ライフサイエンスコーパス: nuclear layer

1 ells) have their soma displaced to the inner nuclear layer.

2 plexiform, and to a lesser extent the outer nuclear layer.

3 y distinct and appropriately polarized outer nuclear layer.

4 man retina exhibited positivity in the inner nuclear layer.

5 bnormal vasculature extending into the outer nuclear layer.

6 r plexiform layer (OPL) and invade the outer nuclear layer.

7 ate measure of INL thickness), and the outer nuclear layer.

8 but accurately detected the undamaged outer nuclear layer.

9 SD-OCT were localized deeper than the inner nuclear layer.

10 ence of a single layer of cones in the outer nuclear layer.

11 histologically with a thinning of the inner nuclear layer.

12 f the RPE in the presence of an intact outer nuclear layer.

13 inimally preserved paracentral photoreceptor nuclear layer.

14 both individuals, and thinning of the outer nuclear layer.

15 ells and some but not all cells of the inner nuclear layer.

16 al ganglion cells and the cells of the inner nuclear layer.

17 n both the ganglion cell layer and the inner nuclear layer.

18 ell nuclei to the upper portion of the inner nuclear layer.

19 ving retinal rhythms is located in the inner nuclear layer.

20 numbers of TUNEL-positive cells in the outer nuclear layer.

21 hx10-positive bipolar cells within the inner nuclear layer.

22 i of ganglion cells and neurons in the inner nuclear layer.

23 ation, and nearly complete loss of the outer nuclear layer.

24 layers or cell number in the inner and outer nuclear layer.

25 emonstrated subsequent thinning of the inner nuclear layer.

26 ithelium disruption and/or loss of the outer nuclear layer.

27 of ganglion cell, inner plexiform, and inner nuclear layers.

28 receptors, occupy the ganglion cell or inner nuclear layers.

29 cells in the retinal ganglion cell and inner nuclear layers.

30 ory retina between outer plexiform and outer nuclear layers.

31 To our knowledge, microcystic inner nuclear layer abnormalities have not been investigated i

32 tinal deposits that extend through the outer nuclear layer, affect photoreceptor integrity, and are a

33 neuron (RIN)--horizontal cells (HCs), inner nuclear layer amacrine cells (iACs) and displaced amacri

34 cells), proximal parts of neuroblastic/inner nuclear layer (amacrine cells) and distal part of neurob

35 rizontal cells to inner aspects of the inner nuclear layer, among the retinal amacrine cells.

36 a retraction of rod spherules into the outer nuclear layer and a sprouting of rod bipolar cell dendri

37 s were associated with thinning of the inner nuclear layer and abnormalities of both layers were asso

38 ly result in permanent thinning of the inner nuclear layer and are critical to identify in order to d

39 ctivity (LI) was found in somas in the inner nuclear layer and as punctate staining in the inner and

40 l progenitor cells that migrate to the outer nuclear layer and become rod precursor cells that are co

41 r outer segments extending through the outer nuclear layer and even beyond the outer limiting membran

42 for about 1% of amacrine cells in the inner nuclear layer and for up to 27% of displaced amacrine ce

43 polar cell processes extended into the inner nuclear layer and ganglion cell layer by PNM3.5.

44 by the displacement of the soma to the inner nuclear layer and has morphological similarities with ei

45 bodies in the ganglion cell layer and inner nuclear layer and immunoreactive processes in the inner

46 xiform layer, inner nuclear layer, and outer nuclear layer and in pericytes of postdevelopment day 17

47 uorescein was present in somata in the inner nuclear layer and in synaptic boutons in the inner plexi

48 on of calretinin-positive cells in the inner nuclear layer and in the ganglion cell layer is glutamic

49 The outer nuclear layer and inner retinal thicknesses were normal.

50 -) mice, and transgenics had a thicker outer nuclear layer and less sub-retinal pigment epithelium de

51 omato fluorescence was detected in the inner nuclear layer and localized to type 1, 3b, and 4 OFF bip

52 s were associated with widening of the outer nuclear layer and loss of the foveal depression.

