in normal mouse embryonic development.

92 Treatment of normal mouse embryonic fibroblasts (MEF) with H2O2 resul

93 ute quantification of FAK phosphorylation in normal mouse embryonic fibroblasts (MEFs).

94 normal human embryonic fibroblast SW-38 and normal mouse embryonic fibroblasts.

95 increase in levels of p53 was the same as in normal mouse embryonic fibroblasts.

96 Our results showed that normal mouse embryonic stem (ES) cells had a very low fr

97 To clarify this, we studied normal mouse embryos (days 10 to 15 of gestation), which

98 nes tested, including lines established from normal mouse embryos and tumors, expressed very low or u

99 The normal mouse enamel matrix pH was generally higher and m

100 er cells with less cytotoxic effects against normal mouse epidermal cells.

101 During normal mouse epidermal development, Claudin-1 plasma mem

102 tures containing CRES are a component of the normal mouse epididymal lumen without any apparent cytot

103 Normal mouse eyeballs or whole-mount corneas encompassin

104 Corneas and irises from normal mouse eyes constitutively expressed TSP-1 and -2

105 of orthotopic corneal allografts placed into normal mouse eyes correlates directly with the generatio

106 sisted for 7 weeks, although the mean IOP in normal mouse eyes treated with 2 or 4.0 mg/kg verteporfi

107 DMXAA did not augment the damage to normal mouse feet after low-dose PDT (1.5 mg.kg(-1) Phot

108 oth in a human colon cancer cell line and in normal mouse fibroblast cells; the inhibition of cellula

109 Exposure of normal mouse fibroblasts (MEF3T3) to ionizing radiation

110 bacterial pathogen Listeria monocytogenes in normal mouse fibroblasts and in fibroblasts lacking inte

111 ie3 deletion mutants failed to replicate on normal mouse fibroblasts even when a high multiplicity o

112 can occur in the cytosol and is observed in normal mouse fibroblasts, as well as a variety of human

113 able to replicate with wild-type kinetics in normal mouse fibroblasts, providing evidence that the de

114 athematical modeling, we demonstrate that in normal mouse footpad epidermis, transition of KCs from b

115 n, while minimally affecting normal cell and normal mouse functions, markedly sensitized mouse skin a

116 The activation of Akt1 signaling in normal mouse gastrocnemius muscle was sufficient to prom

117 We show here that during normal mouse germ cell development, hnRNP G-T protein is

118 However, CIP2A is not essential for normal mouse growth and development.

119 our ability to address the role of Dicer1 in normal mouse growth and development.

120 st demonstrated that in competition with the normal mouse gut flora, an EHEC O157:H7 strain with a no

121 Although widely expressed in the developing normal mouse head, Opa3 expression was restricted after

122 sed very rapidly (at rates comparable to the normal mouse heart and to human atrial fibrillation), th

123 nstrated that focal ectopies require, in the normal mouse heart, the simultaneous depolarization of a

124 ces myocardial contraction and relaxation in normal mouse hearts during inotropic stimulation with is

125 NA and protein were present at low levels in normal mouse hearts, but were upregulated after cardiac

126 ty of an MLL-ENL fusion protein to transform normal mouse hematopoietic cells.

127 to chronic myelogenous leukemia (CML) caused normal mouse hematopoietic progenitor cells to divide mo

128 In normal mouse hematopoietic stem cells, the process of se

129 f PHB1 in murine nontransformed AML12 cells (normal mouse hepatocyte cell line) raised cyclin D1 expr

130 Similar changes were found in a "normal" mouse hepatocyte cell line (AML12), thus confirm

131 In PET experiments with normal mouse, high uptake of activity was obtained in th

132 Images of normal mouse histology and strain-dependent background l

133 The numbers of normal mouse HSCs were preserved whereas normal progenit

134 taiotaomicron, a prominent inhabitant of the normal mouse/human gut.

135 of either (111)In*aICAM-1 or (111)In-labeled normal mouse IgG ((111)In*nmIgG) and were imaged and kil

136 tes (PMNs), (111)In-aICAM-1, (111)In-labeled normal mouse IgG (nmIgG), and (111)In-labeled rat serum

137 Control groups were given saline or normal mouse IgG.

138 ecificity, 4B7 was tested in the presence of normal mouse IgG.

