ライフサイエンスコーパス: nothing

1 bles with constant outcome values (0.5 ml or nothing).

2 g the Nash equilibrium action, namely giving nothing).

3 onder rejects the proposal, both players get nothing.

4 for 90 d, whereas control subjects received nothing.

5 The control group received nothing.

6 rable even when in vivo examination detected nothing.

7 sus the negative control PEU films releasing nothing.

8 ects swallow up whatever comes near and emit nothing.

9 ter than those who were punished or received nothing.

10 vy financial burden while arguably achieving nothing.

11 in which starvation corresponded to winning nothing.

12 atory reverberations create something out of nothing: A third flash where there are only two.SIGNIFIC

13 to indicate only which region is nearer, but nothing about how much the regions are separated in dept

14 Understanding its workings, to say nothing about intervening in them, is among the hardest

15 I knew nothing and had thought nothing about parasites until 1971.

16 A few decades ago, we knew next to nothing about the behavior of our closest animal relativ

17 However, we know nothing about the endogenous control of hemocyte develop

18 We assume nothing about the Markov chain beyond reversibility and

19 anging behavioral states, yet we know almost nothing about the mechanisms of such regulation.

20 We know almost nothing about the N form preferentially transferred to t

21 thenticity of submitted items while learning nothing about them.

22 al the onset of a light step with 1-3 all-or-nothing action potentials that attain a peak amplitude o

23 I knew nothing and had thought nothing about parasites until 19

24 A fourth option, a 'do-nothing' approach, is wholly inconsistent with the well-

25 t steering can be detected by the all-versus-nothing argument experimentally even in the presence of

26 -Podolsky-Rosen steering based on all-versus-nothing argument, offering a strong condition to witness

27 1), popular fiction (experiments 2 to 5), or nothing at all (experiments 2 and 5).

28 Multivariate analysis revealed that all-or-nothing behaviour was the key predictor for the onset of

29 ions, stress, anxiety, depression and all-or-nothing behaviour were associated with the risk of PCS.

30 o create mirrors for space telescopes, using nothing but microscopic particles held in place by light

31 Native assemblies are obtained using nothing but their simulated profiles as restraints and e

32 Thus, nothing can be affirmed a priori concerning the binding

33 One in three survivors cited "nothing can be done" as a reason for not discussing dail

34 Nothing can compare with the eureka moments when, togeth

35 energy recovery scenarios compared to a "do nothing" case.

36 ic theories that allow everything but demand nothing, cases where seeing and thinking conflict, menta

37 binding triggers a highly localized, all-or-nothing change of water accessibility to the transport s

38 eceive cash payments (intervention group) or nothing (control group).

39 ene guests was observed to effect the all-or-nothing cooperative templation of an S6-symmetric host s

40 ered at FLC in individual cells in an all-or-nothing (digital) manner or is continuously varying (ana

41 n, such that the group identifies itself and nothing else in a search of GenBank.

42 When nothing else is known, the base rate principle favors Jo

43 to prefer to be doing something rather than nothing, even if that something is negative.

44 llowing CRAC channel activation is an all-or-nothing event over a range of stimulus intensities.

45 h, we demonstrate that MOMP is not an all-or-nothing event.

46 bit of information to coordinate the all-or-nothing expression of early genes, but also over a longe

47 ure, cells respond autonomously in an all-or-nothing fashion, with the fraction of cells that stably

48 he complex at the cost of its normal, all-or-nothing functionality.IMPORTANCECaudovirales encode cell

49 However, nothing further is known about the activation process.

50 ss from their assets or opt for a "double-or-nothing" gamble that would either avoid or double it.

51 sands of genes of which we understand almost nothing: genomics has failed biology.

52 e neural and muscular systems, and virtually nothing has been reported about their embryogenesis.

53 Almost nothing has been reported on longer term prognosis follo

54 Nothing, however, is known about the molecular identity

55 If vaccine action is all-or-nothing (ie, a proportion of vaccine recipients receive

56 Disagreement primarily occurred in the "nothing in bed" versus "in-bed activity" categories beca

57 ty scale was collapsed into four categories: nothing in bed, in-bed activity, out-of-bed activity, an

58 partner could not reciprocate, and expected nothing in return.

59 nts might argue that we are advocating doing nothing instead, but we are not; we are only advocating

60 nion transporters has flourished from almost nothing into a rapidly maturing field of research.