53 ter disease stages were accompanied by inner nuclear layer and nerve fiber layer abnormalities.

54 psin transgene lost the entire retinal outer nuclear layer and no longer responded to illumination.

55 Microscopically, the inner nuclear layer and outer plexiform layer were the most af

56 rence in the thickness of the combined outer nuclear layer and outer plexiform layer when we compared

57 parafoveal thickness, presence of the inner nuclear layer and outer segment, gestational age at birt

58 vity (IR) was present primarily in the inner nuclear layer and the ganglion cell layer.

59 especially the separation between the outer nuclear layer and the Henle fiber layer.

60 ar layer; the hypointense layer 2, the outer nuclear layer and the inner and outer segments; and the

61 dition, cone opsin mislocalized to the outer nuclear layer and the outer plexiform layer in the CNGB3

62 Between the nuclear layer and the yolk, the cytoplasm was homogeneou

63 cells located in the ganglion cell and inner nuclear layers and did not alter NMDA-induced PARP-1 hyp

64 multipotent progenitors that migrate to all nuclear layers and differentiate into layer-specific cel

65 (sGC), was in somata in the inner and outer nuclear layers and in both plexiform layers.

66 , inner plexiform layer, and inner and outer nuclear layers and within the spindle-shaped cell popula

67 lized rosette-like arrangements in the outer nuclear layer, and develop abnormal vascularization, bro

68 ontinue to proliferate, migrate to the outer nuclear layer, and differentiate into photoreceptors.

69 mRNA is found in cells of the retinal inner nuclear layer, and immunofluorescent confocal microscopy

70 ll and bipolar cell processes into the outer nuclear layer, and mislocalized synaptic complexes.

71 ganglia cells, outer plexiform layer, inner nuclear layer, and outer nuclear layer and in pericytes

72 especially in the outer nuclear layer, inner nuclear layer, and photoreceptors.

73 ocalized predominantly to the inner segment, nuclear layer, and synapse in dark-adapted rods, whereas

74 the outer plexiform layer (OPL) or the inner nuclear layer, and while present in the mature retina, a

75 ed to identify A amacrine cells in the inner nuclear layer as well as widefield amacrine and small bi

76 spike in apoptosis was observed in KO outer nuclear layer at P25.

77 poptotic activity in the photoreceptor outer nuclear layer at postnatal week 2 and highly disorganize

78 ent wave of Smad1/5/8 signaling in the inner nuclear layer at the end of the first postnatal week, fr

79 tions trended to have retention of the outer nuclear layer at the fovea and macular thickening, espec

80 studies used hosts with a preexisting outer nuclear layer at the time of treatment.

81 the lesions had resolved into areas of inner nuclear layer atrophy with persistence of scotomas.

82 After birth the foveal outer nuclear layer became much thicker as cone packing occurr

83 form ectopic synapses with rods in the outer nuclear layer but lack bipolar dendrites.

84 th arrestin1 in the inner segments and outer nuclear layer, but remained in the inner segments when a

85 om amacrine cells, were present in the inner nuclear layer by PNM9.5.

86 plaquelike lesions at the level of the inner nuclear layer by spectral-domain OCT and showed correspo

87 pithelial cell integrity and prevented outer nuclear layer cell death as examined by histopathologic

88 rs primarily affects the generation of inner nuclear layer cell types, resulting in complete loss of

89 of nearly all differentiated inner and outer nuclear layer cell types.

90 duced number of rod photoreceptors and inner nuclear layer cells.

91 SCs had fewer amacrine cells and other inner nuclear layer cells.

92 t growth factor (FGF)-2 in the photoreceptor nuclear layer coincided with the inflammatory response i

93 layers and increased thickness in the inner nuclear layer compared with healthy subjects (P < .05).

94 number of photoreceptor nuclei in the outer nuclear layer compared with WT controls.