139 In comparison to the control normal mouse immunoglobulin G, passive immunization of B

140 When normal-mouse infection site tissue Mphi were adoptively

141 lyzed, including nontransformed human cells, normal mouse intestinal epithelia and adenomas, human ca

142 Uptake of D-FAC is a prominent trait of normal mouse intestinal epithelial cells, which is usefu

143 Immunohistochemistry of the normal mouse intestine detected AGR2 expression in proli

144 Cells were isolated from normal mouse intestine using DCAMKL-1 and fluorescence-a

145 ts indicate that induced otitis media in the normal mouse is in most respects comparable to that obse

146 cate that class-selective DIs interfere with normal mouse ischemic hindlimb regeneration and suggest

147 nedione activation (72 h) of this pathway in normal mouse islets caused a threefold increase of GIP-R

148 nsulin granule dynamics in insulinoma cells, normal mouse islets, and primary pancreatic beta cells.

149 alcium-induced terminal differentiation in a normal mouse keratinocyte model system.

150 The studies described here use normal mouse keratinocytes (C5N cells), and spindle carc

151 ow cytometry to analyze MPCs residing in the normal mouse kidney, and identified five discrete subpop

152 In the normal mouse kidney, the vast majority of 19 different W

153 probed with Invs cDNA detected four bands in normal mouse kidney.

154 pathway is active specifically in the SZ in normal mouse knee joints and colocalizes with HMGB2.

155 o seek sex differences in mRNA expression in normal mouse lacrimal glands.

156 level of ICSBP expression is very low in the normal mouse lens, in transgenic mice with constitutive

157 ponsins (U58883) was found to be abundant in normal mouse lenses.

158 lly expressed genes in Pax6 heterozygous and normal mouse lenses.

159 ing the transgene was used to isolate from a normal mouse library overlapping genomic DNA segments th

160 Thus, Dicer is required for the formation of normal mouse limbs.

161 n of mGSTA4-4 was examined in hepatocytes of normal mouse liver and in transfected HepG2 cells by flu

162 with deletions in env were nearly silent in normal mouse liver and showed substantial derepression i

163 tectable transgene expression in NHPHs or in normal mouse liver tissues.

164 derived from RDA using c-Myc transgenic and normal mouse liver.

165 shed rIFN-beta-induced CXCL-10 production in normal mouse livers.

166 Gene expression analysis comparing normal mouse lung fibroblasts and mouse lung CAFs identi

167 FGF-18 mRNA was detected in normal mouse lung in stromal cells surrounding proximal

168 Normal mouse lung tissue contained minute amounts of M-s

169 -1 RNA was detected at significant levels in normal mouse lung, intestinal epithelium, liver and thym

170 In normal mouse lung, the messenger RNAs (mRNAs) for SP-A,

171 ant enzyme that biosynthesizes PGE(2) in the normal mouse lung; (b) PGHS-1 and PGHS-2 products limit

172 A-1 in retention of activated CD8 T cells in normal mouse lungs and in the progression of lethal inju

173 Normal mouse lungs lack appreciable numbers of mast cell

174 ce and bronchiolar Clara cells isolated from normal mouse lungs, but was undetectable in whole lung e

175 g kinetics of f-PPE in the alveolar walls of normal mouse lungs.

176 Here, we show that for normal mouse lymph node T cells, PI 3-kinase activity is

177 r findings show that the ObR is expressed on normal mouse lymphocyte subsets, that leptin plays a rol

178 tively for human CD45RO surface receptor and normal mouse lymphocytes stained negatively confirming t

179 HC11 cells (a normal mouse mammary cell line) were grown to confluence

180 cal have distinct effects on the proteome of normal mouse mammary cultures, some estrogen-like but mo

181 -treated NMuMG cells, which are derived from normal mouse mammary epithelia.

182 stimulated beta-casein promoter activity in normal mouse mammary epithelial cells and also cooperate

183 of 157 metabolites during the progression of normal mouse mammary epithelial cells to an isogenic ser

184 ssing mouse mammary carcinoma cells (but not normal mouse mammary epithelial cells) undergo apoptosis

185 d differentiation and activation of Stat5 in normal mouse mammary epithelial cells.

186 Any epithelial portion of a normal mouse mammary gland can reproduce an entire funct

187 rmine the function of ErbB2 signaling during normal mouse mammary gland development, we expressed a c

188 To investigate the functions of ErbB2 during normal mouse mammary gland development, we transplanted

189 ithelial, and ductal epithelial cells in the normal mouse mammary gland, and becomes progressively re

190 In the normal mouse mammary gland, Tcf3 is highly expressed in

191 rmine the function of ErbB4 signaling in the normal mouse mammary gland, we inactivated ErbB4 signali

192 1DeltaE15 in epithelial and stromal cells in normal mouse mammary gland.

