ライフサイエンスコーパス: normal pregnancy

1 reater at each gestational age compared with normal pregnancies).

2 V and V and alpha6-fucosyltransferase during normal pregnancy.

3 two subsets also changed in PE compared with normal pregnancy.

4 > 100 kDa) were similar in pre-eclampsia and normal pregnancy.

5 blood pressure, throughout the range seen in normal pregnancy.

6 hes in order to characterize this process in normal pregnancy.

7 onic gonadotropin between days 40 and 120 of normal pregnancy.

8 n their DNA methylation status compared with normal pregnancy.

9 ee DNA reach the maternal circulation during normal pregnancy.

10 expression of angiogenic factors throughout normal pregnancy.

11 ibute to the physiologic sodium retention of normal pregnancy.

12 antly raised in PE and IUGR as compared with normal pregnancy.

13 or placental bed in PE or FGR compared with normal pregnancy.

14 aternal insulin resistance, which occurs in "normal" pregnancy.

15 counts>400/mm3, no AIDS-defining illness and normal pregnancies.

16 FBP-1 levels were higher in diabetic than in normal pregnancies.

17 analyzed these glycosylation changes in five normal pregnancies.

18 n, with evidence of placental adaptations in normal pregnancies.

19 levels were significantly lower than that in normal pregnancies.

20 lampsia compared with plasma from women with normal pregnancies.

21 mannose across the umbilical circulation in normal pregnancies.

22 n artery resistance between preeclamptic and normal pregnancies.

23 respectively, in preeclampsia compared with normal pregnancies.

24 as significantly higher than in samples from normal pregnancies.

25 than it does among women with chromosomally normal pregnancies.

26 s 1 in 300 or higher in 7907 (8.3%) of 95476 normal pregnancies, 268 (82-2%) of 326 with trisomy 21,

27 in-1 level was not different in women during normal pregnancy (57 37-85 microg/L) compared to the unc

28 sitive to increased insulin concentration in normal pregnancy (96% suppression), but is less sensitiv

29 In normal pregnancy, a subpopulation of placental cytotroph

30 mmune and metabolic changes occurring during normal pregnancy also describe metabolic syndrome.

31 m 24 control women (20 with current or prior normal pregnancies and 4 who were nulligravid) was analy

32 mes of platelet activation and early in both normal pregnancies and in pregnancy-induced hypertension

33 nts were done on six healthy volunteers with normal pregnancies and nine with pregnancies complicated

34 preeclampsia plasma and plasma samples from normal pregnancies and nonpregnant women (294+/-110, 186

35 agnetic resonance (MR) imaging parameters in normal pregnancies and those complicated by fetal growth

36 eltaPo2 were significantly different between normal pregnancies and those complicated by severe FGR.

37 ofile of PdEs in maternal plasma of GDM with normal pregnancies and to determine the effect of exosom

38 tanes for placentas obtained from women with normal pregnancies and women with preeclampsia, a hypert

39 ons about physiologic changes present during normal pregnancy and after brain death, and the critical

40 describes the routine over-medicalisation of normal pregnancy and birth.

41 Insulin resistance is a cardinal feature of normal pregnancy and excess growth hormone (GH) states,

42 We hypothesized that NHGU is reduced during normal pregnancy and in a pregnant diet-induced model of

43 accommodating fetal nutrient requirements in normal pregnancy and in gestational diabetes mellitus (G

44 les (STBM), into the maternal circulation in normal pregnancy and in increased amounts in pre-eclamps

45 y modulate the maternal immune system during normal pregnancy and in the presence of an intrauterine

46 a preparation of freshly isolated CPASMCs of normal pregnancy and investigated K(+) channel expressio

47 53.7% and 56.8% of plasma S-nitrosothiols in normal pregnancy and preeclampsia, respectively.

48 uman chorionic gonadotropin, produced during normal pregnancy and that secreted by three human chorio

49 mpathetic output was increased in women with normal pregnancy and was even greater in the hypertensiv

50 artum hemorrhage, 2) uncomplicated labor, 3) normal pregnancy, and 4) non-pregnant patients with acut

51 21 women with preeclampsia and 21 women with normal pregnancy, and plasma samples were also obtained

52 of SHG endomicroscopy technology for staging normal pregnancy, and suggest its potential application

53 metabolic profile that develops as a part of normal pregnancy, and that when lactation does not occur

54 e pulsatile arterial load alterations during normal pregnancy are adaptive in that they help to accom

55 n maternal O(2) transport in third trimester normal pregnancy are unlikely to be causally associated

56 5 women at PE diagnosis and in 64 women with normal pregnancies at a similar stage.

57 its regulatory mechanisms in placentas from normal pregnancies at high (3100 m), moderate (1600 m),

58 ined from non-pregnant women, and women with normal pregnancies, before or after the spontaneous onse

59 nt in induced regulatory T cells (iTregs) in normal pregnancy but not in preeclampsia, implicating iT

60 r resistance measured in vivo and ex vivo in normal pregnancy, but not in FGR.

