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1 ak1 (p21-activated kinase -1) --> NF-kappaB (nuclear factor-kappa B).
2 -1, tribbles 3, reactive oxygen species, and nuclear factor kappa B)?
3 phosphorylation and nuclear translocation of nuclear factor kappa B.
4 opolysaccharide (LPS) -induced activation of Nuclear Factor kappa B.
5 togen-activated protein kinase and activates nuclear factor kappa B.
6 n of both interferon regulatory factor 3 and nuclear factor kappa B.
7 bition of glycogen synthase kinase-3beta and nuclear factor kappa B.
8 bition of glycogen synthase kinase-3beta and nuclear factor kappa B.
9 s both C and T alleles were found to bind to nuclear factor kappa B.
10 D91 triggers signalling cascades to activate nuclear factor-kappa B.
11 rivative of dithiocarnamate, an inhibitor of nuclear factor- kappa B.
12 hanism for induction of CDX2 is dependent on nuclear factor kappa B, a crucial transcription factor i
14 bic acid and dehydroascorbic acid attenuated nuclear factor kappa B activation and normalized coagula
16 e course plots of Lipopolysaccharide-induced Nuclear Factor kappa B activation in mouse embryo fibrob
17 ardiac mitochondrial respiratory efficiency, nuclear factor kappa B activation, and caspase activitie
19 nhibited tumor necrosis factor-alpha induced nuclear factor-kappa B activation by 27.5% at 2 mg/ml, w
20 cyclophosphamide with mecamylamine suggested nuclear factor-kappa B activation in the chronic inflamm
21 tinamide effectively attenuated postischemic nuclear factor-kappa]B activation and exhibited robust a
22 ects that were accompanied by a reduction in nuclear factor kappa B activity and cell surface levels
24 m was associated with increased inhibitor of nuclear factor-kappa B alpha (IkappaBalpha) expression,
25 location of transcription factors, including nuclear factor kappa B, although its role in PCa was lar
26 ecule 1-dependent and leads to activation of nuclear factor kappa B and extracellular signal-regulate
27 nate attenuated the HS-induced activation of nuclear factor kappa B and reduced the expression of pro
28 nate attenuated the HS-induced activation of nuclear factor kappa B and reduced the expression of pro
29 Immunofluorescence showed colocalization of nuclear factor kappa-B and OCN in diseased and normal va
30 reby enabling constitutive activation of the nuclear factor-kappa B, and contributing to chronic infl
31 ity of activated glucocorticoid receptor and nuclear factor kappa-b appeared to join pre-existing P30
32 Our data demonstrate that miR-146a controls nuclear factor kappa B-dependent inflammatory responses
34 (Hz), signals NOS induction through ERK- and nuclear factor kappa B-dependent pathways and has been d
35 human peripheral blood mononuclear cells and nuclear factor kappa B-dependent transcription in human
38 t HEK293 cells, HEK293-TLR2 cells had marked nuclear factor-kappa B-driven luciferase activity, had m
39 in inflammatory and cancer pathways, such as nuclear factor kappa B, EGF, Wnt, and B-cell lymphoma 2.
