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1 multiple nuclei, consistent with unbalanced nuclear division.
2 rmation of delocalized septa did not require nuclear division.
3 likely to be involved in control of parasite nuclear division.
4 th retention of nuclear Clb5p at the time of nuclear division.
5 only after the nucleus enters the bud during nuclear division.
6 otype, where septation is completed prior to nuclear division.
7 inately cycle multiple times without causing nuclear division.
8 c nuclear division and the first postzygotic nuclear division.
9 lular polarity, without affecting growth and nuclear division.
10 aughter nucleus in cells that have undergone nuclear division.
11 dc18 fail to enter S phase but still undergo nuclear division.
12 point to a central role in the regulation of nuclear division.
13 hat ensures proper genome segregation during nuclear division.
14 cannot rereplicate between bud formation and nuclear division.
15 CDK activity to allow bud formation prior to nuclear division.
16 ng cells, is normally restrained until after nuclear division.
17 t is required for chromosome segregation and nuclear division.
18 sembly of kinetochores and are essential for nuclear division.
19 cid replication is followed by two rounds of nuclear division.
20 are required for Rec8 cleavage and meiosis I nuclear division.
21 did restore JM resolution, CO formation, and nuclear division.
22 These JMs persist, preventing nuclear division.
23 ediated progression beyond the first meiotic nuclear division.
24 er checkpoint-inducing conditions that block nuclear division.
25 eres, eliminating the possibility of mitotic nuclear division.
26 Constructs that chelate InsP3 also prevent nuclear division.
27 additional round of DNA replication without nuclear division.
28 e SPB and that this hub coordinates cell and nuclear division.
29 to their requirement for IME1 induction and nuclear division.
30 efects can be detected as early as the first nuclear divisions.
31 branch formation reinitiates tip growth and nuclear divisions.
32 ty of the meiotic chromosomes during the two nuclear divisions.
33 s and circadian clock-dependent synchronized nuclear divisions.
34 a maternal effect during the early embryonic nuclear divisions.
35 omatin prior to each of the three prezygotic nuclear divisions.
36 delayed and reduced execution of the meiotic nuclear divisions.
37 and Cdt1 during the interval between meiotic nuclear divisions.
38 ription program and execution of the meiotic nuclear divisions.
39 tions in the syncytial blastoderm after 9-10 nuclear divisions.
40 activity blocks bipolar spindle assembly and nuclear divisions.
41 s that arrest development after two or three nuclear divisions.
42 ource is necessary only prior to the meiotic nuclear divisions.
43 itical role of Cep164 in G2/M checkpoint and nuclear divisions.
44 embryo and remains stable during subsequent nuclear divisions.
45 eplication (meiS) followed by two successive nuclear divisions.
48 break repair and reduced DNA damage-related nuclear division abnormalities, a phenotype associated w
49 romatid cohesion (SSC) during two successive nuclear divisions allows the production of haploid gamet
51 ultiple nuclei indicative of a G2/M delay in nuclear division and also a defect in cytokinesis and/or
54 ur data indicate that glo1-1 plays a role in nuclear division and cytokinesis in the developing seed.
62 emperature-sensitive phoA allele caused both nuclear division and polarity defects at restrictive tem
63 PfCRK4 depletion led to a complete block in nuclear division and profoundly inhibited DNA replicatio
64 ements for remodeling the NE during "closed" nuclear division and provide insight into the structure
65 Syncytial embryos develop through cycles of nuclear division and rearrangement within a common cytop
66 results suggest that the ability to separate nuclear division and septum formation depends upon a thr
68 cnj7 and cnj8 eliminate the third prezygotic nuclear division and the first postzygotic nuclear divis
69 F-actin in one of the daughter nuclei after nuclear division and the formation of septum; and (2) di
70 function in a pathway that controls meiotic nuclear divisions and coordinates meiosis and spore form
71 of Mtrm in the early embryo caused aberrant nuclear divisions and developmental defects, and these w
72 hogenesis checkpoint in budding yeast delays nuclear division (and subsequent cell cycle progression)
75 cts septation and conidiation in addition to nuclear division, and cloning and biochemical analysis o
78 ear fusions occur in the early cycles of the nuclear divisions, and become more prominent at later st
81 meiotic prophase progression and subsequent nuclear division are delayed if these haploids carry an
85 e presence of many cells undergoing aberrant nuclear division, as well as many anucleate cells, demon
86 dc1 depletion might affect bud formation and nuclear division, as well as other cellular processes, b
87 tant displays defects in cell morphology and nuclear division at 26 degrees C which are exaggerated a
89 as initially implicated in bud formation and nuclear division because cdc1(Ts) cells arrested with a
91 rminating conidia undergo multiple rounds of nuclear division before the formation of the first septu
92 e fertilized egg undergoes a series of rapid nuclear divisions before the syncytial blastoderm starts
93 1 or Rad17 partially bypass the block to the nuclear divisions but do not form spores, while mutants
94 ntial specifically for spindle formation and nuclear division, but not for DNA replication, once CDKA
95 protozoans, GPIs appear to be essential for nuclear division, but not for mitochondrial duplication.
