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1 s known, whereas the pathogenesis of ONHD is not clear.
2 HCV); however, the impact of nonheavy use is not clear.
3 Ss), but the mechanism of their selection is not clear.
4 but a sex difference in this association was not clear.
5 s (MSC), but their relationship with MSCs is not clear.
6 or patients with sentinel-node metastases is not clear.
7 tosis, although widely studied, is currently not clear.
8 thies, or tau-mediated neurodegeneration, is not clear.
9 hronic hyperglycemia on host alloimmunity is not clear.
10 ny organisms, its functional significance is not clear.
11 IBD), but the mechanisms of this process are not clear.
12 diated ischemic muscle neovascularization is not clear.
13 ible for their formation and maintenance are not clear.
14 o treatment with systemic corticosteroids is not clear.
15 wide spatial-temporal activity sequences is not clear.
16 er, the efficacy of AMD in either outcome is not clear.
17 ent AFP (pt-AFP) in non-viral HCC (nvHCC) is not clear.
18 ing mechanisms in tropical forests are still not clear.
19 ever, how HR-deficient cells process DSBs is not clear.
20 mmune responses following such encounters is not clear.
21 rmined by the tissue in which they reside is not clear.
22 rstanding of T cell population dynamics, are not clear.
23 ssembly, stability, and maintenance is still not clear.
24 e ILC lineages develop and are maintained is not clear.
25 binding regulates transcriptional changes is not clear.
26 nce, but the nature of this heterogeneity is not clear.
27 ; however, the connection between the two is not clear.
28 cholerae surface, but its specific role was not clear.
29 However, their roles in plants are not clear.
30 causing variation from person to person are not clear.
31 n the chemotaxis of bacterial populations is not clear.
32 driving organ precursor formation are often not clear.
33 Treg cells proliferate in these patients is not clear.
34 gy and physiology on annual GPP variation is not clear.
35 n immunodeficiency virus (HIV) or frailty is not clear.
36 fficient to drive cellular transformation is not clear.
37 on local circuit and network computations is not clear.
38 ons are affected in ALS/FTLD pathogenesis is not clear.
39 ugh the mechanisms underlying cell death are not clear.
40 ting, which can at times lead to obesity, is not clear.
41 e exact relationship between DLG5 and YAP is not clear.
42 lation elongation speed in animal systems is not clear.
43 tibodies are produced in memory responses is not clear.
44 e mechanisms underlying this T cell loss are not clear.
45 selectively adhere to different surfaces is not clear.
46 (ICH), but how this function is regulated is not clear.
47 stics of quiescent epithelial stem cells are not clear.
48 mites feed on adult bees for a few days, is not clear.
49 , but the explanation for this preference is not clear.
50 ty associated with these poly-dipeptides are not clear.
51 optimal choice for AVR in each age group is not clear.
52 ctional significance of this interaction are not clear.
53 ns also function to inhibit other viruses is not clear.
54 ssociation and the amount of dairy needed is not clear.
55 with women but the underlying factor(s) are not clear.
56 FGF23 leads to hypophosphatemic rickets are not clear.
57 ism of its involvement in these processes is not clear.
58 ion and their role, if any, in cell death is not clear.
59 the protonation-driven insertion process is not clear.
60 f senescent cells within distinct tissues is not clear.
61 but the mechanisms of their interaction are not clear.
62 e in endothelium-dependent vasodilatation is not clear.
63 evant allergen and the "allergic" disease is not clear.
64 how the TLR9 pathway is activated in NASH is not clear.
65 during bone metastasis of breast cancer are not clear.
66 h effector T cells, though its role is still not clear.
67 ies commonly affecting obese individuals, is not clear.
68 diatric cancer whose exact cell of origin is not clear.
69 unity, but the mechanism by which it acts is not clear.
70 changes, and reduction in H3N2 virulence is not clear.
71 promoters (P1 and P2) active in the colon is not clear.
72 (EDC) and how PTBP1 might modulate them are not clear.
73 son for this difference in susceptibility is not clear.
74 but their roles in 60S biogenesis are still not clear.
75 the in vivo relevance of such differences is not clear.
76 nuclear export of HDV ribonucleoprotein are not clear.