61 Nothing is currently known about interactions between th

62 Almost nothing is currently known about the maturation of funct

63 e their known wide distribution in primates, nothing is currently known about the occurrence, frequen

64 e cell types including conventional T cells, nothing is currently known concerning Notch function in

65 plained by cold winters or hot summers, then nothing is explained.

66 own for its role in olfaction, but virtually nothing is known about a clade of approximately 35 membe

67 However, nothing is known about a potential functional interactio

68 gulate CIITA in B cells have been described, nothing is known about additional distal elements that m

69 Meanwhile, almost nothing is known about App's function in glia, where it

70 of established asthma in schoolchildren, but nothing is known about ASM in preschool wheezers.

71 on by bacteria is well described, but almost nothing is known about bacterial iron export even though

72 are our closest living relatives but almost nothing is known about bonobo internal anatomy.

73 nhibitors are in clinical trials, yet almost nothing is known about Brd4 function in the brain.

74 hat adults are broadly exposed to OPFRs, but nothing is known about children's exposure.

75 However, almost nothing is known about enediyne core biosynthesis, and t

76 However, nothing is known about epigenetic regulation of this Ins

77 However, almost nothing is known about events leading to this islet auto

78 However, little or nothing is known about expression in moderately-sized (1

79 Nothing is known about fetal-placental signaling in resp

80 However, nothing is known about FucP conformational dynamics.

81 ted traffic to and from the plasma membrane, nothing is known about GORK, its distribution and traffi

82 s do not transfer to other tasks, and almost nothing is known about how activation in IC-related brai

83 Yet little to nothing is known about how coacervates evolve into intri

84 s of DISC1-binding proteins reported, almost nothing is known about how DISC1 interacts with other pr

85 e Gi and abrogate PI3K-Akt signals; however, nothing is known about how GIV's GEF function is regulat

86 ied camphor elicits a sensation of cool, but nothing is known about how it affects cold temperature s

87 ories on aging on the cellular scale, nearly nothing is known about how microscopic failures cascade

88 Virtually nothing is known about how phages impact the establishme

89 ons as a modulator of inflammation; however, nothing is known about how Rc3h1 expression is regulated

90 s the formation of cohesion, and essentially nothing is known about how sister chromatid cohesion is

91 signaling remain incompletely understood, as nothing is known about how superficial dorsal horn neuro

92 For example, nothing is known about how these receptors bind odorants

93 Almost nothing is known about how these three-dimensional topol

94 portance of these binding regions and almost nothing is known about how they work together.

95 Golgi apparatus and the plasma membrane, but nothing is known about how this cycle is regulated.

96 a decrease in FSAP expression but, to date, nothing is known about how this might be regulated.

97 Surprisingly, nothing is known about how visually guided insects proce

98 To date, nothing is known about how wounding activates DUOX.

99 contributor to periodontal bone loss, almost nothing is known about immune responses to this organism

100 However, virtually nothing is known about intracellular signaling proteins

101 However, nothing is known about its effects on infarction after P

102 cell wall alpha(1-3)glucan is essential, but nothing is known about its localization and function in

103 sent in all examined vertebrate genomes, but nothing is known about its molecular or cellular functio

104 lso used for wine and cider fermentation but nothing is known about its natural history.

105 muscle cell contraction in several tissues, nothing is known about its possible role in tracheal smo

106 eceptor CXCR1 shares ligands with CXCR2, yet nothing is known about its potential role in liver patho

107 in this process have been characterized, but nothing is known about its regulation.

108 However, nothing is known about its role in memory retrieval afte

109 or (HIF) in vitro and in cultured cells, but nothing is known about its roles in mammalian erythropoi

110 Almost nothing is known about Kv2 channel functions in medial e

111 al infections has only recently emerged, and nothing is known about MDSC function in the context of S

112 , 2 and 4 have been characterized, virtually nothing is known about members of cluster 3 (ACA12 and A

113 tion targets are well established, virtually nothing is known about PKG targets in LTD.

114 However, nothing is known about SAMHD1 posttranslational modifica

115 studied in extant and fossilized organisms, nothing is known about secondary cartilage in fossils.

116 However, almost nothing is known about strike performance of viperid sna

117 a number of biological functions, virtually nothing is known about structure-function relationships

118 However, nothing is known about the alteration and role of MEG3 i

119 ins are essential for this process; however, nothing is known about the arrangement of the subunits i

120 extracellular electron transfer, and almost nothing is known about the associated mechanisms.