42 chanism of action is partly mediated through nuclear factor-kappa B inhibition, resulting in apoptosi
43 nhibitor PD98059, the Src inhibitor pp2, the nuclear factor- kappa B inhibitor caffeic acid phenethyl
45 ylated and thereby inhibited by inhibitor of nuclear factor kappa B kinase subunit beta (IKKbeta) to
46 howed that Hsp90 interacts with inhibitor of nuclear factor kappa-B kinase (IKK) together with cochap
47 Pharmacological inhibition of inhibitor of nuclear factor kappa-B kinase subunit beta (IKKbeta), bu
49 ncreased expression of receptor activator of nuclear factor kappa B ligand (RANK-L) due to pro-inflam
50 on and inactivation of Receptor Activator of Nuclear factor Kappa B Ligand (RANKL) and osteoprotegeri
52 clast (OC) response to receptor activator of nuclear factor kappa B ligand (RANKL) and thus bone meta
53 ng factor (M-CSF), and receptor activator of nuclear factor kappa B ligand (RANKL) expression, but th
54 osis factor (TNF), and receptor-activator of nuclear factor kappa B ligand (RANKL) were significantly
55 nonoclonal antibody to receptor activator of nuclear factor kappa B ligand, has completed its major f
56 nsing receptor and the receptor activator of nuclear factor kappa B ligand/receptor activator of nucl
57 that stimulation with receptor activator of nuclear factor kappa-B ligand (RANKL) resulted in a robu
58 oxidase (MPO), and the cytokine receptor for nuclear factor kappa-B ligand (RANKL) were significantly
59 ze salivary levels of receptor activator for nuclear factor kappa-B ligand (RANKL), osteoprotegerin,
60 ted T cells to produce receptor activator of nuclear factor kappa-B ligand (RANKL), which, in turn, p
61 TNF-alpha), as well as receptor activator of nuclear factor kappa-B ligand (RANKL)-induced osteoclast
62 tal tissue and induces receptor activator of nuclear factor kappa-B ligand (RANKL)-RANK-osteoproteger
63 human monoclonal anti-receptor activator of nuclear factor kappa-B ligand antibody, with zoledronic
64 Change in circulating receptor activator of nuclear factor kappa-B ligand was higher in the interven
65 chemotactic protein 1, receptor activator of nuclear factor kappa-B ligand, and macrophage colony-sti
66 lcin, osteoprotegerin, receptor activator of nuclear factor kappa-B ligand, vascular endothelial grow
67 factor (TNF)-alpha and receptor activator of nuclear factor kappa-B ligand; these cytokines were also
68 issue homogenates, and receptor activator of nuclear factor-kappa B ligand (RANKL) activation was ana
70 , expression of activated receptor activator nuclear factor-kappa B ligand (RANKL) and bone density i
72 ) PBMCs was induced by receptor activator of nuclear factor-kappa B ligand (RANKL), either alone or i
73 histochemistry for the receptor activator of nuclear factor-kappa B ligand (RANKL), osteoprotegerin (
74 ranscription factor-2, receptor activator of nuclear factor-kappa B ligand (RANKL), sclerostin, and D
75 LR-4, osteoprotegerin, receptor activator of nuclear factor-kappa B ligand (RANKL), tumor necrosis fa
76 osorbent assay for: 1) receptor activator of nuclear factor-kappa B ligand (RANKL); 2) osteoprotegeri
77 (IL)-6, IL-8, soluble receptor activator of nuclear factor-kappa B ligand (sRANKL), osteoprotegerin
78 mmunoreactivity to the receptor activator of nuclear factor-kappa B ligand at day 7 post-treatment, s
79 nuclear factor-kappa B-receptor activator of nuclear factor-kappa B ligand interaction for osteoclast
80 interleukin-1beta and receptor activator of nuclear factor-kappa B ligand than group EP-HN019 (P <0.
82 lectrophoresis; RANKL, receptor activator of nuclear factor kappa B-ligand; NFATc1, nuclear factor of
83 eracting protein 2) to coordinate NF-kappaB (nuclear factor kappa B)-mediated cytokine responses.
84 ulting in de novo lipogenesis, and increased nuclear factor kappa B-mediated inflammation act in conc
85 enomena at the organismal level, we assessed nuclear factor kappa B (NF-kappaB) activation (indicativ
86 ction in cultured hepatoma cells, leading to nuclear factor kappa B (NF-kappaB) activation and the pr
88 intestinal epithelial cells and also induced nuclear factor kappa B (NF-kappaB) activation in HEK293T
90 at least three different pathways leading to nuclear factor kappa B (NF-kappaB) activation, apoptosis
95 the expression of genes reflecting enhanced nuclear factor kappa B (NF-kappaB) activity were noted i
