ライフサイエンスコーパス: normoxia

1 7%, glucagon 48.6 +/- 2.1%, all P < 0.05 vs. normoxia).

2 however, commonly performed in ambient air (normoxia).

3 BK/Kv were mostly closed at rest in normoxia.

4 tamine to lipogenesis in hypoxia, but not in normoxia.

5 Ia up-regulated the HIFalpha Ia levels under normoxia.

6 is limited by the lability of the protein in normoxia.

7 eases basal expression of GM-CSF in cells in normoxia.

8 y reduced sRBC adhesion to levels seen under normoxia.

9 ts HIFs to the proteasome for degradation in normoxia.

10 CSH rats but had no effect on ventilation in normoxia.

11 ivalent relative and absolute intensities in normoxia.

12 iferation of Leishmania promastigotes during normoxia.

13 ow that ERRs also stimulate glycolysis under normoxia.

14 after exposure to IH in vitro compared with normoxia.

15 m for multilevel regulation of HIF-1alpha in normoxia.

16 m mice exposed to either 24 hours hypoxia or normoxia.

17 se tissue is insufficient to maintain tissue normoxia.

18 ntrol); and (3) to the limit of tolerance in normoxia.

19 ood flow during hypoxic exercise relative to normoxia.

20 pal water content was increased, compared to normoxia.

21 knockdown of DEGS1 or DEGS2 by siRNA during normoxia.

22 ed relative to the same level of exercise in normoxia.

23 tant inhibitory effect on its activity under normoxia.

24 and oxidative function to values observed in normoxia.

25 a abolishes the myogenic effect of IGF under normoxia.

26 postal day 0-5) were exposed to either IH or normoxia.

27 iated with increased mortality compared with normoxia.

28 al mortality compared with either hypoxia or normoxia.

29 ress constitutive levels of HIF-1alpha under normoxia.

30 , suggesting enhanced cluster disassembly in normoxia.

31 ession by post-transcriptional regulation in normoxia.

32 ith mES cells that were differentiated under normoxia.

33 95% oxygen for 4 days, followed by return to normoxia.

34 riptional and post-transcriptional levels in normoxia.

35 ts and mice exposed either to 10 d of CIH or normoxia.

36 esponsible for suppressing HIF levels during normoxia.

37 nt, proteasomal degradation of HIF-1alpha in normoxia.

38 ed retina to a level higher than that during normoxia.

39 ) that were exposed to 1-4 days of CIH or to normoxia.

40 arkedly induced differentiation of CFs under normoxia.

41 mice were exposed to 75% oxygen followed by normoxia.

42 vity and extends replicative life span under normoxia.

43 tion of hypoxia-inducible factor (HIF) under normoxia.

44 growth recovery when cells were returned to normoxia.

45 d Experiment 3) Lutein supplemented media in normoxia.

46 posure to hypoxia, continuing upon return to normoxia.

47 ced by over 2-fold under hypoxia compared to normoxia.

48 means that these afferents are active under normoxia.

49 ygen sensor that controls HIF activity under normoxia.

50 evels of l-lactate after its formation under normoxia.

51 real time are limited to areas of hypoxia or normoxia.

52 he P wave in V1 was lower in hypoxia than in normoxia.

53 occurring during neuronal regeneration under normoxia.

54 0001) and hyperoxia (P=0.0006) compared with normoxia.

55 enerally showed a more favorable outcome for normoxia.

56 (18%), 3999 had hypoxia (63%), and 1171 had normoxia (19%).

57 3D pellets of human primary chondrocytes in normoxia (20% oxygen) and hypoxia (2.5% oxygen) and empl

58 to the limit of tolerance (T(lim)), once in normoxia (20.9% O(2); CON) and twice in hypoxia (14.5% O

59 in hypoxia (1 and 0.2% oxygen) compared with normoxia (20.9%).

