ライフサイエンスコーパス: noxious stimuli

1 us stimuli (learned anorexia associated with noxious stimuli).

2 hyperalgesia (exaggerated responsiveness to noxious stimuli).

3 or cell viability during exposure to various noxious stimuli.

4 Both DRR and AR can be evoked by noxious stimuli.

5 e healing and neoangiogenesis in the face of noxious stimuli.

6 10, were persistently activated by transient noxious stimuli.

7 uring evaluation of the spatial locations of noxious stimuli.

8 actions of CO in modulating the response to noxious stimuli.

9 tentiation of endogenous opioid responses to noxious stimuli.

10 either protein display altered responses to noxious stimuli.

11 for optimizing their role in transmission of noxious stimuli.

12 t, Klf7(-/-) mice have deficient response to noxious stimuli.

13 lator of the responses of sensory neurons to noxious stimuli.

14 ne of defense against invading pathogens and noxious stimuli.

15 allodynia-a painful response to normally non-noxious stimuli.

16 and upregulate c-Fos protein in response to noxious stimuli.

17 responding preferentially to threatening or noxious stimuli.

18 nto how EGFR integrates responses to diverse noxious stimuli.

19 myelinated nerve fibers (C-fibers) following noxious stimuli.

20 (JNK) is activated when cells are exposed to noxious stimuli.

21 O mice responded normally to a wide array of noxious stimuli.

22 central nociceptive neurons to innocuous and noxious stimuli.

23 st cells with this receptor are activated by noxious stimuli.

24 ion whilst attention is distracted away from noxious stimuli.

25 e fields (RFs) excited by non-noxious and/or noxious stimuli.

26 sensory neurons and is activated by multiple noxious stimuli.

27 ing the response of sensory nerve endings to noxious stimuli.

28 uli and a mild increased sensitivity to some noxious stimuli.

29 seline hindpaw sensitivity to non-noxious or noxious stimuli.

30 , to significantly attenuated sensitivity to noxious stimuli.

31 ation without diminishing responses to other noxious stimuli.

32 rn and is released in response to painful or noxious stimuli.

33 eficient in their responses to each of these noxious stimuli.

34 ne signals to the brain and in responding to noxious stimuli.

35 g in a decreased pain response to peripheral noxious stimuli.

36 ugmented glucose mobilization in response to noxious stimuli.

37 vous system analyzes cutaneous innocuous and noxious stimuli.

38 d a high firing rate but responded weakly to noxious stimuli.

39 ition is dynamically activated by peripheral noxious stimuli.

40 P N-terminal activity in mice not exposed to noxious stimuli.

41 prolonged the after-discharge in response to noxious stimuli.

42 een strongly implicated in the processing of noxious stimuli.

43 sent a way for dopamine to modulate incoming noxious stimuli.

44 unicate to us their perceived levels of such noxious stimuli.

45 or for heat, acidic pH, capsaicin, and other noxious stimuli.

46 v1.7 plays a crucial role in transmission of noxious stimuli.

47 rain region mediating affective responses to noxious stimuli.

48 ment neuronal sensitivity (sensitization) to noxious stimuli.

49 racolumbar skin and is selectively driven by noxious stimuli.

50 ferent nociceptors and is activated by other noxious stimuli.

51 retinal cells die in response to a range of noxious stimuli.

52 o associated with the perception of pain and noxious stimuli.

53 ating homeostatic responses against external noxious stimuli.

54 n the magnitude and duration of responses to noxious stimuli.

55 ses hyperalgesia, an enhanced sensitivity to noxious stimuli.

56 arly stage of lung inflammatory responses to noxious stimuli.

57 ne if methylene blue could protect RGCs from noxious stimuli.

58 part of the inflammatory response to inhaled noxious stimuli.

59 nilloid 1(TRPV1), an important transducer of noxious stimuli.

60 ody's way of combating invading pathogens or noxious stimuli.

61 in protecting the cell against stressful and noxious stimuli.

62 Localized noxious stimuli (55 degrees C or intradermal injection o

63 ify and process distinct sensory features of noxious stimuli according to top-down task demands.

