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1 own for its role in olfaction, but virtually nothing is known about a clade of approximately 35 membe
3 gulate CIITA in B cells have been described, nothing is known about additional distal elements that m
6 on by bacteria is well described, but almost nothing is known about bacterial iron export even though
8 the life cycle of plants is well documented, nothing is known about boron homeostasis and function in
17 euronal gene RNAi in Caenorhabditis elegans, nothing is known about flp function in nematode parasite
19 ted traffic to and from the plasma membrane, nothing is known about GORK, its distribution and traffi
21 the presenilin proteins therein, but almost nothing is known about how Abeta is degraded and cleared
22 s do not transfer to other tasks, and almost nothing is known about how activation in IC-related brai
23 been partially elucidated in a higher plant, nothing is known about how algae make DMSP except that t
25 s of DISC1-binding proteins reported, almost nothing is known about how DISC1 interacts with other pr
26 o the membrane cytoskeleton, but essentially nothing is known about how Drosophila glutamate receptor
29 e Gi and abrogate PI3K-Akt signals; however, nothing is known about how GIV's GEF function is regulat
30 ied camphor elicits a sensation of cool, but nothing is known about how it affects cold temperature s
32 ns as a Ras-GTPase activating protein (GAP), nothing is known about how it is normally regulated or i
35 endocrine representation of day length, but nothing is known about how long it takes to establish ph
36 ories on aging on the cellular scale, nearly nothing is known about how microscopic failures cascade
40 o have occurred over millennia or centuries, nothing is known about how quickly reproductive isolatio
41 ons as a modulator of inflammation; however, nothing is known about how Rc3h1 expression is regulated
42 ing taxa that are deeply diverged, virtually nothing is known about how regeneration loss occurs.
43 ells receive afferent terminal contacts, yet nothing is known about how reinnervation progresses or w
45 s the formation of cohesion, and essentially nothing is known about how sister chromatid cohesion is
46 signaling remain incompletely understood, as nothing is known about how superficial dorsal horn neuro
47 th sexes throughout most of development, but nothing is known about how SxlPm is expressed during the
48 f fiber and solid wood products, essentially nothing is known about how the biological processes lead
50 edress against discrimination, yet virtually nothing is known about how the intended beneficiaries ha
54 ans-Golgi network (TGN) in living cells, but nothing is known about how these vesicles identify and d
56 esis has been studied extensively, virtually nothing is known about how this axis can be established
61 trast to the well-studied Min system, almost nothing is known about how Z ring assembly is blocked in
62 contributor to periodontal bone loss, almost nothing is known about immune responses to this organism
64 achieves its quaternary structure in the ER, nothing is known about its acquisition of tertiary struc
65 a molecule has been lacking and essentially nothing is known about its basic biochemical properties.
66 that Bordetella can form biofilms in vitro, nothing is known about its biofilm mode of existence in
69 cell wall alpha(1-3)glucan is essential, but nothing is known about its localization and function in
70 sent in all examined vertebrate genomes, but nothing is known about its molecular or cellular functio
73 muscle cell contraction in several tissues, nothing is known about its possible role in tracheal smo
74 eceptor CXCR1 shares ligands with CXCR2, yet nothing is known about its potential role in liver patho
78 or (HIF) in vitro and in cultured cells, but nothing is known about its roles in mammalian erythropoi
81 al infections has only recently emerged, and nothing is known about MDSC function in the context of S
83 , 2 and 4 have been characterized, virtually nothing is known about members of cluster 3 (ACA12 and A
86 ve only partially been defined, while almost nothing is known about nutritional requirements of other
87 er, this hypothesis has not been tested, and nothing is known about other mechanisms that control sub
91 G on DNA polymerases have been well studied, nothing is known about possible effects of this lesion o
93 studied in extant and fossilized organisms, nothing is known about secondary cartilage in fossils.