96 r 4), interferon regulatory factor-3 (IRF3), nuclear factor kappa B (NF-kappaB) activity, and proinfl
97 ecific lipin-1 deficiency diminished hepatic nuclear factor kappa B (NF-kappaB) activity, limited liv
99 uclear export sequence (NES) of inhibitor of nuclear factor kappa B (NF-kappaB) alpha (IkappaBalpha)
100 (TRAF1-6) links the TNFR superfamily to the nuclear factor kappa B (NF-kappaB) and activator protein
101 vestigated the role of transcription factors nuclear factor kappa B (NF-kappaB) and signal transducer
105 -protein interaction (PPI) interface between nuclear factor kappa B (NF-kappaB) essential modulator (
107 TRF also showed a greater inhibition on the nuclear factor kappa B (NF-kappaB) expression than T and
108 alling pathways leading to activation of the nuclear factor kappa B (NF-kappaB) family of transcripti
113 a 12,14- Prostaglandin J2 (15dPGJ2) inhibits Nuclear factor kappa B (NF-kappaB) in human myocytes and
117 e report that systemic administration of the nuclear factor kappa B (NF-kappaB) inhibitor Bay 11-7085
127 We utilized a transgenic mouse model where nuclear factor kappa B (NF-kappaB) is selectively inhibi
129 (IL)12 or IL18 as well as inhibition of the nuclear factor kappa B (NF-kappaB) pathway reversed NK c
130 rse cellular and biological functions of the nuclear factor kappa B (NF-kappaB) pathway, together wit
132 ) 3' untranslated region (UTR) and activates nuclear factor kappa B (NF-kappaB) pathways that contrib
133 cycle progression and I-kappa B kinase (IKK)/nuclear factor kappa B (NF-kappaB) signaling contributes
135 MRV miRNAs, we screened for their effects on nuclear factor kappa B (NF-kappaB) signaling in the pres
136 biquitinated chains from key proteins in the nuclear factor kappa B (NF-kappaB) signaling pathway.
139 Here we demonstrate that the dynamics of Nuclear Factor kappa B (NF-kappaB) signalling and the ce
143 CD47 expression was found to be regulated by nuclear factor kappa B (NF-kappaB) through a specific re
145 phate (NAD(P)H)-oxidase and translocation of nuclear factor kappa B (NF-kappaB) were also evaluated.
146 Recently, VHL loss was shown to activate nuclear factor kappa B (NF-kappaB), a family of transcri
148 acts as an upstream activator of astroglial nuclear factor kappa B (NF-kappaB), leading to the relea
149 is is strongly associated with inhibition of nuclear factor kappa B (NF-kappaB), proapoptotic activat
151 rophages enhance myofibroblast survival in a nuclear factor kappa B (NF-kappaB)-dependent manner and
152 impaired by more than 95%, but no defect in nuclear factor kappa B (NF-kappaB)-induced gene expressi
155 cellular DNA microarrays, we found that many nuclear factor kappa B (NF-kappaB)-responsive genes were
164 ated the hepatocellular damage by inhibiting nuclear factor kappa B (NF-kappaB)/cyclooxygenase 2 sign
166 decrease in PKC activity, and inhibition of nuclear factor kappa B (NF-kB) translocation, were attri
168 cluding TNF-alpha and IL-6 and activation of nuclear factor kappa B (NF-kB/p65), p38 mitogen-activate
170 gulators linked to TNF-alpha/TNFR signaling, nuclear factor kappa-B (NF-kappaB) activation, autophagy
171 ll receptor (BCR) signaling and constitutive nuclear factor kappa-B (NF-kappaB) activation, which is
172 response element-binding protein (CREB) and nuclear factor kappa-B (NF-kappaB) are two ubiquitous tr
173 of this study is to investigate the role of nuclear factor kappa-B (NF-kappaB) in regulating IL-6 ex
174 NOS1 is complex but recent data suggest that nuclear factor kappa-B (NF-kappaB) is an important trans
175 recent Immunity articles probe the roles of Nuclear Factor kappa-B (NF-kappaB) pathways in survival
176 egulated kinase (ERK), calcium (Ca(2+)), and nuclear factor kappa-B (NF-kappaB) pathways, response dy
179 A key antiapoptotic transcription factor, nuclear factor kappa-B (NF-kappaB), is known to be criti
183 gical inhibitors of either IL-8 secretion or nuclear factor- kappa B (NF-kappa B) activation on IL-8
184 d the binding of the p65 (RelA) component of nuclear factor-kappa B (NF-kappa B) to the endogenous E-
185 a (IkappaBalpha) expression, which inhibited nuclear factor-kappa B (NF-kappaB) activation and induct
186 ad of colonic tumors and increased STAT3 and nuclear factor-kappa B (NF-kappaB) activation in colon.
