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1 ough effectors such as calcineurin and NFAT (nuclear factor of activated T cells).
2 biting the transcriptional activity of NFAT (nuclear factor of activated T cells).
3 PPAR gamma and suppression of NF-kappaB and nuclear factor of activated T cells.
4 , cAMP response element-binding protein, and nuclear factor of activated T cells.
5 )/Ca(2+)-dependent manner but independent of nuclear factor of activated T cells.
6 uclear translocation of the Ca(2+)-regulated nuclear factor of activated T cells.
7 n of the transcription factors NF-kappaB and nuclear factor of activated T cells.
8 II subset, leading to phosphorylation of the nuclear factor of activated T cells.
9 ires calcium flux and activation of PI3K and nuclear factor of activated T cells.
10 signals downstream of the TCR, including the nuclear factor of activated T cells.
11 ibit its dephosphorylation and activation of nuclear factor of activated T cells.
12 appaB, AP-1 (activator protein 1), and NFAT (nuclear factor of activated T) cells.
13 play enhancement of Orai1, STIM1, STIM2, and nuclear factor of activated T cells 1 (NFAT1) expression
14 phosphorylation and nuclear translocation of nuclear factor of activated T cells 1 (NFAT1), a substra
15 or of kappaB (IkBalpha) phosphorylation, and nuclear factor of activated T cells 1 (NFATc1) expressio
17 the expression and nuclear translocation of nuclear factor of activated T cells 1, a master transcri
20 have identified an autocrine pathway linking nuclear factor of activated T cells 2 (NFAT1), autotaxin
21 The Ca(2+) -dependent transcription factors, nuclear factor of activated T cells 2 (NFAT2) and NFAT4,
22 egression, the intronic rs6021191 variant in nuclear factor of activated T cells 2 (NFATC2) had the s
23 CaMKIV) activation, and interactions between nuclear factor of activated T cells 3 (NFATc3) and FosB
24 slocation of the transcription factor NFAT3 (nuclear factor of activated T cells 3) in cultured myocy
27 hy indicate that DRBP76 heterodimerizes with nuclear factor of activated T cells, 45 kDa (NF45), in n
28 that the osmoprotective transcription factor nuclear factor of activated T cells 5 (NFAT5) critically
29 ions activate the p38/MAPK pathway involving nuclear factor of activated T cells 5 (NFAT5; also calle
31 ion from the IL-4 promoter and from isolated nuclear factor of activated T cells/activating protein-1
32 e T cell receptor-mediated calcium response, nuclear factor of activated T cells activation, and cyto
34 cancer cells has been associated with NFAT1 (nuclear factor of activated T cells) activation and expr
35 1beta (10 ng/ml) recruited calcineurin/NFAT (nuclear factor of activated T cells) activation in prima
36 phospholipase C-gamma 1 and decreases NFAT (nuclear factor of activated T cells) activation upon eng
37 e GM-CSF enhancer that are recognized by the nuclear factor of activated T cells.activator protein 1
38 in-associated protein-70 phosphorylation and nuclear factor of activated T cells/activator protein 1
39 or of nuclear factor kappa B-ligand; NFATc1, nuclear factor of activated T-cells; ADAM, a disintegrin
42 receptor-dependent signaling events, such as nuclear factor of activated T cells and AP-1 transcripti
43 uent activation of the transcription factors nuclear factor of activated T cells and hypoxia-inducibl
44 the activation of the transcription factors nuclear factor of activated T cells and NF-kappaB is imp
45 ription factors crucial for IL-2 expression, nuclear factor of activated T cells and nuclear factor k
46 or-1alpha and hypoxia-induced factor-2alpha, nuclear factor of activated T cells and survivin, and si
47 ted transcription factor activation, such as nuclear factor of activated T-cells and serum response e
48 ession of alpha-actin and transactivation of nuclear factors of activated T cells and serum response
49 ng pathway, which in turn, activates NFATc1 (nuclear factor of activated T-cells) and downstream gene
50 ses [ERKs]), inhibition of calcineurin-NFAT (nuclear factor of activated T cells), and induction of B
52 o immunoglobulins, B cells, T-cell receptor, nuclear factor of activated T cells, and cytokine and ch
53 tained in time by the activation of miR-204, nuclear factor of activated T cells, and hypoxia-inducib