60 Eighteen male lowlanders were examined in normoxia (21% O(2)) and following 6 h passive exposure t

61 prague-Dawley rats were preexposed to either normoxia (21% O(2)) or hyperoxia (85% O(2)) for up to 21

62 Newborn rats (6 days) were subjected to normoxia (21% O(2)) or PC (8% O(2)) for 3h followed by 2

63 Pregnant ewes were exposed to normoxia (21% O2 ) or hypoxia (10% O2 ) from 105 to 138

64 exposed to normobaric hypoxia (12% O2 ) and normoxia (21% O2 ).

65 In contrast, normoxia (21% O2) prevented the expression of these inhi

66 sulin was decreased in hypoxia compared with normoxia (225 +/- 23 vs. 128 +/- 30 nmol (kg fat free ma

67 -1); P < 0.05), but this was not the case in normoxia (289 +/- 15 ml min(-1); P = 0.33).

68 ime was reduced by 54%in hypoxia compared to normoxia (3.6 +/- 1.3 vs. 8.1 +/- 2.9 min; P<0.001).

69 Male Sprague-Dawley rats were given normoxia (30% O(2)) or NBO (95% O(2)) during 10, 30, 60

70 n of exercise in hypoxia was greater than in normoxia (345 +/- 21 ml min(-1) vs. 297 +/- 18 ml min(-1

71 FAT-luciferase reporter mice were exposed to normoxia (630 torr) or hypoxia (380 torr) for 2, 7, or 2

72 s pretreated with IH-1 (2 min hypoxia, 2 min normoxia; 8 h) or sham normoxia and allowed 16 h for rec

73 R 1.5, 95% CI 1.1-2.5, p=.01) as compared to normoxia (87/403 [23%]).

74 .001) with hypoxia (63 +/- 2%) compared with normoxia (96 +/- 0%), and was unaffected by sympathetic

75 addition of a blue light-blocking filter in normoxia, a significant increase in angiogenin levels wa

76 layed aerobic glycolysis when cultured under normoxia, accompanied by increased free NADH and NADH/NA

77 ntly, regeneration of dopamine neurons under normoxia also depends on ROS-production.

78 In normoxia, AMPA increased ventilation 25% and 50% in CON

79 In normoxia, AMPK induces PGC-1alpha, but how HIF is activa

80 compared with wild-type PASMCs, during both normoxia and after acute hypoxia.

81 (2 min hypoxia, 2 min normoxia; 8 h) or sham normoxia and allowed 16 h for recovery.

82 cular density between young and aged mice in normoxia and at 2 and 3weeks of hypoxia.

83 H23390 increased ventilation during baseline normoxia and did not affect ventilation during exposure

84 an explicit delineation between physiologic normoxia and genuine hypoxia is defined here, with impli

85 es were acquired in the rats at steady-state normoxia and hyperoxia and then during dynamic gas chall

86 a glucose concentrations were similar during normoxia and hyperoxia at baseline (5.52 +/- 0.15 vs. 5.

87 ell death in wild-type and GGT(enu1) mice in normoxia and hyperoxia.

88 tion in ECs suppressed Dll4 expression under normoxia and hypoxia and inhibited Dll4-induced Notch si

89 t multiple RTKs may regulate the HIF axis in normoxia and hypoxia and suggest that multikinase inhibi

90 were to compare ECG at moderate exercise in normoxia and hypoxia at the same heart rate, to provide

91 asurement of inner retinal OEF in rats under normoxia and hypoxia based on vascular oxygen tension (P

92 oxidation or Amplex Red, was similar during normoxia and hypoxia but markedly increased during reoxy

93 a cells in the presence/absence of OCs under normoxia and hypoxia conditions and did protein profilin

94 ily member in breast cancer cells under both normoxia and hypoxia conditions.

95 posure, compared to dark conditions, in both normoxia and hypoxia conditions.

96 verexpression decreases HIF-1alpha levels in normoxia and hypoxia in both pVHL-competent and -deficie

97 induces significant tumor cell apoptosis in normoxia and hypoxia in p53-positive cells.