121 Almost nothing is known about the basis for recombination partn

122 characterized to a limited extent and almost nothing is known about the behavior of these materials u

123 Curiously, nothing is known about the behavior of this cellular mac

124 Nothing is known about the binding mode, however.

125 periplasmic domain of the binding protomer, nothing is known about the binding of many other ligands

126 ence suggests that IscS and Fdx interact but nothing is known about the binding site and the role of

127 However, nothing is known about the biochemical or mechanical pro

128 nt biochemical function between GNC and GNL, nothing is known about the biological role of the four o

129 However, nothing is known about the cell surface dynamics of Kv7.

130 In contrast, almost nothing is known about the circuitry or laminar features

131 ains with short incubation periods; however, nothing is known about the conversion of the long-incuba

132 Yet nothing is known about the effects of calcineurin when i

133 Virtually nothing is known about the effects on fetal physiology o

134 However, virtually nothing is known about the epigenetic transition of a ce

135 n numerous nonerythroid cells, but virtually nothing is known about the expression and potential sign

136 Nothing is known about the expression levels of the gene

137 However, nothing is known about the factors that ensure effective

138 Almost nothing is known about the final (completion) step becau

139 Despite their importance, virtually nothing is known about the folding and transport of TRP

140 nello import studies and in silico analyses, nothing is known about the function and properties of pr

141 rane domains have been analyzed extensively, nothing is known about the function of CAR-D2 and the ex

142 e been investigated extensively, practically nothing is known about the function of NRG2, the closest

143 Essentially nothing is known about the functions of SEIPIN homologs

144 ishing reproductive mode remain unknown, and nothing is known about the genetic basis of postzygotic

145 However, almost nothing is known about the heterochromatin's structural

146 However, essentially nothing is known about the identity of these inhibitory

147 ) triggers prominent motivation deficits but nothing is known about the impact of this exposure in th

148 y to deletion in P. falciparum in vitro, but nothing is known about the in vivo functions of PAT in P

149 However, nothing is known about the influence of Jak/Stat-signali

150 o-inflammation and -degeneration, but almost nothing is known about the interactions between the peri

151 produce nitrous acid, HONO, but essentially nothing is known about the isomeric nitrosyl-O-hydroxide

152 nd ability to transmit vertically, virtually nothing is known about the life history of these endobac

153 ortant to achieving high toxicity, virtually nothing is known about the matrix that binds the toxin i

154 Almost nothing is known about the mechanism by which a magnetic

155 However, nothing is known about the mechanism of hepcidin binding

156 Virtually nothing is known about the mechanisms and enzymes respon

157 e leading causes of chronic pain, but almost nothing is known about the mechanisms and molecules that

158 Almost nothing is known about the mechanisms of dissimilatory m

159 ntially important physiological role, almost nothing is known about the mechanisms responsible for th

160 chwann cell development during this process, nothing is known about the mechanisms that mediate the m

161 Despite the importance of DGs, nothing is known about the mechanisms underlying their t

162 ealth of information about archaeal biology, nothing is known about the molecular basis of DNA segreg

163 nd heart in humans and mice; however, almost nothing is known about the molecular downstream targets

164 However, for most of these mutations, nothing is known about the molecular mechanism linking K

165 core management strategy for this pathogen, nothing is known about the nature of resistance.

166 o known as hypocretin) signaling, but almost nothing is known about the neural mechanisms through whi

167 spite these far-reaching behavioral effects, nothing is known about the neural processes impacted by

168 Unfortunately, almost nothing is known about the normal functions of this gene

169 Nothing is known about the pathways that regulate gameto

170 ssion has been observed in cancer cells, but nothing is known about the piRNA partners or the functio

171 lease contributes to the pathogenesis of AF, nothing is known about the potential role of JPH2 in atr

172 ed vaccine (LAV) to protect against measles, nothing is known about the primary cells in which the vi

173 linked to the flagellar basal body; however, nothing is known about the proteins encoding the collar

174 ngage a pseudo-ELR and an N-like loop motif, nothing is known about the regions of CXCR4 and MIF that

175 However, almost nothing is known about the regulation of the central gen

176 Virtually nothing is known about the relevance of glycolysis in pa

177 Virtually nothing is known about the role of mechanotransductive s

178 However, to date, nothing is known about the role of miRNAs during develop

179 the other post-translational modifications, nothing is known about the role of SUMO conjugation to h

180 However, nothing is known about the role of SUMOylation in regula

181 However, nothing is known about the role of the PinX1-TRF1 intera

182 Virtually nothing is known about the role of these nonproteolytic

183 However, nothing is known about the role or importance of CtpA.

184 However, nothing is known about the roles of HPATs in basal plant

185 However, nothing is known about the single T. brucei CDS gene (Tb

186 ion in other environmental compartments, and nothing is known about the stability of high-molecular-w

187 Apart from their identification, almost nothing is known about the structure and function of PRO

188 multimeric channel complex, although almost nothing is known about the structure or subunit composit

189 To our knowledge, nothing is known about the survival of plasmacytoid dend

190 ell-established clinical syndrome, virtually nothing is known about the tumor cells responsible for t

191 er for sufferers and their families, and yet nothing is known about the underlying mechanism.