193 dem mass spectrometry (LC-MS/MS) analyses of normal mouse mammary glands or mammary tumors derived fr

194 J3Z cells also increase the proliferation of normal mouse mammary luminal cells adjacent to tumors.

195 Enforced expression of miR-142 or miR-150 in normal mouse mammary stem cells resulted in the regenera

196 a p53 null genotype on the transcriptome of 'normal' mouse mammary epithelium using a unique in vivo

197 Although we did not detect its expression in normal mouse melanocytes, Grm1 was ectopically expressed

198 ody-mediated effect required exposure to the normal mouse microbial flora, since mice raised under ge

199 Irradiated mice inoculated with normal mouse MLN macrophages (M) were shown to be resist

200 . faecalis Ag in cultures of I-MLNM, whereas normal mouse MLNM were converted to M1M in response to t

201 LL/2 epithelial cells but went undetected in normal mouse muscle, liver, spleen, or kidney.

202 ath, was more abundant in Lama2-null than in normal mouse muscles.

203 ns of other cytokines to enhance survival of normal mouse myeloid progenitors.

204 In normal mouse myocardium, galectin-3 was constitutively e

205 This effect was confirmed on normal mouse NCTC hepatocytes, but not on hepatoma cells

206 embryonic stem cells, human fibroblasts, and normal mouse neural differentiation and, using proteomic

207 tinct concentration optima for chemotaxis of normal mouse neutrophils, but only the high concentratio

208 Spinorphin induced calcium flux in normal mouse neutrophils, but was inactive in neutrophil

209 her control cortical wounds nor transplanted normal mouse or human cells were targeted, indicating vi

210 in g/mL. min) for (18)F-FTT was assessed in normal mouse organs before and after treatment with olap

211 arcoma cell lines compared with its level in normal mouse osteoblasts.

212 Intriguingly, normal mouse pancreas also contains a small population o

213 g leads to Ngn3 protein stabilization in the normal mouse pancreas explants.

214 dykinin-elicited Ca(2+) signal generation in normal mouse pancreatic lobules.

215 c-Met is a marker of normal mouse pancreatic stem and progenitor cells; we in

216 characterized transgenic line expressing the normal mouse peripherin/Rds (NMP) was crossed with a hig

217 Remnant lipoproteins (d < 1.019 g/ml) from normal mouse plasma were iodinated and injected into nor

218 In vitro, we demonstrate that normal mouse platelets, but not P-selectin-deficient pla

219 gical and phenotypic features of ERG gain in normal mouse prostate cells, including expansion of the

220 PDGF ligand switch from PDGF B to PDGF D in normal mouse prostate epithelial cells, further demonstr

221 idization analysis revealed LO expression in normal mouse prostate epithelium but, in most cases, pro

222 Here, we show that both normal mouse prostate tissue and human LuCaP35 prostate

223 us in human prostate cancer cells and in the normal mouse prostate.

224 cells but not in the epithelial cells of the normal mouse prostates.

225 mbryonic stem (ES) cells that also expressed normal mouse RAD51 from the other chromosome.

226 antly to multivesicular bodies of neurons in normal mouse, rat, and human brain.

227 n and localization of COX-1 and COX-2 in the normal mouse, rat, and human retina.

228 Normal mouse, rat, and rabbit whole eyes and rabbit ocul

229 ion datasets (Illumina WG 6.2 arrays) in the normal mouse retina and 2 days after ONC.

230 The normal mouse retina consists predominantly of rods, but

231 Normal mouse retina expressed very low levels of Bcl-2 a

232 In the normal mouse retina, COX-1 immunoreactivity is present i

233 In the normal mouse retina, Drp1 is expressed in the ganglion c

234 In the normal mouse retina, photoreceptoral and post-photorecep

235 population of 33D1(+) DCs was identified in normal mouse retina.

236 exceed by only approximately 23% that of the normal mouse retina.

237 ess rd mouse retinal cDNAs from the cDNAs of normal mouse retina.

238 ansport of folate, has been characterized in normal mouse retinal pigment epithelium (RPE) and in cul

239 ified human rhodopsin-GFP fusion gene at the normal mouse rhodopsin locus.

240 Normal mouse sera were shown to contain low levels of Ab

241 compared with stimulation in the presence of normal mouse sera.

242 observed in the presence of purified IgM and normal mouse serum (NMS), but not serum from Rag-2(-/-)

243 t Helicobacter felis activates complement in normal mouse serum but not in serum from Rag2(-/-) mice,

244 in down-regulation of ospA, but transfer of normal mouse serum depleted of immunoglobulin did not in

245 nimals given specific antiviral pIgA but not normal mouse serum eliminated virus from the airway and

246 nto the PspA(-) pneumococcus when exposed to normal mouse serum from wild-type mice as compared with

247 M (IgM) from normal mouse serum, or IgG from normal mouse serum into infected C3H-scid mice resulted

248 ys, parenterally fed animals received either normal mouse serum or antiviral pIgA before viral challe

249 ncubated isolated neutrophils for 3 hours in normal mouse serum or minimum essential medium buffer.