95 cells progressively accumulate in the outer nuclear layer concurrently with photoreceptor degenerati

96 GFA mRNA signal was located within the inner nuclear layer corresponding to CRALBP-labeled Muller cel

97 ions revealed hyperreflectivity of the outer nuclear layer; disruption of the external limiting membr

98 bserved in the ganglion cell layer and inner nuclear layer during development.

99 migrate towards the apical side of the outer nuclear layer during early postnatal retinal development

100 ute retinal changes in PPM involve the outer nuclear layer, external limiting membrane, ellipsoid lay

101 reactivity in the plexiform layers and outer nuclear layer fell into at least three patterns dependin

102 jury paradigms that target different retinal nuclear layers for cell ablation.

103 whose nuclei are scattered across the outer nuclear layer, had no effect on the positioning of their

104 the deeper retinal layers, such as the outer nuclear layer, has not been previously described in mult

105 The cell bodies in the inner nuclear layer, however, were larger in the peripheral re

106 (19%), and loss of ellipsoid zone and outer nuclear layer in 3 (19%).

107 reatment preserved ERG b-waves and the outer nuclear layer in Bbs1(M390R/M390R) mice, and prevented o

108 of microcystoid macular changes in the inner nuclear layer in eyes with concomitant epiretinal membra

109 owed a substantial preservation of the outer nuclear layer in most parts of the treated retina only.

110 OCT was less accurate at detecting the inner nuclear layer in ouabain-damaged retinas, but accurately

111 of microcystoid macular changes in the inner nuclear layer in patients with idiopathic epiretinal mem

112 preservation of the ellipsoid zone and outer nuclear layer in the fovea.

113 mislocalize to the inner segments and outer nuclear layer in the Nphp4(nmf192/nmf192) mutant retina.

114 reatment preserved ERG b-waves and the outer nuclear layer in the rd10 mice to P30.

115 ied out a quantitative analysis of the inner nuclear layer in the retina of the marmoset (Callithrix

116 n was localized to some neurons of the inner nuclear layer, in the inner plexiform layer, and along t

117 in CCDKO mice develops in the inferior outer nuclear layer independently of light around postnatal da

118 orizontal and bipolar cells toward the outer nuclear layer indicating impaired rod transmitter releas

119 ies formed a regular mosaic within the inner nuclear layer, indicating they represent a single amacri

120 ti fluorescent cell bodies were in the inner nuclear layer (INL) and a few cell bodies were in the ga

121 ne and displaced amacrine cells in the inner nuclear layer (INL) and ganglion cell layer (GCL), respe

122 in (HPC-1)-immunoreactive cells in the inner nuclear layer (INL) and GCL, consistent with their ident

123 The MME was restricted to the inner nuclear layer (INL) and had a characteristic perifoveal

124 monostratified cells with somas in the inner nuclear layer (INL) and medium-field monostratified cell

125 unit distributed on cell bodies in the inner nuclear layer (INL) and on processes within both the inn

126 evaluated by TUNEL and measurement of inner nuclear layer (INL) and outer nuclear layer (ONL) thickn

127 ce of ectopic neuronal clusters in the inner nuclear layer (INL) and regions of disrupted retinal lam

128 s of cholinergic amacrine cells in the inner nuclear layer (INL) and the ganglion cell layer (GCL) di

129 in Henle's fiber layer (HFL) than the inner nuclear layer (INL) and was highly associated with hyper

130 iogenesis were detected in the retinal inner nuclear layer (INL) before morphologic neoplastic change

131 s with outer retinal changes, isolated inner nuclear layer (INL) cysts were found in 6 of 131 eyes wi

132 cell layer (GCL) and inner part of the inner nuclear layer (INL) from 3-9 dpf; after 14 dpf, it was r

133 ic macular oedema (MMO) of the retinal inner nuclear layer (INL) has been identified in patients with

134 macular splitting were present in the inner nuclear layer (INL) in all 11 eyes and in the outer nucl

135 dantly expressed in the neurons of the inner nuclear layer (INL) of the retina.