192 Nothing is known about the upstream regulators of SND1.

193 icrobial functions of porcine cathelicidins, nothing is known about their ability to promote pDC acti

194 eptors have immunomodulatory properties, but nothing is known about their antiviral effects.

195 ined unprecedented popularity, but virtually nothing is known about their cardiovascular risks.

196 ave been identified in the zebrafish genome, nothing is known about their expression pattern.

197 (11-42) make up one fifth of plaque load yet nothing is known about their interaction with full-lengt

198 lity, cell trafficking/adhesion, and cancer, nothing is known about their involvement in liver pathol

199 water bodies is relatively well studied, but nothing is known about their presence in most of the com

200 However, nothing is known about their relationship.

201 are rapidly spreading in Southeast Asia, yet nothing is known about their transmission.

202 mbrane, the phagophore; however, essentially nothing is known about this process including the molecu

203 ociated factor 2 (TRAF2); however, virtually nothing is known about TRAF2 signaling in the adult mamm

204 nt arterialization of endothelial cells, yet nothing is known about what regulates COUP-TFII expressi

205 ough the RST is present postnatally in cats, nothing is known about when rubrospinal projections coul

206 However, nothing is known about whether a DNA damage response is

207 hance neuronal form and function, but almost nothing is known about whether and how it alters the bra

208 However, nothing is known about whether this equivalence extends

209 Even for sets of proteins for which nothing is known beyond unannotated sequences and struct

210 However, virtually nothing is known concerning the amino acid residues that

211 corridors impact animal movement, virtually nothing is known for species dispersed by wind, which ar

212 By contrast, almost nothing is known in plants accumulating specialized isop

213 Without an accurate classification, nothing is known of diagnosis, prognosis, or treatment b

214 sing and consuming foodstuffs, but virtually nothing is known of how early pots were used.

215 Since nothing is known of how mRNA structural elements factor

216 in secretion, and social behavior in adults, nothing is known of its role during embryonic developmen

217 gical role is not completely understood, and nothing is known of the biology of these enzymes in Arch

218 ith the exception of one inconclusive study, nothing is known of the genetic basis of this phenomenon

219 l interactions, and in particular, virtually nothing is known of the molecular mechanisms that contro

220 eral inflammatory response to sepsis, almost nothing is known of the neuronal changes that cause asso

221 However, nothing is known of the role of Mical or its redox activ

222 However, almost nothing is known of the skeleton's physiology, and myria

223 ically split across the plasma membrane, yet nothing is known of the transporters required for the tr

224 acaques on the binding of human (hu)IgG, and nothing is known of this interaction in the pig-tailed m

225 Alas, virtually nothing is known on how the gustatory thalamus (VPMpc) p

226 the molecular mechanism remains elusive and nothing is known on PIP(2) effect on other Kv such as th

227 ctors of hypersensitivity to betalactam, but nothing is known on the influence of NOD genes, despite

228 ell types such as atrial myocytes, virtually nothing is known regarding endosomal transport.

229 However, nothing is known regarding the mechanism of langerin ass

230 th human cardiovascular disease, essentially nothing is known regarding the molecular identity or fun

231 Currently, nothing is known regarding the structural basis or molec

232 ated with deleterious clinical outcomes, but nothing is known to date about tissue protein carbamylat

233 question in cell biology about which almost nothing is known.

234 he cellular function of C9ORF72, about which nothing is known.

235 ry nonverbal information, about which almost nothing is known.