61 loss of VSMCs from SpAs during remodeling in normal pregnancy, but VSMCs appear to migrate away from

62 ion between pregnancy complicated by FGR and normal pregnancy by using DeltaPo2, baseline R1, and bas

63 Normal pregnancy can be associated with a decline in ene

64 ly higher level of IgM-AECA was found during normal pregnancy compared with that in healthy nonpregna

65 This review examines sleep in normal pregnancy; discusses the physiologic bases for al

66 ospective outpatient study of 90 consecutive normal pregnancies during routine obstetric services at

67 vasion is essential for the establishment of normal pregnancy, dysregulation of this process may cont

68 During normal pregnancy, elevated angiotensin II (Ang II) conce

69 This finding has implications for normal pregnancy, for obstetric complications that incre

70 t was generally 61.5% lower than that in the normal pregnancy group (P = .003).

71 plicated by IUGR differed significantly from normal pregnancies in patterns of perfusion within the p

72 s in sTNFR-I levels during the later half of normal pregnancies indicate that sTNFR-I may be importan

73 d 1 year postpartum compared with women with normal pregnancies, indicating increased cardiovascular

74 ntly higher in pre-eclampsia than in control normal pregnancy (inhibin A 3.05 [1.8] vs 0.36 [0.14] ng

75 t have indicated that insulin action in late normal pregnancy is 50-70% lower than in nonpregnant wom

76 The discovery that normal pregnancy is a controlled state of inflammation,

77 Normal pregnancy is also accompanied by sodium retention

78 Normal pregnancy is associated with a reversible fall in

79 Normal pregnancy is characterized by an early expansion

80 Normal pregnancy is characterized by decreased venous se

81 Normal pregnancy is characterized by the presence of inn

82 ), which is expressed in the placenta during normal pregnancy, is down regulated in pre-eclampsia, a

83 g finding was that while P(4) at higher than normal pregnancy levels conferred PR signaling sufficien

84 ic hyperactivity during the latter months of normal pregnancy may help to return the arterial pressur

85 thinner media, and higher I/M ratio than in normal pregnancy (mean I/M difference, 0.21; 95% confide

86 Resistance was measured in normal pregnancies (n = 10) and FGR (n = 10) both in viv

87 (preeclamptic plasma, N = 12) or plasma from normal pregnancies (normal plasma, N = 12).

88 women with late-onset preeclampsia (LPE) or normal pregnancy (NP) and investigated its underlying me

89 uring pregnancy and postpartum in women with normal pregnancy (NP) and PIH and in normotensive nonpre

90 s of plasma from women with preeclamptic and normal pregnancies on the transcriptome of an immortaliz

91 In normal pregnancies only, the highest level of ex vivo FM

92 iated specifically with malaria and not with normal pregnancy or even pregnancy with low birth weight

93 significantly higher in preeclampsia than in normal pregnancy or nonpregnancy plasma (6.3+/-1.4, 5.1+

94 the transient hypotension that occurs during normal pregnancy or that is induced by treatment with li

95 of NKG2D in poly(I:C)-treated WT mice led to normal pregnancy outcome.

96 nd/or localization may have consequences for normal pregnancy outcome.

97 ied 109 amniotic fluid samples of women with normal pregnancy outcomes (n = 28) and women with (n = 3

98 e plastic for induced Foxp3 expression, with normal pregnancy outcomes.

99 were exposed for 24 hours to preeclamptic or normal pregnancy plasma and their transcriptome was anal

100 d 186% compared with normal nonpregnancy and normal pregnancy plasma, respectively).

101 t variation of GPBB concentrations occurs in normal pregnancy, pre-eclampsia, and SGA pregnancies.

102 and second trimester placental tissues from normal pregnancies predominantly expressed IL-10, wherea

103 Fms-related tyrosine kinase 3 ligand, led to normal pregnancy rates in a spontaneous abortion-prone m

104 reased excretion of 3-HIA seen frequently in normal pregnancy reflects reduced biotin status.

105 but the exact nature of the differences from normal pregnancy remain elusive.

106 ority of the women with eating disorders had normal pregnancies, resulting in healthy babies.

107 vical tissue sections at different stages of normal pregnancy reveal progressive, quantifiable change

108 al S-nitrosothiols (11.1+/-2.9 nmol/mL) than normal pregnancy samples (9.4+/-1.5 nmol/mL).

109 showed a four-fold increase in sVEGFR-1 than normal pregnancies, suggesting that villous explants in

110 nd greater aldosterone concentrations during normal pregnancy than Caucasians.

111 the uterus stays remarkably quiescent during normal pregnancy to allow sufficient time for developmen

112 For normal pregnancy, tumor necrosis factor-alpha and deplet

113 Normal pregnancy was associated with a lower preference

114 mimic aspects of the uterine environment in normal pregnancy, we added PRL and IGFBP1, which enhance

115 Differentiating features from normal pregnancy were left ventricular wall thickness of

116 nown whether sympathetic drive is altered in normal pregnancy, when arterial blood pressure can be no

117 tochondria leads to oxidative stress even in normal pregnancy which is exacerbated further in IUGR, d

118 exhibited reduced neutrophil activation and normal pregnancies, which indicates that PAR2 plays an i

119 as the relative amounts of these glycans in normal pregnancy will be determined by gestational age.

120 Forty-eight normal pregnancies with a gestational age (GA) of 25 to

121 tprandial state is delayed and suppressed in normal pregnancy, with concomitant reduction in glycogen