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2 tudy reveals that miR-144 directly regulates nuclear factor-erythroid 2-related factor 2, a central r
3 vels of estrogen receptor-alpha activity and nuclear factor erythroid 2-related factor 2 activity, an
4 regulate Slc7a11 expression by activation of nuclear factor erythroid 2-related factor 2 and transcri
5 e master antioxidant regulatory factor Nrf2 (nuclear factor erythroid 2-related factor 2) and its tar
6 xygenases, 12/15-LOs, nuclear factor-kappaB, nuclear factor-erythroid 2-related factor-2, and signal
8 ctivator of p62 through competition of Nrf2 (nuclear factor erythroid 2-related factor 2) for Keap1 b
9 induced production of HO-1 via activation of nuclear factor erythroid 2-related factor 2-, IL-10-, an
10 ween RAC1 and the transcription factor NRF2 (nuclear factor erythroid 2-related factor 2), master reg
11 none 1; constitutive androstane receptor; or nuclear factor erythroid 2-related factor 2 mRNA express
12 eases, liver X receptor/retinoid X receptor, nuclear factor erythroid 2-related factor 2, notch, and
14 ion through reactive oxygen species-mediated nuclear factor erythroid 2-related factor 2 (NRF2) activ
15 ICAM) (P < 0.001, respectively) and improved nuclear factor erythroid 2-related factor 2 (Nrf2) and h
16 increased ROS, and subsequent activation of nuclear factor erythroid 2-related factor 2 (Nrf2) antio
18 ified Sesn2 and the antioxidant gene inducer nuclear factor erythroid 2-related factor 2 (Nrf2) as po
19 nd colleagues demonstrated that induction of nuclear factor erythroid 2-related factor 2 (NRF2) enhan
20 nalyses have uncovered an essential role for nuclear factor erythroid 2-related factor 2 (Nrf2) in re
21 tified an increased nuclear translocation of nuclear factor erythroid 2-related factor 2 (NRF2) in th
28 xidant production through down-regulation of nuclear factor erythroid 2-related factor 2 (Nrf2) pathw
29 rough the antioxidant response element (ARE)-nuclear factor erythroid 2-related factor 2 (Nrf2) pathw
30 her the redox-sensitive transcription factor nuclear factor erythroid 2-related factor 2 (Nrf2) plays
31 of the redox-sensitive transcription factor nuclear factor erythroid 2-related factor 2 (Nrf2) plays
33 tudy sought to determine factors involved in nuclear factor erythroid 2-related factor 2 (Nrf2) regul
36 on in renal proximal tubule cells (RPTCs) on nuclear factor erythroid 2-related factor 2 (Nrf2) stimu
37 were uniquely characterized by activation of nuclear factor erythroid 2-related factor 2 (NRF2) targe
38 response element (ARE) binding activity from nuclear factor erythroid 2-related factor 2 (Nrf2) to c-
39 by activating receptor/transcription factor nuclear factor erythroid 2-related factor 2 (Nrf2) to co
44 ivating the aryl hydrocarbon receptor (AhR), nuclear factor erythroid 2-related factor 2 (Nrf2), and
45 cancer cells attenuated GSH/GSSG, total GSH, nuclear factor erythroid 2-related factor 2 (Nrf2), and
46 ion of FST transcription was mediated by the nuclear factor erythroid 2-related factor 2 (Nrf2), as e
50 lecule activator of the transcription factor nuclear factor erythroid 2-related factor 2 (NRF2), was
52 show that vitiligo melanocytes have impaired nuclear factor erythroid 2-related factor 2 (Nrf2)-antio
56 ess induced by S. pneumoniae and the role of nuclear factor erythroid 2-related factor 2 (Nrf2)-media
57 ts of GDM on the proteome, redox status, and nuclear factor erythroid 2-related factor 2 (Nrf2)-media
58 ardoxolone methyl, a potent activator of the nuclear factor erythroid 2-related factor 2 (Nrf2)-media
60 Lack of NO formation resulted in impaired nuclear factor erythroid 2-related factor-2 (Nrf2) expre
64 KEAP1), which targets transcriptional factor nuclear factor erythroid-2-related factor 2 (NRF2) for d
68 e present study, we investigated the role of nuclear factor erythroid-2-related factor 2 (Nrf2), a ma
69 ancer cell lines via focal amplification and nuclear factor erythroid-2-related factor 2 (NRF2)-media
70 r advanced glycation end products (RAGE) via nuclear factor erythroid-2-related-factor-2 (Nrf2)-depen
73 is described based on the Keap1-independent Nuclear Factor-erythroid 2-related factor 2 (Nrf2) signa
76 sue of the JCI, Thimmulappa et al. show that nuclear factor-erythroid 2-related factor 2 (Nrf2), a me
80 xidative stress suppressed the expression of nuclear factor-erythroid-2-related factor 2 (Nrf2), an i
82 rotective activity through the activation of nuclear factor erythroid 2-related factor 2 pathway, sca
83 Together, these data suggest that suboptimal nuclear factor erythroid 2-related factor 2-regulated de
84 -activated protein kinase (AMPK) pathway and nuclear factor-erythroid 2-related factor 2 target genes
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