188 d mitogen-activated protein (MAP) kinase and nuclear factor-kappa B (NF-kappaB) activation, and the s
191 carcinoma cells (T24 cells), and TPA-induced nuclear factor-kappa B (NF-kappaB) activity with human h
192 idermis of mice lacking transcription factor nuclear factor-kappa B (NF-kappaB) activity, primary hai
193 the activation of the transcription factors nuclear factor-kappa B (NF-kappaB) and activator protein
194 ed PHB expression on TNF-alpha activation of nuclear factor-kappa B (NF-kappaB) and epithelial barrie
195 ithelia in Hashimoto's thyroiditis activates nuclear factor-kappa B (NF-kappaB) and induces pro-infla
196 GF), inducible nitric oxide synthase (iNOS), nuclear factor-kappa B (NF-kappaB) and intercellular adh
198 ctivates typical cytokine signal pathways of nuclear factor-kappa B (NF-kappaB) and p38 mitogen-activ
200 genotoxic and oncogenic stress activates the nuclear factor-kappa B (NF-kappaB) and p53 proteins; how
201 amic interplay of two transcription factors, nuclear factor-kappa B (NF-kappaB) and peroxisome prolif
202 UPR, there was transcriptional regulation by nuclear factor-kappa B (NF-kappaB) and RNA stabilization
203 t-7b cluster and identified several putative nuclear factor-kappa B (NF-kappaB) consensus elements.
204 P-ribose polymerase, alteration in c-myc and nuclear factor-kappa B (NF-kappaB) expression, regulatio
206 tion of proinflammatory transcription factor nuclear factor-kappa B (NF-kappaB) in 7-week-old mdx mic
207 imately 40,000 genes, and tested the role of nuclear factor-kappa B (NF-kappaB) in maintaining an act
208 ic proteins and the proinflammatory molecule nuclear factor-kappa B (NF-kappaB) in the retina of Lewi
210 we have shown that the transcription factor nuclear factor-kappa B (NF-kappaB) is activated by BCR-A
212 One of the primary physiological roles of nuclear factor-kappa B (NF-kappaB) is in the immune syst
213 affects expression of receptor activator of nuclear factor-kappa B (NF-kappaB) ligand (RANKL), a pot
215 constitutive activation of the antiapoptotic nuclear factor-kappa B (NF-kappaB) pathway, which can in
216 ne of these main signaling mechanisms is the nuclear factor-kappa B (NF-kappaB) pathway, which integr
219 d activators of transcription 1 (STAT1), and nuclear factor-kappa B (NF-kappaB) signaling in Lilrb3(-
220 dy, we show that the activation of classical nuclear factor-kappa B (NF-kappaB) signaling increases t
221 nvading pathogens, their ability to regulate nuclear factor-kappa B (NF-kappaB) signaling, interleuki
225 kine is dependent upon transcription factors nuclear factor-kappa B (NF-kappaB), stimulating protein-
226 eading to activation of transcription factor nuclear factor-kappa B (NF-kappaB), which is a central r
227 like receptor 4 (TLR4)-dependent pathway via nuclear factor-kappa B (NF-kappaB), while simultaneously
228 so stimulated IFN-beta promoter activity and nuclear factor-kappa B (NF-kappaB)-dependent transcripti
234 cell cycle and prosurvival pathways (such as nuclear factor kappa B [NF-kappaB], AKT), pathways regul
235 monstrated a signaling pathway from HIF-1 to nuclear factor kappa B (NFkappaB) activation to enhanced
237 f intrapancreatic trypsinogen activation and nuclear factor kappa B (NFkappaB) activation, the two ma
240 in terms of interleukin (IL)-8 promoter and nuclear factor kappa B (NFkappaB) activity, were observe
241 IkappaBalpha enhances the rate of release of nuclear factor kappa B (NFkappaB) from DNA target sites
243 tion by laminar shear stress is dependent on nuclear factor kappa B (NFkappaB) subunits p50 and p65 b
244 a) is a potent cytokine that signals through nuclear factor kappa B (NFkappaB) to activate a subset o
245 or (TRPV1) and Ca(2+) to microglial IL-6 and nuclear factor kappa B (NFkappaB) translocation with ele
247 nt protein deacetylase sirtuin-6 (SIRT6) and nuclear factor kappa B (NFkappaB), sequesters GR express
248 infection of hepatic cells by HCV stimulates nuclear factor kappa B (NFkappaB)-dependent production o
251 d to dramatically reduce the level of active nuclear factor kappa-B (NFkappaB) in the cell; NFkappaB
252 ivator of transcription factor 6 (STAT6) and nuclear factor-kappa B (NFkappaB) in mice and cells.