98 al oxygen metabolism (MO2_IR) under systemic normoxia and hypoxia in rat.

99 nner retinal OEF measurements obtained under normoxia and hypoxia were compared.

100 In both normoxia and hypoxia, ACZ resulted in higher arterial P(

101 y impaired in conidia germination, growth in normoxia and hypoxia, and displayed attenuated virulence

102 , drives breast cancer cell proliferation in normoxia and hypoxia, and epigenetically regulates the e

103 Both in normoxia and hypoxia, dietary nitrate suppressed cardiac

104 tochondria of these cells, shuttling between normoxia and hypoxia, maintain bioenergetic efficiency a

105 In both normoxia and hypoxia, PASMC HIF-1alpha maintains low pul

106 chondrial respiration in breast cancer under normoxia and hypoxia, which correlates with decreased mi

107 opy or healthy controls were incubated under normoxia and hypoxia, with or without glucocorticoids.

108 ndent reduction in HIF-1 activity under both normoxia and hypoxia.

109 as detected in MMP-1-transfected cells under normoxia and hypoxia.

110 e, inducing a mortality by about 50% in both normoxia and hypoxia.

111 argely sequestered in breast cancer cells at normoxia and hypoxia.

112 ity and decreases polyubiquitination in both normoxia and hypoxia.

113 led with decreased GSK3beta activation under normoxia and hypoxia.

114 r for glycolysis and energy production under normoxia and hypoxia.

115 sponse elements, are regulated by ERalpha in normoxia and hypoxia.

116 beta-adrenergic-stimulated lipolysis in both normoxia and hypoxia.

117 by heterogeneous palladium chemistry both in normoxia and hypoxia.

118 ort term culture under hypoxia compared with normoxia and in response to interleukin 15 (IL-15) primi

119 n both the immature and mature neurons under normoxia and in the mature neurons under hypoxic conditi

120 Inner retinal OEF was 0.46 +/- 0.13 under normoxia and increased significantly to 0.67 +/- 0.16 un

121 on yeast exhibited growth defects under both normoxia and low oxygen conditions.

122 DO2_IR was similar under normoxia and moderate hypoxia (P = 0.7), but significant

123 Likewise, MO2_IR under normoxia and moderate hypoxia was similar (P = 0.1), but

124 0.01) than that observed at task failure in normoxia and moderate hypoxia.

125 arm exercise (10% and 20% of maximum) during normoxia and normocapnic hypoxia (80% arterial O(2) satu

126 on early after cardiac arrest such that both normoxia and normocarbia were documented in only 25 pati

127 tion guidelines that advocate maintenance of normoxia and normoventilation after pediatric cardiac ar

128 HIF-1alpha restores the microvascular airway normoxia and prevents airway fibrosis highlight a novel

129 dipocytes exhibited lower basal lipolysis in normoxia and reduced beta-adrenergic-stimulated lipolysi

130 sion tomography (15O-PET) were undertaken at normoxia and repeated at hyperoxia (FiO2 increase of bet

131 1) min(-)(1); P =0.03), and unchanged during normoxia and sympathetic inhibition (219 +/- 19; P =0.86

132 oxia (FIO(2) =0.21), hypoxia (FIO(2) =0.11), normoxia and sympathetic inhibition (via 48 h transderma

133 glycogen provisioning when they experienced normoxia and to decrease embryo glycogen provisioning wh

134 re compared with wild-type littermates under normoxia and with exposure to either acute or chronic hy

135 t in normoxia, during hypoxia (P<0.05 versus normoxia), and especially during exercise (P<0.05 versus

136 ) was injected into the arterial line during normoxia, and during early and late hypoxia, and their h

137 ected into the vitreous after restoration to normoxia, and its effects on vascular growth were analyz

138 omotor exercise in acute hypoxia compared to normoxia, and that such change would be related to reduc

139 ioning underlies adaptation to a fluctuating normoxia-anoxia hatching environment by increasing embry

140 , populations facing irregularly fluctuating normoxia-anoxia hatching environments failed to evolve r

141 )) clamps 1 week apart, randomized to either normoxia (arterial P(O2) (P(aO2)) 111 +/- 6.3 mmHg) or h

142 lternated between 1.0 (hyperoxia) and 0.209 (normoxia) as follows: hyperoxia for 180 s, normoxia for

143 tylcysteine (NAC), HBO and NAC, and control (normoxia at sea level).