95 ted Smad phosphorylation is well understood, nothing is known about Smad phosphatases that turn off S
97 a number of biological functions, virtually nothing is known about structure-function relationships
101 ins are essential for this process; however, nothing is known about the arrangement of the subunits i
104 characterized to a limited extent and almost nothing is known about the behavior of these materials u
107 periplasmic domain of the binding protomer, nothing is known about the binding of many other ligands
108 ence suggests that IscS and Fdx interact but nothing is known about the binding site and the role of
111 ts in double-strand break repair, but almost nothing is known about the biochemical role of Rad52 pro
112 t to the recent studies on GDF-9 and BMP-15, nothing is known about the biological function of BMP-6
114 nt biochemical function between GNC and GNL, nothing is known about the biological role of the four o
116 kout of Nrf1 is lethal in mouse embryos, but nothing is known about the cell types that absolutely re
120 ains with short incubation periods; however, nothing is known about the conversion of the long-incuba
123 as been the focus of most studies, virtually nothing is known about the effect of G protein-coupled r
124 ic map refinement in mammals is limited, and nothing is known about the effect of spontaneous retinal
130 enases has only recently been described, and nothing is known about the evolutionary origin of the en
131 n numerous nonerythroid cells, but virtually nothing is known about the expression and potential sign
135 well understood in the insect retina, almost nothing is known about the extraretinal blue light photo
139 nello import studies and in silico analyses, nothing is known about the function and properties of pr
141 rane domains have been analyzed extensively, nothing is known about the function of CAR-D2 and the ex
143 e been investigated extensively, practically nothing is known about the function of NRG2, the closest
144 e was especially interesting, because almost nothing is known about the function of OC factors in int
146 l ribosomal proteins L16 and L33, but almost nothing is known about the function or enzymology of thi
149 ishing reproductive mode remain unknown, and nothing is known about the genetic basis of postzygotic
152 ) triggers prominent motivation deficits but nothing is known about the impact of this exposure in th
153 the recent studies on Ron in breast cancer, nothing is known about the importance of this protein du
154 y to deletion in P. falciparum in vitro, but nothing is known about the in vivo functions of PAT in P
157 o-inflammation and -degeneration, but almost nothing is known about the interactions between the peri
158 produce nitrous acid, HONO, but essentially nothing is known about the isomeric nitrosyl-O-hydroxide
160 nd ability to transmit vertically, virtually nothing is known about the life history of these endobac
161 ortant to achieving high toxicity, virtually nothing is known about the matrix that binds the toxin i
165 a sufficient supply of RF to the retina, but nothing is known about the mechanism of the vitamin upta
167 o the highly metabolically active retina but nothing is known about the mechanism, regulation or biol
169 e leading causes of chronic pain, but almost nothing is known about the mechanisms and molecules that
171 ith a variety of disease states, practically nothing is known about the mechanisms employed by this b
173 by helping recruit core binding factors, but nothing is known about the mechanisms of transcription a
174 rofound effects on neuronal function, almost nothing is known about the mechanisms responsible for co
175 ntially important physiological role, almost nothing is known about the mechanisms responsible for th
177 chwann cell development during this process, nothing is known about the mechanisms that mediate the m
181 be due to changes in gene regulation, almost nothing is known about the modes of natural selection ac
182 cardiogenic mesoderm in this process, almost nothing is known about the molecular aspects of avian li
184 ealth of information about archaeal biology, nothing is known about the molecular basis of DNA segreg
185 nd heart in humans and mice; however, almost nothing is known about the molecular downstream targets
186 e been localised to kinetochores, yet almost nothing is known about the molecular events which take p
190 es in the CA1 region of the hippocampus, but nothing is known about the nature of thalamic influence
193 located within the capsid domain of Gag, but nothing is known about the necessary CyPA residues.
