ライフサイエンスコーパス: noxious stimulus

1 ance learning in the absence of a peripheral noxious stimulus.

2 conditioning blocked learning elicited by a noxious stimulus.

3 ous stimulus on the reaction to a subsequent noxious stimulus.

4 oxious stimulus that closely follows another noxious stimulus.

5 rsion to formalin injection, an inflammatory noxious stimulus.

6 cal threat to an individual is exposure to a noxious stimulus.

7 spinal cord and in the brainstem to a tonic noxious stimulus.

8 the response measure, test time, and type of noxious stimulus.

9 Off food, animals reverse away from a noxious stimulus.

10 ee hind limb withdrawal reflexes of an acute noxious stimulus (20 % mustard oil) applied to a number

11 atory, we identified and selectively labeled noxious-stimulus-activated PBL neurons and performed com

12 ify neurons according to their response to a noxious stimulus and map their location based on stainin

13 nctional ASICs, are insensitive to acid as a noxious stimulus and show diminished avoidance of acidic

14 n can be unmasked by the administration of a noxious stimulus and that it is manifested as increased

15 ve examined the variables of gender, type of noxious stimulus, and the origin of nociceptive input as

16 ed to the onset of allodynia, in which a non-noxious stimulus becomes painful.

17 reflects the susceptibility to a subsequent noxious stimulus being perceived as painful.

18 utes an anatomical substrate through which a noxious stimulus can activate 5HT neurons of the NRM and

19 log of a form of arousal induced by a mildly noxious stimulus can promote two antagonistic responses,

20 To determine if administration of a noxious stimulus can unmask a sensitization of dorsal ho

21 t reprogram mouse and human fibroblasts into noxious stimulus-detecting (nociceptor) neurons.

22 ns to prevent central sensitization when the noxious stimulus does not produce inflammation and it is

23 625, 0.125, and 0.25 mg/kg, i.v.) suppressed noxious stimulus-evoked activity of VPL neurons in a dos

24 Before morphine, the magnitude of the noxious stimulus-evoked burst in ON cells correlated wit

25 Noxious stimulus-evoked firing was affected more than sp

26 we found that morphine had little effect on noxious stimulus-evoked internalization of the NK-1 rece

27 nerve injury and inflammation and prevented noxious stimulus-evoked neuronal activation in spinal co

28 udied how the nervous system selects between noxious stimulus-evoked withdrawals and micturition, mov

29 between a neutral stimulus (tone [CS]) and a noxious stimulus (foot shock [US]).

30 ic sensory dysfunction following a transient noxious stimulus in the neonatal period and a potentiall

31 ect of chronic administration of morphine on noxious stimulus-induced antinociception (NSIA) produced

32 GBR-12935 also produced a reduction in noxious stimulus-induced c-fos expression in nociceptive

33 ciceptive neurons was obtained by monitoring noxious stimulus-induced c-fos expression in rats having

34 induced by activation of an opioid-mediated, noxious stimulus-induced endogenous pain control circuit

35 Morphine significantly reduced noxious stimulus-induced Fos expression in lamina I, but

36 viously observed sites of ADX enhancement of noxious stimulus-induced Fos-like immunoreactivity.

37 f the magnitude and duration of decreases in noxious stimulus intensity.

38 Additionally, we show that, after a noxious stimulus (intradermal capsaicin injection), thes

39 ere examined after treatment with a visceral noxious stimulus (intraperitoneal injection of dilute la

40 ggests that movement of the limb away from a noxious stimulus is a sensitive indication of nociceptiv

41 ancement of an unhabituated response after a noxious stimulus is applied outside of the reflex recept

42 ty.SIGNIFICANCE STATEMENT The intensity of a noxious stimulus is encoded by the frequency of action p

43 ned stimulus that was previously paired to a noxious stimulus leads to the gradual disappearance of c

44 ively, such neurons may signal itch, whereas noxious stimulus levels recruit these and a larger popul

45 To fulfill these functions, a noxious stimulus might induce a percept which, in turn,

46 sts that the brain can distinguish different noxious stimulus modalities from the earliest stages of

47 ors are polymodal (i.e., respond to multiple noxious stimulus modalities, such as mechanical and ther

48 predictive of the modulatory effects of one noxious stimulus on the reaction to a subsequent noxious

49 hieved optogenetically, without any external noxious stimulus or injury.

50 active change of the retina in response to a noxious stimulus or to RGC death.

51 nockout larvae, whereas responses to another noxious stimulus or touch were not affected.

52 nonassociative learning in which a strong or noxious stimulus persistently enhances the response prod

53 g primary afferents and their responses to a noxious stimulus (subcutaneous formalin injection).

54 prolonged (up to 48 h) stress response to a noxious stimulus such as UVB.

55 Prior exposure to a noxious stimulus (tailshock) has selective effects on th

56 on and off cells in enhancing reactions to a noxious stimulus that closely follows another noxious st

57 open question is whether the property of the noxious stimulus underlying the genetic correlation is h

58 Five minutes later, a noxious stimulus was delivered to the paw.

59 reported significantly higher pain when the noxious stimulus was preceded by the high-intensity visu

60 inates from visceral organs in response to a noxious stimulus which, if prolonged, may lead to chroni