144 3.5% higher (P < 0.01) during hyperoxia than normoxia at steady state during the clamp (28.2 +/- 0.15

145 relative to control exposures breathing air (normoxia) at each light level.

146 lyzed VHL-R167Q proteostasis and function at normoxia, at hypoxia with different oxygen pressure, and

147 increased levels under hypoxia compared with normoxia both in vitro and in vivo.

148 mplex for driving RTA expression not only in normoxia but also in hypoxia.

149 pensable for muscle stem cell function under normoxia but are required for maintaining satellite cell

150 , which express F77 antigen moderately under normoxia but at an elevated level under hypoxia.

151 and von Hippel-Lindau protein (pVHL) during normoxia but not in hypoxia.

152 H(2)S decreased in the presence of NO under normoxia but not under anoxia indicating that H(2)S does

153 conclusion, CYGB revealed TSG properties in normoxia but promoted tumourigenic potential of the cell

154 hose alpha subunit is rapidly degraded under normoxia but stabilized when O2-dependent prolyl hydroxy

155 to be critical in cardiac hypertrophy under normoxia, but its role in the heart under hypoxia is poo

156 ation of sodium nitrite had little effect in normoxia, but produced significant vasodilation and incr

157 C1 neurons have low activity under normoxia, but their activation is important to BP stabil

158 as a suppressor of tumor angiogenesis under normoxia by simultaneously down-regulating potent pro-an

159 e activity of these kinases is stimulated in normoxia by the oxygen-sensing prolyl hydroxylase PHD1 (

160 ow that chondrocytes cultured in hypoxia and normoxia can be differentiated by their lipid profiles.

161 ls' ATP content was similar to control under normoxia, cellular ATP did not change significantly in I

162 e and body weight were reduced compared with normoxia, cIH induced systemic insulin resistance in a h

163 Compared with normoxia, CO increased approximately 30% phagocytosis of

164 To test this, we treated rats held in normoxia (CON) or 10% O2 (CSH) for 7 days and measured v

165 t in liver mitochondria at a low level under normoxia conditions.

166 te hypoxia; (2) for the same duration but in normoxia (control); and (3) to the limit of tolerance in

167 of normal blood vessel maturation similar to normoxia controls.

168 Bifurcated BACH2 controls during hypoxia and normoxia coordinate not only MCL tumor dispersal but als

169 gher in the hyperoxia group as compared with normoxia (crude odds ratio 1.7 [95% CI 1.3-2.1]; p < 0.0

170 cantly during chronic hypoxia but not during normoxia (Delta: 4.8 +/- 1.6 vs. 0.5 +/- 1.4 mumol l(-1)

171 od flow after 2 hours of hypoxia (hypoxia vs normoxia: Delta148ml/min(-1) , 95% confidence interval [

172 and near-physiological concentrations via a normoxia-dependent mechanism that is associated with cGM

173 None of the mice exposed to normoxia developed atherosclerosis.

174 entiated phenotype, whereas CSCs cultured at normoxia did not.

175 rmation (venous>arterial; P<0.05) at rest in normoxia, during hypoxia (P<0.05 versus normoxia), and e

176 hat ERF-VII N-terminal cysteine oxidation in normoxia enables arginylation followed by proteasomal de

177 cultured for 24 h as follows: Experiment 1) Normoxia, Experiment 2) Hypoxia, and Experiment 3) Lutei

178 ts of i.v. vasodilators in Sugen5416/hypoxia/normoxia-exposed PAH rats.

179 a very late stage of the Sugen 5416/hypoxia/normoxia-exposed rat is accompanied by the formation of

180 on, and DNA synthesis compared to cells from normoxia-exposed rats.

