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1 m for cargo protection during passage of the nuclear pore.
2 he unique possibility of blocking EMT at the nuclear pore.
3 solutions, mimicking the environment of the nuclear pore.
4 nuclear periphery via interactions with the nuclear pore.
5 ites required for ratcheting mRNA across the nuclear pore.
6 ) that is critical for capsid routing to the nuclear pore.
7 level of its nuclear entry and exit via the nuclear pore.
8 K) and the transport of viral capsids to the nuclear pore.
9 rt receptors ferry protein cargo through the nuclear pore.
10 ne-glycine dipeptides (FG domains) fills the nuclear pore.
11 he genome once the capsid interacts with the nuclear pore.
12 ent NPC module, forms the outer rings of the nuclear pore.
13 G-nucleoporins to facilitate passage through nuclear pores.
14 -nucleoporins to facilitate movement through nuclear pores.
15 sing the viral genome from capsids docked at nuclear pores.
16 shuttles between unattached kinetochores and nuclear pores.
17 ribution of specific nucleoporins within the nuclear pores.
18 otherwise arrest CRM1 transport complexes at nuclear pores.
19 or increasing flux of molecules through the nuclear pores.
23 is (ALS) binds to the central channel of the nuclear pore and inhibits the movement of macromolecules
26 gans germline, cytoplasmic P granules at the nuclear pores and perinuclear Mutator foci contribute to
27 y depended on the number and permeability of nuclear pores and the availability of nuclear binding si
30 NPC, structural arrangements of Nups in the nuclear pore, and mechanisms of nucleocytoplasmic transp
31 to the cytoplasm, docking of the capsid at a nuclear pore, and release of the viral genome into the n
32 f mRNAs are more than 0.5 microm away from a nuclear pore, and we do so for the first time accounting
33 uclear periphery, shortens residency time at nuclear pores, and results in frequent release of mRNAs
34 nal regulation, key components of C. elegans nuclear pores are required for processing of a subset of
35 plex, components of mitotic kinetochores and nuclear pores, are blocked from binding to kinetochores
36 stent with the Mlp1/2 role in gene gating to nuclear pores, artificial tethering to the nuclear perip
37 orts a model in which animal genomes use the nuclear pore as an organizing scaffold for inducible poi
38 agues describe a surveillance pathway during nuclear pore assembly and, in doing so, identify a new r
40 that mitotic spindle, nuclear membrane, and nuclear pore assembly occur exclusively around chromatin
41 abilize the Nup82-Nsp1-Nup159 complex of the nuclear pore assembly through its interaction with nucle
43 f a fraction of Vps15-GFP and Vps34-GFP with nuclear pores at nucleus-vacuole (NV) junctions in live
51 architectures ranging from bacteriophages to nuclear pores, cilia, and synaptonemal complexes in larg
54 uncover two metformin response elements: the nuclear pore complex (NPC) and acyl-CoA dehydrogenase fa
55 ding yeast, targeting of active genes to the nuclear pore complex (NPC) and interchromosomal clusteri
58 generated nanobodies against the vertebrate nuclear pore complex (NPC) and used them in STORM imagin
59 e associated with shedding of NUP62 from the nuclear pore complex (NPC) and/or retention of NUP62 in
62 show that PfSR1 is localized adjacent to the Nuclear Pore Complex (NPC) clusters in the nucleus of ea
67 mechanism to explain how a component of the nuclear pore complex (NPC) could cause Htx/CHD was undef
68 esized membrane proteins traffic through the nuclear pore complex (NPC) en route to the inner nuclear
70 /RanGAP1*SUMO1/Ubc9 localizes at cytoplasmic nuclear pore complex (NPC) filaments and is a docking si
71 nterest and represents a central paradigm to nuclear pore complex (NPC) function, where nuclear trans
72 nucleocytoplasmic information transfer, the nuclear pore complex (NPC) has been studied in great det
75 lluring proposal outlining functions for the nuclear pore complex (NPC) in transcription and nuclear
78 nups) that line the transport channel of the nuclear pore complex (NPC) is investigated by means of c
88 iated Esc1, the SUMO E3 ligase Siz2, and the nuclear pore complex (NPC) protein Nup170-physically and
89 enterovirus 2A protease directly cleaves the nuclear pore complex (NPC) protein, Nup98, at amino acid
90 ring Aspergillus nidulans mitosis peripheral nuclear pore complex (NPC) proteins (Nups) disperse from
94 in receptor (p75(NTR)) is a component of the nuclear pore complex (NPC) required for glial scar forma
100 nsport, however, is tightly regulated by the nuclear pore complex (NPC) with the hydrophobic transpor
101 novirus (AdV) to the cytoplasmic face of the nuclear pore complex (NPC), a key step during delivery o
102 etic modifiers that encode components of the nuclear pore complex (NPC), as well as the machinery tha
104 ls involves regulatory interactions with the nuclear pore complex (NPC), followed by translocation to
105 2, a component of the central channel of the nuclear pore complex (NPC), for forced dimerization by t
106 g, transport, and cytoplasmic release from a nuclear pore complex (NPC), is fast ( approximately 200