54 NF-kappaB (nuclear factor kappaB) and NFAT (nuclear factor of activated T-cells), as well as potenti
55 r oncogenes, which are overexpressed in PAH: nuclear factor of activated T cells, B-cell lymphoma 2,
56 motes the expression of oncogenes, including nuclear factor of activated T cells, B-cell lymphoma 2,
57 ve pathway probably includes calcineurin and nuclear factor of activated T cells, because A(2B) recep
59 acellular-regulated kinases 1/2 (ERK1/2) and nuclear factor of activated T cells c1 (NF-ATc1), consis
60 ammation depends on T-cell receptor-mediated Nuclear Factor of Activated T cells c1 (NFATc1) activati
63 onstrate that cartilage-specific ablation of Nuclear factor of activated T cells c1 (Nfatc1) in Nfatc
65 he essential osteoclast transcription factor nuclear factor of activated T cells c1 (NFATc1) or its n
66 sulting in selective and robust induction of nuclear factor of activated T cells c1 (NFATc1), a Ca(2+
67 clasts was caused by decreased expression of nuclear factor of activated T cells c1 (NFATc1), a maste
68 eoclastogenesis by differentially regulating nuclear factor of activated T cells c1 (NFATc1), activat
69 sphatase (TRAP) gene promoter and suppresses nuclear factor of activated T cells c1 (NFATc1)-induced
72 a enhanced CD154 transcription mediated by a Nuclear Factor of Activated T cells c1 (NFATc1)/Nuclear
73 ivation, IL-25 potentiates expression of the nuclear factor of activated T cells c1 and JunB transcri
74 with other noncytokine receptors to modulate nuclear factor of activated T cells c1 and to promote op
75 y with CD28, is essential for high levels of nuclear factor of activated T cells c1 to accumulate in
77 hat cooperates with the transcription factor nuclear factor of activated T cells, c1 (NFATc1) to driv
78 the expression of the OC genes encoding the nuclear factor of activated T-cells, c1 (NFATc1), cathep
79 scription factors and intronic activation by nuclear factor of activated T-cells-c2 act in concert to
80 E-1) and increased expression and binding of nuclear factor of activated T-cells-c2 to consensus sequ
81 determinant of BKCa channel remodeling, CaN/nuclear factor of activated T-cells c3 (NFATc3) activati
82 cineurin (CaN), and the transcription factor nuclear factor of activated T-cells c3 (NFATc3) as requi
83 lian target of rapamycin complex 1 (mTORC1), nuclear factor of activated T cells -c3 (NFATc3), and br
86 ast, only 3 of 5 were able to activate NFAT (nuclear factor of activated T cells); chimpanzee and ora
88 r-TCR and increased nuclear translocation of nuclear factor of activated T cells, consistent with TCR
89 g the master regulator of osteoclastogenesis nuclear factor of activated T cells cytoplasmic 1 (NFATc
91 1 during formation of HFs and those encoding nuclear factor of activated T-cells cytoplasmic calcineu
92 t to be mediated by the transcription factor nuclear factor of activated T cells, cytoplasmic 1 (NFAT
93 eated OCs by up-regulating the expression of nuclear factor of activated T cells, cytoplasmic 1 (Nfat
94 the master regulator of osteoclastogenesis (nuclear factor of activated T cells, cytoplasmic 1) by a
96 at suppresses the expression and activity of nuclear factor of activated T cells, cytoplasmic, calcin
97 NFI switch program via activation of the CaN/nuclear factor of activated T-cells, cytoplasmic (NFATc)
98 tion stimulated nuclear translocation of the nuclear factor of activated T-cells, cytoplasmic 1 (NFAT
99 (Ca(2+)) oscillations are indispensable for nuclear factor of activated T-cells, cytoplasmic 1 (NFAT
100 )-induced osteoclast markers, such as c-Fos, nuclear factor of activated T-cells, cytoplasmic, calcin
101 6) and/or c-fos signaling to induce NFATc1 (nuclear factors of activated T cells, cytoplasmic 1).
102 TL were signaled via a Ca(2+), p56(lck), and nuclear factor of activated T cells-dependent pathway.
103 ds, and CNB1, the essential component of the nuclear factor of activated T cells dephosporylation com
104 1, protein kinase Czeta, calcium-calcineurin-nuclear factor of activated T cells, epidermal growth fa
107 e leads to the efficient activation of NFAT (nuclear factor of activated T cells) family transcriptio
108 compounds, antiviral 6 (AV6), which required nuclear factor of activated T cells for early mRNA expre
109 blast fusion and associated with compromised nuclear factor of activated T cells import and activity.
