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1 to the mitotic cytoplasm around the time of nuclear envelope breakdown.
2 ssembly of the nuclear lamina and subsequent nuclear envelope breakdown.
3 destroyed in prometaphase within minutes of nuclear envelope breakdown.
4 T) capture typically occur within minutes of nuclear envelope breakdown.
5 sassembly was not due to a general defect in nuclear envelope breakdown.
6 nuclear protein import and plays roles after nuclear envelope breakdown.
7 in interfaces to promote NPC disassembly and nuclear envelope breakdown.
8 es at the nuclear periphery and an arrest of nuclear envelope breakdown.
9 dely used in vitro to study the mechanics of nuclear envelope breakdown.
10 upon nuclear entry of cyclin B1, but before nuclear envelope breakdown.
11 o localizes to unattached kinetochores after nuclear envelope breakdown.
12 ow that centrosomes have a role in promoting nuclear envelope breakdown.
13 chromosome condensation from late G2 through nuclear envelope breakdown.
14 followed by accumulation in the nucleus then nuclear envelope breakdown.
15 re disassembly begins several minutes before nuclear envelope breakdown.
16 change in turn is approximately 30 s before nuclear envelope breakdown.
17 ctivate nuclear centering, DNA synthesis, or nuclear envelope breakdown.
18 cated by lack of chromosome condensation and nuclear envelope breakdown.
19 gets critical for chromatin condensation and nuclear envelope breakdown.
20 microtubules and F-actin, disassembles upon nuclear-envelope breakdown.
21 maintain long centrosomal microtubules after nuclear-envelope breakdown.
22 ization with perinuclear microtubules before nuclear envelope breakdown, after which it congresses in
25 the sperm chromatin after pronuclear fusion, nuclear envelope breakdown and formation of a bipolar sp
26 vertebrates, the addition of HEI10 inhibits nuclear envelope breakdown and mitotic entry in Xenopus
27 sis inhibitor, injection of CycB accelerates nuclear envelope breakdown and mitotic remodeling of the
28 iation with kinetochores appeared soon after nuclear envelope breakdown and persisted until late anap
30 Golgi, has been implicated in the process of nuclear envelope breakdown and requires interactions at
31 uring mitosis, fluorescent tracers that mark nuclear envelope breakdown and the subsequent reformatio
32 cyte cytoskeleton during prophase I prior to nuclear envelope breakdown, and (2) extensive depolymeri
33 on of the retinoblastoma protein and lamins, nuclear envelope breakdown, and duplication of centrosom
34 DNA damage can occur either before or after nuclear envelope breakdown, and provides an effective bl
35 leus during interphase, the cortex following nuclear envelope breakdown, and the cleavage furrow duri
36 s and orients a pair of centrosomes prior to nuclear envelope breakdown, and the spindle assembles be
37 hroughout prophase, but is disassembled upon nuclear-envelope breakdown as the mitotic spindle forms.
38 ould then be in a position to participate in nuclear envelope breakdown, as described in recent studi
39 characterized by chromosome recondensation, nuclear envelope breakdown, assembly of microtubules int
40 WAVE1 redistributes to the cytoplasm upon nuclear envelope breakdown at mitosis, and concentrates
41 before fertilization caused arrest prior to nuclear envelope breakdown at much lower concentrations
42 pombe undergoes closed mitosis but 'virtual nuclear envelope breakdown' at anaphase of meiosis II, i
43 cell cycle and a tendency to arrest prior to nuclear envelope breakdown, at metaphase and at cytokine
44 s: pronuclear meeting occurred normally, but nuclear envelope breakdown, centrosome separation, and c
45 not coupled to nuclear osmolytes released by nuclear envelope breakdown, chromatin condensation, or c
46 clusters appeared transiently at the time of nuclear envelope breakdown, disappeared at the time of f
47 r data further show that PLK-1 is needed for nuclear envelope breakdown during early embryogenesis.
48 ins, and Ce-lamin to determine the timing of nuclear envelope breakdown during mitosis in C. elegans.
50 port a role for the COPI coatomer complex in nuclear envelope breakdown, implicating vesiculation as
51 COPI and dominantly inhibits progression of nuclear envelope breakdown in an assay that robustly rec
54 ystem that recapitulates CDK1 activation and nuclear envelope breakdown in response to mitotic cyclin
55 depletion of Myt1 in Xenopus oocytes causes nuclear envelope breakdown in vitro, we found that deple
56 We propose a new model for the mechanism of nuclear envelope breakdown in which disassembly of the n
57 membrane is compartmentalized shortly before nuclear envelope breakdown into an anterior and a poster
60 apturing microtubules, signals released upon nuclear envelope breakdown may activate proteins like Su
61 sis biosensor that monitors the time between nuclear envelope breakdown (NEB) and re-formation using
62 , these cells display mitotic collapse after nuclear envelope breakdown (NEB) characterized by defect
64 ems, the possibility of poleward flux before nuclear envelope breakdown (NEB) has not been examined.
73 sed the nuclear envelope and interfered with nuclear envelope breakdown (NEBD) during cell division.
74 microtubules gain access to chromatin after nuclear envelope breakdown (NEBD) during meiotic maturat
75 pendent role for Aurora A and centrosomes in nuclear envelope breakdown (NEBD) during the first mitot
76 irectly observed from prophase to just after nuclear envelope breakdown (NEBD) in early prometaphase.
80 positioning of duplicated centrosomes after nuclear envelope breakdown (NEBD), thereby preventing th
81 C first targets cyclin A2 for degradation at nuclear envelope breakdown (NEBD), we find that in zygot
86 a role for this pore protein in coordinating nuclear envelope breakdown, Nup358-specific antibodies i
90 thways, defined by centrosome maturation and nuclear envelope breakdown, plays any role in spindle as
91 of Golgi spots begins to decline soon after nuclear envelope breakdown, reaches a minimum soon after
92 KIF11 further fragments the MTOCs following nuclear envelope breakdown so that they can be evenly di
93 h-18 mutant oocytes exhibit defects prior to nuclear envelope breakdown, suggesting that they are phy
94 her Nup153 or Nup358 for inhibition perturbs nuclear envelope breakdown, supporting a model in which
95 aster labeling was constant from the time of nuclear envelope breakdown, the kinetochore labeling fir
96 ted pronuclear centering, DNA synthesis, and nuclear envelope breakdown; there appeared to be a thres
97 G2 phase and activated at a set time before nuclear envelope breakdown, thereby initiating the event
98 nucleus during late prophase (<30 min before nuclear envelope breakdown) they progress normally throu
100 of metaphase cells, and traversal times from nuclear envelope breakdown to anaphase, and an override
102 sphodynein associates with kinetochores from nuclear envelope breakdown to metaphase, but bioriented
103 rocesses ranging from vesicular transport to nuclear envelope breakdown to mitotic spindle alignment.
104 ng the first meiotic division, shortly after nuclear envelope breakdown, translational hotspots devel
107 in mitosis, KAP-GFP moved into nuclei before nuclear envelope breakdown, was again present in nuclei
108 the nuclear envelope, a process analogous to nuclear envelope breakdown, which occurs at the onset of
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