64 Transient noxious stimuli also enhanced MCC excitability, and this

65 (KO) mice had normal responses to acute non-noxious stimuli and a mild increased sensitivity to some

66 s that assemble in response to infectious or noxious stimuli and activate the CASPASE-1 protease.

67 Laminae that are responsive to non-noxious stimuli and activated by walking, IIi, nucleus p

68 are responsive to a variety of modalities of noxious stimuli and can signal pain even when activated

69 and activity mediates a hypersensitivity to noxious stimuli and causes the inhibition of opiate effi

70 By sensing noxious stimuli and contributing to the necessary reacti

71 rsal root ganglion (DRG) neurons that detect noxious stimuli and elicit pain.

72 that the sexes differ in their perception of noxious stimuli and in their responsivity to exogenous a

73 l A1 (TRPA1) ion channel senses a variety of noxious stimuli and is involved in nociception.

74 coeruleus are involved in the processing of noxious stimuli and may contribute to anti-nociceptive m

75 but markedly attenuated responses to highly noxious stimuli and mechanical and thermal hyperalgesia.

76 avioural responses to thermal and mechanical noxious stimuli and morphine-induced antinociception.

77 Responses to normally noxious stimuli and motor function were unchanged in SCI

78 sured by nerve conduction, gait, response to noxious stimuli and nerve histology.

79 cortex potently suppresses SpVc responses to noxious stimuli and produces behavioral hypoalgesia.

80 s by which the brain protects itself against noxious stimuli and recovers from ischaemic damage are a

81 it permits responses such as withdrawal from noxious stimuli and regulation of body temperature.

82 Spinal cord neurons respond to peripheral noxious stimuli and relay this information to higher bra

83 he sea slug Pleurobranchaea are responses to noxious stimuli and replace orienting turns to food stim

84 rons in the peripheral nervous system detect noxious stimuli and report the information to the centra

85 olution are critical in ensuring disposal of noxious stimuli and return to homeostasis.

86 orsal root ganglia (DRG) neurons that detect noxious stimuli and signal pain.

87 etween supraspinal and spinal sites to acute noxious stimuli and suggest possibility that compounds a

88 and mark dying cells in response to diverse noxious stimuli and suggest that elaborate, multilayered

89 n in preventing endothelial cell death after noxious stimuli and suggest that the ICE pathway may med

90 a pivotal role in the transmission of highly noxious stimuli and the maintenance of hyperalgesia.

91 ) is implicated in the affective response to noxious stimuli and the motivational properties of condi

92 sion that underlies increased sensitivity to noxious stimuli and the perception of non-noxious stimul

93 estigated whether it increases resistance to noxious stimuli and the role of changes in intracellular

94 e disorder characterized by insensitivity to noxious stimuli and variable intellectual disability (ID

95 TRPA1 is a unique sensor of noxious stimuli and, hence, a potential drug target for

96 Finally, we show that the neural coding of noxious stimuli, and consequent experience of pain, are

97 ressed in neurons that sense temperature and noxious stimuli, and TrkC is expressed in proprioceptive

98 encounters with predators, competitors, and noxious stimuli, animals have evolved defensive response

99 taneous activity and responded vigorously to noxious stimuli applied to any part of the body surface.

100 sula, and ACC when comparing the response to noxious stimuli applied to control and placebo cream-tre

101 induced siphon withdrawal reflex by repeated noxious stimuli applied to one of three sites: the (1) i

102 whether stimulation of peripheral nerves by noxious stimuli applied to their receptive fields activa

103 Aggression and responsiveness to noxious stimuli are adaptable traits that are ubiquitous

104 ulation and nocifensive responses to thermal noxious stimuli are not modified.

105 n as to how such distinct sensory aspects of noxious stimuli are processed by the human brain.

106 Noxious stimuli are sensed and carried to the spinal cor

107 to noxious stimuli and the perception of non-noxious stimuli as painful.

108 the ACC to increase the aversive response to noxious stimuli at anatomically unrelated sites.