111 ineurin-dependent transcription factor NFAT (nuclear factor of activated T-cells) is implicated in re
114 GF), E-cadherin, SRY-box 7 (SOX7), and NFAT (nuclear factor of activated T cells) kinase dual-specifi
116 in (CN)-dependent transcription factor NFAT (Nuclear Factor of Activated T cells) mediates deleteriou
117 ZBTB25 functions as a negative regulator of nuclear factor of activated T cells (NF-AT) activation,
118 active Ad-IkappaB, and in some cases by the nuclear factor of activated T-cells (NF-AT) inhibitor, c
119 the antigen receptor response element (ARRE)/nuclear factor of activated T-cells (NF-AT) target DNA s
121 transgenic mice showed increased calcineurin-nuclear factor of activated T cells (NFAT) activation in
122 d to leukemogenesis through dysregulation of nuclear factor of activated T cells (NFAT) activation.
123 utation of this residue enhanced TCR-induced nuclear factor of activated T cells (NFAT) activation.
124 ibrosis, cardiac dysfunction and calcineurin-nuclear factor of activated T cells (NFAT) activity afte
125 effect of DLEU7 expression on NF-kappaB and nuclear factor of activated T cells (NFAT) activity.
126 4/MKK7 pathway, resulting in greater cardiac nuclear factor of activated T cells (NFAT) activity.
127 demonstrated that ATRAP knockdown increased nuclear factor of activated T cells (NFAT) activity.
129 beta cells was dependent on calcineurin (CN)/nuclear factor of activated T cells (NFAT) and MAPK sign
130 from scurfy mice have a dramatic increase in nuclear factor of activated T cells (NFAT) and NF-kappa
131 through ROS and NF-kappaB without affecting nuclear factor of activated T cells (NFAT) and PKC, demo
132 activation to produce mROS for activation of nuclear factor of activated T cells (NFAT) and subsequen
133 nished capacity to inhibit the activation of nuclear factor of activated T cells (NFAT) and suppress
134 d mutation reporter constructs revealed that nuclear factor of activated T cells (NFAT) and the cycli
135 tabolic regulators through the activation of nuclear factor of activated T cells (NFAT) and the PI3K-
136 in turn, augmented nuclear translocation of nuclear factor of activated T cells (NFAT) and Treg prol
137 oduced a truncated fluorescent derivative of nuclear factor of activated T cells (NFAT) as a real-tim
138 c hypertrophy involves the activation of the nuclear factor of activated T cells (NFAT) because GRK5
140 oth calcium release and dephosphorylation of nuclear factor of activated T cells (NFAT) but maintaine
142 involving the oncogenic transcription factor nuclear factor of activated T cells (NFAT) c2 targeting
144 ions of the regulatory domain of the protein nuclear factor of activated T cells (NFAT) caused by pho
145 s an oncogene, is under negative calcineurin/nuclear factor of activated T cells (NFAT) control and i
146 rom controls and lacked Ca(2+) oscillations, nuclear factor of activated T cells (NFAT) dephosphoryla
147 activation of protein synthesis involves the nuclear factor of activated T cells (NFAT) family of tra
149 osphatase required for the activation of the nuclear factor of activated T cells (NFAT) family of tra
151 ty regulates the nuclear localization of the nuclear factor of activated T cells (NFAT) family of tra
155 d pharmacological suppression of calcineurin/nuclear factor of activated T cells (NFAT) function prom
157 d calcineurin-dependent transcription factor nuclear factor of activated T cells (NFAT) in developmen
158 in WT cells is prevented by the calcineurin-nuclear factor of activated T cells (NFAT) inhibitor, cy
160 merous transcriptional activators, including nuclear factor of activated T cells (NFAT) isoforms, the
161 ut had no effect on G1-mediated signaling to nuclear factor of activated T cells (NFAT) luciferase.
163 ease in [Ca2+]i correlates with constitutive nuclear factor of activated T cells (NFAT) nuclear trans
164 dependent on protein kinase A (PKA) and the nuclear factor of activated T cells (NFAT) P1 transcript
165 hrough the T-cell receptor (TCR)/calcineurin/nuclear factor of activated T cells (NFAT) pathway impai
167 sing the Dunlop strain of BKV, we found that nuclear factor of activated T cells (NFAT) plays an impo
168 phatase calcineurin and transcription factor nuclear factor of activated T cells (NFAT) plays an impo
174 ic and extrinsic pathways and is involved in nuclear factor of activated T cells (NFAT) signaling and
175 sporine A (CsA) stems from the inhibition of nuclear factor of activated T cells (NFAT) signaling in
177 Here we show that S. pneumoniae activates nuclear factor of activated T cells (NFAT) signaling pat
178 ites is strongly involved in CaV1.2-mediated nuclear factor of activated T cells (NFAT) signaling, lo
180 itogenic effect of Ex-4 requires calcineurin/nuclear factor of activated T cells (NFAT) signaling.