236 Nothing is more intuitive, yet more complex, than the co

237 However, virtually nothing is presently known about the post-translational

238 t important sources of human arsenic intake, nothing is published about uptake, toxicity, or toleranc

239 Hence, nothing is yet known of how amygdala subregional network

240 ions of TNF mediated by engagement of TNFR1, nothing is yet known of their role in CD40-mediated even

241 Additionally, there is essentially nothing known about the genetic relationships between po

242 this circuit to decision making, and almost nothing known about the whether any contribution is via

243 Currently, there is nothing known about transcriptional regulation of the SL

244 As a result, Mars's growth is stunted, and nothing large in the asteroid belt can accumulate.

245 explain the experimentally observed 'all-or-nothing' LAX3 spatial expression pattern in cortical cel

246 ticated catalytic centre shows that there is nothing magical about the catalytic activities or mechan

247 eceptors are thought to operate in an all-or-nothing manner, existing in an immunologically active or

248 l displacement threshold governing an all-or-nothing mechanically-induced calcium response.

249 RP promoters to provide more than an all-or-nothing mode of transcriptional regulation.

250 vital in animals that reproduce in an all-or-nothing mode, such as bristle worms: females committed t

251 The majority of cases require nothing more than a combination of symptomatic care and

252 Sign was once viewed as nothing more than a system of pictorial gestures without

253 h vigor and imagination, our pinnacle may be nothing more than an inflection point leading to decline

254 appears premature; populations may entertain nothing more than shared adaptiveness.

255 a row), what's at work is random variation, nothing more.

256 rimination that are characteristic of all-or-nothing Na(+) spikes.

257 Here, we show that this all-or-nothing neuronal differentiation results from Hippo path

258 t has been done will be done again; there is nothing new under the sun.

259 To date however, we know nothing of a role for Ketamine in modulating both struct

260 The Netherlands, against the base case, "do-nothing" option.

261 Nothing or little is known about how eukaryotic mariner

262 r's contention that a belief in free will is nothing other than a continuing belief in vitalism--some

263 alue of virus-based vectors is that they add nothing permanently to the environment.

264 6:1 at the branches, we observed the "all-or-nothing" phenomenon with plasma only entering the low fl

265 ctive and matures through a symmetric all-or-nothing process.

266 In this work, we show that the all-versus-nothing proof of Einstein-Podolsky-Rosen steering can be

267 nt (CABG surgery, PCI, medical treatment, or nothing) recommended by the catheterization laboratory c

268 Although initially considered an all-or-nothing reflex [1], numerous studies on freely diving ma

269 en identified in diverse organisms; however, nothing regarding their physiology in the context of cel

270 on of fliT or fliD did not lead to an all-or-nothing response in FlhD(4) C(2) activity.

271 reater control of the emergence of an all-or-nothing response of a cell.

272 izable lactose analogues generates an all-or-nothing response, where some cells express the lac genes

273 ., nitazoxanide consistently shows an all-or-nothing response.

274 rse responses, ranging from graded to all-or-nothing responses and combinations thereof.

275 ical control of the source of pathology, and nothing should be allowed to delay transfer to the opera

276 its the important neural functions of all-or-nothing spiking with signal gain and diverse periodic sp

277 it comes to microtubules, kinetochores leave nothing to chance.

278 6 to 15 minutes in a room by themselves with nothing to do but think, that they enjoyed doing mundane

279 ction, irrelevant events that government has nothing to do with and for which no government response

280 hould be an alternative explanation that has nothing to do with genetic continuity, but stresses the

281 and one-off ivory sales in 1999 and 2008 did nothing to halt elephant poaching.

282 ng DSEK to as many patients as possible (and nothing to others), rather than providing PK.

283 body to the 2F5 ELDKWA epitope but little or nothing to the 4E10 determinant.

284 e loading of a cofactor represents an all-or-nothing transition in regard to the enzymatic function a

285 ation are often assumed to involve an all-or-nothing two-state dissociation pathway, but deviations f

286 This all-or-nothing two-state solution is a hallmark of positive coo

287 However, virtually nothing was known about how the pathway was regulated.

288 Almost nothing was known about it.

289 ssion of CTnDOT transfer; however, virtually nothing was known about RteR.

290 However, nothing was known about the difference between these two

291 However, nothing was known about the mechanism by which PVR and D

292 Until relatively recently, almost nothing was known about the molecular pathogenesis of ei

293 However, until now nothing was known about the subsequent step in Fap1 glyc

294 Prior to this work, very little to nothing was known about these mechanisms at the genomic

295 bon, nitrogen and phosphorous, but virtually nothing was previously known about how A. tumefaciens ac

296 nventional pathway enrichment tests detected nothing, while our approach discovered biological proces

297 ies are measured, and reported on regularly, nothing will be done explicitly to reduce them.

298 , which delivered either double the wager or nothing with 50:50 odds.

299 To do nothing would allow thousands of pregnant women to expos

300 have it, not much else matters, and there is nothing you can do about it.