ライフサイエンスコーパス: nuclear envelope

1 n that are consistent with disruption of the nuclear envelope.

2 ntial for the attachment of telomeres to the nuclear envelope.

3 lumen of the endoplasmic reticulum (ER) and nuclear envelope.

4 hifts many chromosomal regions closer to the nuclear envelope.

5 age-like" structure inside the disassembling nuclear envelope.

6 eicosanoids by 5-lipoxygenase (5-LOX) on the nuclear envelope.

7 tion of nuclear pore complexes (NPCs) at the nuclear envelope.

8 highlighting the remarkable dynamics of the nuclear envelope.

9 - and KASH-domain-containing proteins in the nuclear envelope.

10 ls that mediate nuclear transport across the nuclear envelope.

11 or translocation of large cargoes across the nuclear envelope.

12 eals that perinuclear inclusions disrupt the nuclear envelope.

13 surfaces of the ER and inner membrane of the nuclear envelope.

14 rect association of dNesp1 isoforms with the nuclear envelope.

15 chain fatty acids and the maintenance of the nuclear envelope.

16 NF-kappaB shuttles back and forth across the nuclear envelope.

17 ay link the central plaque of the SPB to the nuclear envelope.

18 ly localized delays in the reassembly of the nuclear envelope.

19 RanGAP and delocalization of RanGAP from the nuclear envelope.

20 nal vesicle-mediated trafficking through the nuclear envelope.

21 lso the only known viruses that bud into the nuclear envelope.

22 of phospholipase C (PLC), PLCepsilon, at the nuclear envelope.

23 the MTOCs into fragmented ribbons along the nuclear envelope.

24 lasm, mRNPs are frequently confined near the nuclear envelope.

25 zation of various centrosome proteins to the nuclear envelope.

26 r HMGR isoform, causes ER aggregation at the nuclear envelope.

27 nt mechanism during calcium release from the nuclear envelope.

28 um and also to its extensions comprising the nuclear envelope.

29 einaceous transport channels embedded in the nuclear envelope.

30 ent depend on attachment of telomeres to the nuclear envelope.

31 bnormal distribution of SUN1 proteins on the nuclear envelope.

32 nes are repositioned toward or away from the nuclear envelope.

33 onal TAN line reporter construct, within the nuclear envelope.

34 lation to the lamin expression levels in the nuclear envelope.

35 s and F-actin did not uniformly decorate the nuclear envelope.

36 7p/CHMP7 and downstream ESCRT factors to the nuclear envelope.

37 he transport of progeny viral capsids to the nuclear envelope.

38 mple physical perturbation of a pre-existing nuclear envelope.

39 wnregulation of the diffusion barrier in the nuclear envelope.

40 GRXS17 resides in both the cytoplasm and the nuclear envelope.

41 ficking along microtubules targets it to the nuclear envelope.

42 nge between nucleus and cytoplasm across the nuclear envelope.

43 which are localized to the outer face of the nuclear envelope.

44 ates nucleocytoplasmic transport through the nuclear envelope.

45 physical process mediated by tension in the nuclear envelope.

46 translocating it from the nucleoplasm to the nuclear envelope.

47 hrough the nuclear pores or by rupturing the nuclear envelope.

48 and cells from these patients have aberrant nuclear envelopes.

49 SYNE1 (synaptic nuclear envelope 1) encodes multiple isoforms of Nesprin

50 Chromatin and nuclear envelope A-type lamin proteins are known to be k

51 to growth signals, activation of Pah1 at the nuclear envelope acts as a switch to control the balance

52 s are unable to undergo ciliogenesis and the nuclear envelope adopts the function as cellular microtu

53 composed of a single bilayer that forms the nuclear envelope, along with a network of sheets and dyn

54 r tagging fusion (NTF) protein with an outer nuclear-envelope-anchored domain.

55 e endoplasmic reticulum (ER) consists of the nuclear envelope and a reticulated interconnected networ

56 nucleotide-gated channels are located at the nuclear envelope and are permeable to Ca(2+) We demonstr

57 le into a filamentous meshwork, bridging the nuclear envelope and chromatin.

58 Lamin proteins form a meshwork beneath the nuclear envelope and contribute to many different cellul

59 pha translocated from the nucleoplasm to the nuclear envelope and cytosol but did not associate with

60 d predicts thresholds for the rupture of the nuclear envelope and for nuclear plastic deformation dur

61 ells S1R was identified predominantly in the nuclear envelope and found in the ER as well.