109 he response of the brain reward circuitry to noxious stimuli at baseline before opioid administration

110 Containment of the glial HO-1 response to noxious stimuli at strategic points of the life cycle ma

111 The ion channel TRPA1 detects noxious stimuli at the plasma membrane of neurons and el

112 whereby noradrenaline may suppress incoming noxious stimuli at the primary synaptic afferents in the

113 redicted by the neuronal response to painful noxious stimuli before infusion in key structures of the

114 e studied for sensitivity to non-noxious and noxious stimuli, before and after induction of experimen

115 distal limbs have a high spatial acuity for noxious stimuli but a low density of pain-sensing neurit

116 espond to beta-alanine, heat, and mechanical noxious stimuli but do not respond to histamine.

117 peripheral and spinal nociceptive neurons to noxious stimuli but only when the joint was inflamed.

118 fit trait that defends against pathogens and noxious stimuli but whose overactivation can result in i

119 ally cause pain and pain usually arises from noxious stimuli, but exceptions to these apparent truism

120 hibited normal behavioral responses to acute noxious stimuli, but subsequent to partial sciatic nerve

121 within the vasculature and tissue respond to noxious stimuli by sending out coordinated signals that

122 ain results from the detection of intense or noxious stimuli by specialized high-threshold sensory ne

123 underlying the detection and transmission of noxious stimuli by the peripheral nervous system.

124 All three types of noxious stimuli caused a depletion of CGRP from corneal

125 The nociceptive system of detecting noxious stimuli comprises two classes of peripheral affe

126 These findings suggest that multiple noxious stimuli converge upon a peptidase-driven, core s

127 firmed that the escape/avoidance response to noxious stimuli corresponds to changes in neural activat

128 cterized by hypersensitivity to innocuous or noxious stimuli during sustained opiate administration.

129 d by heat >42 degrees C in addition to other noxious stimuli, e.g. acids and vanilloids.

130 b neurons augments the autonomic response to noxious stimuli, ensuring sufficient glucose mobilizatio

131 ttle is known about the proteins that detect noxious stimuli, especially those of a physical nature.

132 nent of the cellular mechanism through which noxious stimuli evoke pain.

133 al nerves, indicating that all modalities of noxious stimuli evoked peptide release.

134 We found that a variety of noxious stimuli evoked strong responses in ASH including

135 1, a channel implicated in detecting several noxious stimuli, exhibiting a 50% effective concentratio

136 sensation, as well as in hypersensitivity to noxious stimuli following tissue injury.

137 olved highly efficient mechanisms to "flush" noxious stimuli from airway surfaces by selective activa

138 he mechanisms underlying the transduction of noxious stimuli from the viscera, suggest that the inves

139 ds to profoundly increased pain sensitivity: noxious stimuli generate a greater response and stimuli

140 hronic pain: it is initiated by a variety of noxious stimuli, has indefinite duration, and pain appea

141 t long-lasting changes in reflexes evoked by noxious stimuli have evolved to enhance reflex protectio

142 Noxious stimuli have motivational power and can support

143 orphine on evoked responses of LC neurons to noxious stimuli have not been systematically examined.

144 on neurons: the majority of these respond to noxious stimuli, however some are activated by cooling.

145 ions) modulate behavioral responses to acute noxious stimuli; however, little is known about the endo

146 persistent pain, an exacerbated response to noxious stimuli (hyperalgesia), and a lowered pain thres

147 We found that intense noxious stimuli (i.e., subdermal injection of capsaicin

148 opioid release in the spinal cord evoked by noxious stimuli in anesthetized rats.

149 sation and neural circuitry that result from noxious stimuli in early life.

150 ascending pain pathway occurred during acute noxious stimuli in healthy individuals.

151 tive suckling produce analgesia to transient noxious stimuli in infant rats and humans.

152 induced suppression of neuronal responses to noxious stimuli in key structures of the descending pain

153 the receptor by setting sensitivity to other noxious stimuli in response to changes in extracellular

154 havioral and neurophysiological responses to noxious stimuli in rodents when administered systemicall

155 al and subcortical responses to auditory and noxious stimuli in sedated and unresponsive states.

156 rol the transduction of inflammation-induced noxious stimuli in sensory neurons.

157 tive cation channels that integrate multiple noxious stimuli in sensory neurons.

158 e amygdala did not affect responses to acute noxious stimuli in the absence of inflammation, indicati

159 anism for noradrenaline to modulate incoming noxious stimuli in the dorsal horn of the spinal cord.

