ライフサイエンスコーパス: normal retina

1 genin-6 during degeneration when compared to normal retina.

2 ctivity in the rd1 retina was similar to the normal retina.

3 her than in the lesion but lower than in the normal retina.

4 e regions with functionally and structurally normal retina.

5 pericyte-containing microvasculature of the normal retina.

6 t of non-neurogenic environment of the adult normal retina.

7 ions of the empirically observed patterns in normal retina.

8 in CRB1 mutations resembles that of immature normal retina.

9 ld yield photoreceptor deficits in otherwise normal retina.

10 achments, increased to 180% of the amount in normal retina.

11 lowed by functional maturation by P30 in the normal retina.

12 d the development of retinal function in the normal retina.

13 d significant differences between AMD versus normal retinas.

14 ere more frequent in the AMD retinas than in normal retinas.

15 reased mtDNA control region SNPs compared to normal retinas.

16 reased blood flow due to vasodilation in the normal retinas.

17 ound in photoreceptor and RPE cell layers in normal retinas.

18 ntrast to the lightly stained glial cells in normal retinas.

19 ger blood vessels, compared with age-matched normal retinas.

20 clear layers of diabetic retinas, but not in normal retinas.

21 munoreactivity; these swellings were rare in normal retinas.

22 vertheless, significantly lower than that of normal retinas.

23 urvey for novel peroxidases expressed within normal retinas.

24 cular membranes in comparison with levels in normal retinas.

25 s no detectable level of the active forms in normal retinas.

26 gher levels in neovascular membranes than in normal retinas.

27 as switched to base medium preconditioned by normal retinas.

28 er between the junctional zone and adjacent "normal" retina.

29 The effect of PDT on normal retina and choroid (with no CNV) was studied in 9

30 Forty spots on the normal retina and choroid were treated with PDT and were

31 Molecular biological analyses of RNA from normal retina and from proliferative and myofibroblastic

32 Full-length CRMP-5 protein was identified in normal retina and optic nerve by Western blot analyses.

33 on and three-dimensional (3D) imaging of the normal retina and optic nerve head were performed.

34 In the normal retina and optic nerve, immunocytochemical staini

35 f, are more crucial than others in effecting normal retina and pigment epithelium development.

36 ed in innate immunity that is present in the normal retina and that is activated by optic nerve crush

37 Here, we determine which cell types in the normal retina and the optic nerve express OPA1.

38 serve as a marker for RGC quantification in normal retinas and for estimation of RGC loss in ocular

39 ility to modulate cell proliferation in both normal retinas and retinas with lesions.

40 overall pattern of immunoreactivity between normal retinas and retinas with severe loss of ganglion

41 tion at the tissue level is regulated in the normal retina, and how retinal diseases may affect oxyge

42 holesterol metabolism for maintenance of the normal retina, and suggest new targets for diseases affe

43 but the intrinsic survival mechanisms in the normal retina are poorly understood.

44 of the compensatory dilation observed in the normal retina, arterioles constrict in response to an ox

45 An additional Zone 4 was defined as "normal" retina, at least 500 mum from the edge of the GA

46 33D1(+) dendritic cells were present in the normal retina but were MHC class II(low/-).

47 e demonstrate that BBS2 retinopathy involves normal retina development followed by apoptotic death of

48 , it is imperative that we better understand normal retina development.

49 CS retinas were significantly larger than in normal retinas due to lower basal blood flow in the RCS

50 survival pathway in retinal neurons and that normal retina expresses a highly active basal insulin re

51 ed a custom retinal microarray to analyze 33 normal retinas from 19 donors, aged 29-90 years.

52 We found that the normal retina has a baseline expression of the LPA recep

53 ogy, diabetic patients with angiographically normal retinas have been found to exhibit subtle defects

54 egment optical coherence tomography showed a normal retina in all patients, but the subfoveal choroid

55 ells, when retrospectively enriched from the normal retina, like their radial glial counterparts in t

56 scribed here that attempt to identify in the normal retina of goldfish neuronal progenitors intrinsic

57 I (+) dendriform cells could be found in the normal retinas of highly EAU-susceptible B10.RIII mice.

58 but revealed no evidence of retinal waves in normal retina or during regeneration.

59 Within normal retina, OX2R was not detected on myeloid-derived

60 ntly longer for OFF RGCs than for ON RGCs in normal retinas (P = 0.025).

61 ot statistically different from those in the normal retinas (P>0.05).

62 In normal retinas, reflectance spectra were similar along t

63 images of the retinas of mice (B6/SJLF2 for normal retina, rhodopsin-deficient Rho(-/-) for photorec

64 unohistochemistry and electron microscopy of normal retinas showed that Mueller cells, which synthesi

65 inoblastomas was more similar to that of the normal retina than that of retina-derived iPSCs, and we

66 confined to the apical retinal surface as in normal retinas, the planar organization of cone photorec

67 X binding and dynamic epigenetic profiles in normal retinas, the up-regulated cone-enriched genes do

68 e produced a rapid and prolonged decrease in normal retina TrkB.FL.

69 Myeloid-derived cells from normal retina were programmed similar to TGF-beta-stimul

70 Expression profiles of mutant and normal retinas were compared by using canine retinal cus

71 l significance of Hmgb1 release, ischemic or normal retinas were treated with the neutralizing anti-H

72 to cone photoreceptor outer segments in the normal retina, whereas no Tgamma c immunoreactivity was

73 understanding of these signaling pathways in normal retina will provide a firm basis for targeting th

74 CD81 was found in all layers of the normal retina with a distinct absence of labeling in the

75 ave regions of structurally and functionally normal retina with definable transitions to severe lamin