136 hyporeflective spaces was lower in the inner nuclear layer (INL) than in the complex formed by the ou

137 Fourier-domain-OCT, there was apparent inner nuclear layer (INL) thickening in regions with ONL thinn

138 A small degree of inner nuclear layer (INL) thickening occurred in MSON eyes com

139 of age, outer nuclear layer (ONL) and inner nuclear layer (INL) thicknesses were measured.

140 Although type 1 lesions lead to inner nuclear layer (INL) thinning, type 2 lesions resulted in

141 and inner plexiform layer (GCIPL) and inner nuclear layer (INL) volumes were tested for association

142 of the outer plexiform layer (OPL) and inner nuclear layer (INL), and development of a hyporeflective

143 isual thresholds; total nuclear layer, inner nuclear layer (INL), and outer nuclear layer (ONL) thick

144 well as the inner plexiform layer, the inner nuclear layer (INL), and the outer plexiform layer (OPL)

145 cell layer (GCL) and the other in the inner nuclear layer (INL), that together comprise approximatel

146 nner plexiform layers (RGCL+), and the inner nuclear layer (INL).

147 in the inner plexiform layer (IPL) and inner nuclear layer (INL).

148 cell layer, with 23% displaced to the inner nuclear layer (INL).

149 the middle retina, extending from the inner nuclear layer (INL)/outer plexiform layer junction to in

150 Visual acuity and visual thresholds; total nuclear layer, inner nuclear layer (INL), and outer nucl

151 essed in the retina, especially in the outer nuclear layer, inner nuclear layer, and photoreceptors.

152 e detected in the ganglion cell layer, inner nuclear layer, inner/outer plexiform layers, photorecept

153 c arrestin 1 (tet-ARR1), stored in the outer nuclear layer/inner segments in the dark, modulates phot

154 plexiform layer irregularity (8%), and inner nuclear layer irregularity (6%).

155 immunoreactive cells is located in the inner nuclear layer, is immunopositive for glycine transporter

156 Significant retinal thinning and outer nuclear layer loss occurred in Rpe65(-)(/-)/Nrl(-)(/-) m

157 inner plexiform layer (mIPL), macular inner nuclear layer (mINL), macular outer plexiform layer (mOP

158 outer plexiform layer (mOPL), macular outer nuclear layer (mONL), photoreceptors (PR), and retinal p

159 The inner nuclear layer/Muller cell localization of the key protei

160 n = 1), outer plexiform layer (n = 4), outer nuclear layer (n = 12), or inner segment/outer segment j

161 mRNAs were present in photoreceptors, inner nuclear layer neurons, and ganglion cells in C57BL/6 mou

162 orescent, cystoid macular edema in the inner nuclear layer, no light rise in the electro-oculography,

163 d glass pipettes and inserted into the inner nuclear layer of fixed whole-mount retinas.

164 eptors nuclei at the outer edge of the outer nuclear layer of mammalian retinas.

165 own if a degenerate retina lacking the outer nuclear layer of photoreceptor cells would allow the sur

166 ted the presence of Plk1s1 mRNA in the outer nuclear layer of the mouse retina.

167 tive donor cells integrated within the outer nuclear layer of the recipient and differentiated into n

168 stic oedema predominantly involved the inner nuclear layer of the retina and tended to occur in small

169 icrocystic abnormalities involving the inner nuclear layer of the retina occurs in a subset of patien

170 inal ganglion cells (RGCs) through the inner nuclear layer of the retina with glutamate, a primary ne

171 ression is dramatically induced in the inner nuclear layer of the retina, suggesting that PGC-1alpha

172 is predominantly expressed within the outer nuclear layer of the retina.

173 found within the sclerad region of the inner nuclear layer of the retina.