62 We observed localization of HypE at the nuclear envelope and further identified histones H2-H4,

63 nuclear proteins are transported through the nuclear envelope and how the import processes are regula

64 sing a poroelastic material representing the nuclear envelope and inner nucleoplasm, respectively.

65 this process, active G9a is recruited to the nuclear envelope and interacts with lamin B1 during T-ce

66 sociated polypeptide 1 (LAP1) resides at the nuclear envelope and interacts with Torsins, poorly unde

67 the vicinity of the nucleus that encased the nuclear envelope and interfered with nuclear envelope br

68 al how relaxation of external tethers to the nuclear envelope and internal chromatin-chromatin tether

69 ultiple polymers that transmit forces to the nuclear envelope and into the nuclear interior.

70 is for LD biogenesis by translocation to the nuclear envelope and not cytoplasmic LDs.

71 nance of nuclear compartmentalization by the nuclear envelope and nuclear pore complexes (NPCs) is es

72 chromatin remodelers, histone modifiers, and nuclear envelope and pore proteins.

73 na is a filamentous structure subtending the nuclear envelope and required for chromatin organization

74 Nuclear pore complexes (NPCs) perforate the nuclear envelope and serve as the primary transport gate

75 leus and chromatin via physical links on the nuclear envelope and the lamin meshwork.

76 pplying atomic force microscopy (AFM) to the nuclear envelope and the nuclear pore complexes, we demo

77 Telomeres become mobilized from sites on the nuclear envelope and the pericentromere expands after ex

78 n and Cytoskeleton (LINC) complexes span the nuclear envelope and transduce force from dynamic cytosk

79 also promoted CCTalpha translocation to the nuclear envelope and/or cytoplasm but not LDs.

80 SUN/KASH bridges serve as bolts through the nuclear envelope, and nucleoskeleton components LMN-1 an

81 umulates in nuclei, leading to disruption of nuclear envelope architecture, partial sequestration of

82 lear structure, chromosomal organization and nuclear envelope architecture.

83 nsmitted through the cytoskeleton and to the nuclear envelope are important for mechanosensing, inclu

84 of the nuclear lamina (NL) found beneath the nuclear envelope, are known to interact with most of the

85 ucleoplasm, or during somatic and nonsomatic nuclear envelope assembly.

86 in segregation during mitosis and subsequent nuclear envelope assembly.

87 stranded breaks, expanded CAG repeats at the nuclear envelope associate with pores but not with the i

88 ent MTOCs in the division plane (eMTOCs) and nuclear-envelope associated MTOCs in interphase cells (i

89 y a marked decrease in expression of several nuclear envelope-associated components (Lamin B1, Lamin

90 gene expression in keratinocytes and suggest nuclear envelope-associated genes as important targets m

91 ations in the nuclei shape and expression of nuclear envelope-associated proteins were accompanied by

92 P has an N-terminal WPP domain, required for nuclear envelope association and several mitotic locatio

93 stribution of BICD2 and p150 dynactin on the nuclear envelope at prophase, which results in inefficie

94 s caused by a defect in forming a gap in the nuclear envelopes at the interface between the two pronu

95 The integrity of the nuclear envelope barrier relies on membrane remodeling b

96 ent are necessary for the disassembly of the nuclear envelope between the two pronuclei, ultimately a

97 also resulted in failure to disassemble the nuclear envelope between the two pronuclei.

98 ted Myosin on the neuroblast cortex prior to nuclear envelope breakdown (NEB).

99 ribution of the mRNAs to the cytoplasm after nuclear envelope breakdown (NEBD) at prometaphase.

100 sed the nuclear envelope and interfered with nuclear envelope breakdown (NEBD) during cell division.

101 In animal cells, nuclear envelope breakdown (NEBD) is required for proper

102 Nuclear envelope breakdown (NEBD) serves as a major regu

103 C first targets cyclin A2 for degradation at nuclear envelope breakdown (NEBD), we find that in zygot

104 erated exclusively by the kinetochores after nuclear envelope breakdown (NEBD).

105 r data further show that PLK-1 is needed for nuclear envelope breakdown during early embryogenesis.

