ライフサイエンスコーパス: novel object recognition

1 memory was assessed by Morris water maze and novel object recognition.

2 lidation in contextual fear conditioning and novel object recognition.

3 vivo efficacy in auditory sensory gating and novel object recognition.

4 sufficient to produce subsequent deficits in novel object recognition.

5 t memory enhancing effects in a rat model of novel object recognition.

6 vivo model to assess cognitive performance, novel object recognition.

7 om WT in swimming ability, cued learning and novel object recognition.

8 nse to whisker deprivation, impaired texture novel object recognition and altered social behavior.

9 icacy in rodent behavioral cognition models (novel object recognition and auditory sensory gating).

10 ered faster than memory formation, impacting novel object recognition and cued fear conditioning but

11 lesion reduced long-term but not short-term novel object recognition and decreased long-term potenti

12 BI) induced lasting cognitive impairments in novel object recognition and less severe deficits in Y-m

13 prevented the ability of SKF81297 to rescue novel object recognition and long-term potentiation.

14 long-term potentiation, superior memory for novel object recognition and more persistent remote cont

15 adiation, cognitive function was assessed by novel object recognition and Morris water maze.

16 learning as shown by improved performance in novel object recognition and Morris water maze.

17 lpha (PPT) or ERbeta (DPN) agonists enhanced novel object recognition and object placement memory in

18 n hippocampal behavioral assays, it prevents novel object recognition and placement without affecting

19 Impaired novel object recognition and rotarod performance were co

20 In contrast, castration impaired novel object recognition and spatial memory retention in

21 Short-term memory of mice was assessed by novel object recognition and Y-maze tests.

22 recognition and spatial memory, measured by novel object recognition and Y-maze tests.

23 rols, PV-M1 knockout mice exhibited impaired novel object recognition and, to a lesser extent, impair

24 evaluated by Y-maze spontaneous alternation, novel object recognition, and Barnes maze spatial memory

25 sts: Light Dark Latency, Elevated Plus Maze, Novel Object Recognition, and Barnes Maze.

26 luding social interaction deficits, impaired novel object recognition, and behavioral inflexibility.

27 Ts65Dn mice in the novel place recognition, novel object recognition, and contextual fear conditioni

28 ent working memory in the Morris water maze, novel object recognition, and contextual fear-conditioni

29 its in radial-arm water maze performance and novel object recognition as early as 8 months, outcrosse

30 ovement in cognitive abilities, as seen with novel object recognition as well as spatial learning and

31 rofile with procognitive efficacy in the rat novel object recognition assay.

32 term effects were assessed by Open-field and Novel-Object-Recognition at P30 and P120.

33 8 weeks using zero maze, locomotor activity, novel object recognition, cued, hidden and reduced Morri

34 tial tasks dependent on hippocampus (Y-maze, novel object recognition, dual solution cross-maze) and

35 Multiple short training trials also rescued novel object recognition in Fmr1 KOs.

36 studies of contextual fear conditioning and novel object recognition in I-2 heterozygous mice sugges

37 sion of DPFE into perirhinal cortex restored novel object recognition in long-access meth rats.

38 e applied, including tasks to assess memory (novel object recognition in open field and V-maze paradi

39 ssary for 17beta-estradiol (E(2)) to enhance novel object recognition in young ovariectomized mice.

40 However, both lesions left novel object recognition intact.

41 se inhibition, contextual fear conditioning, novel object recognition, locomotor, and social choice p

42 lted in normal adult synaptic plasticity and novel object recognition memory in mice exposed to ethan

43 se inhibition, improved social behavior, and novel object recognition memory in NMDA receptor hypofun

44 In the present study, rats were given a novel object recognition memory task in which initial en

45 on, it induced cognitive improvements in the novel object recognition memory test in NR1-KD animals,

46 tion in the open field, restore PPI, improve novel object recognition memory, partially normalize soc

47 At three doses tested in the mouse novel object recognition model (1, 3, and 10 mg/kg s.c.)

48 havioral tests including locomotor activity, novel object recognition, Morris water maze (cued, hidde

49 C57BL/6J mice were tested using the novel object recognition (NOR) assay at various time poi

50 yclidine (PCP), induces enduring deficits in novel object recognition (NOR) in rodents.