174 rosette-like structures located in the outer nuclear layer of the retinae of the 4 older patients wer

175 No pathology was seen in the inner or outer nuclear layers of eyes with optic neuritis, suggesting t

176 The Mdm1 transcript is localized to the nuclear layers of neural retina.

177 er show that cells in the ganglion and inner-nuclear layers of the retina constitutively express IRF-

178 helium, choroidal thinning, undifferentiated nuclear layers of the retina, and a perivascular inflamm

179 , ganglion cells, inner plexiform, and inner nuclear layers) of eyes with previous optic neuritis (P

180 PE loss on the same day, and a reduced outer nuclear layer on day 7.

181 flectance of outer plexiform layer and inner nuclear layer on spectral-domain OCT was observed to imp

182 laque-like lesions at the level of the inner nuclear layer on spectral-domain OCT, with no fluorescei

183 loss occurred on day 14 with a reduced outer nuclear layer on the same day.

184 f cells in the ganglion cell layer and inner nuclear layer on whole-retinal sections.

185 inner plexiform layer (IPL) (nasally), outer nuclear layer (ONL) (nasally), and inner segment (centra

186 ally by measuring the thickness of the outer nuclear layer (ONL) and functionally by electroretinogra

187 cquisition, the boundaries between the outer nuclear layer (ONL) and Henle's fiber layer (HFL) were w

188 When the rats were 12 weeks of age, outer nuclear layer (ONL) and inner nuclear layer (INL) thickn

189 Ellipsoid zone (EZ) width (EZW) and outer nuclear layer (ONL) and inner retinal layer (IRL) thickn

190 Overall retinal thickness and outer nuclear layer (ONL) and inner retinal parameters across

191 ctroretinograms were recorded, and the outer nuclear layer (ONL) and the choroidal thickness and area

192 timulated Muller cell migration to the outer nuclear layer (ONL) and to differentiate into photorecep

193 ion in whole retinal thickness and the outer nuclear layer (ONL) at 3 and 8 weeks (P < 0.05), along w

194 ased apoptotic nuclei in their retinal outer nuclear layer (ONL) at postnatal day (P) 22.

195 One month after separation, outer nuclear layer (ONL) cell counts were significantly lower

196 ces donor cells beneath an intact host outer nuclear layer (ONL) containing host photoreceptors.

197 ment (IS/OS) band, and thinning of the outer nuclear layer (ONL) have been identified in association

198 layer (INL) in all 11 eyes and in the outer nuclear layer (ONL) in 4 eyes.

199 h disorganized POS and thinning of the outer nuclear layer (ONL) in addition to the anomaly at the PO

200 e outer plexiform layer (OPL) into the outer nuclear layer (ONL) in aged retinas.

201 OCT there were small foveal islands of outer nuclear layer (ONL) in those with preserved acuity.

202 The outer nuclear layer (ONL) of 1-month-old Rs1h-KO mice was diso

203 the measurement, the thickness of the outer nuclear layer (ONL) of central horizontal B-scans increa

204 The outer nuclear layer (ONL) of KO retinas became 20% thinner bet

205 hT17M Rho was localized in the outer nuclear layer (ONL) of T17M(+/-)ERAI(+/-) photoreceptors

206 ursors were able to integrate into the outer nuclear layer (ONL) of the Rd9 retina.

207 e were small foveal islands of thinned outer nuclear layer (ONL) surrounded by thick delaminated reti

208 /rd3 strains were measured for retinal outer nuclear layer (ONL) thickness from 5 to 12 weeks of age.

209 AF, A2E bisretinoid concentration, and outer nuclear layer (ONL) thickness in mice of different genot

210 Outer nuclear layer (ONL) thickness in T(-/-) mice was -70% of

211 s were recorded after OKT testing, and outer nuclear layer (ONL) thickness measurements were then obt

212 h, one eye was fixed and processed for outer nuclear layer (ONL) thickness measurements.

213 ificant reduction of the photoreceptor outer nuclear layer (ONL) thickness overlying 92% of the druse

214 Outer nuclear layer (ONL) thickness was mapped topographically

215 Foveal and outer nuclear layer (ONL) thickness was measured and presence

216 Outer nuclear layer (ONL) thickness was measured from fluoresc

217 cone photoreceptor packing density and outer nuclear layer (ONL) thickness within the central 15 degr

218 nd labeling (TUNEL) and measurement of outer nuclear layer (ONL) thickness.