160 or a lateralized response to noxious and non-noxious stimuli in the human spinal cord.

161 ulation of orienting and attack by low-level noxious stimuli in the hungriest animals may reflect ris

162 eby serving important functions in detecting noxious stimuli in the sensory system and pathological s

163 vel NPVF-vRN functional circuit modulated by noxious stimuli in vertebrates.

164 ial vanilloid 1 (TRPV1), a key integrator of noxious stimuli, in the pathogenesis of pancreatic pain

165 VR1 responds to noxious stimuli including capsaicin, the pungent compone

166 the capsaicin receptor, is an integrator of noxious stimuli including heat and extracellular acidic

167 lion neurones and is activated by a range of noxious stimuli including irritant chemicals, acids and

168 rapid and complex physiological reaction to noxious stimuli including microbial pathogens.

169 A variety of noxious stimuli, including balloon angioplasty, may comp

170 nel that can be activated by a wide range of noxious stimuli, including capsaicin, acid, and heat.

171 nel that can be activated by a wide range of noxious stimuli, including capsaicin, acid, and heat.

172 TRPA1 is activated by a variety of noxious stimuli, including cold temperatures, pungent na

173 Polymodal nociceptors detect noxious stimuli, including harsh touch, toxic chemicals

174 The TRPV1 channel is a detector of noxious stimuli, including heat, acidosis, vanilloid com

175 TRPV1 functions as a molecular integrator of noxious stimuli, including heat, low pH, and chemical li

176 iple different infectious, inflammatory, and noxious stimuli, including pneumococcal pneumonia.

177 that detect odors, tastants, pheromones, and noxious stimuli, including receptors of the odor recepto

178 We tested the hypothesis that noxious stimuli induce pain modulation by activation of

179 results indicate that lumbar opiates inhibit noxious stimuli-induced neurotransmitter release from pr

180 Noxious stimuli inhibit inflammation by activating neuro

181 ential functions of nociceptors--transducing noxious stimuli into depolarizations that trigger action

182 Hyperalgesia to noxious stimuli is accompanied by increased spinal cyclo

183 creasingly greater pain evoked by repetitive noxious stimuli, is highly variable between individuals.

184 fibrosis airways are recurrently exposed to noxious stimuli, leading to epithelial injury.

185 ors, for example), especially in response to noxious stimuli (learned anorexia associated with noxiou

186 s, sensory pathways mediating appetitive and noxious stimuli may have dual access to neural networks

187 s, which reflect sensory properties of acute noxious stimuli, NAc activity in humans encodes its pred

188 he activation of sensory neurons that detect noxious stimuli (nociceptors).

189 r thoracic spinal neurons responding to both noxious stimuli of the heart and lungs in rats.

190 The effect of various noxious stimuli on sensory and enteric neural function i

191 In response to invading microbes, noxious stimuli, or tissue injury, an acute inflammatory

192 in the ICU in order for patients to tolerate noxious stimuli, particularly mechanical ventilation.

193 d dorsal horn neuron activity in response to noxious stimuli, possibly through a release of GABA.

194 ts keratinocyte proliferation in response to noxious stimuli, possibly through p53 activation, which

195 ndogenous ion channels that are activated by noxious stimuli, primarily TRPV1.

196 The ability to detect noxious stimuli, process the nociceptive signal, and eli

197 Hyperalgesia is an exaggerated response to noxious stimuli produced by peripheral or central plasti

198 Cutaneous sensory neurons that detect noxious stimuli project to the dorsal horn of the spinal

199 eased by the olfactory epithelium (OE) after noxious stimuli provides a physiological source for a ne

200 NGF in behavioral responses of adult rats to noxious stimuli, providing high titers of antibody are p

201 on, in healthy mice increases sensitivity to noxious stimuli (referred to as 'pain') without general

202 evention of harm in the presence of novel or noxious stimuli, regardless of whether such stimuli are

203 eurons and vRN are suppressed and excited by noxious stimuli, respectively.