51 d access meth self-administration results in novel object recognition (NOR) memory deficits in rats.

52 n reversing the effect of scopolamine in the novel object recognition (NOR) paradigm with a minimum e

53 enting sleep after the learning phase of the novel object recognition (NOR) task significantly decrea

54 se hippocampus at distinct stages during the novel object recognition (NOR) task: during object memor

55 Memory function was assessed using the novel object recognition (NOR) test, administered 3 days

56 n (SND), which relies on olfactory cues, and novel object recognition (NOR), a visual-recognition tas

57 oligomer-dose-dependent anxiety and impaired novel object recognition (NOR).

58 e acetyltransferase, in long-term memory for novel object recognition (NOR).

59 ning, cued learning, locomotor activity, and novel object recognition (NOR).

60 (T-maze) and a hippocampus-independent task (Novel Object Recognition, NOR).

61 r memory in tests of cued fear conditioning, novel object recognition, object location recognition, c

62 Nonsocial behaviors or memories, including novel object recognition or fear conditioning, were not

63 hibit impairment of learning and memory in a novel object recognition paradigm.

64 and aged (26 months) mice were tested in the novel object recognition task (NORT).

65 imulation of LC-NE enhanced performance in a novel object recognition task and reduced hyperactivity

66 o procognitive activity (1 mg/kg, ip) in the novel object recognition task as a model of memory defic

67 The IDUA(-/-) mice performed normally in a novel object recognition task at younger ages until 8 mo

68 , we used different strength of training for novel object recognition task in mice.

69 t-term (STM) and long-term memory (LTM) in a novel object recognition task, but exhibit impairments d

70 Mutant mice showed deficits in the novel object recognition task, suggesting hippocampal dy

71 ognitive alterations, including defects in a novel object recognition task.

72 ted plus maze, holeboard, light-dark box and novel object recognition task.

73 only mice outperformed wild-type mice in the novel object recognition task.

74 Object recognition was tested using a novel object recognition task.

75 We used the novel object-recognition task as a model of nonemotional

76 short-, but not the long-term memory in the novel-object recognition task.

77 adian modulation of performance in a delayed novel-object recognition task.

78 the first experiment, mice performed both a novel-object-recognition task identical to that performe

79 Bl/6J mice were tested in the water maze and novel object recognition tasks.

80 ificant alterations in an open field test, a novel object recognition test and in a T-maze task.

81 We assessed cognition with the novel object recognition test and stained for amyloid pr

82 gnitive profile as it improved memory in the novel object recognition test but had no antidepressant

83 ognition tests, and their memory loss in the novel object recognition test is associated with high le

84 light-dark box test) and cognitive function (novel object recognition test).

85 dopamine-lesioned mice were subjected to the novel object recognition test, and long-term potentiatio

86 ith cognitive impairment, as assessed by the novel object recognition test, but not signs of brain in

87 r cognitive impairment when examined using a novel object recognition test.

88 he water maze, Y-maze reversal task, and the novel object recognition test.

89 -term memories were robustly improved in the novel-object recognition test and Morris water-maze spat

90 ced-swim, Y-maze spontaneous alternation and novel-object recognition test performance that developed

91 Notably, the novel-object recognition test showed that the treatment

92 tle and prepulse inhibition, open field, and novel object recognition tests to evaluate behavior in f

93 he memory retention in passive avoidance and novel object recognition tests, and their memory loss in

94 al fear-conditioning, Morris water maze, and novel object recognition tests.

95 e in fear conditioning, object location, and novel object recognition tests.

96 d improved memory performance of CES rats in novel-object recognition tests and in the Morris water m

97 Novel object recognition was not impaired in R192Q mice;

98 locomotor activity, pre-pulse inhibition and novel object recognition were not impaired.

99 noise were modestly or minimally impaired in novel object recognition, whereas similar-duration multi