96 )/M, prp17Delta and prp22-1 strains, permits nuclear division, but suppression of the cell cycle bloc
97 he loss of linkages between chromosomes with nuclear division by restraining two other phosphatases,
102 uring megagametogenesis, including syncitial nuclear divisions, cellularization, nuclear migration an
103 as zygotic cnn expression display defects in nuclear division, chromosome alignment, and microtubule
104 osophila, induced a reversible arrest of the nuclear divisions comparable with that induced by hypoxi
109 tivity and is essential for coordinating the nuclear division cycle with cytokinesis through the cyto
114 ishment and tip growth, but they have normal nuclear division cycles and can complete the asexual gro
116 processes such as cytokinesis and syncytial nuclear division cycles in Drosophila Pseudocleavage fur
119 t dfmr1 embryos display defects in the rapid nuclear division cycles that precede gastrulation in nuc
120 elanogaster embryos that supports successive nuclear division cycles with native characteristics.
125 also show inefficient nuclear migration and nuclear division, defects in the organization of the sec
126 or proceed through mitosis in the absence of nuclear division, demonstrating an uncoupling of proper
127 e wild-type cells, clb5 clb6 mutants undergo nuclear division despite the presence of nuclear non-deg
129 cing can persist through DNA replication and nuclear divisions, disrupting uniform gene expression in
130 -sensitive snxA1 mutation leads to arrest of nuclear division during G(1) or early S. snxB1 causes hy
132 ions are asynchronous and lag behind somatic nuclear divisions during syncytial cycles 9 and 10.
133 rther, the checkpoint is capable of delaying nuclear division even when SWE1 transcription is deregul
135 ut instead triggered DNA synthesis, abnormal nuclear divisions, failure of cytokinesis, and emergence
137 sac development begins with a phase of free nuclear division followed by cellularization and differe
138 leus and GFP-Cdc24p levels diminished before nuclear division followed by its reappearance in divided
139 e endosperm originates with a series of free-nuclear divisions, followed by cellularisation and subse
140 osis, a diploid cell undergoes two rounds of nuclear division following one round of DNA replication
141 owth of actin and microtubule networks after nuclear division generates reordering forces that counte
142 cytokinesis, and binucleated cells in which nuclear division had occurred without cell division.
143 lus nidulans become competent for growth and nuclear division in a process called conidial germinatio
146 The pachytene checkpoint prevents meiotic nuclear division in cells that fail to complete meiotic
148 unctions in a checkpoint pathway that delays nuclear division in response to defects in these events.
154 cyclin E (cye-1) expression is critical for nuclear divisions in the intestine and is elevated in do
156 eLa cells blocked cellular cleavage, but not nuclear division, indicating a functional role for PRC1
157 Rereplication occurs prior to the meiotic nuclear divisions, indicating that this process is diffe
160 lins in mitosis and meiosis I, in which each nuclear division is coupled with a peak of expression of
163 growth, induced mitotic arrest and aberrant nuclear division leading to decreased survival, and incr
164 gametophytes have a prolonged phase of free nuclear divisions leading to a variety of embryo sac abn
167 core of the meiotic process is a specialized nuclear division (meiosis I) in which homologs pair with
168 ave been shown to have roles in coordinating nuclear division, membrane trafficking and organizing th
170 ated, spo12 mutants undergo a single meiotic nuclear division most closely resembling meiosis II.
172 us, Aspergillus nidulans, multiple rounds of nuclear division occur before cytokinesis, allowing an u
179 n used a histone hH1-GFP reporter to observe nuclear divisions over time, and show that a large numbe
180 pronuclear fusion, and anchoring postzygotic nuclear division products to the posterior cell cortex.
181 The homologous-pairing protein2/meiotic nuclear division protein1 (HOP2/MND1) protein complex ha
182 that the ability of this checkpoint to delay nuclear division requires the SWE1 gene, encoding a prot
185 o arrest by serum-withdrawal, EBNA3C induces nuclear division that is often divorced from cytokinesis
187 gnu (GNU embryos) under DNA synthesis but no nuclear division; this leads to the formation of a small
190 entally regulated, reaching a maximum during nuclear division towards the end of the intraerythrocyti
194 stances, the morphogenesis checkpoint delays nuclear division via the mitosis-inhibitory Wee1-family
195 n typical nucleosomal structures, defects in nuclear divisions were restricted to mitotically dividin
196 Sic1 provides a failsafe mechanism promoting nuclear division when levels of mitotic cyclins are low.
197 is the exit from prophase and entry into the nuclear divisions, which in the yeast Saccharomyces cere
199 on in mitosis, Nop15p-depleted cells undergo nuclear division with wild-type kinetics, activate the m
200 the mutant mother frequently show disrupted nuclear divisions with defects in chromosome condensatio
201 re, mutant cells arrest in the cell cycle at nuclear division, with a large bud, a single nucleus wit
202 opmental arrest, usually during the first 13 nuclear divisions, with a nuclear phenotype of chromatin
204 with multiple rounds of DNA replication and nuclear division without cytokinesis, resulting in a mul
206 y binucleate, trimeras underwent coordinated nuclear division yielding four daughter nuclei, two of w
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