77 ogenic role of T cells in lupus nephritis is not clear.
78 perthermia induces DNA damage sensitivity is not clear.
79 but whether a causal relationship exists is not clear.
80 vascular and neuropsychiatric morbidity, are not clear.
81 w this is achieved at the molecular level is not clear.
82 10 in intestinal epithelial tumorigenesis is not clear.
83 rhinovirus influences the immune response is not clear.
84 individual subunits play in this process is not clear.
85 blings of patients with advanced adenomas is not clear.
86 ges in maternal weight affect these risks is not clear.
87 ts of these redundant network structures are not clear.
88 However, the mechanisms are still not clear.
89 , the connection between FXR1 and disease is not clear.
90 e in coinfection with respiratory viruses is not clear.
91 nching patterns are precisely established is not clear.
92 mogenic, yet their underlying mechanisms are not clear.
93 he interplay between these two mechanisms is not clear.
94 malaria molecular interactions in nature are not clear.
95 s in net loss of hydride, via steps that are not clear.
96 progression, and end-stage renal disease is not clear.
97 ructural rationale for these observations is not clear.
98 tion following ischemic insult are presently not clear.
99 lization is linked to polarity disruption is not clear.
100 The reasons for this are not clear.
101 and function of these distinct processes are not clear.
102 cana, hereafter mexicana) to modern maize is not clear.
103 arities in survival of children with ESRD is not clear.
104 to compulsive drug taking and abstinence is not clear.
105 his promotes or suppresses translocations is not clear.
106 their relation to the cGAS-STING pathway is not clear.
107 ical significance of this interplay is still not clear.
108 h these mutations promote disease in vivo is not clear.
109 omplex social behaviors more specifically is not clear.
110 er, the effects of p53 on LMP1 expression is not clear.
111 and time of day impact school performance is not clear.
112 n with Clav (referred as Augmentin [AG]) are not clear.
113 sm on supporting RCC carcinogenesis is still not clear.
114 The role of mutations in GP and NP is not clear.
115 animals and type 1 diabetes in childhood is not clear.
116 these changes play a causative role in AD is not clear.
117 ns acutely under physiological conditions is not clear.
118 of accuracy of the FIT in this population is not clear.
119 ation may impact alcohol intake in humans is not clear.
120 s in the context of embryonic development is not clear.
121 significance of INS-membrane interaction is not clear.
122 t the information to the apical membrane are not clear.
123 chanism by which disease is induced is still not clear.
124 intestines under physiological conditions is not clear.
125 evel of accuracy of the FIT in population is not clear.
126 but the chemical state of these surfaces is not clear.
127 as targets for therapeutic intervention are not clear.
128 are predicted in the other genome, and it is not clear a priori from sequence comparison and similari
129 influences AIDS progression, but it is still not clear about the exact role of pC/EBPbeta and the det
132 hrough which FAK functions in the complex is not clear, and in this study we examined the role of thi
134 their exact roles during invasive growth are not clear because pmk1 and mps1 mutants are defective in
135 ptic mechanisms underlying these effects are not clear, because the postsynaptic responses of differe
138 ion is ubiquitous in most populations; it is not cleared by the host after primary infection but pers
139 ea-cycle enzymes, while approximately 30% is not cleared by the liver, independent of portal ammonia
140 3 and/or calcium in hair follicle cycling is not clear despite their impact on keratinocyte different
141 mRNA and protein from the disease allele is not clear due to the lack of sensitive allele-specific q
142 ivity in U.S. will increase in the future is not clear, due to the large uncertainty in dust modeling
143 isual information from their environments is not clear, even as the use of mice for studying visually
144 ation of aducanumab in 22-month-old mice did not clear existing plaques, calcium overload was amelior
151 ablished in high-income countries, but it is not clear how behavioural risk factors are distributed w
157 nformation on the system's components, it is not clear how chemoreceptor clusters are reliably target
158 (AD) and Down syndrome (DS); however, it is not clear how dysfunction in this pathway influences the
160 commonly prescribed for children, but it is not clear how exposure to antibiotics early in life affe
163 n occurs during diabetes development, it was not clear how human islets of T2D patients would respond
166 nesis of Alzheimer's disease; however, it is not clear how individual amyloid-beta species accumulate
168 to be important to maintain cohesion, it is not clear how many neighbors each individual can simulta
170 la larvae against wasp infection, but it was not clear how muscles could affect the immune response.