194 tance and pervasiveness of marketing, almost nothing is known about the neural mechanisms through whi
195 o known as hypocretin) signaling, but almost nothing is known about the neural mechanisms through whi
196 spite these far-reaching behavioral effects, nothing is known about the neural processes impacted by
197 quences for our thoughts and actions, almost nothing is known about the neuronal substrates governing
199 form oligomeric rings termed sliding clamps, nothing is known about the oligomeric state of BMRF1 or
201 ssion has been observed in cancer cells, but nothing is known about the piRNA partners or the functio
202 r (PM) can produce inflammatory effects, but nothing is known about the possible proliferative and ca
203 lease contributes to the pathogenesis of AF, nothing is known about the potential role of JPH2 in atr
204 ed vaccine (LAV) to protect against measles, nothing is known about the primary cells in which the vi
206 highly conserved recombination signals, but nothing is known about the protein-DNA complexes that ex
207 linked to the flagellar basal body; however, nothing is known about the proteins encoding the collar
209 proteasome by a p97-based complex, currently nothing is known about the proteins that initially selec
212 gonadotropin release in prepubertal animals, nothing is known about the receptor subunits that may be
213 ponse against these organisms, and virtually nothing is known about the receptors involved in host re
214 interaction is well characterized; however, nothing is known about the region of FtsA involved in th
215 ngage a pseudo-ELR and an N-like loop motif, nothing is known about the regions of CXCR4 and MIF that
216 ectron donor to the MoFe protein, but almost nothing is known about the regions of the protein that a
217 multiple signaling molecules, but virtually nothing is known about the regulation of FAK phosphoryla
219 lagen-induced platelet activation, virtually nothing is known about the regulatory elements that modu
221 ase has not been reconstituted in vitro, and nothing is known about the requirements for this reactio
227 the other post-translational modifications, nothing is known about the role of SUMO conjugation to h
235 well conserved among clinical isolates, but nothing is known about the sequence variability of the g
237 ell characterized in brain and other organs, nothing is known about the signal transduction pathway o
241 n displaying marked oscillations, and almost nothing is known about the specific features of these Ca
242 ion in other environmental compartments, and nothing is known about the stability of high-molecular-w
246 Apart from their identification, almost nothing is known about the structure and function of PRO
248 multimeric channel complex, although almost nothing is known about the structure or subunit composit
251 described to infect mature oligodendrocytes, nothing is known about the susceptibility of glial precu
252 hway of primary visual cortex, but virtually nothing is known about the synaptic influence of single
255 of AC133 to the field of stem cell biology, nothing is known about the transcriptional regulation of
257 ell-established clinical syndrome, virtually nothing is known about the tumor cells responsible for t
260 icrobial functions of porcine cathelicidins, nothing is known about their ability to promote pDC acti
264 RPTPs are important in axon pathfinding, but nothing is known about their function on a cellular leve
265 (11-42) make up one fifth of plaque load yet nothing is known about their interaction with full-lengt
266 lity, cell trafficking/adhesion, and cancer, nothing is known about their involvement in liver pathol
267 lutionary success of the elements, virtually nothing is known about their mode of regulation within g
268 water bodies is relatively well studied, but nothing is known about their presence in most of the com
270 ife these factors exert their influence, and nothing is known about their specificity to aggressive b
271 GEFs have also been described, but little or nothing is known about their structure, mechanism of act
273 within the echinoderm + hemichordate clade, nothing is known about these mechanisms in several other
274 e SCN of nocturnal laboratory rats, however, nothing is known about these projections in diurnal rode
275 mbrane, the phagophore; however, essentially nothing is known about this process including the molecu
276 ociated factor 2 (TRAF2); however, virtually nothing is known about TRAF2 signaling in the adult mamm
278 retina and is active during sleep; however, nothing is known about VLPO function in day-active (diur
279 a variety of species, including humans, but nothing is known about what dance reveals about the unde
280 nt arterialization of endothelial cells, yet nothing is known about what regulates COUP-TFII expressi
281 ough the RST is present postnatally in cats, nothing is known about when rubrospinal projections coul
283 hance neuronal form and function, but almost nothing is known about whether and how it alters the bra
284 eat deal is known about vir gene activation, nothing is known about whether or how the vir gene regul
286 PMT structures have not yet been solved, and nothing is known about which amino acids in these enzyme
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