106 Following nuclear envelope breakdown during mitosis, the viral DNA

107 membrane is compartmentalized shortly before nuclear envelope breakdown into an anterior and a poster

108 KIF11 further fragments the MTOCs following nuclear envelope breakdown so that they can be evenly di

109 From nuclear envelope breakdown until anaphase onset, GTSE1 b

110 on of the retinoblastoma protein and lamins, nuclear envelope breakdown, and duplication of centrosom

111 DNA damage can occur either before or after nuclear envelope breakdown, and provides an effective bl

112 s: pronuclear meeting occurred normally, but nuclear envelope breakdown, centrosome separation, and c

113 not coupled to nuclear osmolytes released by nuclear envelope breakdown, chromatin condensation, or c

114 thways, defined by centrosome maturation and nuclear envelope breakdown, plays any role in spindle as

115 ng the first meiotic division, shortly after nuclear envelope breakdown, translational hotspots devel

116 ssembly of the nuclear lamina and subsequent nuclear envelope breakdown.

117 in interfaces to promote NPC disassembly and nuclear envelope breakdown.

118 destroyed in prometaphase within minutes of nuclear envelope breakdown.

119 ows anillin to be rapidly available when the nuclear envelope breaks down to remodel the cellular arc

120 in photoreceptor cells S1R was found in the nuclear envelope but not localized in the endoplasmic re

121 , we propose that LAP1 targets Torsin to the nuclear envelope by forming an alternating, heterohexame

122 requires Ca(2+) mobilization from the inner nuclear envelope by nuclear InsP3 receptors.

123 -compounded by the occasional rupture of the nuclear envelope-can have important functional consequen

124 sphorylated MDC1 is dynamically localized to nuclear envelopes, centrosomes, kinetochores, and midbod

125 nd nuclear membrane, but it is distinct from nuclear envelope changes that occur during apoptosis and

126 intralumenal vesicle formation, HIV budding, nuclear envelope closure, and cytokinetic abscission.

127 The nuclear envelope, composed of two lipid bilayers and num

128 Unlike other membrane structures, the nuclear envelope comprises two concentric membrane shell

129 egradation of substrates in the ER membrane, nuclear envelope, cytoplasm, and nucleoplasm.

130 nts, FMN2 promotes cell survival by limiting nuclear envelope damage and DNA double-strand breaks.

131 ts phosphorylation state results not only in nuclear envelope defects, including mislocalization of L

132 (KASH) domain to displace nesprins from the nuclear envelope did not abolish Ca(2+)-dependent perinu

133 n but is prompted by release of GCL from the nuclear envelope during mitosis.

134 anizing centers move from centrosomes to the nuclear envelope during muscle development.

135 explain how forces are dissipated across the nuclear envelope during nuclear migration.

136 membrane-tethered AAA+ ATPase implicated in nuclear envelope dynamics as well as the nuclear egress

137 s that localize to the endoplasmic reticular/nuclear envelope (ER/NE) lumen.

138 to play a pivotal role in the maintenance of nuclear envelope/ER homeostasis.

139 ere all telomeres cluster to one pole on the nuclear envelope, facilitating chromosomal pairing and m

140 Nuclear envelope fluctuations are suppressed on the stif

141 w chromosome segregation is coordinated with nuclear envelope formation (NEF), we examined the dynami

142 lar processes: nucleo-cytoplasmic transport, nuclear envelope formation and mitotic spindle assembly.

143 We further found that, during nuclear envelope formation, recruitment of the ESCRT fac

144 smic transport, mitotic spindle assembly and nuclear envelope formation.

145 The nuclear envelope forms a barrier between the cytoplasm,

146 ope with the capability of globally altering nuclear envelope functions in the infected host cell and

147 ed a heterozygous splice site variant in the nuclear envelope gene SYNE1 in a child with severe dilat

148 tomics with high-resolution DamID mapping of nuclear envelope-genome contacts, we show that three mus

149 In recent decades, the nuclear envelope has emerged as a global gene regulatory

150 d vesicle until mitosis is completed and the nuclear envelope has reformed.

151 t vesicle until mitosis is completed and the nuclear envelope has reformed.

152 embrane remodeling by the ESCRTs, which seal nuclear envelope holes and contribute to the quality con

153 A predominant localization of S1R in the nuclear envelope in all three retinal neurons implicates

154 cal abnormalities in the architecture of the nuclear envelope in cells expressing expanded G4C2 repea

155 proteins have been implicated in sealing the nuclear envelope in mammals, spindle pole body dynamics

156 n replication progression associate with the nuclear envelope in sap1 mutant cells.