181 (means +/- SEM)), in a random order, during normoxia (FIO(2) =0.21), hypoxia (FIO(2) =0.11), normoxi

182 C57BL/6J mice were exposed to CIH or normoxia for 10 weeks while being treated with either SC

183 (normoxia) as follows: hyperoxia for 180 s, normoxia for 120 s, hyperoxia for 120 s, normoxia for 12

184 s, normoxia for 120 s, hyperoxia for 120 s, normoxia for 120 s, hyperoxia for 60 s and normoxia for

185 , normoxia for 120 s, hyperoxia for 60 s and normoxia for 120 s.

186 ays in comparison to both mild hyperoxia and normoxia for all metrics except for the worst PaO2.

187 They were then returned to normoxia for an additional 10 to 11 weeks.

188 were exposed to repeated hypoxia or repeated normoxia from P1 to P3.

189 IV 4) and mature (DIV 20) neurons; 3) during normoxia GABA, glycine and taurine decreased GABA(A)Ralp

190 e interval {CI}, 60%-66%]) compared with the normoxia group (532/1171 [45%; 95% CI, 43%-48%]; proport

191 e small intestine and ascending colon of the normoxia group.

192 sh injected intraperitoneally with FLX under normoxia had resting cardiovascular and ventilatory para

193 At task failure in normoxia (haemoglobin saturation (SpO2) approximately 94

194 control value in the detached retina during normoxia; hyperoxia increased Q(av) to 68% +/- 17% of co

195 Compared with normoxia, hypoxia significantly increased palmitate-indu

196 reased ventilation of EH hamsters exposed to normoxia, hypoxia, and hypercapnia.

197 HAF knockdown increases HIF-1alpha levels in normoxia, hypoxia, and under epidermal growth factor sti

198 hibited lower ventilation during exposure to normoxia, hypoxia, or hypercapnia, but comparable ventil

199 h activation (10 s) in conscious rats during normoxia, hypoxia, or hyperoxia.

200 Under normoxia, IGF activates the Akt-mTOR, p38, and Erk1/2 MA

201 rols; i.e. acute hypoxia in CON and CSH, and normoxia in CSH.

202 1alpha (HIF-1alpha), which accumulates under normoxia in LKB1-deficient cells and is antagonized by i

203 ption factor to drive HK2 gene expression in normoxia in these cells.

204 AGO1 knockdown increased angiogenesis under normoxia in vivo.

205 allyl glycine, a stabilizer of HIF-1alpha at normoxia, increased IL-22 expression.

206 s as a sensor of glucose availability during normoxia, inducing apoptosis in response to glucose depl

207 Hypoxia favors stem cell quiescence, whereas normoxia is required for stem cell activation, but wheth

208 sts that culturing cells in ambient air, or "normoxia," is far from physiological or "normal." In fac

209 IGF promotes myoblast differentiation under normoxia, it stimulates proliferation under hypoxia.

210 ry in four groups of ewes: normoxic control, normoxia + ketamine, hypoxic control and hypoxia + ketam

211 Recurrent periods of hypoxia and normoxia lasted for a duration of 3h.

212 roup) and 3 weeks of hypoxia plus 2 weeks of normoxia (late group).

213 ate that the higher cholesterol levels under normoxia might regulate fibroblast growth factor 1 (FGF-

214 In normoxia, miR-574-3p, acting as a decoy, binds cytoplasm

215 we report that cytosolic acidification under normoxia moderately elevated 2-HG in cells, and boosting

216 80%, and were exposed to 13% O2 (hypoxia) or normoxia (n = 10 per group) for 14 days.