219 ement of inner nuclear layer (INL) and outer nuclear layer (ONL) thickness.

220 layer, inner nuclear layer (INL), and outer nuclear layer (ONL) thicknesses; and horizontal extent o

221 The outer nuclear layer (ONL) thinned with eccentricity and was no

222 antly blocked photoreceptor apoptosis, outer nuclear layer (ONL) thinning, and retinal gliosis.

223 of Muller glia nuclei migrated to the outer nuclear layer (ONL) to divide.

224 ion, external limiting membrane (ELM), outer nuclear layer (ONL), and outer plexiform layer (OPL).

225 eceptor inner-outer segment junction), outer nuclear layer (ONL), and total retinal thickness were me

226 The internal limiting membrane, outer nuclear layer (ONL), external limiting membrane (ELM), i

227 outer segments plus RPE (OS+) and the outer nuclear layer (ONL).

228 D95 and mGluR6 was mislocalized in the outer nuclear layer (ONL).

229 rmed by counting rows of nuclei in the outer nuclear layer (ONL).

230 (RGCL), inner nucleus layer (INL), and outer nuclear layer (ONL).

231 after 14 dpf, it was restricted to the outer nuclear layer (ONL).

232 layer (GCL+IPL), RNFL, outer plexiform/inner nuclear layers (OPL+INL), and outer nuclear/photorecepto

233 ere observed in the number of cells in inner nuclear layer or in ganglion cells at 12 months of age.

234 t all correlated with thickness of the inner nuclear layer or total retina.

235 ell complex (GCC), and some sectors of outer nuclear layer (P- values </=0.05) was found with no sign

236 Patients with inner and outer nuclear layer pathology have more rapid disability progr

237 Microcystic inner nuclear layer pathology occurs in a proportion of patien

238 Identification of microcystic inner nuclear layer pathology on spectral-domain optical coher

239 and some plaques were observed in the outer nuclear layer, photoreceptor outer segment, and optic ne

240 cells) and distal part of neuroblastic/outer nuclear layer (photoreceptors).

241 ell layer, the anterior portion of the inner nuclear layer, photoreceptors, and choroidal stroma.

242 hickness, inner retinal thickness, and outer nuclear layer plus Henle fiber layer (ONL+HFL) thickness

243 ns at which the outer segment (OS) and outer nuclear layer plus outer plexiform layer (ONL+) thicknes

244 The thicknesses of the outer nuclear layer plus outer plexiform layer (ONL+), outer s

245 s of the outer segment (OS) layer, the outer nuclear layer plus outer plexiform layer (ONL+), the ret

246 ed with GCIP (r = -0.30; p = 0.02) and inner nuclear layer (r = -0.25; p = 0.04) atrophy rates.

247 aluation revealed complete loss of the outer nuclear layer, remodeling of the inner retina, loss of r

248 gressive thinning of the photoreceptor outer nuclear layer resulted from apoptotic cell death.

249 The lesions extended to the outer nuclear layer, resulting in focal retinal degeneration,

250 -/-) exhibit a progressive loss of the outer nuclear layer, retinal physiology deficits, and a higher

251 serially over years, the outer photoreceptor nuclear layer showed progressive thinning.

252 of choriocapillaris flow (CC-slab) or outer nuclear layer structure (ONL-slab) were generated from O

253 d ganglion cell layer, but also in the inner nuclear layer, suggesting that retinal injury is more wi

254 r plexiform layer (S6 and N6 sectors), inner nuclear layer (T6 and N6 sectors), and outer plexiform l

255 yers of the retina at the level of the inner nuclear layer that may develop in response to ischemia o

256 mitotic nuclei remaining in the basal inner nuclear layer, the region where Muller glia typically re

257 ted of nerve fiber, ganglion cell, and inner nuclear layer; the hypointense layer 2, the outer nuclea

258 in several cell layers, including the inner nuclear layer; they are present in primary mouse Muller

259 tinal damage was assessed by measuring outer nuclear layer thickness and by electroretinogram (ERG).