107 this study we used BioID to study the human nuclear pore complex (NPC), one of the largest macromole
108 ic reticulum to the Golgi apparatus, and the nuclear pore complex (NPC), which facilitates nucleo-cyt
118 Grima et al. (2017) describe defects in the nuclear pore complex and impaired nucleocytoplasmic tran
119 ependent on the Nup107-160 subcomplex of the nuclear pore complex and is modulated through interactio
120 t of nucleoporins (Nups) can detach from the nuclear pore complex and move into the nuclear interior
121 s, might help vaults safely pass through the nuclear pore complex and potentiate their role as self-r
125 e nuclear periphery and interaction with the nuclear pore complex are prerequisites for gene clusteri
128 nk between the Torsin/cofactor system and NE/nuclear pore complex biogenesis or homeostasis and estab
129 east, some inducible genes interact with the nuclear pore complex both when active and for several ge
133 The NIMA kinase is required for mitotic nuclear pore complex disassembly and potentially control
135 Here, we genetically show that an intact nuclear pore complex is important for cell survival and
142 that the Arabidopsis (Arabidopsis thaliana) nuclear pore complex protein Nup88/MOS7 is essential for
144 provided a high-resolution understanding of nuclear pore complex structure and transport, revealing
145 rate complicated molecular gates such as the nuclear pore complex to control the transport of biologi
148 onal hsp-16.2/41 promoter interacts with the nuclear pore complex upon activation by heat shock in th
149 entary studies that Plk1 is recruited to the nuclear pore complex upon mitotic entry, where it acts w
152 ous intracellular compartments including the nuclear pore complex, COPII-coated vesicles, and inside
153 omolecular complexes with an emphasis on the nuclear pore complex, holding great potential for applic
154 , which is shared by several proteins of the nuclear pore complex, including those in the central cha
155 uding capsid transport, decapsidation at the nuclear pore complex, particle assembly, and secondary e
157 ssical nuclear import pathway, involving the nuclear pore complex, the small GTPase Ran, and cellular
158 the cellular substrates, particularly in the nuclear pore complex, used by these proteases were indee
159 are stacked ER-derived membranes containing nuclear pore complex-like structures whose fate and func
160 Analysis of a large dynamic structure-the nuclear pore complex-revealed variations detectable at t
174 lasmic reticulum (ER), translocation through nuclear pore complexes (NPCs) and retention on nuclear p
175 ic systems such as Saccharomyces cerevisiae, nuclear pore complexes (NPCs) and the spindle pole body
176 eraction of non-chromosomal DNA circles with nuclear pore complexes (NPCs) and thereby promotes their
177 hp1:Sem1:Sus1:Cdc31 (TREX2) complex binds to nuclear pore complexes (NPCs) and, in addition to integr
183 long-term protein persistence, we found that nuclear pore complexes (NPCs) are maintained over a cell
185 e nucleus, possibly due to delocalization of nuclear pore complexes (NPCs) at the nuclear envelope.
190 of mitosis and modulates distribution of the nuclear pore complexes (NPCs) during mitotic NE expansio
191 Nucleocytoplasmic transport is mediated by nuclear pore complexes (NPCs) embedded in the nuclear en
196 es can lead to an uneven distribution of the nuclear pore complexes (NPCs) in the interphase nuclear
199 artmentalization by the nuclear envelope and nuclear pore complexes (NPCs) is essential for cell func
200 epletions suggest that translocation through nuclear pore complexes (NPCs) is rate-limiting and restr
201 Passive macromolecular diffusion through nuclear pore complexes (NPCs) is thought to decrease dra
202 cleus and cytoplasm, is tightly regulated by nuclear pore complexes (NPCs) made up of nucleoporins (N
207 to p53-SUMO-1 and their accumulation in the nuclear pore complexes (NPCs), as well as their persiste
208 horylation-driven partial disassembly of the nuclear pore complexes (NPCs), increasing their permeabi
209 ucleocytoplasmic transport are maintained by nuclear pore complexes (NPCs), large structures composed
210 known as the constituent building blocks of nuclear pore complexes (NPCs), membrane-embedded channel
211 ucleocytoplasmic transport is facilitated by nuclear pore complexes (NPCs), which are massive protein
216 les and contribute to the quality control of nuclear pore complexes (NPCs); whether these processes a
217 vealing the octameric arrangement of Xenopus nuclear pore complexes and by quantifying the diffusion
218 istic step in Gle1's mRNA export function at nuclear pore complexes and directly implicate altered ex
219 compaction that facilitates movement through nuclear pore complexes and the length of transcript poly
223 rdered Phe-Gly nucleoporins (FG Nups) within nuclear pore complexes exert multivalent interactions wi
226 entional fluorophores, we have imaged single nuclear pore complexes in the nuclear membrane and aggre
229 ion of identical daughter nuclei by coupling nuclear pore complexes to the segregating chromosomes.