217 s of inspired oxygen (FiO2) to induce either normoxia (n = 10), moderate hypoxia (n = 14), or severe

218 centa from near-term C57BL/6J mice housed in normoxia (n = 8) or hypoxia (10% oxygen, n = 7-9) from d

219 c chain by different patterns of impulses in normoxia (N) and systemic hypoxia (H: breathing 8% O(2))

220 Upon return to normoxia, Ndufs4 KO mice die within days.

221 In normoxia, neither lateral ventricular volume (R(2) = 0.0

222 l infarction, exosomes derived from neonatal normoxia, neonatal hypoxia, infant hypoxia, and child hy

223 cise at 20% maximal voluntary contraction in normoxia (NormEx) and isocapnic hypoxia (HypEx; O2 satur

224 experiments, under controlled conditions of normoxia, normocarbia, and normothermia, spontaneously b

225 uential exposure to hypoxia/hypoglycemia and normoxia/normoglycemia (H/H-N/N).

226 2) to fraction of inspired oxygen <300); and normoxia, not classified as hyperoxia or hypoxia.

227 O2/FIO2 ratio less than or equal to 300, and normoxia, not defined as hyperoxia or hypoxia.

228 g (7.99 kPa) or PaO2/FiO2 ratio </= 300, and normoxia, not defined as hyperoxia or hypoxia.

229 Hg (7.99 kPa) or PaO2/FiO2 ratio </=300 and normoxia, not defined as hyperoxia or hypoxia.

230 cardiopulmonary resuscitation compared with normoxia (odds ratio, 1.77; 95% CI, 1.03-3.30).

231 e 2 diabetic rats were housed for 3 weeks in normoxia or 11% oxygen.

232 (beginning at P90) were subjected to either normoxia or CCH for durations of 14 or 28 days.

233 en or vehicle followed by exposure to either normoxia or chronic hypoxia (10% O2) for 30 days before

234 ppm) diet for 14 weeks and were continued in normoxia or exposed to hypoxia (8% O2) for the last 4 we

235 eurons contribute little to resting BP under normoxia or hypercapnia, C1 neuron discharge is restrain

236 embryos (n = 11 per group) were incubated in normoxia or hypoxia (14% O2 ) from day 1 and treated wit

237 ncy, rats (n = 20 per group) were exposed to normoxia or hypoxia +/- vitamin C.

238 Neonatal mice exposed to normoxia or hypoxia were randomly assigned to receive da

239 transcriptional activity than controls under normoxia or hypoxia, and pulmonary arteries from affecte

240 In normoxia or hypoxia, RTN inhibition produced a more sust

241 th in-hospital death as compared with either normoxia or hypoxia.

242 CF-7 extract prepared from cells grown under normoxia or hypoxia.

243 mouse brain endothelial cells maintained in normoxia or hypoxia.

244 nished the level of VEGF under conditions of normoxia or hypoxia.

245 F4E is the dominant cap-binding protein (21% normoxia or standard cell culture conditions), where eIF

246 RASF) under hypoxic conditions but not under normoxia or TNF-alpha treatment.

247 (1) Under normoxia, overexpression of N-end-rule-insensitive Delta

248 (2) decreased significantly as compared with normoxia (P < 0.001; n = 10).

249 ionships between hypoxemia (PaO2 < 60mm Hg), normoxia (PaO2 60-100mm Hg), moderate hyperoxia (PaO2 10

250 tion during anoxia alone or by NO2(-) during normoxia places constraints on how S-nitrosation occurs

251 ned implants remained cartilaginous, whereas normoxia-preconditioned implants readily underwent calci

252 Exogenous hydrogen peroxide under normoxia prevented hydroxylation of HIF-1alpha protein a

253 We postulate that hydroxylation of ASB4 in normoxia promotes binding to and degradation of substrat

254 tors antimycin A and potassium cyanide under normoxia promotes transient MPK6 and MPK3 activation.

255 ed a high incidence of mortality compared to normoxia rats.

256 icient to confer susceptibility to postnatal normoxia, reminiscent of bronchopulmonary dysplasia.