260 , producing significant improvement in outer nuclear layer thickness and ERG activity.

261 Cell death peaked at 1 day, and outer nuclear layer thickness declined from 1 to 5 days.

262 26 significantly increased the retinal outer nuclear layer thickness from 6.34 +/- 1.6 to 11.7 +/- 0.

263 Outer nuclear layer thickness in CNGA3-/- retina was reduced b

264 ns were examined by TUNEL staining and outer nuclear layer thickness measurements.

265 paralleled reported losses of photoreceptor nuclear layer thickness over the same age range.

266 on, and there was normal outer photoreceptor nuclear layer thickness retina-wide.

267 Histological studies showed that the outer nuclear layer thickness was dramatically reduced in the

268 calized visual sensitivity and photoreceptor nuclear layer thickness were measured across the central

269 1040 mum allowed the quantification of outer nuclear layer thickness, a direct correlate of photorece

270 Inner nuclear layer thickness, also in eyes without a history

271 f CME, central foveal thickness (CFT), inner nuclear layer thickness, and foveal-to-parafoveal thickn

272 sessed by TUNEL and measurement of the outer nuclear layer thickness.

273 eptor cell layer with variable loss of outer nuclear layer thickness.

274 Inner and outer nuclear layer thicknesses in patients with non-macular t

275 velopment, whereas ganglion cell loss, inner nuclear layer thinness, and early onset of glaucoma were

276 Amelioration of outer nuclear layer thinning indicated that vitamin E treatmen

277 scence, decreased HPLC-quantified A2E, outer nuclear layer thinning, and increased methylglyoxal (MG)

278 ation with severe GCL loss, borderline inner nuclear layer thinning, and less prominent photoreceptor

279 retinal loss, generally involving the outer nuclear layer to photoreceptors, occasionally with a cha

280 Our data indicate that regardless of which nuclear layer was damaged, MG respond by generating mult

281 he loss of photoreceptor nuclei in the outer nuclear layer was further accentuated and the number of

282 Among the intraretinal layers, the inner nuclear layer was identified as the best indicator of CR

283 The inner nuclear layer was normal or had borderline thinning.

284 y 21, but no significant damage to the outer nuclear layer was observed.

285 luding en face OCT segmentation of the inner nuclear layer was performed in all patients.

286 outer segments were preserved, and the outer nuclear layer was significantly thicker in the treated B

287 After 2 months, the outer nuclear layer was significantly thicker, and the photore

288 Within the nuclear layer we found an increase of the elastic and vi

289 d horizontal cells of all cells in the inner nuclear layer were comparable in central and peripheral

290 Microcystoid macular changes in the inner nuclear layer were diagnosed in 52 out of 264 eyes with

291 .3%), and hyperreflective spots in the outer nuclear layer were observed in 5 eyes (16.7%).

292 s of the retinal outer (ONL) and inner (INL) nuclear layers were evaluated at 9 weeks of age.

293 ificant thinning of both the inner and outer nuclear layers, when compared with other patients with m

294 eration reveals a proliferative advantage in nuclear layers where neurons were ablated.

295 th equals peripheral; on SDOCT, foveal outer nuclear layer (which includes HFL) and IS/OS thickens.

296 roportionate thinning of the inner and outer nuclear layers, which may be occurring as a primary proc

297 ron microscopy illustrated degenerated inner nuclear layer with disintegration of cells and loss of c

298 Neuronal loss was noted in the inner nuclear layer with focal reduction in cell density.

299 age, resulting in a thinner but intact outer nuclear layer with residual cones expressing S- and M-op

300 Substantial cell loss occurred in the inner nuclear layer, with a smaller reduction in photoreceptor

301 mislocalizes to the inner segment and outer nuclear layer, with only approximately 20% in rod outer