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1 Not surprising, there is a plethora of hereditary conditions
4 ing the unique enzootic life cycle of B. burgdorferi, it is not surprising that a large proportion of its genome is compo
6 undamental role in many essential cellular functions, it is not surprising that autophagic dysfunction is associated with
8 ic alternations associated with orofacial clefts; so, it is not surprising that CL/P is among the most common of all birt
16 microbiota in regulating host immunity to pathogens, it is not surprising that microbiota are also capable of regulating
17 in the regulation of molecular networks in the brain, it is not surprising that miRNA dysregulation causes neurodegenerat
19 Given the central metabolic function of mitochondria, it is not surprising that mitochondrial dynamics and bioenergetics
21 scope of LRP1 ligands and its multifunctional nature, it is not surprising that numerous biological activities have been
22 lationship between Mtb and the human immune response, it is not surprising that our ability to vaccinate against tubercul
25 nature of cardiac differentiation and morphogenesis, it is not surprising that some form of congenital heart disease is
27 biquitination in the regulation of the immune system, it is not surprising that the ubiquitination network is a common ta
28 d in this study showed a high correlation; therefore, it is not surprising that there was no significant difference to pr
29 heir ancient origin and high structural conservation, it is not surprising that tRNAs have been hijacked during evolution
33 every cellular gene-regulatory pathway, and it therefore is not surprising that viruses have found ways to subvert this p
35 as toxic or nutritionally harmful to human health, which is not surprising since the seeds are not known to produce toxic
36 -cadherin is normally involved in cell-cell adhesion, so it not surprising that individuals with this syndrome are predis
40 Against this backdrop, it is perhaps not surprising that computer science, the science of building
49 he packaging of glycoprotein E (gE) was observed, which was not surprising, since it has been reported that UL16 interact
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