230 ants, and 3) transcripts being enriched near nuclear pore complexes when components of the mRNA expor
232 stration between fluorescently labeled mRNA, nuclear pore complexes, and chromatin, we obtained globa
234 ddition to its well-defined interaction with nuclear pore complexes, here we find that Gle1 is enrich
235 lo-like kinase 1 (PLK-1) is recruited to the nuclear pore complexes, just prior to NEBD, through its
237 oscopy (AFM) to the nuclear envelope and the nuclear pore complexes, we demonstrate that disposition
244 air; neuron pruning; extraction of defective nuclear pore complexes; nuclear envelope reformation; pl
245 ins and exportins, Ran-GTP cycle regulators, nuclear pore components, and arginine methylases in medi
246 cipitation of NPP-13 and NPP-3, two integral nuclear pore components, and importin-beta IMB-1 provide
247 pecific transcription complexes and show how nuclear pore composition changes can be exploited to reg
248 e standards was demonstrated by showing that nuclear pores contain 32 copies of the Nup107 complex.
249 scission in response to lagging chromosomes, nuclear pore defects, and tension forces at the midbody.
250 echanosensing mechanism mediated directly by nuclear pores, demonstrated for YAP but with potential g
252 gh the cytoplasmic environment and dock with nuclear pores for transport of their genomes into the nu
253 the expression of multiple genes involved in nuclear pore formation and is required for nuclear impor
255 ur results demonstrate a requirement for the nuclear pore function of Sec13 in development of the ret
258 ced the number of viral capsids reaching the nuclear pore if added at the time of viral entry and tha
261 cells of sec13(sq198) failed to form proper nuclear pores, leading to a nuclear accumulation of tota
262 tor binding, rather than permeability of the nuclear pores, may be rate-limiting for nucleo-cytoplasm
263 envelope (NE) specifically during G2 via two nuclear pore-mediated mechanisms involving RanBP2-BicD2
264 high-resolution structure of the cytoplasmic nuclear pore-mRNA export holo-complex, challenging our t
266 us and exit it either by passing through the nuclear pores or by rupturing the nuclear envelope.
268 y confirmed that the relocation of damage to nuclear pores plays an important role in a naturally occ
277 n HeLa cells, we detect cleavage of specific nuclear pore proteins known to be cleaved during poliovi
278 tained in the nucleoplasm, requires distinct nuclear pore proteins, and is regulated differently thro
279 e NPC and depends critically on unstructured nuclear pore proteins, and is therefore not well underst
283 signaling and programmed cell death require nuclear pore rearrangement and release of sequestered cy
284 leads to nuclear flattening, which stretches nuclear pores, reduces their mechanical resistance to mo
286 e of nucleocytoplasmic transport through the nuclear pore, revealing a novel mechanism of neurodegene
287 paired nuclear import kinetics, although the nuclear pore-size exclusion barrier was maintained.
288 lates the nuclear entry rates of YAP/TAZ via nuclear pore stretching, clarifying how forces affect ge
289 ry mechanisms might control septal pores and nuclear pores such that they are opened and closed out o
290 n microscopy revealed capsids accumulated at nuclear pores that retained the viral genome for at leas
291 hey are also found in the central channel of nuclear pores, the nexus points of intermediate filament
295 t cold-stress responses, associates with the nuclear pores to regulate mRNA export, and regulates the
296 (Y-complex), a major scaffold module of the nuclear pore, together with its partner Elys, colocalize
298 teins too large to passively diffuse through nuclear pores were readily imported into the nucleus thr
299 mutations caused a depletion of hGle1 at the nuclear pore where it carries out an essential role in n
300 nd both proteins localize to the nucleus and nuclear pores, where they interact with Tpr (translocate
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