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1 eractions with nuclear hormone receptors and nuclear respiratory factors.
3 tochrome c promoter mutants showed that both nuclear respiratory factor 1 (NRF-1) and cAMP-response e
4 re conserved in EWG homologs including human nuclear respiratory factor 1 (NRF-1) and the sea urchin
11 The present study tested our hypothesis that nuclear respiratory factor 1 (NRF-1) serves such a role
12 othesis, binding of the transcription factor nuclear respiratory factor 1 (NRF-1) to the cytochrome c
13 expression of a dominant negative allele of nuclear respiratory factor 1 (NRF-1), and glucose depriv
14 regulated by the same transcription factor, nuclear respiratory factor 1 (NRF-1), found recently by
15 in other COX genes, namely binding sites for nuclear respiratory factor 1 (NRF-1), nuclear respirator
17 oregulated by the same transcription factor, nuclear respiratory factor 1 (NRF-1), which regulates al
19 nscriptional activation [c-fos, c-jun, JunB, nuclear respiratory factor 1 (NRF-1)], mitochondrial pro
20 ing of EglN2 under hypoxic conditions reveal nuclear respiratory factor 1 (NRF1) motif enrichment in
21 X gene minimal promoter-binding activity as nuclear respiratory factor 1 (NRF1), a previously known
22 ecific DNA-binding transcriptional activator nuclear respiratory factor 1 (NRF1), in promoting contex
23 ed receptor-gamma coactivator-1alpha [PGC1], nuclear respiratory factor 1 [NRF1], mitochondrial trans
25 encoded by nrf is a close homologue of human Nuclear Respiratory Factor 1 and avian Initiation Bindin
26 ry mitochondrial biogenesis (no induction of nuclear respiratory factor 1 and mitochondrial transcrip
27 ear localization of 2 transcription factors, nuclear respiratory factor 1 and nuclear factor-E2-relat
28 vator 1alpha, the transcriptional activators nuclear respiratory factor 1 and nuclear respiratory fac
29 eceptor gamma coactivator 1alphaand 1betaand nuclear respiratory factor 1 mRNA and mitochondrial DNA
30 al transduction and recruitment of the NRF1 (nuclear respiratory factor 1) and THRB to the promoters
31 that the gene encoding the homolog of human Nuclear respiratory factor 1, erect wing (ewg), is auton
32 activator 1alpha, PGC1 related co-activator, nuclear respiratory factor 1, transcription factor A of
33 ors including estrogen-related receptors and nuclear respiratory factor 1, yet its physiological role
37 reduced levels of the PGC-1alpha downstream nuclear respiratory factors 1 and 2, mitochondrial trans
38 vated receptor-gamma coactivator 1 alpha and nuclear respiratory factors 1 and 2, or to enhanced expr
39 responsive to reactive oxygen species, i.e. nuclear respiratory factor-1 (NRF-1) and mitochondrial t
40 t I RE1-like silencer and functional Sp1 and nuclear respiratory factor-1 (NRF-1) elements within a G
43 he polio virus receptor that is bound by the nuclear respiratory factor-1 (NRF-1) transcription facto
44 We reported that estradiol (E(2)) induces nuclear respiratory factor-1 (NRF-1) transcription throu
45 that control the expression of these genes - nuclear respiratory factor-1 (NRF-1) was significantly u
46 ssociate with reduced expression of multiple nuclear respiratory factor-1 (NRF-1)-dependent genes enc
51 inding algorithms predicted the existence of nuclear respiratory factor-1 (NRF1) binding sites in E2F
52 own to orchestrate mitochondrial biogenesis, nuclear respiratory factor-1 and peroxisome-proliferator
55 gene expression and nuclear accumulation of nuclear respiratory factor-1, -2alpha (Gabpa), and perox
56 ptor-gamma coactivator-1alpha (PGC-1 alpha), nuclear respiratory factor-1, and mitochondrial transcri
57 activated receptor-gamma coactivator 1-beta, nuclear respiratory factor-1, and transcription factor A
58 c mitochondrial biogenesis by enhancement of nuclear respiratory factor-1, PGC-1alpha (peroxisome pro
62 o analysis revealed tandem binding sites for nuclear respiratory factor 2 (NRF-2) on the promoter of
63 at a transcription factor of the Ets family, nuclear respiratory factor 2 (NRF-2), can activate in vi
65 rresponded with induction of the ISR and the nuclear respiratory factor 2 antioxidant program but not
68 activators nuclear respiratory factor 1 and nuclear respiratory factor 2, mitochondrial transcriptio
70 es for nuclear respiratory factor 1 (NRF-1), nuclear respiratory factor 2/GA binding protein (NRF-2/G
71 beta/delta mRNA and protein (5 sessions) and nuclear respiratory factor-2 protein (3 sessions) while
72 els of mitochondrial transcription factor A, nuclear respiratory factor-2a, and peroxisome proliferat
73 ion of the PGC-1alpha pathway, PQQ increased nuclear respiratory factor activation (NRF-1 and NRF-2)
75 92-bp basal promoter contains sites for the nuclear respiratory factors NRF-1 and NRF-2, which have
78 box, two potential GC boxes, and a potential nuclear respiratory factor (NRF)-1 binding site, which w
81 inding analysis of VSNL1 promoter identified nuclear respiratory factor (NRF)-1/alpha-PAL as a major
82 a promoters, which for IL10 included Nfe2l2, nuclear respiratory factor (NRF)-2 (Gabpa), and MEF2, an
84 e Tfam expression through phosphorylation of nuclear respiratory factor (NRF-1) and binding to the Tf
85 or binding within multiple DH sites detected nuclear respiratory factors (NRF's) and YY1 specifically
86 man TFB1M and TFB2M promoters is governed by nuclear respiratory factors (NRF-1 and NRF-2), key trans
91 1alpha, oestrogen-related receptor alpha and nuclear respiratory factors, resulting in a decrease in
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