257 ol (47.7+/-5.7 vs. 20.1+/-4.3%) (hypoxia vs. normoxia, respectively; P<0.01), a finding mimicked in n

258 er the curve values (expressed as percentage normoxia response) for counterregulatory hormones during

259 der the noradrenaline curve (relative to the normoxia response) was increased with hypoxia (137 +/- 1

260 c hearts that were metabolically abnormal in normoxia, resulting in glycolytic rates 30% lower, and f

261 the other hand, knockdown of miR-199a during normoxia results in the upregulation of Hif-1alpha and S

262 pithelial cells after exposure to hypoxia or normoxia revealed dramatic changes in ECM regulators.

263 ption, and neither hypoxia nor atpenin A5 in normoxia robustly stabilizes hypoxia-inducible factor (H

264 4 atm) for 21 days and allowed to recover at normoxia (room air) for up to 21 days.

265 s exposed to hypoxia (3 days in 1% of O2) or normoxia (room air).

266 s(-1)) on a level gradient under normobaric normoxia (room air, 21% O2), moderate hypoxia (15% O2),

267 Under normoxia, SCD mice display bone loss and bone impairment

268 ecular oxygen and 2-oxoglutarate that, under normoxia, selectively hydroxylate proline residues of HI

269 In vivo studies confirm normoxia silencing, hypoxic induction, and cell specific

270 rformed on cancer cells grown in hypoxia and normoxia strongly suggest that DNQ undergoes bioreductio

271 1) expression were reduced compared with the normoxia/SU5416 control group.

272 ic subunits caused cardiac restriction under normoxia that are further aggravated under hypoxia.

273 ting mir-210 represses genes expressed under normoxia that are no longer necessary to adapt and survi

274 itric oxide (NO) in hypoxia (P < 0.05 versus normoxia), the normoxic baseline plasma and red blood ce

275 Compared with normoxia, the breathing frequency reduction (Deltaf(R))

276 At normoxia, the levels are kept low as a consequence of th

277 Under normoxia, the oxygen-sensitive alpha subunit of HIF1 is

278 In normoxia, three isoHbs predominated (Hbalpha-3.1, -3.2,

279 table during hypoxia but destabilized during normoxia through their targeting to the N-end rule pathw

280 dazole increased from 0.44% +/- 0.17% during normoxia to 2.24% +/- 0.08% during hypoxia.

281 A shorter protocol with normoxia to hyperoxia was also performed (five levels of

282 if mitochondria were rapidly converted from normoxia to near-anoxia ([O(2)], <1 micromol/L), the inc

283 response to hypoxic gradients extending from normoxia to severe hypoxia, and therapy responsiveness,

284 We conclude that across the range of normoxia to severe hypoxia, the major determinants of ce

285 ase into the medium was 428% greater than in normoxia-treated controls (45-min normoxic incubation) a

286 Whereas normoxia-treated KO mice die from neurodegeneration at a

287 During modest hypothermia and normoxia, TVR was high and CVA unit activity was present

288 hypoxia inducible factor (HIF) pathway under normoxia using a prolyl-4-hydroxylase inhibitor, dimethy

289 under physiologically normal oxygen levels (normoxia) vaccinia virus (VACV) infection leads to a rap

290 9.6 +/- 43.2% vs. 296.0 +/- 43.9% LF/HFHRV , normoxia vs. hypercapnia, respectively), incidence of ca

291 Normoxia was documented in 34% and hypoxia in 22% of pat

292 In normoxia, we detected increased expression of the microR

293 urements of inner retinal OEF obtained under normoxia were compared between nasal and temporal retina

294 as ventilation and blood gases at rest under normoxia were normal.

295 ntributes to the low sensory activity during normoxia, whereas H2S is excitatory and mediates sensory

296 awake, during normal ventilation, and during normoxia, which contributed to hypertension and organ da

297 m, and induction of cell death compared with normoxia, which was attenuated by NOS inhibition.

298 n of downstream protein-coding regions under normoxia, which was further modulated by hypoxia.

299 d flow and femoral blood flow (P<0.05 versus normoxia) with further, more pronounced increases observ

300 In normoxia, Yoh attenuated the responses evoked by high fr