250 to that of controls with passive transfer of normal mouse serum or polyclonal immunoglobulin M (IgM),

251 Treatment of infected muMT(-CD4) mice with normal mouse serum spiked with hemagglutinin-specific Ig

252 Normal mouse serum supported more C3 deposition on pneum

253 required the presence of Mg(2+), whereas in normal mouse serum the presence of divalent cations was

254 resence of C1q-deficient serum compared with normal mouse serum, and the addition of purified C1q rec

255 gs identify not only the presence of FAAH in normal mouse serum, but also its critical role in the tu

256 n wt mice and in Rag1(-/-) mice administered normal mouse serum, demonstrating that a single Ab react

257 -specific PTEC that had been pretreated with normal mouse serum, heat-inactivated mouse serum, or com

258 nnii resistance to killing by NHS but not by normal mouse serum, highlighting the human species speci

259 Passive transfer of normal mouse serum, immunoglobulin M (IgM) from normal m

260 Following incubation in normal mouse serum, label-free quantitative proteomics i

261 mal mouse serum, immunoglobulin M (IgM) from normal mouse serum, or IgG from normal mouse serum into

262 lations of myogenic cells were isolated from normal mouse skeletal muscle based on their adhesion cha

263 enhanced permeability barrier homeostasis in normal mouse skin by the following mechanisms: (i) stimu

264 Human skin grafts of control mice and normal mouse skin on the test mice and control mice did

265 ed mitogen-induced cellular proliferation in normal mouse skin primary keratinocytes and in the C50 k

266 That is, it is not detected in normal mouse skin, however, a significant increase in ex

267 vivo, whereas little if any was detected in normal mouse skin.

268 t a hyperplastic response similar to that of normal mouse skin.

269 pidermal morphology and thickness similar to normal mouse skin.

270 and hypomethylation as a function of age in normal mouse small intestine tissues and a strong tissue

271 yR completely blocked ZP initiation of AR in normal mouse sperm.

272 nal antibody (mAb) (SCH79.08) raised against normal mouse spinal cord homogenate, which reacts with m

273 chaperone function in a cell-free extract of normal mouse spinal cords.

274 They are present at a high frequency in normal mouse spleen and bone marrow, are Ag dependent, a

275 y, we used the WEHI-231 mouse B lymphoma and normal mouse spleen B lymphocytes to elucidate the mecha

276 pts to identify their histologic location in normal mouse spleen have proven difficult due to both th

277 ulting in elevation of MK expression in both normal mouse splenic B and chronic lymphocytic leukemia

278 mulus of this phenotypic shift is culture of normal mouse splenic B lymphocytes with splenic fibrobla

279 ted excellent biocompatibility of LipoLLA to normal mouse stomach.

280 p upregulation was detected in the reference normal mouse strain (C57BL/6), which can develop signifi

281 fer of Ia-incompatible spleen cells from one normal mouse strain (such as B6.C-H2(bm12)/KhEg (bm12))

282 lar levels in both the preautoimmune and the normal mouse strains.

283 Genome-wide expression analysis of normal mouse tail skin and benign proliferative lesions

284 athology, hyperphosphorylated and aggregated normal mouse tau, significant neuron loss, and cognitive

285 ered laminin alpha5 chain showed that in the normal mouse, the protein had a restricted distribution

286 ymphoma lines S49.A2 and WEHI7.2 but also in normal mouse thymocytes following dexamethasone treatmen

287 ne T-cell lymphoma line WEHI7.2, but also in normal mouse thymocytes.

288 Many dendritic cells (DCs) in the normal mouse thymus are generated intrathymically from c

289 nal cord, and immunohistochemical studies of normal mouse tissue detected strong staining primarily i

290 s with microinsertions) have been studied in normal mouse tissues using the Big Blue lacI transgenic

291 These findings were confirmed in normal mouse tissues with anti-podoplanin antibody 8.1.1

292 ves tumor regression, but has no toxicity in normal mouse tissues.

293 observed in the non-transcribed lacI gene in normal mouse tissues.

294 s but not in endothelial cells isolated from normal mouse tissues.

295 enic Aurora A protein is readily degraded in normal mouse tissues.

296 Z in tumor cells but not in normal cells and normal mouse tissues.

297 rred in appropriate subcellular fractions of normal mouse tissues.

298 n XII-encoded sequence, has been detected in normal mouse tissues.

299 Normal mouse V1 neurons are dominated by contralateral e

300 ximal in the UV spectrum, in contrast to the normal mouse, which is most sensitive in the green regio