253 onally activated by mTOR complex 2 (mTORC2): nuclear factor-kappa B (NFkappaB) signaling cascade.
254 essing fibroblasts induced the activation of nuclear factor-kappa B (NFkappaB), a regulator fibronect
258 ese genes were involved in interferon alpha, nuclear factor-kappa B (NFkB), extracellular signal-rela
259 onase exerts an effect via downregulation of nuclear factor kappa B (NFKbeta) by specific microRNAs (
260 cally regulated by p38delta isoform, whereas nuclear factor kappa B (NFsmall ka, CyrillicB) pathway r
262 factors, such as glucocorticoid receptor and nuclear factor kappa-b, on the three-dimensional organiz
263 Cs), a phenomenon not mediated by changes to nuclear factor-kappa B or hydrogen sulfide but that occu
264 in the presence of antagonists of NFkappaB (nuclear factor kappa B) or iNOS activity, NO synthesis a
265 factor kappa B ligand/receptor activator of nuclear factor kappa B/osteoprotegerin system involved i
266 ich also correlated with increased levels of nuclear factor kappa B p52 and decreased levels of c-Jun
269 a4beta1, which resulted in signaling through nuclear factor kappa B p65, protein kinase B1, and p38 m
270 vo that depends on Ser536 phosphorylation of nuclear factor kappa B p65; this pathway may hold valuab
271 -0.59 (-0.8, -0.3) mmol/L; P < 0.001], total nuclear factor kappa-B p65 [-0.016 (-0.022, -0.011) comp
273 e pathway that, together with the Rho-kinase nuclear factor kappa B pathway (NF-kB), are required for
274 om platelets, particularly those part of the nuclear factor kappa B pathway, are associated with BMI,
276 ied activating mutations in the noncanonical nuclear factor-kappa B pathway could give rise to ibruti
277 as 18q21 amplification or activation of the nuclear factor-kappa B pathway, as reported previously,
278 of the mitogen-activated protein kinase and nuclear factor kappa B pathways in MDSCs was MyD88 depen
279 rough regulating the activation of canonical nuclear factor kappa B, phosphatidylinositol-4,5-bisphos
280 phosphoinositide 3-kinase/phosphorylated Akt/nuclear factor kappa B (PI3K/pAkt/NFkappaB)-signaling pa
281 e immune system signaling pathways including nuclear factor-kappa B profoundly alter intestinal epith
282 Chronic alcohol consumption inhibited the nuclear factor kappa B proinflammatory cytokine pathway
285 id-state nanopore has been used to recognize Nuclear Factor-kappa B proteins (NF-kappaB), that are in
286 lonal antibody against receptor activator of nuclear factor kappa B (RANK) ligand, with zoledronic ac
287 omeostatic conditions, receptor activator of nuclear factor kappa-B (RANK) ligand on osteoblasts driv
288 uent bone loss via the receptor activator of nuclear factor-kappa B (RANK)-RANK ligand (RANKL)-osteop
289 eractions, such as the receptor activator of nuclear factor-kappa B-receptor activator of nuclear fac
290 d transcription factors, such as Bcl-2, Bax, nuclear factor kappa B, regulate the decision-making pow
292 occurs through direct targeting of upstream nuclear factor kappa B signal transducers caspase recrui
293 es the proinflammatory transcription factors nuclear factor-kappa B, signal transducer and activators
295 K/NFkappaB (mitogen-activated protein kinase/nuclear factor-kappa B) signals participate in regulatin
296 -responsive element (HRE)) and inflammation (nuclear factor kappa B) to obtain a chimeric promoter na
298 is known to coamplify with EGFR and regulate nuclear factor-kappa B transcriptional activity) had hig
299 diac tumor necrosis factor-alpha expression, nuclear factor kappa B translocation, obesity-induced pe
300 ession of TWEAK increased, the activation of nuclear factor-kappa B without affecting the activation
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