204 is normally attenuated after elimination of noxious stimuli, restoration of homeostasis and initiati

205 We also observed that repetitive noxious stimuli resulted in adaptation of the signal.

206 During noxious stimuli, serotonergic neurons activation was due

207 sic nocebo responses to identical calibrated noxious stimuli showed signal increases in brain regions

208 to maintain pancreatic health in response to noxious stimuli such as alcohol.

209 o recorded L-ITCcs are strongly activated by noxious stimuli, such as hindpaw pinches or electrical f

210 begins when sensory neurons are activated by noxious stimuli, such as stepping on a tack.

211 ome c oxidase and protect RGCs against these noxious stimuli supports its suggested mechanism of acti

212 any endogenous opioids that are released by noxious stimuli target presynaptic MORs or delta-opioid

213 pain intensity ratings of acute experimental noxious stimuli than age-matched control subjects.

214 nderlie detection, coding, and modulation of noxious stimuli that generate pain.

215 gerated hyperalgesic behavior in response to noxious stimuli that may model aspects of painful diabet

216 in nociceptive processing during persistent noxious stimuli that resemble pathological pain conditio

217 Inflammation is a protective response to noxious stimuli that unavoidably occurs at a cost to nor

218 expectancy modulates activations produced by noxious stimuli through a distinct modulatory network th

219 in, and suppress the overall withdrawal from noxious stimuli through a pathway requiring the opioid-l

220 of pain is initiated by the transduction of noxious stimuli through specialized ion channels and rec

221 n sensation is initiated by the detection of noxious stimuli through specialized transduction ion cha

222 the spinal cord following intense peripheral noxious stimuli, tissue injury or nerve damage.

223 in cocoa for 14days prior to an injection of noxious stimuli to cause acute or chronic excitation of

224 atiation state interacts with appetitive and noxious stimuli to determine feeding and avoidance respo

225 All animals must detect noxious stimuli to initiate protective behavior, but the

226 nal activity is necessary and sufficient for noxious stimuli to produce an aversive teaching signal.

227 Neurodegenerative diseases and noxious stimuli to the brain enhance transcription of se

228 ensory neurons that convey information about noxious stimuli to the central nervous system.

229 ays an important role in the transduction of noxious stimuli to the spinal cord.

230 elimination of evoked pain; (v) although non-noxious stimuli to the unaffected limb were perceived as

231 In addition, several noxious stimuli trigger the release of intracellular spe

232 As a sensor for noxious stimuli, TRPV1 channel has been described as a k

233 dies) and cellular reactions to a variety of noxious stimuli (ubiquitinated dystrophic neurites, astr

234 This treatment left responses to mild noxious stimuli unchanged, but markedly attenuated respo

235 We found, however, that noxious stimuli, under normal or inflammatory conditions

236 Noxious stimuli usually cause pain and pain usually aris

237 sture (e.g., escape behaviors in response to noxious stimuli vs freezing in response to fear-evoking

238 The response of GiA cells to noxious stimuli was abolished by transection of only the

239 ailure to perceive or respond to auditory or noxious stimuli was associated with a reduction in the f

240 A series of auditory and noxious stimuli was presented to eight healthy volunteer

241 ptor potential channel that is responsive to noxious stimuli, was required for the fructose response.

242 PV channels inhibits behavioral responses to noxious stimuli, we found that both mechanoreceptor curr

243 For both innocuous and noxious stimuli, we observed a signal change in the prim

244 OFF cells) by noxious cutaneous stimulation, noxious stimuli were applied during evoked BAT temperatu

245 The cytoprotective effects of rhPRL against noxious stimuli were assessed by flow cytometry (tetrame

246 Acute illness and noxious stimuli were associated with 24% and 23% of the

247 The noxious stimuli were heel lances performed for routine b

248 Application of noxious stimuli, which often signal the need to mobilize

249 ting responses from being directly evoked by noxious stimuli while also facilitating the ability of f

250 Many organisms respond to noxious stimuli with defensive maneuvers.

251 Many organisms respond to noxious stimuli with defensive maneuvers.

252 sonance imaging, we compared self-controlled noxious stimuli with physically identical stimuli that w

253 the preinfusion period neuronal response to noxious stimuli within the ventral tegmentum.

254 Comparison of the noxious and non-noxious stimuli yielded several new insights into neural