157 the apical membrane in epithelial cells, the nuclear envelope in skeletal muscle, and down the length

158 n bipolar cells S1R was detected only in the nuclear envelope, in ganglion cells S1R was identified p

159 ER junctions, and mainly build up beside the nuclear envelope, indicating conserved OSER biogenesis i

160 association of a single gene locus with the nuclear envelope influences the surrounding chromosome a

161 is to promote spindle assembly by modulating nuclear envelope integrity at the onset of mitosis.

162 Hallmarks of aged cells include compromised nuclear envelope integrity, impaired nucleocytoplasmic t

163 endogenous DNA release upon perturbation of nuclear envelope integrity.

164 Herpesvirus nucleocapsids traverse the nuclear envelope into the cytoplasm in a process called

165 ear size and less frequent intrusions of the nuclear envelope into the nuclear lumen indicated altere

166 surviving neurons feature numerous and deep nuclear envelope invaginations, a hallmark of cellular s

167 The nuclear envelope is a unique topological structure forme

168 Dynein anchored on the nuclear envelope is known to be important for centrosome

169 stable, recent studies demonstrate that the nuclear envelope is prone to rupture.

170 Dynein recruitment to the nuclear envelope is required for pre-mitotic nucleus-cen

171 onstitutively active kinesin-1 motors to the nuclear envelope is sufficient to prevent the nuclear ag

172 eyond its role in providing structure to the nuclear envelope, lamin A/C is involved in transcription

173 bilities can define the global topology of a nuclear envelope-like structure.

174 urthermore, we demonstrate a requirement for nuclear envelope LINC (linker of nucleoskeleton and cyto

175 dent of their kinetochore, spindle pole, and nuclear envelope localization.

176 In this study, we report that the nuclear envelope-localized AAA+ (ATPase associated with

177 s bud at the inner nuclear membrane into the nuclear envelope lumen.

178 disease, and suggest that dysfunction of the nuclear envelope may be an under-recognized component of

179 with findings in mice, marked alterations in nuclear envelope morphology, abnormal localization of Ra

180 microtubule (MT) nucleation activity at the nuclear envelope (NE) [1-4].

181 The nuclear envelope (NE) and endoplasmic reticulum (ER) are

182 NPCs penetrate the nuclear envelope (NE) and regulate the nucleocytoplasmic

183 d spindle pole body (SPB) is embedded in the nuclear envelope (NE) at fusion sites of the inner and o

184 Using nuclear envelope (NE) blebbing as a phenotypic measure,

185 me propagation requires coordination between nuclear envelope (NE) breakdown, spindle formation, and

186 that differ in the timing and the extent of nuclear envelope (NE) breakdown.

187 ink a novel mechanism for RNA export through nuclear envelope (NE) budding [4, 5] that requires A-typ

188 te for nuclear escape, a process referred to nuclear envelope (NE) budding.

189 The nuclear envelope (NE) consists of two evenly spaced bila

190 d cytoskeleton (LINC) complexes spanning the nuclear envelope (NE) contribute to nucleocytoskeletal f

191 ly been shown to seal holes in the reforming nuclear envelope (NE) during mitotic exit [2, 3].

192 mechanisms regulating nuclear morphology and nuclear envelope (NE) expansion are poorly understood.

193 Nuclear deformation caused localized loss of nuclear envelope (NE) integrity, which led to the uncont

194 The nuclear envelope (NE) is critical for numerous fundament

195 f green fluorescent protein (GFP)-BAF at the nuclear envelope (NE) is elevated, suggesting that prolo

196 vide evidence that PLK-1 localization to the nuclear envelope (NE) is required for efficient NEBD.

197 The nuclear envelope (NE) of eukaryotic cells not only serve

198 The nuclear envelope (NE) presents a physical boundary betwe

199 During telophase, the nuclear envelope (NE) reforms around daughter nuclei to

200 fungi undergo a closed mitosis in which the nuclear envelope (NE) remains intact.

201 treated cells are protected from ETO-induced nuclear envelope (NE) rupture and DNA leakage through in

202 Repeated rounds of nuclear envelope (NE) rupture and repair have been obser

203 In each case, dynein is recruited to the nuclear envelope (NE) specifically during G2 via two nuc

204 ombe undergoes "closed" mitosis in which the nuclear envelope (NE) stays intact throughout chromosome

205 envelope spectrin 1) that associate with the nuclear envelope (NE) through a C-terminal KASH (Klarsic

206 , in that it translocates from the ER to the nuclear envelope (NE) to recruit chromatin-remodeling mo

207 the outer nuclear membrane that connect the nuclear envelope (NE) to the cytoskeleton.