183 uppression, 15d-PGJ(2)-G treatment decreased nuclear factor of activated T cells (NFAT) transcription
185 nt phosphatase calcineurin, which signals to nuclear factor of activated T cells (NFAT) transcription
188 idney but not other regions colocalized with nuclear factor of activated T cells (NFAT) transcription
189 cantly decreased TCR-induced Ca2+ influx and nuclear factor of activated T cells (NFAT) translocation
190 owever, these differences in Ca2+ influx and nuclear factor of activated T cells (NFAT) translocation
195 ficantly reduced activity in the calcineurin-nuclear factor of activated T cells (NFAT), a known Ca(2
196 dy induced Ca2+ oscillations, activating the nuclear factor of activated T cells (NFAT), a prosurviva
197 ne oxidase was detected by activation of the nuclear factor of activated T cells (NFAT), an establish
198 f the Ca(2+)-dependent transcription factor, nuclear factor of activated T cells (NFAT), and cyclospo
199 a ligand, Frizzled 5 receptor, beta-catenin, nuclear factor of activated T cells (NFAT), and major si
201 ignaling activates the transcription factors nuclear factor of activated T cells (NFAT), nuclear fact
203 n both mcip1/2 showed impaired activation of nuclear factor of activated T cells (NFAT), suggesting t
204 ion of transcriptional regulators, including nuclear factor of activated T cells (NFAT), which contro
205 we have identified an ncRNA repressor of the nuclear factor of activated T cells (NFAT), which intera
206 ur results further showed that a mutation in nuclear factor of activated T cells (NFAT)-binding sites
207 channels activates expression of c-fos- and nuclear factor of activated T cells (NFAT)-dependent gen
208 induced cyclin D1 expression in a time- and nuclear factor of activated T cells (NFAT)-dependent man
209 el activity, and downstream Ca(2+)-dependent nuclear factor of activated T cells (NFAT)-driven gene e
210 hat encodes a known regulator of calcineurin/nuclear factor of activated T cells (NFAT)-mediated grow
220 the calcium-dependent transcription factor, nuclear factor of activated T cells (NFAT)c1, which medi
221 D4 cells acted on the same cells, maintained nuclear factor of activated T cells (NFAT)c2 in the nucl
222 that mice deficient in both calcineurin and nuclear factor of activated T cells (NFAT)c2/c3 lack a p
223 as measured by nuclear translocation of the nuclear factor of activated T cells (NFAT)c4 transcripti
224 in remodeling, bind the transcription factor nuclear factor of activated T cells (NFAT)p in vitro and
225 discover that FTY-720 appreciably inhibited nuclear factor of activated T-cells (NFAT) activity.
226 ysically and functionally interacts with the nuclear factor of activated T-cells (NFAT) and is requir
227 is of these regions identified two consensus nuclear factor of activated T-cells (NFAT) binding sites
228 ranslocation and transcriptional activity of nuclear factor of activated T-cells (NFAT) c1, whereas N
229 endent phosphatase calcineurin, resulting in nuclear factor of activated T-cells (NFAT) dephosphoryla
231 ceptor function by activating members of the nuclear factor of activated T-cells (NFAT) family of tra
233 lso interrupts the activation of calcineurin/nuclear factor of activated T-cells (NFAT) pathway and t
234 oid hormone (PTH), oxytocin, and calcineurin/nuclear factor of activated T-cells (NFAT) pathways.