171 netic cause has long been known, it is still not clear how mutations at this locus produce this array
175 F-beta functions in hepatic cells, but it is not clear how STATs are activated under these circumstan
182 themselves are inherently active, and it is not clear how the active, energy-consuming processes tha
188 which encodes a Notch ligand, although it is not clear how these contribute to disease development.
189 dback from antennal mechanosensors but it is not clear how these inputs combine to elicit flight-rela
190 iron-regulatory proteins in fungi, but it is not clear how these proteins function in mammalian syste
191 nt target for neutralizing antibodies, it is not clear how these viruses avoid cross-neutralization.
193 es the stable flipped-out complex, but it is not clear how this complex differs from nonspecific comp
210 ations are superior in SSI prevention, it is not clear if different alcohol-based preparations are eq
212 roughout T cell development; however, it was not clear if Tce1 is required for Gata3 transcription in
214 tors bind transiently to chromatin but it is not clear if the duration of these binding interactions
217 migrate in response to the 3D matrix, it is not clear if they can switch between high- and low-press
218 e excited in a semiconductor, it is a priori not clear if they form a plasma of unbound fermionic par
221 duces the risk of several cancers, but it is not clear if use of these drugs is associated with risk
225 e existence of PRC1 variants; however, it is not clear in how many processes these components partici
228 hese infections reflects that they often are not cleared properly by the immune response or rather is
229 of mutations constrains evolution, but it is not clear that constraints exist, and, even if they do,
231 onditions is impacted by calcium addition is not clear, the formation of more photolabile weakly comp
235 eir relationship to other early Eurasians is not clear, we generated genome-wide data from a 40,000-y
236 n blood pressure and endothelial function is not clear.We sought to determine the effects of adhering
240 that apoE4 enhances amyloid pathology, it is not clear what the critical stage(s) is during amyloid d
241 hile GluA1 is important for cognition, it is not clear what the role of GluA1 is in hedonic responses
243 al fluids has not been determined, and it is not clear whether a Widom line can be identified for bin
246 rol content in the lipid bilayer, but it was not clear whether changes in permeability were due to di
248 a quantitative global synthesis, it is still not clear whether drought-induced mortality rates differ
249 hough ECRs regulate receptor function, it is not clear whether ECRs play a direct regulatory role in
251 ssociated with ovarian carcinomas, but it is not clear whether it is a cause or a consequence of the
252 as far as we know, on our planet, but it is not clear whether it must hold throughout the cosmos.
253 mary gland development during puberty, it is not clear whether its overexpression alters normal mamma
254 the pathogenesis of human leprosy, yet it is not clear whether Mycobacterium leprae, which has a dist
265 nd thereby induce cell death; however, it is not clear whether synthetic ion transporters can also be
269 embles in each daughter cell; however, it is not clear whether the disassembly process per se is impo
270 ween NAFLD in children and adults, and it is not clear whether the disease observed in children is th
272 o self-generated sounds is well known, it is not clear whether the learned action-sound association m
273 proteins from infected to naive cells, it is not clear whether the viral genome is also transferred v
274 s vary over the course of the day, but it is not clear whether these changes affect the activity of t
277 ely related organisms or lineages, but it is not clear whether they are maintained by selection or ne
278 4A3-5 variants were found as well, but it is not clear whether they should be considered isolated FSG
280 e in many bird and mammal species, but it is not clear whether this correlation evolved convergently
281 ve regulators of memory formation, but it is not clear whether this function depends on HDAC enzymati
283 a mechanism of kin discrimination, but it is not clear whether this killing is sufficient to explain
288 blation also impaired metabolism, but it was not clear whether this was a cause or consequence of hea
291 ty in breast cancer and lymphomas, but it is not clear which additional Rb-positive cancers might ben
294 as been implicated in social behavior, it is not clear which neurons are relevant or how they contrib
299 sm of Cu-Cy nanoparticles on cancer cells is not clear yet and Cu-Cy nanoparticles as novel radiosens
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