208 the changes that occur in cell adhesion and nuclear envelope (NE) topography, during necrosis and ap

209 Although a plethora of nuclear envelope (NE) transmembrane proteins (NETs) have

210 trimers or partially disassembled AdV at the nuclear envelope (NE) was observed in digitonin-permeabi

211 Here we extend brightness analysis to the nuclear envelope (NE), a double membrane barrier separat

212 ilar to MX2 but not MX1, can localize to the nuclear envelope (NE), linking HIV-1 inhibition with MX

213 The half bridge is a SPB substructure on the nuclear envelope (NE), playing a key role in SPB duplica

214 Interactions occurring at the nuclear envelope (NE)-chromatin interface influence both

215 teins predominantly known to function at the nuclear envelope (NE).

216 ate important morphogenesis processes at the nuclear envelope (NE).

217 lminate in insertion of the new SPB into the nuclear envelope (NE).

218 n assemblies at the plasma membrane (PM) and nuclear envelope (NE).

219 -Cytoskeleton (LINC) bridging complex at the nuclear envelope (NE).

220 calized to the ER membrane, primarily to the nuclear envelope (NE).

221 er SPB (mSPB) on the cytoplasmic side of the nuclear envelope (NE).

222 ed to prevent the aggregation of NPCs in the nuclear envelope near centrosomes in late G2 and prophas

223 nimal cells disassemble and reassemble their nuclear envelopes (NEs) upon each division.

224 specific defects in F-actin coupling to the nuclear envelope, nuclear movement, and the ability of c

225 ganelles or regions, including nucleoli, the nuclear envelope, nuclear speckles, centrosomes, mitocho

226 gen Chlamydia psittaci, uniquely targets the nuclear envelope of C. psittaci-infected cells and uninf

227 rm gateways for material transfer across the nuclear envelope of eukaryotic cells.

228 Before the first zygotic division, the nuclear envelopes of the maternal and paternal pronuclei

229 tion approach that uses fluorescently tagged nuclear envelope or endoplasmic reticulum membrane marke

230 ata demonstrate functional links between the nuclear envelope organization, chromatin architecture, a

231 und the nucleoli to be stiffer than both the nuclear envelope (p < 0.0001) and the surrounding cytopl

232 Cell death events including acidification, nuclear envelope permeabilization, and DNA fragmentation

233 Specifically, the nuclear envelope plays a critical role in gene regulatio

234 Mutations in the synaptic nuclear envelope protein 1 (SYNE1) gene have been report

235 als of cells that overexpress the GFP-tagged nuclear envelope protein lamin A.

236 ease cell stiffness by overexpression of the nuclear envelope protein lamin A.

237 Here, we identified the nuclear envelope protein nesprin-2 as a binding partner

238 des lamina-associated protein LAP-1, myocyte nuclear envelope protein Syne1, BetaM itself, heme oxida

239 ol transcription of the gene encoding LBR, a nuclear envelope protein that is required for the charac

240 ute in part, to improper localisation of the nuclear envelope protein TPR.

241 We show that the Arabidopsis nuclear envelope protein, CPR5, negatively regulates ETI

242 nesin light chain (KLC)-binding motif in the nuclear envelope proteins nesprin-1 and nesprin-2, and s

243 oted through chromosome movement mediated by nuclear envelope proteins, microtubules, and dynein.

244 rs (PCs), cis-acting loci that interact with nuclear envelope proteins, such as SUN-1, to access cyto

245 In so doing, the nuclear envelope provides a physical barrier between chr

246 o anchor the viral PIC in the nucleus as the nuclear envelope re-forms postmitosis.

247 sical separation of sister chromatids before nuclear envelope reassembly (NER).

248 f cytokinesis and its duration is coupled to nuclear envelope reassembly and the nuclear sequestratio

249 chromatid separation checkpoint" that delays nuclear envelope reassembly and, consequently, Pebble nu

250 Trailing chromatids induce a delay in nuclear envelope reassembly concomitant with prolonged c

251 here is a decrease in the frequency of local nuclear envelope reassembly delays, resulting in an incr

252 Localized delays of nuclear envelope reassembly require Aurora B kinase acti

253 actions, we explored the role of LEM2 during nuclear envelope reformation in human cells.