235 MEKK3 is capable of stimulating calcineurin/nuclear factor of activated T-cells (NFAT) signaling in
236 MEKK3 was capable of activating calcineurin/nuclear factor of activated T-cells (NFAT) signaling in
237 suggested a specialized role for calcineurin-nuclear factor of activated T-cells (NFAT) signaling in
238 as cyclosporin A indicates that calcineurin/nuclear factor of activated T-cells (NFAT) signalling mi
240 rrent with a loss of microcluster formation, nuclear factor of activated T-cells (NFAT) transcription
241 dent) signaling in a cell line model using a nuclear factor of activated T-cells (NFAT) transcription
243 ear localization of the transcription factor nuclear factor of activated T-cells (NFAT), and that thi
244 ion is mediated at least in part through the nuclear factor of activated T-cells (NFAT), as NFAT occu
245 e also led to significant transactivation of nuclear factor of activated T-cells (NFAT), but not acti
246 ult feline myocytes induced calcineurin (Cn)-nuclear factor of activated T-cells (NFAT)-mediated hype
252 X1 and P2X4 receptors inhibits Ca(2+) entry, nuclear factors of activated T cells (NFAT) activation,
253 K1A) as the likely target of harmine and the nuclear factors of activated T cells (NFAT) family of tr
255 ent (MAPK, JNK) and non-catalytic-dependent (nuclear factor of activated T cells, NFAT) VAV1 effector
256 ulatory factor 4 (IRF4) mRNA expression, and nuclear factor of activated T cells (NFAT1) protein expr
257 ment and its substrate transcription factor, nuclear factor of activated T cells (NFAT4), appear in a
258 des a calcineurin inhibitor that inactivates nuclear factor of activated T cells (NFATc) activity.
259 B (NF-kappaB) and the calcineurin-dependent nuclear factor of activated T cells (NFATc) control spec
260 Interestingly, the calcineurin substrate nuclear factor of activated T cells (NFATc) was highly e
261 ve active protein kinase G, dephosphorylated nuclear factor of activated T cells (NFATc1) and phospho
262 ified novel LMO2 interacting partners: ELK1, nuclear factor of activated T-cells (NFATc1), and lympho
263 and the subsequent nuclear translocation of nuclear factor of activated T cells (NFATs) in mDA neuro
265 increased nuclear factor-kappaB (NF-kappaB), nuclear factor of activated T cells (NFATs), and activat
266 h requires calcineurin (CN), which activates Nuclear Factor of Activated T cells (NFATs), but the gen
267 tin beta component of the ncRNA repressor of nuclear factor of activated T cells (NRON) ribonucleopro
268 ed growth factor) and transcriptional (e.g., nuclear factor of activated T cells or NFAT) factors in
269 the chromosome 21 gene DYRK1A suppresses the nuclear factor of activated T cells pathway and promotes
270 ity of the integrins for their ligands NFAT: nuclear factor of activated T cells PI3K: phosphoinositi
271 of microRNA-204 and has binding sites on the nuclear factor of activated T cells promoter region.
272 pt levels and nuclear occupancy of the NFAT (nuclear factor of activated T cells) regularly fluctuate
273 sitol trisphosphate production, activated an nuclear factor of activated T cells-responsive reporter
274 activation of the transcription factor NFAT (Nuclear Factor of Activated T cells) selectively, we hav
276 in PPR patients upregulates the calcineurin/nuclear factor of activated T cells signaling triggering
278 gene transcription through calcineurin/NFAT (nuclear factor of activated T cells) signaling through a
279 nvestigated its effects on calcineurin-NFAT (nuclear factor of activated T cells) signaling, an impor
280 th the immune/inflammatory calcineurin/NFAT (nuclear factor of activated T-cells) signaling pathway,
282 ized VSMC showed 39-fold higher Rgs16 (NFAT (nuclear factor of activated T-cells) target; MAPK inhibi
283 ysically with the transcription factor NFAT (nuclear factor of activated T-cells) that binds to the A
284 s (IL-10, Cox2, Egr1, and Egr2) regulated by nuclear factor of activated T cells, the best characteri
285 ooperate in a DNA-binding complex with NFAT (nuclear factor of activated T cells) to regulate the tra
287 n of members of the cytoplasmic component of nuclear factor of activated T cells transcription factor
288 calcineurin and its downstream effector, the nuclear factor of activated T cells transcription factor
289 lated by calcineurin inhibitors and involved nuclear factor of activated T cells transcription pathwa
291 s, DYRK3 proved to effectively inhibit NFAT (nuclear factor of activated T cells) transcriptional res
292 Expression of TAK1DeltaN also promoted NFAT (nuclear factor of activated T-cells) transcriptional act
293 cium signaling, cytosolic calcium activating nuclear factor of activated T cells translocation direct
294 tic proteins; longer exposure leads to NFAT (nuclear factor of activated T-cells) translocation to th
295 phage colony-stimulating factor induction of nuclear factor of activated T cells type c-1 and catheps
296 of the osteoclast transcriptional activator nuclear factor of activated T cells type c-1, associated
299 Ca(2+)-dependent transcription factor NFAT (nuclear factor of activated T-cells), which has been imp
300 increased nuclear localization of endogenous nuclear factor of activated T cells, which could be bloc
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