254 am, thereby influencing spindle disassembly, nuclear envelope reformation, and cytokinesis.

255 burst of transcription that occurs following nuclear envelope reformation.

256 raction of defective nuclear pore complexes; nuclear envelope reformation; plus-stranded RNA virus re

257 nalling, RanBP2 co-localizes with SHP at the nuclear envelope region and mediates SUMO2 modification

258 However, isoforms that function beyond the nuclear envelope remain poorly examined.

259 ng the correct polarity of LD budding at the nuclear envelope, restricting it to the outer membrane.

260 ned contractility causes nuclear dysmorphia, nuclear envelope rupture and genome instability.

261 fter anaphase, the chromatin bridges induced nuclear envelope rupture in interphase, accumulated the

262 We detect actin filaments at nuclear envelope rupture sites and define the Rho-ROCK p

263 R12A or PPP1CB causes nuclear fragmentation, nuclear envelope rupture, nuclear compartment breakdown

264 sting that the sealing of defective NPCs and nuclear envelope ruptures could proceed through similar

265 Cells exhibit transient nuclear envelope ruptures during interphase, but the res

266 repeat (SR) proteins, predominantly known as nuclear envelope scaffolds.

267 ed kinetochores were not associated with the nuclear envelope, so Mad1 does not anchor them to nuclea

268 UN2, which interact transluminally to form a nuclear envelope-spanning linker molecular bridge known

269 We implicate a Schizosaccharomyces pombe nuclear envelope-spanning linker of nucleoskeleton and c

270 pe 1) encodes multiple isoforms of Nesprin1 (nuclear envelope spectrin 1) that associate with the nuc

271 SCRT-II/III chimera, Chm7, is recruited to a nuclear envelope subdomain that expands upon inhibition

272 nuclei was redesigned by replacing the outer nuclear-envelope-targeting domain of the nuclear tagging

273 nt experiments, may result in rupture of the nuclear envelope that can lead to cell death, if not pre

274 These delays result in a gap in the nuclear envelope that facilitates the inclusion of laggi

275 s could play a role in the remodeling of the nuclear envelope that takes place during the mitotic cyc

276 matin channels capsids are able to reach the nuclear envelope, the site of their nuclear egress.

277 ng for the passage of progeny viruses to the nuclear envelope, their site of nuclear egress.

278 In addition to the nuclear envelope, there exists a perinuclear region (PNR

279 highly condensed heterochromatic DNA to the nuclear envelope, thereby establishing the three-dimensi

280 on of lamin A, a structural component of the nuclear envelope, thereby promoting the release of DNA i

281 nic cells, AL are rapidly delivered into the nuclear envelope through fenestrations, highlighting the

282 in-dependent recruitment of kinesin-1 to the nuclear envelope through the interaction of a conserved

283 omoting the formation of a passageway in the nuclear envelope through which late-segregating acentric

284 how that LINC complexes also signal from the nuclear envelope to critical regulators of the actin cyt

285 at an ensemble of Kif5B motors acts from the nuclear envelope to distribute nuclei throughout the len

286 s from activated immune receptors across the nuclear envelope to intranuclear targets.

287 ay from PI production in the vicinity of the nuclear envelope to prevent excess ER sheet formation an

288 can exit the division without reforming the nuclear envelope, to uncover an intriguing role of the n

289 The normal distribution of nuclear envelope transmembrane proteins (NETs) is disrup

290 ulating gene positions through targeting the nuclear envelope transmembrane proteins (NETs) that dire

291 nuclear membrane during HSV-1 infection and nuclear envelope vesiculation in NEC-transfected cells.

292 Morphology of the nuclear envelope was assessed with immunofluorescence on

293 SINC localization at the nuclear envelope was blocked by importazole, confirming

294 ibe its RNA genome to DNA and traffic to the nuclear envelope, where the viral genome is translocated

295 lear pore complexes (NPCs) in the interphase nuclear envelope, whereas deletion of B-type lamins resu

296 The nuclear envelope, which can be modeled as a double lipid

297 the proteasome and its anchor, Cut8, at the nuclear envelope, which in turn regulates proteostasis o

298 t factors (TFs) and their cargoes across the nuclear envelope, while blocking the passage of other ma

299 s and worms, utilizes in vivo tagging of the nuclear envelope with biotin and the subsequent affinity

300 s a novel bacterial protein that targets the nuclear envelope with the capability of globally alterin